Search results for "TAGL"

showing 10 items of 296 documents

Clonidine induces nitric oxide- and prostaglandin-mediated vasodilation in healthy human skin

2005

Sustained sympathetic activation not only leads to vasoconstriction but also might induce paradox vasodilation. This study was performed to explore whether and how alpha(2)-receptor stimulation mediates this vasodilation. We investigated 11 healthy subjects in 33 dermal microdialysis (MD) sessions. After nerve trunk blockade, MD fibers were inserted and perfused with physiological saline until skin trauma-related vasodilation subsided. Thereafter, fibers were perfused with either clonidine solutions (10(-3), 5 x 10(-4), 10(-4) mol/l), N(G)-monomethyl-l-arginine (L-NMMA; nitric oxide synthase blocker), acetylsalicylic acid (ASA; cyclooxygenase blocker), or combinations of these. Laser-Dopple…

AdultMalemedicine.medical_specialtySympathetic nervous systemPhysiologyImidazoline receptorProstaglandinVasodilationStimulationNitric OxideClonidineNitric oxidechemistry.chemical_compoundReference ValuesSkin Physiological PhenomenaPhysiology (medical)Internal medicineAdrenergic alpha-2 Receptor AgonistsmedicineHumansSkinDose-Response Relationship DrugChemistryVasodilationEndocrinologymedicine.anatomical_structureEicosanoidProstaglandinsmedicine.symptomAdrenergic alpha-AgonistsBlood Flow VelocityVasoconstrictionJournal of Applied Physiology
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Clopidogrel and aspirin in the prevention of thromboembolic complications after mechanical aortic valve replacement (CAPTA)

2003

Axel Schlitt*, Ralf S. von Bardeleben, Anne Ehrlich, Antje Eimermacher, Dirk Peetz, Manfred Dahm, Hans J. Rupprecht Department of Medicine II, Johannes Gutenberg-University Mainz, Langenbeckstr. 1, 55131 Mainz, Germany Coordination Center for Clinical Studies, Johannes Gutenberg-University Mainz, Mainz, Germany Clinic for Clinical Chemistry and Laboratory Medicine, Johannes Gutenberg-University Mainz, Mainz, Germany Department of Cardiothoracic and Vascular Surgery, Johannes Gutenberg-University Mainz, Mainz, Germany

AdultMalemedicine.medical_specialtyTiclopidineTreatment outcomeAdministration OralHemorrhagePilot ProjectsValvula aorticaProstaglandin-Endoperoxide SynthaseThromboembolismmedicineHumansOral anticoagulationAgedAspirinAspirinbusiness.industryHematologyMiddle AgedClopidogrelClopidogrelSurgeryTreatment OutcomeAortic ValveHeart Valve ProsthesisAortic valve surgeryPhenprocoumonDrug Therapy CombinationFemalebusinessVenous diseasemedicine.drugThrombosis Research
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The effect of two low doses of aspirin on whole blood thromboxane and prostacyclin generation in healthy subjects

1983

SummaryThe effects of two low doses of aspirin (20 mg and 100 mg) on prostacyclin and thromboxane formation during whole blood clotting were studied in 8 healthy volunteers.A single 100 mg aspirin dose caused more than 90% reduction of both serum TXB2 and 6-keto-PGF1α; a single 20 mg dose of aspirin inhibited serum TXB2 more than 6-keto-PGF1α but effects on these two products could not be completely dissociated.However, the effect of a single 20 mg aspirin dose on serum TXB2, was of much longer duration than its inhibitory effect on PGI2 synthesis during whole blood clotting.

AdultMalemedicine.medical_specialtyTime Factorsmedicine.drug_classThromboxaneAlpha (ethology)Prostacyclin6-Ketoprostaglandin F1 alphaInternal medicinemedicineHumansCyclooxygenase InhibitorsBlood CoagulationWhole bloodAspirinAspirinChemistryAnticoagulantLow doseThromboxanesHematologyHeparinEpoprostenolThromboxane B2EndocrinologyFemalelipids (amino acids peptides and proteins)circulatory and respiratory physiologymedicine.drug
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Efficacy of the treatment with prostaglandin E-1 in venous ulcers of the lower limbs.

