Search results for "SYNERGISM"

showing 10 items of 153 documents

Plexin-B1 and Semaphorin 4D Cooperate to Promote Perineural Invasion in a RhoA/ROK-Dependent Manner

2012

Perineural invasion (PNI) is a tropism of tumor cells for nerve bundles located in the surrounding stroma. It is a pathological feature observed in certain tumors, referred to as neurotropic malignancies, that severely limits the ability to establish local control of disease and results in pain, recurrent growth, and distant metastases. Despite the importance of PNI as a prognostic indicator, its biological mechanisms are poorly understood. The semaphorins and their receptors, the plexins, compose a family of proteins originally shown to be important in nerve cell adhesion, axon migration, and proper central nervous system development. Emerging evidence has demonstrated that these factors a…

Nervous systemPathologymedicine.medical_specialtyCell typeanimal structuresRHOANervous System NeoplasmsTransplantation HeterologousPerineural invasionRetraction NoticeMice NudeNerve Tissue ProteinsReceptors Cell SurfaceSemaphorinsPathology and Forensic Medicine03 medical and health sciencesMice0302 clinical medicineSemaphorinAntigens CDCell MovementCell Line TumorSettore BIO/10 - BiochimicamedicineAnimalsHumansNeoplasm InvasivenessAxonRNA Small InterferingCell adhesion030304 developmental biologyMice Knockout0303 health sciencesbiologyDrug SynergismAxonsTransplantationMice Inbred C57BLmedicine.anatomical_structure030220 oncology & carcinogenesisembryonic structuresbiology.proteinCancer researchperineural invasion tumor cells Rho kinase-dependent manner plexin B1rhoA GTP-Binding ProteinNeoplasm TransplantationSignal TransductionThe American Journal of Pathology
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Untersuchungen zum Mechanismus der blutdrucksteigernden Wirkung von Mineralokortikoiden

1973

1. Es wird uber Untersuchungen zu der Frage berichtet, ob die blutdrucksteigernde Wirkung von Mineralokortikoiden mit einer Anderung der Noradrenalininaktivierung in Zusammenhang steht.

Norepinephrine (medication)Pregnanetriolchemistry.chemical_compoundChemistryDrug DiscoverymedicineMolecular MedicineGeneral MedicineVasoconstrictor AgentsPharmacologyGenetics (clinical)Drug synergismmedicine.drugKlinische Wochenschrift
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Endogenous AhR agonist FICZ accumulates in rainbow trout (Oncorhynchus mykiss) alevins exposed to a mixture of two PAHs, retene and fluoranthene

2022

AbstractMultiple studies have reported synergized toxicity of PAH mixtures in developing fish larvae relative to the additive effect of the components. From a toxicological perspective, multiple mechanisms are known to contribute to synergism, such as altered toxicodynamics and kinetics, as well as increased oxidative stress. An understudied contributor to synergism is the accumulation of endogenous metabolites, for example: the aryl hydrocarbon receptor 2 (AhR2) agonist and tryptophan metabolite 6-Formylindolo(3,2-b)carbazole (FICZ). Fish larvae exposed to FICZ, alongside knock-down of cytochrome p450 (cyp1a), has been reported to induced symptoms of toxicity similar to those observed foll…

PAH-yhdisteetHealth Toxicology and MutagenesisGeneral MedicinePAHManagement Monitoring Policy and LawToxicologymixtureekotoksikologiaFICZReceptors Aryl HydrocarbonkirjolohiLarvasynergismOncorhynchus mykissAnimalsblue sac diseaseaineenvaihduntatuotteetympäristömyrkytalkionkehitysPolycyclic Aromatic Hydrocarbonskalat
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Opioid poorly-responsive cancer pain. Part 3. Clinical strategies to improve opioid responsiveness.

2001

Some pain syndromes may be difficult to treat due to a poor response to opioids. This situation demands a range of alternative measures, including the use of adjuvant drugs with independent effects, such as antidepressants, sodium channel-blocking agents, steroids and anti-inflammatory drugs (NSAIDs); drugs that reduce opioid side effects; and drugs that enhance analgesia produced by opioids, such as N-methyl-D-aspartate (NMDA) antagonists, calcium channel antagonists, and clonidine. Other approaches, including opioid trials, neural blockade when necessary, and psychological interventions, also may be useful.

