Search results for "Shedding"

showing 10 items of 52 documents

Microbiota Depletion Promotes Human Rotavirus Replication in an Adult Mouse Model

2021

Intestinal microbiota-virus-host interaction has emerged as a key factor in mediating enteric virus pathogenicity. With the aim of analyzing whether human gut bacteria improve the inefficient replication of human rotavirus in mice, we performed fecal microbiota transplant (FMT) with healthy infants as donors in antibiotic-treated mice. We showed that a simple antibiotic treatment, irrespective of FMT, resulted in viral shedding for 6 days after challenge with the human rotavirus G1P[8] genotype Wa strain (RVwa). Rotavirus titers in feces were also significantly higher in antibiotic-treated animals with or without FMT but they were decreased in animals subject to self-FMT, where a partial re…

Rotavirus0301 basic medicinePermissivenessmiceQH301-705.5Virus RNA030106 microbiologyPopulationMicrobiologiaMedicine (miscellaneous)Antibiòticsmedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyVirusArticlevirus sheddingMicrobiologyMice03 medical and health sciencesAntigenAntibioticsRotavirusLactobacillusantibioticmedicinemicrobiotaBiology (General)Viral sheddingeducationFecesInfectivityeducation.field_of_studyInnate immune systembiologyMicrobiotaVirus sheddingbiology.organism_classificationSmall intestine030104 developmental biologymedicine.anatomical_structurerotavirusBiomedicines
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Evaluation of reverse transcription and polymerase chain reaction (RT/PCR) for the detection of rotaviruses: applications of the assay.

1996

Summary Our aim was to evaluate the reverse transcription and polymerase chain reaction (RT/PCR) technique for the detection of rotavirus shedding by infected children as a routine diagnostic procedure, in comparison to the enzyme-linked immunosorbent assay (ELISA), electron microscopy (EM) and polyacrylamide gel etectrophoresis (PAGE) of rotavirus double stranded RNA. Two-hundred and twenty stool specimens were collected from infants and young children with diarrhoea, and 10–20% faecal suspensions were made. Several methods of rotavirus dsRNA extraction were assayed. Electrophoretic analysis of viral RNA was carried out on 10% polyacrylamide gols followed by silver staining. RT/PCR was per…

RotavirusRT/PCRTranscription GeneticImmunologyReoviridaeEnzyme-Linked Immunosorbent AssayMicroscopic électroniquemedicine.disease_causeSensibilité.Polymerase Chain ReactionSensitivity and SpecificityRotavirus InfectionsArticlelaw.inventionFecesfluids and secretionsSensitivitylawVirologyRotavirusViral analysismedicineElectron microscopyHumansTypingChildRotavirus RT/PCRPolymerase chain reactionbiologyRNAInfantbiology.organism_classificationVirologyMolecular biologyReverse transcriptaseVirus SheddingPAGEMicroscopy ElectronReal-time polymerase chain reactionEvaluation Studies as TopicAnalyse viraleRNA ViralElectrophoresis Polyacrylamide GelELISARNA extractionResearch in virology
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Inhibitory Activities of Bovine Macromolecular Whey Proteins on Rotavirus Infections In Vitro and In Vivo

2006

Rotavirus is a major cause of infantile viral gastroenteritis and can lead to severe and sometimes lethal dehydration. Previous studies have shown that breast-fed children are better protected against symptomatic infections, and that the milk fat globule protein lactadherin might be at least partly responsible for this effect. In vitro studies have shown that human lactadherin, in contrast to the bovine ortholog, could inhibit rotavirus infectivity, and that bovine MUC1 and a commercially available bovine macromolecular whey protein (MMWP) fraction proved to be effective. The present work describes the versatility of MMWP against the infection of 2 human intestinal cell lines (Caco-2 and FH…

RotavirusWhey proteinvirusesmedicine.medical_treatmentPassive immunityBiologyAntibodies Viralmedicine.disease_causeAntiviral AgentsRotavirus InfectionsVirusCell LineMicrobiologyMicefluids and secretionsRotavirusGeneticsmedicineAnimalsHumansViral sheddingLactadherinInfectivityMice Inbred BALB CSulfhydryl ReagentsMilk ProteinsVirologyDisease Models AnimalWhey ProteinsColostrumCattleAnimal Science and ZoologyCaco-2 CellsFood Science
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Enterovirus Co-infections and Onychomadesis after Hand, Foot, and Mouth Disease, Spain, 2008

