Search results for "SENESCENCE"

showing 10 items of 339 documents

An experimental manipulation of life-history trajectories and resistance to oxidative stress

2006

Optimal investment into life-history traits depends on the environmental conditions that organisms are likely to experience during their life. Evolutionary theory tells us that optimal investment in reproduction versus maintenance is likely to shape the pattern of age-associated decline in performance, also known as aging. The currency that is traded against different vital functions is, however, still debated. Here, we took advantage of a phenotypic manipulation of individual quality in early life to explore (1) long-term consequences on life-history trajectories, and (2) the possible physiological mechanism underlying the life-history adjustments. We manipulated phenotypic quality of a co…

Male0106 biological sciencesSenescenceAgingsenescenceOffspringmedia_common.quotation_subject[SDE.MCG]Environmental Sciences/Global ChangesLongevityZoologyfree radicalsBiology010603 evolutionary biology01 natural sciences[ SDV.EE ] Life Sciences [q-bio]/Ecology environment03 medical and health sciencesGeneticsAnimalsEcology Evolution Behavior and Systematics030304 developmental biologymedia_common[SDV.EE]Life Sciences [q-bio]/Ecology environment0303 health sciences[ SDE.BE ] Environmental Sciences/Biodiversity and EcologyEcologyLongevitysenescence.biology.organism_classificationFecundityBiological Evolution[SDE.ES]Environmental Sciences/Environmental and SocietyBrooddisposable-soma theory of agingOxidative Stress[ SDE.MCG ] Environmental Sciences/Global ChangesFertilityPhenotypeAging/physiology; Animals; Evolution; Female; Fertility/physiology; Finches/physiology; Longevity; Male; Oxidative Stress/physiology; PhenotypeAge at first reproductionFemaleFinchesReproductive valueReproduction[SDE.BE]Environmental Sciences/Biodiversity and EcologyGeneral Agricultural and Biological Sciencesresistance to oxidative stressTaeniopygia[ SDE.ES ] Environmental Sciences/Environmental and Societylife-table response experiment
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Cellular immune activation in Sardinian middle-aged, older adults and centenarians

2017

In addition to viral infections, malignant disorders, autoimmune diseases, and allograft rejection episodes, neopterin increases in older people where it is found to be predictive of overall mortality. Thus, the serum concentrations of this biomarker of systemic immune and inflammation activation, were measured in a small cohort of Sardinian middle-aged, older adults and centenarians. There was a significant positive correlation between neopterin concentrations and age with the subjects in the 95-year-old group with the highest values. Notably, the group of centenarians had neopterin values comparable to those of 80- and 90-year-old groups, and significantly lower than that of 95-year-old g…

Male0301 basic medicineAgingImmunosenescenceLongevityInflammationBiologyNeopterinBiochemistryMonocytes03 medical and health scienceschemistry.chemical_compoundEndocrinologyImmune systemGeneticCentenariansGeneticsmedicineHumansMacrophageCentenarianCytokineMolecular BiologyAgedSettore MED/04 - Patologia GeneraleAged 80 and overMacrophagesPteridinesMonocyteAge FactorsNeopterinCell BiologyImmunosenescenceMacrophage ActivationMiddle AgedInflammaging030104 developmental biologymedicine.anatomical_structureItalychemistryImmunologyCohortPteridineCytokinesBiomarker (medicine)FemaleInflammation Mediatorsmedicine.symptomBiomarkersExperimental Gerontology
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Adverse Effects of Diabetes Mellitus on the Skeleton of Aging Mice

2016

In the present study, the possibility that a diabetic (DM) status might worsen age-related bone deterioration was explored in mice. Male CD-1 mice aged 2 (young control group) or 16 months, nondiabetic or made diabetic by streptozotocin injections, were used. DM induced a decrease in bone volume, trabecular number, and eroded surface, and in mineral apposition and bone formation rates, but an increased trabecular separation, in L1-L3 vertebrae of aged mice. Three-point bending and reference point indentation tests showed slight changes pointing to increased frailty and brittleness in the mouse tibia of diabetic old mice. DM was related to a decreased expression of both vascular endothelial …

