Search results for "Roi"

showing 10 items of 11055 documents

Computational Fluid Dynamics Suggests Ecological Diversification among Stem-Gnathostomes.

2020

Summary The evolutionary assembly of the vertebrate bodyplan has been characterized as a long-term ecological trend toward increasingly active and predatory lifestyles, culminating in jawed vertebrates that dominate modern vertebrate biodiversity [ 1 , 2 , 3 , 4 , 5 , 6 , 7 , 8 ]. This contrast is no more stark than between the earliest jawed vertebrates and their immediate relatives, the extinct jawless, dermal armor-encased osteostracans, which have conventionally been interpreted as benthic mud-grubbers with poor swimming capabilities and low maneuverability [ 9 , 10 , 11 , 12 ]. Using computational fluid dynamics, we show that osteostracan headshield morphology is compatible with a dive…

0301 basic medicineWater flowcomputational fluid dynamicsBiologyDiversification (marketing strategy)General Biochemistry Genetics and Molecular BiologyPassive control03 medical and health sciences0302 clinical medicinebiology.animalAnimalsComputer Simulation14. Life underwatergeometric morphometricsSwimmingKey innovationEcologyFossilsFishesVertebrateBiodiversityFeeding BehaviorSubstrate (marine biology)jawed vertebratesBiological Evolution030104 developmental biologyBenthic zoneOblate spheroidHydrodynamicsecologyGeneral Agricultural and Biological Sciencesstem-gnathostomesHead030217 neurology & neurosurgeryEvolució (Biologia)Current biology : CB
researchProduct

Impact of Lipid Components and Emulsifiers on Plant Sterols Bioaccessibility from Milk-Based Fruit Beverages.

2016

Sterol bioaccessibility (BA) of three plant sterol (PS)-enriched milk-based fruit beverages (MFb) with different fat contents (1.1–2.4%), lipid sources (animal or vegetable), and without or with emulsifiers (whey proteins enriched with milk fat globule membrane (MFGM) or soy lecithin) was evaluated after simulated gastrointestinal digestion. The BA of total PS followed the order 31.4% (MFbM containing milk fat and whey proteins enriched with MFGM) = 28.2% (MFbO containing extra virgin olive oil and soy lecithin) > 8.7% (MFb without fat addition). Total and individual PS content in the bioaccessible fractions followed the order MFbM > MFbO > MFb. Consequently, formulation with MFGM is propos…

0301 basic medicineWhey proteinBeverages03 medical and health scienceschemistry.chemical_compoundLecithinsAnimalsFood scienceGlycoproteins030109 nutrition & dieteticsChromatographyChemistryCholesterolfood and beveragesPhytosterolsGeneral ChemistryLipid DropletsLipidsSterolBioavailabilityGastrointestinal TractMilkMilk fatEmulsifying AgentsFruitCattleDigestionGlycolipidsGeneral Agricultural and Biological SciencesDigestionPlant sterolsOlive oilJournal of agricultural and food chemistry
researchProduct

Impact of polymer-modified gold nanoparticles on brain endothelial cells: exclusion of endoplasmic reticulum stress as a potential risk factor

2016

A library of polymer-coated gold nanoparticles (AuNPs) differing in size and surface modifications was examined for uptake and induction of cellular stress responses in the endoplasmic reticulum (ER stress) in human brain endothelial cells (hCMEC/D3). ER stress is known to affect the physiology of endothelial cells (ECs) and may lead to inflammation or apoptosis. Thus, even if applied at non-cytotoxic concentrations ER stress caused by nanoparticles should be prevented to reduce the risk of vascular diseases and negative effects on the integrity of barriers (e.g. blood-brain barrier). We exposed hCMEC/D3 to twelve different AuNPs (three sizes: 18, 35, and 65 nm, each with four surface-modif…

0301 basic medicineXBP1BiPCell SurvivalPolymersBiomedical EngineeringMetal NanoparticlesApoptosis02 engineering and technologyBiologyEndoplasmic ReticulumToxicologyArticleCell LineProinflammatory cytokine03 medical and health sciencescell stressDownregulation and upregulationRisk FactorsHeat shock proteinAnimalsHumansHSP70 Heat-Shock ProteinsParticle SizeHeat-Shock ProteinsATF6Endoplasmic reticulumInterleukin-8ATF4Endothelial CellsMembrane Proteinsunfolded protein responseEndoplasmic Reticulum Stress021001 nanoscience & nanotechnologyQPActivating Transcription Factor 4Cell biology030104 developmental biologyBlood-Brain Barriertight junction proteinsImmunologyUnfolded protein responseGold0210 nano-technologyTranscription Factor CHOPNanotoxicology
researchProduct

Systematic gene overexpression in Candida albicans identifies a regulator of early adaptation to the mammalian gut.

