Search results for "Synaptic Transmission"

showing 10 items of 178 documents

Functional Hallmarks of GABAergic Synapse Maturation and the Diverse Roles of Neurotrophins

2011

Functional impairment of the adult brain can result from deficits in the ontogeny of GABAergic synaptic transmission. Gene defects underlying autism spectrum disorders, Rett’s syndrome or some forms of epilepsy, but also a diverse set of syndromes accompanying perinatal trauma, hormonal imbalances, intake of sleep-inducing or mood-improving drugs or, quite common, alcohol intake during pregnancy can alter GABA signaling early in life. The search for therapeutically relevant endogenous molecules or exogenous compounds able to alleviate the consequences of dysfunction of GABAergic transmission in the embryonic or postnatal brain requires a clear understanding of its site- and state-dependent …

Review ArticleBiologyNeurotransmissionInhibitory postsynaptic potentialgamma-Aminobutyric acidlcsh:RC321-571tonic inhibitionCellular and Molecular NeurosciencePostsynaptic potentialPresynaptic functionmedicinelcsh:Neurosciences. Biological psychiatry. Neuropsychiatryquantal analysisNGFSynaptic pharmacologyexcitatory–inhibitory balanceGABAergic synaptic transmissionBDNFbiology.proteinGABAergicsynapse developmentNeuroscienceSynapse maturationNeuroscienceNeurotrophinmedicine.drugFrontiers in Cellular Neuroscience
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Targeting Homer genes using adeno-associated viral vector: lessons learned from behavioural and neurochemical studies.

2008

Over a decade of in-vitro data support a critical role for members of the Homer family of postsynaptic scaffolding proteins in regulating the functional architecture of glutamate synapses. Earlier studies of Homer knockout mice indicated a necessary role for Homer gene products in normal mesocorticolimbic glutamate transmission and behaviours associated therewith. The advent of adeno-associated viral vectors carrying cDNA for, or short hairpin RNA against, specific Homer isoforms enabled the site-directed targeting of Homers to neurons in the brain. This approach has allowed our groups to address developmental issues associated with conventional knockout mice, to confirm active roles for di…

Scaffold proteinSubstance-Related DisordersTransgeneEmotionsGenetic VectorsGlutamic AcidMice TransgenicBiologySynaptic TransmissionArticleViral vectorAdenoviridaeSmall hairpin RNAMiceNeurochemicalHomer Scaffolding ProteinsAnimalsGeneGenes Immediate-EarlyPharmacologyMice KnockoutBehavior AnimalGlutamate receptorGene Transfer TechniquesBrainPsychiatry and Mental healthAlcoholismKnockout mouseMutagenesis Site-DirectedArousalCarrier ProteinsNeuroscienceBehavioural pharmacology
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Imaging synaptic zinc release in living nervous tissue

2001

Zinc enriched neurons have a pool of synaptic vesicles which contain free or loosely-bound zinc ions. The movement of the vesicular zinc ions into the synaptic clefts has been previously studied by microdialysis, fluorescence postmortem staining for zinc and radioactive zinc isotope. In this study the zinc fluorescence probe N-6-metoxy-p-toluensulfonamide quinoline (TSQ) has been applied as a tracer of synaptic release of zinc ions. This fluorochrome permeates cell membranes and when exposed to living brain slices gives rise to a staining pattern similar to that seen with autometallography. In the living brain slices, fluorescence emission persists after exposure to calcium saturated ethyle…

TelencephalonMicrodialysisCell Membrane PermeabilitySynaptic cleftSodiumNeurophysiologychemistry.chemical_elementZincSynaptic TransmissionSynaptic vesiclePotassium ChlorideTosyl CompoundsImage Processing Computer-AssistedExtracellularAnimalsEdetic AcidFluorescent DyesElectronic Data ProcessingMicroscopy VideoGeneral NeuroscienceCell MembraneLizardsZincMembraneMicroscopy FluorescencechemistryBiochemistryIsotopes of zincAminoquinolinesBiophysicsRabbitsSynaptic VesiclesJournal of Neuroscience Methods
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A critical role for VEGF and VEGFR2 in NMDA receptor synaptic function and fear-related behavior

2016

Vascular endothelial growth factor (VEGF) is known to be required for the action of antidepressant therapies but its impact on brain synaptic function is poorly characterized. Using a combination of electrophysiological, single-molecule imaging and conditional transgenic approaches, we identified the molecular basis of the VEGF effect on synaptic transmission and plasticity. VEGF increases the postsynaptic responses mediated by the N-methyl-D-aspartate type of glutamate receptors (GluNRs) in hippocampal neurons. This is concurrent with the formation of new synapses and with the synaptic recruitment of GluNR expressing the GluN2B subunit (GluNR-2B). VEGF induces a rapid redistribution of Glu…

