Search results for "Signal Transducing"

showing 10 items of 156 documents

Structural basis of the migfilin-filamin interaction and competition with integrin beta tails.

2008

A link between sites of cell adhesion and the cytoskeleton is essential for regulation of cell shape, motility, and signaling. Migfilin is a recently identified adaptor protein that localizes at cell-cell and cell-extracellular matrix adhesion sites, where it is thought to provide a link to the cytoskeleton by interacting with the actin cross-linking protein filamin. Here we have used x-ray crystallography, NMR spectroscopy, and protein-protein interaction studies to investigate the molecular basis of migfilin binding to filamin. We report that the N-terminal portion of migfilin can bind all three human filamins (FLNa, -b, or -c) and that there are multiple migfilin-binding sites in FLNa. H…

Models MolecularIntegrin beta ChainsMagnetic Resonance SpectroscopyFilaminsIntegrinMolecular ConformationPlasma protein bindingmacromolecular substancesBiologyFilaminLigandsBiochemistryMiceContractile ProteinsFLNAAnimalsHumansCytoskeletonCell adhesionMolecular BiologyActinCytoskeletonDose-Response Relationship DrugMicrofilament ProteinsMechanisms of Signal TransductionSignal transducing adaptor proteinCell BiologyCell biologyCytoskeletal Proteinsbiology.proteinNIH 3T3 CellsCell Adhesion MoleculesProtein BindingThe Journal of biological chemistry
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Mona/Gads SH3C binding to hematopoietic progenitor kinase 1 (HPK1) combines an atypical SH3 binding motif, R/KXXK, with a classical PXXP motif embedd…

2004

Hematopoietic progenitor kinase 1 (HPK1) is implicated in signaling downstream of the T cell receptor. Its non-catalytic, C-terminal half contains several prolinerich motifs, which have been shown to interact with different SH3 domain-containing adaptor proteins in vitro. One of these, Mona/Gads, was also shown to bind HPK1 in mouse T cells in vivo. The region of HPK1 that binds to the Mona/Gads C-terminal SH3 domain has been mapped and shows only very limited similarity to a recently identified high affinity binding motif in SLP-76, another T-cell adaptor. Using isothermal titration calorimetry and x-ray crystallography, the binding of the HPK1 motif to Mona/Gads SH3C has now been characte…

Models MolecularTime FactorsProtein ConformationAmino Acid MotifsMolecular Sequence DataPlasma protein bindingBiologyCalorimetryProtein Serine-Threonine KinasesCrystallography X-RayBiochemistrySH3 domainProtein Structure Secondarysrc Homology DomainsMiceProtein structureAnimalsHumansAmino Acid SequenceMolecular BiologyPeptide sequencePolyproline helixAdaptor Proteins Signal TransducingSequence Homology Amino AcidSignal transducing adaptor proteinIsothermal titration calorimetryCell BiologyPhosphoproteinsCell biologyProtein Structure TertiaryCrystallographyKineticsPXXP MotifCarrier ProteinsPeptidesProtein BindingThe Journal of biological chemistry
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The genetic contribution of the NO system at the glutamatergic post-synapse to schizophrenia: Further evidence and meta-analysis

2013

NO is a pleiotropic signaling molecule and has an important role in cognition and emotion. In the brain, NO is produced by neuronal nitric oxide synthase (NOS-I, encoded by NOS1) coupled to the NMDA receptor via PDZ interactions; this protein-protein interaction is disrupted upon binding of NOS1 adapter protein (encoded by NOS1AP) to NOS-I. As both NOS1 and NOS1AP were associated with schizophrenia, we here investigated these genes in greater detail by genotyping new samples and conducting a meta-analysis of our own and published data. In doing so, we confirmed association of both genes with schizophrenia and found evidence for their interaction in increasing risk towards disease. Our stron…

NOS1Glutamic AcidNitric Oxide Synthase Type IPolymorphism Single NucleotidePrefrontal cortexNitric oxideAssociationSynapsechemistry.chemical_compoundGlutamatergicSDG 3 - Good Health and Well-beingmedicineAnimalsHumansGenetic Predisposition to DiseasePharmacology (medical)Promoter Regions GeneticPrefrontal cortexBiological PsychiatryAdaptor Proteins Signal TransducingPharmacologyGlutamate receptorComputational BiologyNitric oxideCognitionmedicine.diseasePsychiatry and Mental healthPost-synapseNeurologychemistrySchizophreniaSynapsesRC0321SchizophreniaNeurology (clinical)GlutamatePsychologyRC0321 Neuroscience. Biological psychiatry. NeuropsychiatryNeuroscienceSignal TransductionEuropean Neuropsychopharmacology
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Loss of Dishevelleds disrupts planar polarity in ependymal motile cilia and results in hydrocephalus.