2018

International audience; Candida albicans is part of the human gastrointestinal (GI) microbiota. To better understand how C. albicans efficiently establishes GI colonisation, we competitively challenged growth of 572 signature-tagged strains (~10% genome coverage), each conditionally overexpressing a single gene, in the murine gut. We identified CRZ2, a transcription factor whose overexpression and deletion respectively increased and decreased early GI colonisation. Using clues from genome-wide expression and gene-set enrichment analyses, we found that the optimal activity of Crz2p occurs under hypoxia at 37°C, as evidenced by both phenotypic and transcriptomic analyses following CRZ2 geneti…

0301 basic medicine[SDV.MHEP.AHA] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]030106 microbiologyImmunologyMicrobiologyMannosyltransferasesBiological pathwayTranscriptomeFungal ProteinsMannans03 medical and health scienceschemistry.chemical_compoundtranscriptomicsregulatory networksCell WallVirologyGene Expression Regulation FungalCandida albicanssignature‐tagged overexpression[SDV.MHEP.AHA]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]AnimalsGene Regulatory NetworksCandida albicansPromoter Regions GeneticGeneTranscription factorResearch ArticlesFungal proteinMice Inbred BALB CCRZ2chromatin immunoprecipitation‐on‐chipbiologyCRZ2;Candida albicans;chromatin immunoprecipitation-on-chip;gastrointestinal colonisation;regulatory networks;signature-tagged overexpression;transcriptomicsTunicamycinTunicamycinHydrogen-Ion Concentrationbiology.organism_classificationPhenotypeCell biologyGastrointestinal MicrobiomeGastrointestinal Tractchemistrychromatin immunoprecipitation-on-chipFemalesignature-tagged overexpressionMicroorganisms Genetically-Modifiedgastrointestinal colonisationResearch Article
researchProduct

A Multilevel Functional Study of aSNAP25At-Risk Variant for Bipolar Disorder and Schizophrenia

2017

The synaptosomal-associated protein SNAP25 is a key player in synaptic vesicle docking and fusion and has been associated with multiple psychiatric conditions, including schizophrenia, bipolar disorder, and attention-deficit/hyperactivity disorder. We recently identified a promoter variant inSNAP25,rs6039769, that is associated with early-onset bipolar disorder and a higher gene expression level in human prefrontal cortex. In the current study, we showed that this variant was associated both in males and females with schizophrenia in two independent cohorts. We then combinedin vitroandin vivoapproaches in humans to understand the functional impact of the at-risk allele. Thus, we showedin vi…

0301 basic medicine[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology[SDV.NEU.PC] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Psychology and behaviorbrain imagingAmygdala03 medical and health sciences0302 clinical medicineNeuroimagingSynaptic vesicle dockingmedicinegeneticsBipolar disorderAllelePrefrontal cortexComputingMilieux_MISCELLANEOUSbipolar disorder[SDV.NEU.PC]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Psychology and behavior[SCCO.NEUR]Cognitive science/NeuroscienceGeneral Neuroscience[SCCO.NEUR] Cognitive science/Neuroscience[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology[SDV.NEU.SC]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive SciencesSNAP25medicine.diseaseschizophrenia030104 developmental biologymedicine.anatomical_structureSNARESNAP25CohortPsychologyNeuroscience[SDV.NEU.SC] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive Sciences030217 neurology & neurosurgeryThe Journal of Neuroscience
researchProduct

GDF11 exhibits tumor suppressive properties in hepatocellular carcinoma cells by restricting clonal expansion and invasion.

