Search results for "Smooth"

showing 10 items of 710 documents

Characterization of prejunctional muscarinic autoreceptors in the guinea-pig trachea

1991

1. The effects of ten muscarinic antagonists on electrically evoked [3H]-acetylcholine release and muscle contraction were compared in an epithelium-free preparation of the guinea-pig trachea that had been preincubated with [3H]-choline. 2. The M3-selective antagonists UH-AH 37, 4-diphenyl-acetoxy-N-piperidine methobromide and para-fluorohexahydrosiladiphenidol were more potent in reducing the contractile response than in facilitating the evoked [3H]-acetylcholine release. Hexahydrosiladiphenidol did not discriminate between pre- and postjunctional effects. The rank order of the postjunctional potencies of the ten antagonists as well as the postjunctional pA2 values obtained for hexahydrosi…

medicine.medical_specialtyGuinea PigsIn Vitro TechniquesBiologyCholinechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineMethoctramineAnimalsReceptorPharmacologyParasympatholyticsMuscarinic acetylcholine receptor M3Muscle SmoothMuscarinic acetylcholine receptor M2Receptors MuscarinicAcetylcholineElectric StimulationTracheaEndocrinologychemistryAutoreceptormedicine.symptomAcetylcholineMuscle ContractionResearch Articlemedicine.drugMuscle contractionBritish Journal of Pharmacology
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Inhibitory and excitatory muscarinic receptors modulating the release of acetylcholine from the postganglionic parasympathetic neuron of the chicken …

1992

The effects of muscarinic receptor antagonists on ACh release were studied in the absence or presence of cholinesterase (ChE) inhibition using the isolated perfused chicken heart. Presynaptic inhibitory muscarinic autoreceptor were characterized by determining the potency of various antagonists to enhance [3H]-ACh release evoked by field stimulation (3 Hz, 1 min). The order of potencies was: (±)-telenzepine > atropine > 4-DAMP > silahexocyclium > pirenzepine > hexahydro-siladifenidol > AF-DX 116. The comparison with known pA2 values for M1-, M2- and M3-receptors revealed that the presynaptic autoreceptor meets the criteria of an M1-receptor. Basal, not electrically evoked overflow of unlabe…

medicine.medical_specialtyGuinea PigsMuscarinic AntagonistsInhibitory postsynaptic potentialchemistry.chemical_compoundHeart RateInternal medicineMuscarinic acetylcholine receptormedicineMuscarinic acetylcholine receptor M4AnimalsPharmacologyChemistryMyocardiumHeartMuscle SmoothGeneral MedicinePirenzepineMyocardial ContractionAcetylcholineElectric StimulationAtropineEndocrinologyTelenzepineAutoreceptorCholinesterase InhibitorsChickensAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Increase by NO synthase inhibitors of acetylcholine release from guinea-pig myenteric plexus

1994

The effects of nitric oxide (NO) synthase inhibitors on the electrically evoked release of [3H]acetylcholine were studied in guinea-pig myenteric plexus preparations preincubated with [3H]choline. NG-monomethyl-L-arginine (EC50 5.3 mumol l-1) and NG-nitro-L-arginine (EC50 1.3 mumol l-1) concentration-dependently increased the evoked release of [3H]acetylcholine without affecting the basal outflow. The facilitatory effect of NG-mono-methyl-L-arginine was prevented by L-arginine but not by D-arginine. The results suggest that endogenous NO inhibits the depolarisation-evoked release of acetylcholine.

medicine.medical_specialtyGuinea PigsMyenteric PlexusArginineNitric OxideNitroarginineNitric oxideGuinea pigchemistry.chemical_compoundInternal medicinemedicineAnimalsCholineEvoked PotentialsMyenteric plexusPharmacologyomega-N-MethylargininebiologyMuscle SmoothGeneral MedicineAcetylcholineElectric StimulationNitric oxide synthaseEndocrinologychemistryEnzyme inhibitorbiology.proteinLiberationAmino Acid OxidoreductasesNitric Oxide SynthaseAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Two types of neuronal muscarine receptors modulating acetylcholine release from guinea-pig myenteric plexus