2014

Defects in ependymal (E) cells, which line the ventricle and generate cerebrospinal fluid flow through ciliary beating, can cause hydrocephalus. Dishevelled genes (Dvls) are essential for Wnt signaling, and Dvl2 has been shown to localize to the rootlet of motile cilia. Using the hGFAP-Cre;Dvl1(-/-);2(flox/flox);3(+/-) mouse, we show that compound genetic ablation of Dvls causes hydrocephalus. In hGFAP-Cre;Dvl1(-/-);2(flox/flox);3(+/-) mutants, E cells differentiated normally, but the intracellular and intercellular rotational alignments of ependymal motile cilia were disrupted. As a consequence, the fluid flow generated by the hGFAP-Cre;Dvl1(-/-);2(flox/flox);3(+/-) E cells was significant…

Neuroscience(all)Dishevelled ProteinsMice TransgenicBiologyTransgenicArticleMiceEpendymaCell polarityFLOXGeneticsmedicinePsychologyAnimalsCiliaAdaptor Proteins Signal Transducingchemistry.chemical_classificationNeurology & NeurosurgeryGeneral NeuroscienceCiliumSignal TransducingNeurosciencesWnt signaling pathwayAdaptor ProteinsCell PolarityPhosphoproteinsDishevelledCell biologymedicine.anatomical_structurechemistryMotile ciliumCognitive SciencesEpendymaIntracellularHydrocephalusSignal TransductionNeuron
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The association between Mediterranean Diet Score and glucokinase regulatory protein gene variation on the markers of cardiometabolic risk: an analysi…

2014

Consumption of a Mediterranean diet (MD) and genetic variation in the glucokinase regulatory protein (GCKR) gene have been reported to be associated with TAG and glucose metabolism. It is uncertain whether there is any interaction between these factors. Therefore, the aims of the present study were to test the association of adherence to a MD and rs780094 (G>A) SNP in theGCKRgene with the markers of cardiometabolic risk, and to investigate the interaction between genetic variation and MD adherence. We studied 20 986 individuals from the European Prospective Investigation into Cancer (EPIC)-Norfolk study. The relative Mediterranean Diet Score (rMED: range 0–18) was used to assess MD adher…

OncologyMaleMediterranean dietMedicine (miscellaneous)030204 cardiovascular system & hematologyDiet MediterraneanCohort Studies0302 clinical medicineGenotype030212 general & internal medicineProspective StudiesGene–environment interactionProspective cohort studyNutrition and DieteticsGlucokinase regulatory proteinConfoundingDietary Surveys and Nutritional EpidemiologyMiddle AgedFull PapersLipids3. Good healthEnglandCardiovascular DiseasesFemaleAdultRiskmedicine.medical_specialtyBiologyPolymorphism Single Nucleotide03 medical and health sciencesInternal medicineMediterranean dietGenetic variationmedicineSNPHumansGenetic Association StudiesAdaptor Proteins Signal TransducingAgedGlycated HemoglobinCardiometabolic riskEndocrinologyCross-Sectional StudiesApolipoproteinsDiabetes Mellitus Type 2biology.proteinPatient ComplianceGene-Environment InteractionGlucokinase regulatory proteinBiomarkersThe British journal of nutrition
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The giant spectrin βV couples the molecular motors to phototransduction and Usher syndrome type I proteins along their trafficking route.

2013

International audience; Mutations in the myosin VIIa gene cause Usher syndrome type IB (USH1B), characterized by deaf-blindness. A delay of opsin trafficking has been observed in the retinal photoreceptor cells of myosin VIIa-deficient mice. We identified spectrin bV, the mammalian b-heavy spectrin, as a myosin VIIa-and rhodopsin-interacting partner in photoreceptor cells. Spectrin bV displays a polarized distribution from the Golgi apparatus to the base of the outer segment, which, unlike that of other b spectrins, matches the trafficking route of opsin and other phototransduction proteins. Formation of spectrin bV-rhodopsin complex could be detected in the differentiating photoreceptors a…

OpsinRhodopsinLight Signal Transductiongenetic structures[SDV]Life Sciences [q-bio]Cell Cycle Proteinsmacromolecular substancesBiologyMyosinsOpsin transportRetinaMotor protein03 medical and health sciencesMice0302 clinical medicineMyosinotorhinolaryngologic diseasesGeneticsAnimalsHumansSpectrinMolecular BiologyGenetics (clinical)030304 developmental biologyAdaptor Proteins Signal Transducing0303 health sciencesEPB41SpectrinGeneral Medicineeye diseasesCell biologyCytoskeletal ProteinsRhodopsinMyosin VIIabiology.proteinMicrotubule Proteinssense organsUsher Syndromes030217 neurology & neurosurgeryVisual phototransductionHeLa CellsPhotoreceptor Cells VertebrateHuman molecular genetics
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Beneficial Read-Through of aUSH1CNonsense Mutation by Designed Aminoglycoside NB30 in the Retina

2010

PURPOSE. The human Usher syndrome (USH) is the most frequent cause of inherited combined deaf-blindness. USH is clinically and genetically heterogeneous, assigned to three clinical types. The most severe type is USH1, characterized by profound inner ear defects and retinitis pigmentosa. Thus far, no effective treatment for the ophthalmic component of USH exists. The p.R31X nonsense mutation in USH1C leads to a disease causing premature termination of gene translation. Here, we investigated the capability of the novel synthetic aminoglycoside NB30 for the translational read-through of the USH1C-p.R31X nonsense mutation as a retinal therapy option. METHODS. Read-through of p.R31X by three com…