2019

Growth differentiation factor 11 (GDF11) has been characterized as a key regulator of differentiation in cells that retain stemness features, despite some controversies in age-related studies. GDF11 has been poorly investigated in cancer, particularly in those with stemness capacity, such as hepatocellular carcinoma (HCC), one of the most aggressive cancers worldwide. Here, we focused on investigating the effects of GDF11 in liver cancer cells. GDF11 treatment significantly reduced proliferation, colony and spheroid formation in HCC cell lines. Consistently, down-regulation of CDK6, cyclin D1, cyclin A, and concomitant upregulation of p27 was observed after 24 h of treatment. Interestingly,…

0301 basic medicine[SDV]Life Sciences [q-bio]Cyclin ACellChick EmbryoChorioallantoic Membrane0302 clinical medicineCell MovementCyclin D1HCCbiologyNeovascularization PathologicCell DifferentiationHep G2 CellsCell cycleCadherinsHuh7 cells3. Good health[SDV] Life Sciences [q-bio]Gene Expression Regulation NeoplasticGrowth Differentiation Factorsmedicine.anatomical_structure030220 oncology & carcinogenesisBone Morphogenetic ProteinsMolecular MedicineLiver cancerCyclin-Dependent Kinase Inhibitor p27Signal Transduction[SDV.CAN]Life Sciences [q-bio]/CancerCyclin ACell cycleHep3B cells03 medical and health sciencesCyclin D1Downregulation and upregulation[SDV.CAN] Life Sciences [q-bio]/CancerAntigens CDCell Line TumorOccludinSpheroids CellularmedicineAnimalsHumansViability assayMolecular BiologyCell Proliferation[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and GastroenterologyCyclin-Dependent Kinase 6[SDV.MHEP.HEG] Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology030104 developmental biologyCell cultureGDF11biology.proteinCancer researchCyclin-dependent kinase 6Snail Family Transcription FactorsBiochimica et biophysica acta. Molecular basis of disease
researchProduct

N-3 polyunsaturated fatty acids: An innovative strategy against obesity and related metabolic disorders, intestinal alteration and gut microbiota dys…

2019

Obesity is now widely recognized to be associated with low-grade systemic inflammation. It has been shown that high-fat feeding modulates gut microbiota which strongly increased intestinal permeability leading to lipopolysaccharide absorption causing metabolic endotoxemia that triggers inflammation and metabolic disorders. N-3 polyunsaturated fatty acids (PUFAs) have been shown associated with anti-obesity properties, but results still remain heterogeneous and very few studies underlined the metabolic pathways involved. Thus, the use of Fat-1 transgenic mice allows to better understanding whether endogenous n-3 PUFAs enrichment contributes to obesity and associated metabolic disorders preve…

0301 basic medicine[SDV]Life Sciences [q-bio]PopulationGut floradigestive systemBiochemistry03 medical and health sciencesMiceMetabolic DiseasesFatty Acids Omega-3medicineAnimalsHumansObesityeducation2. Zero hungerchemistry.chemical_classificationeducation.field_of_studyIntestinal permeability030102 biochemistry & molecular biologybiologyAkkermansiaGeneral Medicinebiology.organism_classificationmedicine.disease3. Good healthGastrointestinal MicrobiomeMetabolic pathway030104 developmental biologychemistryImmunologyDysbiosisMetabolic syndromeDysbiosisPolyunsaturated fatty acid
researchProduct

Functional comparison of bacteria from the human gut and closely related non-gut bacteria reveals the importance of conjugation and a paucity of moti…

2016

International audience; The human GI tract is a complex and still poorly understood environment, inhabited by one of the densest microbial communities on earth. The gut microbiota is shaped by millennia of evolution to co-exist with the host in commensal or symbiotic relationships. Members of the gut microbiota perform specific molecular functions important in the human gut environment. This can be illustrated by the presence of a highly expanded repertoire of proteins involved in carbohydrate metabolism, in phase with the large diversity of polysaccharides originating from the diet or from the host itself that can be encountered in this environment. In order to identify other bacterial fun…