Palliative carebusiness.industrymedicine.medical_treatmentCalcium channelAnalgesicPalliative CareDrug ResistanceDrug SynergismBioinformaticsClonidineAnalgesics OpioidAnesthesiology and Pain MedicineOpioidAnesthesiaNeoplasmsNeuropathic painMedicineHumansNeurology (clinical)businessCancer painAdjuvantGeneral Nursingmedicine.drugJournal of pain and symptom management
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Exploratory study on the effects of biodegradable nanoparticles with drugs on malignant B cells and on a human/mouse model of Burkitt lymphoma.

2010

The aim of this study was to determine if Rituximab coated Biodegradable Nanoparticles (BNPs) loaded with Chlorambucil and Hydroxychloroquine could induce apoptosis of B-Chronic Lymphocytic Leukemia (B-CLL), MEC-1 and BJAB cells in vitro and evaluate their toxic and therapeutic effects on a Human/Mouse Model of Burkitt Lymphoma at an exploratory, proof of concept scale. We found that Rituximab-Chlorambucil-Hydroxychloroquine BNPs induce a decrease in cell viability of malignant B cells in a dose-dependent manner. The mediated cytotoxicity resulted from apoptosis, and was confirmed by monitoring the B-CLL cells after Annexin V/propidium iodide staining. Additional data revealed that these BN…

Pathologymedicine.medical_specialtyCell Survivalhuman/mouse model of Burkitt lymphoma.human lymphomamodel SCID mouseAntineoplastic Agentschemistry.chemical_compoundAntibodies Monoclonal Murine-DerivedMicerituximabimmune system diseasesAnnexinhemic and lymphatic diseasesnanoparticles; rituximab; human lymphoma; model SCID mouseTumor Cells CulturedMedicineAnimalsHumansPharmacology (medical)Propidium iodideGeneral Pharmacology Toxicology and PharmaceuticsCytotoxicityB-LymphocytesChlorambucilDose-Response Relationship Drugbusiness.industrymalignant B cellnanoparticleDrug SynergismGeneral MedicineBiodegradable nanoparticles with drugmedicine.diseaseBurkitt LymphomaLymphomaMice Inbred C57BLLeukemiaDisease Models AnimalDrug CombinationschemistryApoptosisMonoclonalCancer researchNanoparticlesChlorambucilbusinessRituximabmedicine.drugHydroxychloroquine
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Synergy assessment of fixed combinations of Herba Andrographidis and Radix Eleutherococci extracts by transcriptome-wide microarray profiling

2015

Abstract Background Generally accepted, but insufficiently proved, the concept of synergy is based on an assumption that combining of two biologically active substances is justified because the combination is more active and less harmful than the ingredients. Hypothesis Analysis of RNA microarray of isolated neuroglia cells and the comparison the number of genes deregulated by plant extracts and their fixed herbal formulation might be a useful tool/method for assessment of synergistic and antagonistic interactions of herbal extracts in human organism. Aim The primary aim of this study was to extend a new method of assessment of synergistic and antagonistic interactions of herbal extracts in…

Pharmaceutical ScienceEleutherococcusComputational biologyPharmacologyBiologyVenn diagramsPlant RootsCell LineTranscriptomeInterferonAcanthaceaeDrug DiscoverymedicineHumansAraliaceaeGenePharmacologyRegulation of gene expressionPlant ExtractsMicroarray analysis techniquesGene Expression ProfilingDrug SynergismMicroarray AnalysisFold changeGene expression profilingSynergyGene Expression RegulationComplementary and alternative medicineMolecular MedicineAndrographisSignal transductionPharmacogenomicsNeurogliaNetwork pharmacologymedicine.drugPhytomedicine
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Influence of carbenoxolone on the anticonvulsant efficacy of conventional antiepileptic drugs against audiogenic seizures in DBA/2 mice

2004

Carbenoxolone, the succinyl ester of glycyrrhetinic acid, is an inhibitor of 11beta-hydroxy steroid dehydrogenase and gap junctional intercellular communication. It is currently used in clinical treatment of ulcer diseases. Systemic administration of carbenoxolone (1-40 mg/kg, intraperitoneally (i.p.)) was able to produce a dose-dependent decrease in DBA/2 audiogenic seizure severity score. Glycyrrhizin, an analogue of carbenoxolone inactive at the gap-junction level, was unable to affect audiogenic seizures at doses up to 30 mg/kg. In combination with conventional antiepileptic drugs, carbenoxolone, 0.5 mg/kg, i.p., which per se did not significantly affect the occurrence of audiogenic sei…