Vascular Endothelial Growth Factor A0301 basic medicine[SDV]Life Sciences [q-bio]Cell Culture TechniquesNonsynaptic plasticityBiologyNeurotransmissionHippocampusReceptors N-Methyl-D-AspartateSynaptic TransmissionMice03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicinePostsynaptic potentialAnimalsddc:610Molecular BiologyComputingMilieux_MISCELLANEOUSNeuronsNeuronal PlasticityBehavior AnimalGlutamate receptorExcitatory Postsynaptic PotentialsKinase insert domain receptorFearVascular Endothelial Growth Factor Receptor-2Protein SubunitsPsychiatry and Mental health030104 developmental biologySynaptic fatigueReceptors GlutamateSynapsesSynaptic plasticityNMDA receptorOriginal ArticleNeuroscience030217 neurology & neurosurgery
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Hippocampal responses to electrical stimulation of the major input pathways are modulated by dentate spikes

2022

Dentate gyrus (DG) is important for pattern separation and spatial memory, and it is thought to gate information flow to the downstream hippocampal subregions. Dentate spikes (DSs) are high-amplitude, fast, positive local-field potential events taking place in the DG during immobility and sleep, and they have been connected to memory consolidation in rodents. DSs are a result of signaling from the entorhinal cortex (EC) to the DG, and they suppress firing of pyramidal cells in the CA3 and CA1. To study the effects of DSs to signaling in the hippocampal tri-synaptic loop, we electrically stimulated the afferent fibers of the DG, CA3, and CA1 in adult male Sprague–Dawley rats at different del…

aivokuorihippocampuselektrofysiologialateralizationsynaptic transmissiondentate gyrushippokampuselectrophysiologyaivotmuisti (kognitio)
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AMPA Receptor Auxiliary Proteins of the CKAMP Family

2019

α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors are assembled of four core subunits and several additional interacting proteins. Cystine-knot AMPA receptor-modulating proteins (CKAMPs) constitute a family of four proteins that influence the trafficking, subcellular localization and function of AMPA receptors. The four CKAMP family members CKAMP39/shisa8, CKAMP44/shisa9, CKAMP52/shisa6 and CKAMP59/shisa7 differ in their expression profile and their modulatory influence on AMPA receptor function. In this review, I report about recent findings on the differential roles of CKAMP family members.

glutamate receptorhippocampusGene ExpressionReviewAMPA receptorBiologySynaptic TransmissionCatalysisCell Linelcsh:ChemistryInorganic ChemistryLong term plasticitylateral geniculate nucleusAnimalsHumansAmino Acid SequenceReceptors AMPAAMPA receptorPhysical and Theoretical Chemistrysynaptic functionReceptorlcsh:QH301-705.5Molecular BiologySpectroscopyNeuronal Plasticitymusculoskeletal neural and ocular physiologyOrganic ChemistryGlutamate receptorGeniculate BodiesGeneral MedicineSubcellular localizationlong-term plasticityComputer Science ApplicationsCell biologyProtein TransportSynaptic functionlcsh:Biology (General)lcsh:QD1-999nervous systemauxiliary subunitMultigene FamilySynapsesCarrier ProteinsIon Channel Gatingshort-term plasticityFunction (biology)Protein BindingInternational Journal of Molecular Sciences
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Capsaicin desensitization in vivo is inhibited by ruthenium red.

1990

The effect of systemic administration of Ruthenium Red on the excitatory and desensitizing effect of capsaicin was investigated in rats. Ruthenium Red was injected s.c. 30 min before capsaicin was administered. The excitatory effect of capsaicin on corneal, perivascular and visceral afferents was not influenced by treatment with Ruthenium Red. However, determination of the neuropeptide content and evoked neuropeptide release in peripheral organs and dorsal spinal cord 48 h after treatment showed that Ruthenium Red attenuated the 'desensitizing' effect of capsaicin at peripheral, but not at central, endings of primary afferents. On the other hand, a capsaicin-elicited autonomic reflex mediat…

inorganic chemicalsRuthenium redCalcitonin Gene-Related PeptideInjections SubcutaneousCentral nervous systemUrinary BladderNeuropeptideBlood PressurePharmacologySubstance PSynaptic Transmissionchemistry.chemical_compoundIn vivootorhinolaryngologic diseasesAutonomic reflexmedicineAnimalsNeurons AfferentPharmacologyNerve EndingsNeuronsChemistryNeuropeptidesRats Inbred StrainsRuthenium RedRatsmedicine.anatomical_structureCapsaicinAnesthesiaExcitatory postsynaptic potentiallipids (amino acids peptides and proteins)NeuronCapsaicinEuropean journal of pharmacology
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Interleukin 10 restores lipopolysaccharide-induced alterations in synaptic plasticity probed by repetitive magnetic stimulation