Male0301 basic medicineSenescenceAgingmedicine.medical_specialtyBone densityOsteoporosisDiabetes Mellitus ExperimentalBone remodelingMice03 medical and health scienceschemistry.chemical_compoundSDG 3 - Good Health and Well-beingBone DensityOsteogenesisDiabetes mellitusInternal medicineAnimalsMedicineLumbar Vertebraebusiness.industryStreptozotocinmedicine.diseaseVascular endothelial growth factor030104 developmental biologyEndocrinologymedicine.anatomical_structurechemistryOsteoporosisBone RemodelingBone marrowGeriatrics and GerontologyTomography X-Ray Computedbusinessmedicine.drugThe Journals of Gerontology Series A: Biological Sciences and Medical Sciences
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Calorie Restriction in Adulthood Reduces Hepatic Disorders Induced by Transient Postnatal Overfeeding in Mice

2019

International audience; Impaired early nutrition influences the risk of developing metabolic disorders in later life. We observed that transient postnatal overfeeding (OF) in mice induces long-term hepatic alterations, characterized by microsteatosis, fibrosis associated with oxidative stress (OS), and stress-induced premature senescence (SIPS). In this study, we investigated whether such changes can be reversed by moderate calorie restriction (CR). C57BL/6 male mice pups were maintained during lactation in litters adjusted to nine pups in the normal feeding (NF) group and three pups in the transient postnatal OF group. At six months of age, adult mice from the NF and OF groups were randoml…

Male0301 basic medicineStress-induced premature senescencemedicine.disease_causeMicechemistry.chemical_compound0302 clinical medicineFibrosisLactationoxidative stressCellular Senescence2. Zero hungerNutrition and DieteticsbiologySuperoxideLiver DiseasesDOHaDCatalasemedicine.anatomical_structure030220 oncology & carcinogenesisstress-induced premature senescenceFemalelcsh:Nutrition. Foods and food supplymedicine.medical_specialtyAnimals; Animals Newborn; Caloric Restriction/methods; Catalase/metabolism; Cellular Senescence; Feeding Methods/adverse effects; Female; Liver/metabolism; Liver Diseases/diet therapy; Liver Diseases/etiology; Liver Diseases/physiopathology; Male; Mice; Mice Inbred C57BL; Oxidative Stress; Superoxide Dismutase/metabolism; DOHaD; developmental programming; liver; oxidative stress; reversibility; stress-induced premature senescenceCalorie restrictionlcsh:TX341-641liverArticleLipofuscinFeeding MethodsSuperoxide dismutase03 medical and health sciencesreversibility[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemdevelopmental programmingInternal medicinemedicineAnimalsCaloric RestrictionSuperoxide Dismutasebusiness.industrymedicine.diseaseMice Inbred C57BL030104 developmental biologyEndocrinologyAnimals Newbornchemistrybiology.proteinbusinessOxidative stressFood Science
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Patients experiencing statin-induced myalgia exhibit a unique program of skeletal muscle gene expression following statin re-challenge

2017

Statins, the 3-hydroxy-3-methyl-glutaryl (HMG)-CoA reductase inhibitors, are widely prescribed for treatment of hypercholesterolemia. Although statins are generally well tolerated, up to ten percent of statin-treated patients experience myalgia symptoms, defined as muscle pain without elevated creatinine phosphokinase (CPK) levels. Myalgia is the most frequent reason for discontinuation of statin therapy. The mechanisms underlying statin myalgia are not clearly understood. To elucidate changes in gene expression associated with statin myalgia, we compared profiles of gene expression in skeletal muscle biopsies from patients with statin myalgia who were undergoing statin re-challenge (cases)…