1985

Longitudinal muscle strips of the guinea-pig ileum were incubated with [3H]choline and the effects of muscarinic agonists on smooth muscle contraction and on spontaneous and electrically-evoked outflow of tritium were studied. Muscarine and pilocarpine concentration-dependently increased both muscle contraction and spontaneous outflow of [3H]ACh, and inhibited the electrically-evoked outflow of [3H]ACh. The increase in spontaneous outflow was prevented by tetrodotoxin and scopolamine, but not by hexamethonium. Oxotremorine (1-100 microM) did not increase the spontaneous outflow of tritium. Pirenzepine in concentrations of 10 and 100 nM hardly affected the muscle contractions induced by pilo…

medicine.medical_specialtyGuinea PigsScopolamineMyenteric PlexusIn Vitro Techniqueschemistry.chemical_compoundIleumMuscarineInternal medicineMuscarinic acetylcholine receptormedicineOxotremorineAnimalsPharmacologyBenzodiazepinonesMuscarineOxotremorinePilocarpinePirenzepineGeneral MedicineSmooth muscle contractionReceptors MuscarinicPirenzepineAcetylcholineEndocrinologychemistrycardiovascular systemHexamethoniummedicine.symptomAcetylcholineMuscle ContractionMuscle contractionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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LEUKOTRIENE RECEPTORS ON HUMAN PULMONARY VASCULAR ENDOTHELIUM

1995

1. Cysteinyl-leukotrienes cause contractions and/or relaxations of human isolated pulmonary vascular preparations. Although, the localization and nature of the receptors through which these effects are mediated have not been fully characterized, some effects are indirect and not mediated via the well-described LT1 receptor. 2. In human pulmonary veins (HPV) with an intact endothelium, leukotriene D4 (LTD4) induced contraction above basal tone. This response was observed at lower concentrations of LTD4 in the presence of nitric oxide synthase inhibitor N omega-nitro-L-arginine (L-NOARG). Contractions (in the absence and presence of L-NOARG) were partially blocked by the LT1 antagonists (MK 5…

medicine.medical_specialtyLeukotriene D4EndotheliumVasodilationPulmonary ArteryArginineNitroarginineMuscle Smooth VascularNitric oxideLeukotriene D4Nitroargininechemistry.chemical_compoundNorepinephrineInternal medicinemedicineHumansEnzyme InhibitorsReceptorPharmacologyReceptors LeukotrieneLeukotrieneAnalysis of Variancebiologyrespiratory systemNitric oxide synthaseVasodilationmedicine.anatomical_structureEndocrinologychemistryPulmonary VeinsVasoconstrictionbiology.proteinlipids (amino acids peptides and proteins)Endothelium VascularResearch Article
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Bronchodilator and anti-inflammatory activities of SCA40: studies in human isolated bronchus, human eosinophils, and in the guinea-pig in vivo.

1998

There is currently interest in the use of inhibitors of cyclic nucleotide phosphodiesterases (PDE) as potential anti-asthma agents. In this study we examined the effects of SCA40 (6-bromo-8-methylaminoimidazol-[1,2-a] pyrazine-2-carbonitrile), a preferential inhibitor of PDE 3 also endowed with PDE 4 and 5 inhibitory activities, on isolated bronchus and eosinophil functions and in an animal model of asthma. SCA40 (1 nM-0.1 mM) produced concentration-dependent inhibition of spontaneous and stimulated tone of human isolated bronchus and reached a maximal relaxation similar to that of theophylline (3 mM). The potency (-log EC50 values) of SCA40 against spontaneous tone (6.52 +/- 0.10) was grea…

medicine.medical_specialtyMuscle RelaxationGuinea PigsBronchiIn Vitro Techniqueschemistry.chemical_compoundIn vivoInternal medicinemedicineAnimalsHumansTheophyllineAntigensRolipramPharmacologyLeukotrieneLeukotriene C4Anti-Inflammatory Agents Non-SteroidalImidazolesMuscle SmoothGeneral MedicineEosinophilLeukotriene C4Bronchodilator AgentsEosinophilsmedicine.anatomical_structureEndocrinologychemistryPyrazinesBronchoconstrictionmedicine.symptomBronchial HyperreactivityHistaminemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Intervention of two voltage-dependent calcium-entry pathways in the contractile response to acetylcholine and KCl in rat uterus.

1994

The contractile response of rat uterine smooth muscle was investigated. Verapamil and diltiazem concentration-dependently relax the sustained contractions induced by KCl (56 mmol/l) or acetylcholine (10(-4) mol/l). This inhibitory effect was not not freely reversed by washing the tissue and subsequently no contractile response was obtained in depolarized tissue, but a lower biphasic response (phasic and tonic) to acetylcholine was observed. Addition of cumulative concentrations of CaCl2 (1.2-19.2 mmol/l) induced a partial recovery of the contractile response to acetylcholine or KCl, but addition of MgCl2 (1.2-19.2 mmol/l did not. When the channel was reactivated by a third addition of KCl o…

medicine.medical_specialtyMuscle RelaxationTonic (physiology)Membrane PotentialsPotassium ChlorideDiltiazemUterine ContractionInternal medicinemedicineAnimalsDiltiazemRats WistarPharmacologyMembrane potentialDose-Response Relationship DrugChemistryUterusMuscle SmoothGeneral MedicineAcetylcholineRatsElectrophysiologyDose–response relationshipEndocrinologyVerapamilVerapamilCalciumFemaleCalcium Channelsmedicine.symptomAcetylcholinemedicine.drugMuscle contractionPharmacology
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Comparative study of the rat uterine smooth muscle relaxant activity of three bisbenzyltetrahydroisoquinolines with tetrandrine