2011

Mixed infection of enteroviruses may explain the rare complication of nail shedding. Onychomadesis after HFMD

SerotypeMaleEpidemiologyviruseslcsh:Medicinemedicine.disease_causeCommunicable Diseases Emergingskin and connective tissue diseasesPhylogenybiologyintegumentary systementerovirusvirus diseasesEnterovirus B HumanInfectious DiseasesChild Preschoolnail sheddingFemaleFoot (unit)Microbiology (medical)complicationsCoxsackievirusOnychomadesislcsh:Infectious and parasitic diseasesNail Diseasesco-infectionstomatognathic systemmedicineEnterovirus InfectionsHumansviruseslcsh:RC109-216TypingSerotypingonychomadesisBase SequenceResearchlcsh:ROutbreakInfantand mouth diseasebiology.organism_classificationmedicine.diseaseVirologyHFMDEnterovirus A HumanNail diseaseSpainCase-Control StudiesfootDNA ViralEnterovirushandHand Foot and Mouth DiseaseEmerging Infectious Diseases
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Strategies to Target ADAM17 in Disease: From Its Discovery to the iRhom Revolution

2021

For decades, disintegrin and metalloproteinase 17 (ADAM17) has been the object of deep investigation. Since its discovery as the tumor necrosis factor convertase, it has been considered a major drug target, especially in the context of inflammatory diseases and cancer. Nevertheless, the development of drugs targeting ADAM17 has been harder than expected. This has generally been due to its multifunctionality, with over 80 different transmembrane proteins other than tumor necrosis factor α (TNF) being released by ADAM17, and its structural similarity to other metalloproteinases. This review provides an overview of the different roles of ADAM17 in disease and the effects of its ablation in a n…

TIMPsEGFRiRhomsTNFAnti-Inflammatory AgentsPharmaceutical ScienceInflammationContext (language use)Antineoplastic AgentsDiseaseComputational biologyReviewADAM17 ProteinmetalloproteinasesAnalytical Chemistrylcsh:QD241-44103 medical and health sciences0302 clinical medicineImmune systemlcsh:Organic chemistryIn vivoNeoplasmsDrug DiscoverymedicineDisintegrinTIMPADAM17 ProteinAnimalsHumansPhysical and Theoretical Chemistry030304 developmental biologyInflammation0303 health sciencesADAM17biologyOrganic ChemistryIntracellular Signaling Peptides and ProteinsiRhomChemistry (miscellaneous)030220 oncology & carcinogenesisbiology.proteinADAM17; Ectodomain shedding; EGFR; IRhoms; Metalloproteinases; TIMPs; TNF; ADAM17 Protein; Animals; Anti-Inflammatory Agents; Antineoplastic Agents; Humans; Inflammation; Intracellular Signaling Peptides and Proteins; NeoplasmsMolecular MedicineTumor necrosis factor alphametalloproteinaseectodomain sheddingmedicine.symptomMolecules
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Una tabla inédita de Fernando Yáñez y nueva luz sobre su estancia en Almedina (1518-1525)

2020

The contribution presents a new acquisition for the catalog of Fernando Yáñez, a Holy Family dating back to 1523, shedding new light on the artist?s production in the third decade of the sixteenth century during his return to Almedina, his birthplace, in a time frame that divides the works for Valencia, like the retablo mayor for the city?s cathedral, from his altarpieces for Cuenca.

Tomasso The contribution presents a new acquisition for the catalog of Fernando Yáñezlike the retablo mayor for the city?s cathedralUNESCO::CIENCIAS DE LAS ARTES Y LAS LETRAS:CIENCIAS DE LAS ARTES Y LAS LETRAS [UNESCO]0211-5808 9678 Archivo de arte valenciano 564145 2020 101 7707980 Una tabla inédita de Fernando Yáñez y nueva luz sobre su estancia en Almedina (1518-1525) Mozzatihis birthplaceshedding new light on the artist?s production in the third decade of the sixteenth century during his return to Almedinaa Holy Family dating back to 1523from his altarpieces for Cuenca. 115 126in a time frame that divides the works for Valencia
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ASTROCYTES SHED EXTRACELLULAR VESICLES THAT CONTAIN FIBROBLAST GROWTH FACTOR-2 AND VASCULAR ENDOTHELIAL GROWTH FACTOR.