Male0301 basic medicinemyalgiaGene Expressionlcsh:MedicineApoptosis030204 cardiovascular system & hematologyPathology and Laboratory MedicineBioinformaticsBiochemistry0302 clinical medicineMedicine and Health SciencesGene Regulatory Networkslcsh:ScienceMusculoskeletal SystemEnergy-Producing OrganellesMyositisRegulation of gene expressionMultidisciplinaryCell DeathbiologyMusclesDrugsMiddle AgedMitochondriaCell ProcessesHMG-CoA reductaseFemalelipids (amino acids peptides and proteins)AnatomyCellular Structures and Organellesmedicine.symptomResearch ArticleSenescencemedicine.medical_specialtyStatinmedicine.drug_classPainBioenergeticsPolymorphism Single Nucleotide03 medical and health sciencesSigns and SymptomsDiagnostic MedicineInternal medicineGeneticsmedicineHumansGene Regulationcardiovascular diseasesMuscle SkeletalAgedPharmacologybusiness.industrylcsh:RStatinsBiology and Life SciencesComputational Biologynutritional and metabolic diseasesMyalgiaCell Biologymedicine.disease030104 developmental biologyEndocrinologyGene Expression RegulationSkeletal MusclesLeukocytes Mononuclearbiology.proteinProtein prenylationlcsh:QHydroxymethylglutaryl-CoA Reductase InhibitorsSLCO1B1businessPLOS ONE
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Management of allergic disease in the elderly: key considerations, recommendations and emerging therapies

2015

The number of people over 65 is increasing around the world. At present, between 5 and 10% of allergic diseases affect the elderly. In particular, rhinitis is increasing worldwide; the presence of high comorbidity makes the therapy of asthma even more complicated. With reference to dermatological allergies, the dryness of the skin favors the onset of allergic contact and atopic dermatitis, while the senescence of mucous membranes and the impaired secretion of polymeric IgA could be linked to food allergy. Overcoming the problem of adverse drug reaction is limited by the diagnostic difficulty in patients taking multiple drugs. In addition, some drugs, such as β-blockers, angiotensin-converti…

MaleAgingAllergymedicine.medical_specialtyAIT-allergen immunotherapyHealth Services for the AgedImmunologyDrug allergydermatological allergieanaphylaxiDiseaseSettore MED/10 - Malattie Dell'Apparato RespiratorioelderlyFood allergyHypersensitivitymedicineHumansImmunology and AllergyAsthmaimmunosenescencefood allergybusiness.industryMedicine (all)Atopic dermatitismedicine.diseaseDermatologyPatient ComplianceFemaleallergic rhinitibusinessallergic asthmadrug allergyAnaphylaxisAdverse drug reactionHumanExpert Review of Clinical Immunology
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Sicilian centenarian offspring are more resistant to immune ageing

2018

Background: Immunosenescence constitutes a major indirect cause of morbidity and mortality in the elderly. Previous analysis of immune signatures in a cohort of centenarian offspring showed an intermediate immunophenotype between age-matched and younger controls. Aims: To confirm and extend the previous studies performing further phenotypical analysis in centenarian offspring and controls. Methods: Analysis of Treg cells, γδ T cells, mucosal-associated invariant T cells, and senescent immune T cells was performed in centenarian offspring and controls. Results: We report significant differences between elderly and centenarian offspring in most of the studied subsets, showing that centenarian…

MaleAgingOffspringImmunosenescenceLongevityBiology03 medical and health sciences0302 clinical medicineImmunophenotypingImmune systemT-Lymphocyte SubsetsHumans030212 general & internal medicineCentenarianFlow cytometrySicilyAgedAged 80 and overInflammationSettore MED/04 - Patologia GeneraleImmunity CellularCase-control studyImmunosenescenceImmune senescenceMiddle AgedAgeingAgeingCase-Control StudiesCohortImmunologyAdult ChildrenFemaleCentenarianGeriatrics and Gerontology030217 neurology & neurosurgery
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Is the Mean Blood Leukocyte Telomere Length a Predictor for Sporadic Thoracic Aortic Aneurysm? Data from a Preliminary Study

