Search results for "Review"

showing 10 items of 4492 documents

Localized Interleukin-12 for Cancer Immunotherapy

2020

Interleukin-12 (IL-12) is a potent, pro-inflammatory type 1 cytokine that has long been studied as a potential immunotherapy for cancer. Unfortunately, IL-12's remarkable antitumor efficacy in preclinical models has yet to be replicated in humans. Early clinical trials in the mid-1990's showed that systemic delivery of IL-12 incurred dose-limiting toxicities. Nevertheless, IL-12's pleiotropic activity, i.e., its ability to engage multiple effector mechanisms and reverse tumor-induced immunosuppression, continues to entice cancer researchers. The development of strategies which maximize IL-12 delivery to the tumor microenvironment while minimizing systemic exposure are of increasing interest…

0301 basic medicinelcsh:Immunologic diseases. Allergymedicine.medical_treatmentDrug CompoundingImmunologyGenetic Vectorsinterleukin-12 (IL-12)Antineoplastic AgentsReviewBioinformatics03 medical and health sciences0302 clinical medicineCancer immunotherapyNeoplasmsintratumoral administrationTumor MicroenvironmentImmunology and AllergyMedicineAnimalsHumansTumor microenvironmentDrug Carrierscancer immunotherapyAntitumor immunitybusiness.industryGene Transfer TechniquesCancerImmunotherapyGenetic Therapymedicine.diseaseInterleukin-12Clinical trialcytokine delivery system030104 developmental biologyTreatment OutcomeInterleukin 12Cancer vaccineImmunotherapybusinesslcsh:RC581-607cancer vaccinelocalized delivery030215 immunologyFrontiers in Immunology
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Novel Insight Into the Molecular and Metabolic Mechanisms Orchestrating IL-17 Production in γδ T Cells

2019

Increasing evidence has demonstrated that IL-17-producing γδ T cells (γδ T17) play a tumor-promoting role in a series of cancers via various mechanisms in mice and human cancers, though the relationship between γδ T17 and human tumors has yet to be extensively characterized and established. Molecular signals such as intrinsic cascade, environmental cues and cellular metabolic pathways including nutrient uptake and utilization in γδ T17 cells are significantly important for their activation, differentiation, and function. Understanding the molecular mechanisms and metabolic pathways of γδ T17 cells in both the physiological setting and tumor environment would contribute to the development of…

0301 basic medicinelcsh:Immunologic diseases. Allergymedicine.medical_treatmentMini ReviewMetabolic reprogrammingImmunologyBiology03 medical and health sciences0302 clinical medicineCancer immunotherapyNeoplasmsmedicineTranscriptional regulationTumor MicroenvironmentImmunology and AllergyAnimalsHumansmetabolic reprogrammingtranscriptional regulationinnate immune cellsIntraepithelial Lymphocytescancer immunotherapyMicrobiotaInterleukin-17Immunotherapy3. Good healthCell biologyMetabolic pathway030104 developmental biologyInterleukin 17γδ T17 cellslcsh:RC581-607Function (biology)030215 immunologyFrontiers in Immunology
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Mitochondrial Dynamics: In Cell Reprogramming as It Is in Cancer

2017

Somatic cells can be reprogrammed into a pluripotent cellular state similar to that of embryonic stem cells. Given the significant physiological differences between the somatic and pluripotent cells, cell reprogramming is associated with a profound reorganization of the somatic phenotype at all levels. The remodeling of mitochondrial morphology is one of these dramatic changes that somatic cells have to undertake during cell reprogramming. Somatic cells transform their tubular and interconnected mitochondrial network to the fragmented and isolated organelles found in pluripotent stem cells early during cell reprogramming. Accordingly, mitochondrial fission, the process whereby the mitochond…

0301 basic medicinelcsh:Internal medicineInduced stem cellsSomatic cellReview ArticleCell BiologyBiologyEmbryonic stem cellCell biology03 medical and health sciences030104 developmental biologymitochondrial fusionMitochondrial fissionlcsh:RC31-1245Induced pluripotent stem cellMolecular BiologyCell potencyReprogrammingStem Cells International
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Exosome-Mediated Signaling in Epithelial to Mesenchymal Transition and Tumor Progression