2007

An important component of the pathogenic process of multiple sclerosis (MS) is the blood-brain barrier (BBB) damage. We recently set an in vitro model of BBB, based on a three-cell-type co-culture system, in which rat neurons and astrocytes synergistically induce brain capillary endothelial cells to form a monolayer with permeability properties resembling those of the physiological BBB. Herein we report that the serum from patients with secondary progressive multiple sclerosis (SPMS) has a damaging effect on isolated neurons. This finding suggests that neuronal damaging in MS could be a primary event and not only secondary to myelin damage, as generally assumed. SPMS serum affects the perme…

Vascular Endothelial Growth Factor ACellFluorescent Antibody TechniqueBiologyFibroblast growth factorCulture Media Serum-Freechemistry.chemical_compoundWestern blotSettore BIO/10 - BiochimicaGlial Fibrillary Acidic ProteinGeneticsmedicineAnimalsSecretionFibroblastCells Culturedmedicine.diagnostic_testVesicleIntegrin beta1Secretory VesiclesGeneral MedicineCell biologyRatsVascular endothelial growth factorastrocytesextracellular vesicle sheddingfibroblastic growth factors-2Protein Transportmedicine.anatomical_structureMembrane proteinchemistryAstrocytesFibroblast Growth Factor 2
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NEURONS PRODUCE FGF-2 AND VEGF SECRETE THEM AT LEST IN PART BY SHEDDING EXTRACELLULAR VESCICLES

2007

Abstract We previously found that neurons are able to affect the ability of brain capillary endothelial cells to form in vitro a monolayer with properties resembling the blood-brain barrier. We then looked, by immunofluorescence and western analysis, for factors, produced by neurons, with the potential to influence growth and differentiation of endothelial cells. In the present paper, we report that neurons produce both vascular endothelial growth factor and fibroblast growth factor 2, two well-known angiogenic factors. More interestingly, we gained evidence that both factors are released by neurons, at least in part, by shedding of extracellular vesicles, that contain β1 integrin, a membra…

Vascular Endothelial Growth Factor AFGF-2BiologyFibroblast growth factorchemistry.chemical_compoundsheddingNeurofilament ProteinsGlial Fibrillary Acidic ProteinExtracellularAnimalsSecretionRats WistarCells CulturedNeuronsVesicleIntegrin beta1Secretory VesiclesCell BiologyArticlesVEGFTransport proteinCell biologyRatsVascular endothelial growth factorVascular endothelial growth factor AProtein TransportMembrane proteinchemistryAstrocytesMolecular Medicineneurons vesicles fibroblastic growth factor-2 vascular endothelial growth factorCamptothecinFibroblast Growth Factor 2Extracellular Spaceextracellular vesicles
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Replication of Human Norovirus in Mice after Antibiotic-Mediated Intestinal Bacteria Depletion

2022

Human noroviruses (HuNoVs) are the main cause of acute gastroenteritis causing more than 50,000 deaths per year. Recent evidence shows that the gut microbiota plays a key role in enteric virus infectivity. In this context, we tested whether microbiota depletion or microbiota replacement with that of human individuals susceptible to HuNoVs infection could favor viral replication in mice. Four groups of mice (n = 5) were used, including a control group and three groups that were treated with antibiotics to eliminate the autochthonous intestinal microbiota. Two of the antibiotic-treated groups received fecal microbiota transplantation from a pool of feces from infants (age 1-3 months) or an au…

Virus RNAMicrobiologiaAntibiòticsDNA RibosomalCatalysisInorganic ChemistryFecesMicenorovirus; antibiotic; microbiota; mice; virus sheddingAnimalsHumansPhysical and Theoretical ChemistryMolecular BiologySpectroscopyCaliciviridae InfectionsInterleukin-13BacteriaTumor Necrosis Factor-alphaMicrobiotaNorovirusOrganic ChemistryAntibioticInfantVirus sheddingGeneral MedicineToll-Like Receptor 2Anti-Bacterial AgentsComputer Science ApplicationsInterleukin-4
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SHEDDING OF ANGIOGENIC FACTORS BY BRAIN CELLS

2007

angiogenic factorsbrain cellsshedding
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