2012

Telomeres have been postulated as a universal clock that shortens in parallel with cellular aging. They are specialized DNA-protein structures at the ends of chromosome with remarkable functions--preventing their recognition as double-stranded DNA breaks, protecting their recombination and degradation, and avoiding a DNA damage cellular response. Telomere shortening is currently considered the best aging marker, but is also a predictor for age-related diseases, including cardiovascular diseases. Biological age clearly seems to be a better predictor of vascular risk rather than chronological age. This concept is supported by key assumptions that peripheral blood leukocyte telomere content ac…

MaleAgingPathologymedicine.medical_specialtyThoracicBiological ageVascular riskBiologyBioinformaticsThoracic aortic aneurysmGeneticLeukocytesmedicineHumansSettore MED/05 - Patologia ClinicaAged; Aging; Aortic Aneurysm Thoracic; Case-Control Studies; Cellular Senescence; DNA; DNA Damage; Female; Humans; Leukocytes; Male; Middle Aged; Recombination Genetic; Telomere; Vascular DiseasesVascular DiseasesCellular Senescencevascular ageingAgedRecombination GenetictelomereAortic Aneurysm ThoracicVascular diseaseChromosomeSettore MED/23 - Chirurgia CardiacaDNAMiddle Agedmedicine.diseaseRecombinationPeripheral bloodAortic AneurysmTAATelomereCellular AgingCase-Control StudiesFemaleGeriatrics and GerontologyDNA DamageRejuvenation Research
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HLA, aging, and longevity: a critical reappraisal.

2000

Despite a large number of studies, available data do not allow at present to reach definitive and clear conclusions on role of HLA on longevity, owing to major methodological problems, such as serological and molecular typing of different loci, insufficient sample sizes, different inclusion criteria and age cut-off, inappropriate mixing of data referred to people from 58 to over 100 years of age, inappropriate control matching, and neglected consideration of sex-related effects and the different genetic make-up of studied populations. However, within this confused scenario, some data emerge. First, two studies that do not fit the biases above discussed show that some HLA alleles are associa…

MaleAgingmedia_common.quotation_subjectImmunologyLongevityHuman leukocyte antigenMajor histocompatibility complexEvolution of ageingHLA-B8 AntigenHLA-DR3 AntigenPleiotropyHLA AntigensImmunology and AllergyHumansAllelemedia_commonAgedGeneticsAged 80 and overbiologyHaplotypeHomozygoteLongevityGeneral MedicineImmunosenescenceHaplotypesbiology.proteinFemaleHuman immunology
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Sustained telomere erosion due to increased stem cell turnover during triple autologous hematopoietic stem cell transplantation.

2007

Telomeres cap chromosomal ends and are shortened throughout a lifetime. Additional telomere erosion has been documented during conventional chemotherapy or hematopoietic stem cell transplantation. Previous studies of stem cell transplantation reported variable amounts of telomere shortening with inconsistent results regarding the persistence of telomere shortening. Here we have prospectively studied telomere length and proliferation kinetics of hematopoietic cells in aggressive non-Hodgkin lymphoma patients who underwent a four-course high-dose chemotherapy protocol combined with triple autologous stem cell transplantation. We observed sustained telomere shortening in hematopoietic cells af…

MaleCancer ResearchTransplantation Conditioningmedicine.medical_treatmentHematopoietic stem cell transplantationAntibodies Monoclonal Murine-DerivedAutologous stem-cell transplantationAntineoplastic Combined Chemotherapy ProtocolsGranulocyte Colony-Stimulating FactorLymphocytesProspective StudiesCellular SenescenceEtoposideMyelopoiesisLymphoma Non-HodgkinAntibodies MonoclonalHematologyMiddle AgedTelomereCombined Modality TherapyHaematopoiesisVincristineFemaleStem cellRituximabCell DivisionPrednisoloneTransplantation AutologousDrug Administration ScheduleGeneticsmedicineHumansMolecular BiologyCyclophosphamideChemotherapyPeripheral Blood Stem Cell Transplantationbusiness.industryCell BiologyMyeloablative Agonistsmedicine.diseaseHematopoietic Stem CellsTelomereLymphomaTransplantationClinical Trials Phase III as TopicDoxorubicinImmunologyCancer researchbusinessGranulocytesExperimental hematology
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