ParomomycinUsher syndromeBlotting WesternNonsense mutationCell Culture TechniquesGene ExpressionCell Cycle ProteinsParomomycinBiologyPharmacologyTransfectionRetinaMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineRetinitis pigmentosaIn Situ Nick-End Labelingotorhinolaryngologic diseasesmedicineAnimalsHumansAdaptor Proteins Signal Transducing030304 developmental biologyGenetics0303 health sciencesRetinaDose-Response Relationship DrugAminoglycosideRetinalmedicine.disease3. Good healthMice Inbred C57BLCytoskeletal ProteinsAminoglycosidesElectroporationHEK293 Cellsmedicine.anatomical_structureMicroscopy FluorescencechemistryCodon NonsenseProtein BiosynthesisGentamicinGentamicins030217 neurology & neurosurgerymedicine.drugInvestigative Opthalmology & Visual Science
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Endothelial Wnt/β-catenin signaling inhibits glioma angiogenesis and normalizes tumor blood vessels by inducing PDGF-B expression

2012

Wnt modulates glioma vascularization by regulating PDGF-B expression.

PathologyAngiogenesisCentral Nervous System NeoplasmsMice0302 clinical medicineImmunology and AllergyWnt Signaling Pathwaybeta Catenin0303 health sciencesbiologyNeovascularization PathologicBrain NeoplasmsWnt signaling pathwayIntracellular Signaling Peptides and ProteinsForkhead Transcription FactorsGliomaProto-Oncogene Proteins c-sis3. Good healthmedicine.anatomical_structureBlood-Brain Barrier030220 oncology & carcinogenesiscardiovascular systemIntercellular Signaling Peptides and ProteinsFemalemedicine.medical_specialtyBeta-cateninEndotheliumImmunologyNotch signaling pathwayMice NudeWnt1 ProteinMural cellArticle03 medical and health sciencesGliomamedicineAnimalsHumansddc:610neoplasms030304 developmental biologyAdaptor Proteins Signal TransducingCalcium-Binding ProteinsMembrane Proteinsmedicine.diseaseXenograft Model Antitumor Assaysnervous system diseasesDKK1Cancer researchbiology.proteinEndothelium VascularNeoplasm Grading
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KLEIP Deficiency in Mice Causes Progressive Corneal Neovascular Dystrophy

2012

PURPOSE. The BTB-kelch protein KLEIP/KLHL20 is an actin binding protein that regulates cell-cell contact formation and cell migration. The aim of our study was to characterize KLEIP’s function in ocular health and disease in mice. METHODS. KLEIP -/- mice were generated, and corneas were examined histologically and stained for keratin-1, loricrin, keratin-12, keratin-14, CD31, LYVE-1, F4/80, E-cadherin, and Ki67. Corneal abrasions were performed after eyelid opening. RESULTS. Corneas of KLEIP þ/þ and KLEIP -/- mice were indistinguishable at birth. After eyelid opening corneal epithelial hyperplasia started to manifest in KLEIP -/- mice, showing a progressive epithelial metaplasia leading to …

Pathologymedicine.medical_specialtyStromal cellGenotypeMice TransgenicCorneal dystrophyReal-Time Polymerase Chain ReactionCorneaMiceCorneal OpacityCorneamedicineAnimalsCorneal NeovascularizationRNA MessengerAdaptor Proteins Signal TransducingCorneal epitheliumintegumentary systembusiness.industryDystrophymedicine.diseaseeye diseasesEpitheliumMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureGene Expression RegulationDisease ProgressionEpithelial MetaplasiaLoricrinsense organsCarrier ProteinsbusinessInvestigative Opthalmology & Visual Science
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The MAVS Immune Recognition Pathway in Viral Infection and Sepsis.

2021

Significance: It is estimated that close to 50 million cases of sepsis result in over 11 million annual fatalities worldwide. The pathognomonic feature of sepsis is a dysregulated inflammatory response arising from viral, bacterial, or fungal infections. Immune recognition of pathogen-associated molecular patterns is a hallmark of the host immune defense to combat microbes and to prevent the progression to sepsis. Mitochondrial antiviral signaling protein (MAVS) is a ubiquitous adaptor protein located at the outer mitochondrial membrane, which is activated by the cytosolic pattern recognition receptors, retinoic acid-inducible gene I (RIG-I) and melanoma differentiation associated gene 5 (M…

PhysiologyClinical BiochemistryBiologyBiochemistrySepsisImmune systemInterferonSepsismedicineAnimalsHumansMolecular BiologyGeneral Environmental ScienceMitochondrial antiviral-signaling proteinAdaptor Proteins Signal TransducingImmune EvasionLGP2Pattern recognition receptorSignal transducing adaptor proteinMDA5Cell Biologymedicine.diseaseForum Review ArticlesVirus DiseasesImmunologyHost-Pathogen InteractionsGeneral Earth and Planetary Sciencesmedicine.drugSignal TransductionAntioxidantsredox signaling
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