1993

Abstract The relaxant activity of three bisbenzyltetrahydroisoquinolines—obaberine, popisonine and lindoldhamine—was examined in rat isolated uterus and their inhibitory potencies were compared with that of tetrandrine. All alkaloids tested relaxed KCl-depolarized rat uterus and totally or partially inhibited oxytocin-induced rhythmic contractions. The degree of methylation of the free phenolic hydroxy groups and the loss of one diarylether bridge influence the potency of relaxant action of these alkaloids. Only alkaloids with absolute configuration 1R,1′S or 1R1′R acted intracellularly, promoting relaxation of contractile responses induced by oxytocin or vanadate in a Ca2+-free medium.

medicine.medical_specialtyMuscle RelaxationUterusPharmaceutical Sciencechemistry.chemical_elementIn Vitro TechniquesBiologyCalciumOxytocinBenzylisoquinolinesUterine contractionStructure-Activity RelationshipUterine Contractionchemistry.chemical_compoundAlkaloidsInternal medicinemedicineAnimalsVanadateRats WistarPharmacologyAlkaloidUterusMuscle SmoothCalcium Channel BlockersIsoquinolinesRatsTetrandrinemedicine.anatomical_structureMuscle relaxationEndocrinologyOxytocinchemistryPotassiumCalciumFemaleVanadatesmedicine.symptommedicine.drugJournal of Pharmacy and Pharmacology
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Inhibition of calcium entry induced by cularines and isocrasifoline in uterine smooth muscle.

1991

Abstract The effects of nifedipine, papaverine and four benzylisoquinoline alkaloids (cularine, cularidine, celtisine and isocrasifoline) were studied in isolated rat uterus in order to clarify the mechanism of their relaxant action. All the compounds tested completely relaxed KCl-induced contractions and totally or partially inhibited oxytocin-induced rhythmic contractions. Only papaverine acted intracellularly, promoting relaxation of contractile responses induced by oxytocin or vanadate in a Ca 2+ -free medium. In spite of the structural relationship between papaverine and the other alkaloids, the mechanism of their relaxant action is not the same. The activities of cularine derivatives …

medicine.medical_specialtyNifedipineIn Vitro TechniquesOxytocinchemistry.chemical_compoundUterine ContractionAlkaloidsNifedipineCoumarinsInternal medicinePapaverinemedicineAnimalsVanadateBenzylisoquinolinePharmacologyPapaverineChemistryAlkaloidMuscle SmoothRats Inbred StrainsIsoquinolinesRatsEndocrinologyOxytocinMechanism of actionCalciumFemalemedicine.symptomVanadatesmedicine.drugMuscle contractionMuscle ContractionEuropean journal of pharmacology
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NADPH Oxidase Accounts for Enhanced Superoxide Production and Impaired Endothelium-Dependent Smooth Muscle Relaxation in BKβ1 −/− Mice

2006

Objective— Nitric oxide (NO)-induced vasorelaxation involves activation of large conductance Ca 2+ -activated K + channels (BK). A regulatory BKβ1 subunit confers Ca 2+ , voltage, and NO/cGMP sensitivity to the BK channel. We investigated whether endothelial function and NO/cGMP signaling is affected by a deletion of the β1-subunit. Methods and Results— Vascular superoxide in BKβ1 −/− was measured using the fluorescent dye hydroethidine and lucigenin-enhanced chemiluminescence. Vascular NO formation was analyzed using electron paramagnetic resonance (EPR), expression of NADPH oxidase subunits, the endothelial NO synthase (eNOS), the soluble guanylyl cyclase (sGC), as well as the activity a…

medicine.medical_specialtyNitric Oxide Synthase Type IIIEndotheliumAorta ThoracicNitric OxideMuscle Smooth VascularNitric oxideMicechemistry.chemical_compoundSuperoxidesInternal medicineCyclic GMP-Dependent Protein KinasesmedicineAnimalsHumansProtein IsoformsNADH NADPH OxidoreductasesLarge-Conductance Calcium-Activated Potassium ChannelsMice KnockoutNADPH oxidasebiologySuperoxideMicrofilament ProteinsNADPH OxidasesPhosphoproteinsMolecular biologyVasodilationEndocrinologymedicine.anatomical_structurechemistryGuanylate CyclaseNAD(P)H oxidaseNOX1ApocyninNADPH Oxidase 1biology.proteinEndothelium VascularCardiology and Cardiovascular MedicineSoluble guanylyl cyclaseCell Adhesion MoleculesSignal TransductionArteriosclerosis, Thrombosis, and Vascular Biology
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