2020

Systemic inflammation is associated with alterations in complex brain functions such as learning and memory. However, diagnostic approaches to functionally assess and quantify inflammation-associated alterations in synaptic plasticity are not well-established. In previous work, we demonstrated that bacterial lipopolysaccharide (LPS)-induced systemic inflammation alters the ability of hippocampal neurons to express synaptic plasticity, i.e., the long-term potentiation (LTP) of excitatory neurotransmission. Here, we tested whether synaptic plasticity induced by repetitive magnetic stimulation (rMS), a non-invasive brain stimulation technique used in clinical practice, is affected by LPS-induc…

lcsh:Immunologic diseases. AllergyLipopolysaccharides0301 basic medicinenon-invasive brain stimulationInterleukin-1betaImmunologyTNFα-reporter mouseMice TransgenicStimulationNeurotransmissionHippocampusSynaptic TransmissionneuroinflammationInterferon-gammaMice03 medical and health sciences0302 clinical medicineGenes Reportertranscranial magnetic stimulationAnimalsImmunology and Allergyddc:610NeuroinflammationOriginal ResearchInflammationNeuronsNeuronal Plasticitysynaptic plasticityInterleukin-6Tumor Necrosis Factor-alphaChemistryLong-term potentiationInterleukin-10Mice Inbred C57BLOrganoids030104 developmental biologyBrain stimulationSynaptic plasticityExcitatory postsynaptic potentialTumor necrosis factor alphaMicrogliainterleukin 10lcsh:RC581-607Neuroscience030217 neurology & neurosurgery
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Chelation of synaptic zinc induces overexcitation in the hilar mossy cells of the rat hippocampus.

2004

Complete removal of synaptic zinc by the chelator dietyldithiocarbamate (DEDTC; 500 mg/kg i.p.) in rat was followed by convulsive behaviour including wet dog shakes alternating immobility. Histological analysis 1 day after DEDTC administration detected expression of heat shock protein in the hippocampus restricted to hilar cells. These cells colocalize the marker for neurons and the glutamate receptor GluR2/3 showing that they are excitatory neurons. Additionally, they projected to the contralateral dentate gyrus. Therefore, they correspond to hilar mossy cells. These data show that the synaptic zinc has a role in normal hippocampus avoiding overexcitation, that would impair functionality e…

medicine.medical_specialtyCentral nervous systemPresynaptic TerminalsWheat Germ Agglutinin-Horseradish Peroxidase ConjugateHippocampusAction PotentialsHSP72 Heat-Shock Proteinsc-FosHippocampusSynaptic TransmissionSeizuresInternal medicineNeural PathwaysmedicineAnimalsReceptors AMPAHeat-Shock ProteinsChelating AgentsbiologyGeneral NeuroscienceDentate gyrusGlutamate receptorColocalizationImmunohistochemistryRatsZincEndocrinologymedicine.anatomical_structurenervous systemDentate GyrusMossy Fibers Hippocampalbiology.proteinExcitatory postsynaptic potentialDitiocarbImmediate early geneProto-Oncogene Proteins c-fosNeuroscience letters
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Facilitatory and inhibitory muscarine receptors on the rat phrenic nerve: effects of pirenzepine and dicyclomine

1988

Neuronal transmitter stores of the rat phrenic nerve were labelled by an incubation with [3H]choline. Release of [3H]acetylcholine was elicited either by a short (100 pulses, 5 Hz) or by a long (1500 pulses, 5 or 25 Hz) period of electrical nerve stimulation. Pirenzepine and dicyclomine enhanced transmitter release evoked by the short stimulation period. Both antagonists reduced transmitter release evoked by the long stimulation period. Pirenzepine reduced transmitter release at low concentrations (1 nmol/l) whereas a higher concentration was necessary for the enhancing effect; the opposite pattern was found for dicyclomine. A low concentration of oxotremorine (10 nmol/l) enhanced and a hig…

medicine.medical_specialtyCyclohexanecarboxylic AcidsNeuromuscular JunctionDicyclomineStimulationIn Vitro TechniquesSynaptic TransmissionDicyclominechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineAnimalsEvoked PotentialsPharmacologyMuscarineChemistryRats Inbred StrainsMuscarinic acetylcholine receptor M2PirenzepineGeneral MedicineMuscarinic acetylcholine receptor M1Receptors MuscarinicPirenzepineAcetylcholineRatsPhrenic NerveEndocrinologyAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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