2018

Growing evidence points to exosomes as key mediators of cell–cell communication, by transferring their specific cargo (e.g., proteins, lipids, DNA and RNA molecules) from producing to receiving cells. In cancer, the regulation of the exosome-mediated intercellular communication may be reshaped, inducing relevant changes in gene expression of recipient cells in addition to microenvironment alterations. Notably, exosomes may deliver signals able to induce the transdifferentiation process known as Epithelial-to-Mesenchymal Transition (EMT). In this review, we summarize recent findings on the role of exosomes in tumor progression and EMT, highlighting current knowledge on exosome-mediated inter…

0301 basic medicinelcsh:MedicineReviewExosomeMetastasis03 medical and health sciencesepithelial-mesenchymal plasticity; cancer-derived exosomes; extracellular vesicles; metastasis; tumor nicheGene expressionmedicinemetastasisEpithelial–Mesenchymal plasticityEpithelial–mesenchymal transitionbusiness.industrytumor nicheTransdifferentiationlcsh:RRNAGeneral Medicinemedicine.diseasecancer-derived exosomeMicrovesiclesCell biology030104 developmental biologyepithelial-mesenchymal plasticityTumor progressionmetastasiextracellular vesiclebusinesscancer-derived exosomesextracellular vesicles
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Non-Coding RNAs in Multiple Myeloma Bone Disease Pathophysiology

2020

Bone remodeling is uncoupled in the multiple myeloma (MM) bone marrow niche, resulting in enhanced osteoclastogenesis responsible of MM-related bone disease (MMBD). Several studies have disclosed the mechanisms underlying increased osteoclast formation and activity triggered by the various cellular components of the MM bone marrow microenvironment, leading to the identification of novel targets for therapeutic intervention. In this regard, recent attention has been given to non-coding RNA (ncRNA) molecules, that finely tune gene expression programs involved in bone homeostasis both in physiological and pathological settings. In this review, we will analyze major signaling pathways involved …

0301 basic medicinelcsh:QH426-470Bone diseasenon-coding RNAReviewBiologyBiochemistryBone remodeling03 medical and health sciences0302 clinical medicineOsteoclastmicroRNAGeneticsmedicinetumor microenvironmentMolecular BiologyMultiple myelomamiRNAlong non-coding RNAmedicine.diseaseNon-coding RNALong non-coding RNAmultiple myelomalcsh:Genetics030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisCancer researchbone diseaseBone marrowNon-Coding RNA
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Methods for RNA Modification Mapping Using Deep Sequencing: Established and New Emerging Technologies

2019

New analytics of post-transcriptional RNA modifications have paved the way for a tremendous upswing of the biological and biomedical research in this field. This especially applies to methods that included RNA-Seq techniques, and which typically result in what is termed global scale modification mapping. In this process, positions inside a cell`s transcriptome are receiving a status of potential modification sites (so called modification calling), typically based on a score of some kind that issues from the particular method applied. The resulting data are thought to represent information that goes beyond what is contained in typical transcriptome data, and hence the field has taken to use …

0301 basic medicinelcsh:QH426-470Computer scienceProcess (engineering)Emerging technologieschemical treatmentNext Generation Sequencingengineered Reverse Transcriptase enzymesRNA-SeqReviewcomputer.software_genreDeep sequencingField (computer science)deep sequencing03 medical and health sciences0302 clinical medicineepitranscriptome[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]GeneticsAnimalsHumansRNA-SeqRNA Processing Post-TranscriptionalComputingMilieux_MISCELLANEOUSGenetics (clinical)Sequence Analysis RNAbusiness.industryScale (chemistry)High-Throughput Nucleotide Sequencing[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyRNA modificationTerm (time)lcsh:Genetics030104 developmental biologyAnalyticsRNAData miningbusinesscomputer030217 neurology & neurosurgeryGenes
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RNA Modifications Modulate Activation of Innate Toll-Like Receptors

2019

Self/foreign discrimination by the innate immune system depends on receptors that identify molecular patterns as associated to pathogens. Among others, this group includes endosomal Toll-like receptors, among which Toll-like receptors (TLR) 3, 7, 8, and 13 recognize and discriminate mammalian from microbial, potentially pathogen-associated, RNA. One of the discriminatory principles is the recognition of endogenous RNA modifications. Previous work has identified a couple of RNA modifications that impede activation of TLR signaling when incorporated in synthetic RNA molecules. Of note, work that is more recent has now shown that RNA modifications in their naturally occurring context can have …