PhenytoinAudiogenic seizureGap junctionmedicine.medical_treatmentCarbenoxoloneMotor ActivityLamotriginePharmacologyEpilepsy ReflexFelbamateMicemedicineAnimalsAnticonvulsant potencyPharmacologyValproateEpilepsybusiness.industryDrug SynergismCarbamazepineFelbamateCarbamazepineAnticonvulsantAcoustic StimulationMice Inbred DBAPhenytoinDBA/2CarbenoxoloneAnticonvulsantsPhenobarbitalbusinessDiazepamAntiepileptic drugmedicine.drug
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Possible protective role for C-reactive protein in atherogenesis: complement activation by modified lipoproteins halts before detrimental terminal se…

2004

Background—Previous work indicated that enzymatically remodeled LDL (E-LDL) might activate complement in atherosclerotic lesions via a C-reactive protein (CRP)–dependent and CRP-independent pathway. We sought to substantiate this contention and determine whether both pathways drive the sequence to completion.Methods and Results—E-LDL was prepared by sequential treatment of LDL with a protease and cholesteryl esterase. Trypsin, proteinase K, cathepsin H, or plasmin was used with similar results. Functional tests were used to assess total complement hemolytic activity, and immunoassays were used to demonstrate C3 cleavage and to quantify C3a, C4a, C5a, and C5b-9. E-LDL preparations activated …

PlasminArteriosclerosisLipoproteinsCathepsin HPhysiology (medical)EndopeptidasesmedicineHumansComplement ActivationbiologyC-reactive proteinC4ADrug SynergismComplement System ProteinsSterol EsteraseProteinase KTrypsinImmunohistochemistryComplement systemLipoproteins LDLC-Reactive ProteinBiochemistrybiology.proteinCardiology and Cardiovascular MedicineLipoproteinmedicine.drugCirculation
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SPIONs embedded in polyamino acid nanogels to synergistically treat tumor microenvironment and breast cancer cells.

2018

Abstract The extremely complex tumor microenvironment (TME) in humans is the major responsible for the therapeutic failure in cancer nanomedicine. A new concept of disease-driven nanomedicine, henceforth named “Theranomics”, which attempts to target cancer cells and TME on the whole, represents an attractive alternative. Herein, a nanomedicine able to co-deliver doxorubicin and a tumor suppressive proteolytic protein such as collagenase-2 was developed. We successfully obtained superparamagnetic nanogels (SPIONs/Doco@Col) via the intermolecular azide-alkyne Huisgen cycloaddition. We demonstrated that a local ECM degradation and remodeling in solid tumors by means of collagenase-2 could enha…

Polyamino acidPolyamino acidsCollagenasePharmaceutical ScienceBreast Neoplasms02 engineering and technology030226 pharmacology & pharmacy03 medical and health sciences0302 clinical medicineBreast cancerBreast cancerDrug Delivery SystemsCell Line TumormedicineTumor MicroenvironmentHumansDoxorubicinTargeted cancer therapyAmino AcidsMagnetite NanoparticlesTumor microenvironmentAntibiotics AntineoplasticChemistrySPIONCancerTheranomicDrug Synergism021001 nanoscience & nanotechnologymedicine.diseasenanomedicineNanomedicinesDrug LiberationSPIONsMatrix Metalloproteinase 8DoxorubicinCancer cellCancer researchNanomedicineTheranomicsFemaleBreast cancer cellspolyamino acid0210 nano-technologyGelsmedicine.drugInternational journal of pharmaceutics
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Pre-treatment with high doses of cocaine decreases the reinforcing effects of cocaine in the conditioned place preference paradigm.

2012

The aim of the present study was to determine if pre-exposure to high doses of cocaine can subsequently alter the rewarding effects of this drug. Adult male mice received a pretreatment of physiological saline, or 12.5 or 25 mg/kg of cocaine (one injection a day for five days). After an interval of six days without injections, the rewarding effects of low doses of cocaine (0.5, 1 or 1.5 mg/kg) were evaluated in the conditioned place preference (CPP) paradigm. Doses of 1 and 1.5 mg/kg induced a clear CPP in animals pre-treated with saline but were ineffective in those pre-treated with 25 mg/kg of cocaine. Only the dose of 1.5 mg/kg induced CPP in mice pre-treated with 12.5 mg/kg of cocaine. …

Pre treatmentMaleAdult maleDose-Response Relationship Drugbusiness.industryGeneral Neurosciencemedicine.medical_treatmentLow doseConditioning ClassicalDrug SynergismDrug TolerancePharmacologyConditioned place preferenceMiceCocaineDrug toleranceHigh dosesMedicineAnimalsbusinessSalinePhysiological salineNeuroscience letters
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