0301 basic medicine[SDV]Life Sciences [q-bio]lcsh:MedicineGut floraPathology and Laboratory Medicinemedicine.disease_causeBiochemistryDatabase and Informatics MethodsRNA Ribosomal 16SMedicine and Health SciencesDNA metabolismlcsh:SciencePhylogenyProtein MetabolismClostridium BotulinumMultidisciplinarybiologyChemotaxisGastrointestinal Microbiomedigestive oral and skin physiologyHuman microbiomeGenomicsBacterial Physiological PhenomenaGenomic DatabasesAdaptation PhysiologicalBacterial PathogensNucleic acidsMedical MicrobiologyConjugation GeneticPathogensBacteroides thetaiotaomicronResearch ArticleCell PhysiologyBacterial Physiological PhenomenaResearch and Analysis MethodsBiosynthesisMicrobiologydigestive systemMicrobiology03 medical and health sciencesBacterial ProteinsGeneticsmedicineHumansMicrobial PathogensEscherichia coliClostridiumBacteria030102 biochemistry & molecular biologyGut Bacterialcsh:ROrganismsBiology and Life SciencesComputational BiologyChemotaxisCell BiologyDNAGenome Analysisbiology.organism_classificationGastrointestinal MicrobiomeCell MetabolismBiological DatabasesMetabolism030104 developmental biologyEvolutionary biologylcsh:QGenome BacterialBacteria
researchProduct

Oxidative Stress, Neuroinflammation and Mitochondria in the Pathophysiology of Amyotrophic Lateral Sclerosis

2020

Amyotrophic lateral sclerosis (ALS) is a progressive motor neuron (MN) disease. Its primary cause remains elusive, although a combination of different causal factors cannot be ruled out. There is no cure, and prognosis is poor. Most patients with ALS die due to disease-related complications, such as respiratory failure, within three years of diagnosis. While the underlying mechanisms are unclear, different cell types (microglia, astrocytes, macrophages and T cell subsets) appear to play key roles in the pathophysiology of the disease. Neuroinflammation and oxidative stress pave the way leading to neurodegeneration and MN death. ALS-associated mitochondrial dysfunction occurs at different le…

0301 basic medicineamyotrophic lateral sclerosisPhysiologyClinical BiochemistryReviewDiseaseMitochondrionmedicine.disease_causeBiochemistryneuroinflammationNeurologia03 medical and health sciences0302 clinical medicineoxidative stressMedicineAmyotrophic lateral sclerosisMolecular BiologyNeuroinflammationMicrogliabusiness.industrylcsh:RM1-950NeurodegenerationCell Biologymedicine.diseasePatologiaPathophysiologymitochondrialcsh:Therapeutics. Pharmacology030104 developmental biologymedicine.anatomical_structuremotor neuron diseasebusinessNeuroscience030217 neurology & neurosurgeryOxidative stressAntioxidants
researchProduct

Functions and Therapeutic Potential of Extracellular Hsp60, Hsp70, and Hsp90 in Neuroinflammatory Disorders

2021

Neuroinflammation is implicated in central nervous system (CNS) diseases, but the molecular mechanisms involved are poorly understood. Progress may be accelerated by developing a comprehensive view of the pathogenesis of CNS disorders, including the immune and the chaperone systems (IS and CS). The latter consists of the molecular chaperones; cochaperones; and chaperone cofactors, interactors, and receptors of an organism and its main collaborators in maintaining protein homeostasis (canonical function) are the ubiquitin–proteasome system and chaperone-mediated autophagy. The CS has also noncanonical functions, for instance, modulation of the IS with induction of proinflammatory cytokines. …

0301 basic medicineamyotrophic lateral sclerosislcsh:TechnologychaperonopathiesProinflammatory cytokinelcsh:Chemistrys disease03 medical and health sciences0302 clinical medicinechaperone systemmedicineamyotrophic lateral sclerosiGeneral Materials Sciencelcsh:QH301-705.5InstrumentationchaperonotherapyNeuroinflammationFluid Flow and Transfer Processesbiologylcsh:TMechanism (biology)Process Chemistry and Technologymolecular chaperonesNeurodegenerationAutophagyGeneral EngineeringParkinson’S diseasemolecular chaperonemedicine.diseaseHuntington’ s diseaseHsp90lcsh:QC1-999Computer Science Applications030104 developmental biologylcsh:Biology (General)lcsh:QD1-999lcsh:TA1-2040multiple sclerosiChaperone (protein)Alzheimerbiology.proteinHSP60lcsh:Engineering (General). Civil engineering (General)Alzheimer’s diseaseNeurosciencelcsh:Physics030217 neurology & neurosurgeryHuntington’s diseaseApplied Sciences
researchProduct