0301 basic medicinelcsh:QH426-470EndosomeContext (language use)ReviewBiology03 medical and health sciences0302 clinical medicineRNA modificationsGeneticsAnimalsHumansGenetics(clinical)RNA Processing Post-TranscriptionalReceptorGeneinnate immunityGenetics (clinical)Innate immune systemRNATLR7Immunity InnateCell biologyToll-like receptorslcsh:Genetics030104 developmental biologyTransfer RNAmethylation030215 immunologyGenes
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Epigenetic Regulation in the Pathogenesis of Sjögren Syndrome and Rheumatoid Arthritis

2019

Autoimmune rheumatic diseases, such as Sjögren syndrome (SS) and rheumatoid arthritis (RA), are characterized by chronic inflammation and autoimmunity, which cause joint tissue damage and destruction by triggering reduced mobility and debilitation in patients with these diseases. Initiation and maintenance of chronic inflammatory stages account for several mechanisms that involve immune cells as key players and the interaction of the immune cells with other tissues. Indeed, the overlapping of certain clinical and serologic manifestations between SS and RA may indicate that numerous immunologic-related mechanisms are involved in the physiopathology of both these diseases. It is widely accept…

0301 basic medicinelcsh:QH426-470InflammationReviewmedicine.disease_causeAutoimmunityPathogenesis03 medical and health sciences0302 clinical medicineImmune systemImmunitymicroRNAGeneticsmedicineautoimmune diseasesEpigeneticsepigenetic pathwaysGenetics (clinical)DNA methylationepigeneticshistone modificationsbusiness.industrylcsh:Genetics030104 developmental biology030220 oncology & carcinogenesismiRNAsDNA methylationImmunologyrheumatic diseasesMolecular Medicinemedicine.symptombusinessFrontiers in Genetics
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Environmental epigenetics in zebrafish

2017

Abstract It is widely accepted that the epigenome can act as the link between environmental cues, both external and internal, to the organism and phenotype by converting the environmental stimuli to phenotypic responses through changes in gene transcription outcomes. Environmental stress endured by individual organisms can also enforce epigenetic variations in offspring that had never experienced it directly, which is termed transgenerational inheritance. To date, research in the environmental epigenetics discipline has used a wide range of both model and non-model organisms to elucidate the various epigenetic mechanisms underlying the adaptive response to environmental stimuli. In this rev…

0301 basic medicinelcsh:QH426-470Settore BIO/11 - Biologia MolecolareReviewEnvironmentEpigenesis GeneticEmbryogenesi03 medical and health sciences0302 clinical medicineEnvironmental epigeneticEnvironmental epigeneticsGeneticsAnimalsEpigeneticsToxicantZebrafishMolecular BiologyOrganismZebrafishDNA methylation; Embryogenesis; Environmental epigenetics; Histone modifications; Methylome; Toxicant; Transgenerational inheritance; Zebrafish; Molecular Biology; GeneticsGeneticsDNA methylationbiologyHistone modificationsInheritance (genetic algorithm)Adaptive responseEpigenomebiology.organism_classificationHuman geneticsHistone Codelcsh:Genetics030104 developmental biologyEvolutionary biologyDNA methylationEmbryogenesisMethylomeHistone modification030217 neurology & neurosurgeryTransgenerational inheritanceEpigenetics & Chromatin
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H1.0 Linker Histone as an Epigenetic Regulator of Cell Proliferation and Differentiation

2018

H1 linker histones are a class of DNA-binding proteins involved in the formation of supra-nucleosomal chromatin higher order structures. Eleven non-allelic subtypes of H1 are known in mammals, seven of which are expressed in somatic cells, while four are germ cell-specific. Besides having a general structural role, H1 histones also have additional epigenetic functions related to DNA replication and repair, genome stability, and gene-specific expression regulation. Synthesis of the H1 subtypes is differentially regulated both in development and adult cells, thus suggesting that each protein has a more or less specific function. The somatic variant H1.0 is a linker histone that was recognized…

0301 basic medicinelcsh:QH426-470Somatic cellRNA-binding proteinhistone H1.0RNA-binding proteinsReviewBiologymedicine.disease_cause03 medical and health sciencesSettore BIO/10 - BiochimicaGeneticsmedicineEpigeneticsSettore BIO/06 - Anatomia Comparata E CitologiaGenetics (clinical)linker histonesCell growthChromatinCell biologylcsh:Geneticslinker histone030104 developmental biologyHistoneCancer cellbiology.proteinStem cellextracellular vesiclesCarcinogenesisGenes
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