2005

Background. Venous ulcers represent an important medical problem due to their high prevalence and consequent sanitary costs. In this study we evaluated the effect of Prostaglandin E-1 (PGE-1), a drug that improves district ischemia, on the healing of venous ulcers. Methods. We performed a randomized, placebo-controlled, single blind study, in which 87 patients, carriers of venous leg ulcers, homogeneous for dimensions and characteristics, were treated for 20 days an with infusion of Prostaglandin E-1 or placebo, in association with a topic therapy. The dimension and the number of the ulcers were determined at the beginning of the treatment and then every 20 days up to 4 months or until tota…

AdultMalemedicine.medical_specialtymedicine.medical_treatmentVasodilator AgentsIschemiaHealing timeProstaglandinPlacebolaw.inventionVaricose Ulcerchemistry.chemical_compoundRandomized controlled triallawMedicineHumansSingle-Blind MethodAlprostadilAgedLegUltrasonography Doppler DuplexWound Healingbusiness.industryMiddle Agedmedicine.diseasedigestive system diseasesSurgeryClinical trialchemistryBaseline characteristicsVenous ulcers Prostaglandine E-1SurgeryFemaleCardiology and Cardiovascular MedicinebusinessProstaglandin EJournal of vascular surgery
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In vitro Analysis of Synergistic Effects of Fibrinolytic Agents and Prostacyclin Analogues

1997

We investigated the in vitro thrombolytic effects of streptoki-nase, urokinase, alteplase and saruplase, alone or in combination, with the prostacyclin analogues, iloprost and taprostene. Human platelet-rich plasma was stimulated with collagen (1 μg/ml) to generate thrombi containing platelets and fibrin. Following treatment with fibrinolytic agents, lysis was allowed to proceed for 30 min and was then terminated with aprotinin (2,000 ClU/ml). To evaluate the combinatory effects of fibrinolytic agents and prostacyclin analogues, we used concentrations of fibrinolytic agents which reduced thrombi weight by less than 50%. Neither iloprost nor taprostene alone demonstrated any thrombolytic eff…

AdultMalemedicine.medical_treatmentProstacyclinPharmacologyFibrinolytic AgentsPhysiology (medical)Prostaglandins SyntheticFibrinolysismedicineHumansStreptokinaseIloprostUrokinaseChemistryFibrinolysisDrug SynergismHematologyDrug interactionEpoprostenolUrokinase-Type Plasminogen ActivatorIn vitroTissue Plasminogen Activatorcardiovascular systemFemaleSaruplaseFibrinolytic agentIloprostmedicine.drugPathophysiology of Haemostasis and Thrombosis
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15-prostaglandin dehydrogenase expression alone or in combination with ACSM1 defines a subgroup of the apocrine molecular subtype of breast carcinoma.

2008

Established histopathological criteria divide invasive breast carcinomas into defined groups. Ductal of no specific type and lobular are the two major subtypes accounting for around 75 and 15% of all cases, respectively. The remaining 10% include rarer types such as tubular, cribriform, mucinous, papillary, medullary, metaplastic, and apocrine breast carcinomas. Molecular profiling technologies, on the other hand, subdivide breast tumors into five subtypes, basal-like, luminal A, luminal B, normal breast tissue-like, and ERBB2-positive, that have different prognostic characteristics. An additional subclass termed "molecular apocrine" has recently been described, but these lesions did not ex…

AdultSilver StainingBreast NeoplasmsBiologyProteomicsBiochemistrySubclassAnalytical ChemistryImmunophenotypingCohort StudiesBreast cancerCoenzyme A LigasesmedicineBiomarkers TumorHumansElectrophoresis Gel Two-DimensionalNeoplasm Invasivenessskin and connective tissue diseasesMolecular BiologyAgedAged 80 and overTissue microarrayParaffin EmbeddingApocrineMiddle Agedmedicine.diseaseImmunohistochemistryApocrine GlandsPhenotypeTissue Array AnalysisImmunologyCancer researchDisease ProgressionHydroxyprostaglandin DehydrogenasesImmunohistochemistryFemaleApocrine CellBreast carcinomaMolecularcellular proteomics : MCP
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Data comparison between pharmacological induction of labour and spontaneous delivery. A single centre experience.

2016

Objectives: To assess the differences in the maternal and fetal outcomes between pharmacological induced and sponta­neous labour in nulliparous women. Material and methods: Observational cohort study carried out over a period of 2 years. Inclusion criteria: nulliparous sin­gleton pregnancies, with cephalic fetal presentation, elective labour induction with intra-vaginal prostaglandin E2 (PGE2) gel (Prepidil® 2 mg) at a gestational age of 41 weeks. Control group: patients who entered labour spontaneously at a gestational age of ≥ 40 weeks. The main demographic maternal characteristics and intra- and postpartum data were extracted from computer records and obstetrics diaries and were used for…

Adultmedicine.medical_specialtyTime FactorsProstaglandin E2Cohort Studies03 medical and health sciences0302 clinical medicineObstetrics and gynaecologyPregnancy030225 pediatricsSpontaneous labourOxytocicsInduction of labourMedicineInduction of labour Neonatal outcome Prostaglandin E2 Spontaneous labour Medicine (all) Obstetrics and GynecologyHumans030212 general & internal medicineLabor InducedProstaglandin E2reproductive and urinary physiologyGynecologyFetusbusiness.industryObstetricsInfant NewbornPregnancy OutcomeObstetrics and GynecologyGestational ageSpontaneous labourFetal PresentationNeonatal outcomeDelivery ObstetricSingle centreProstaglandinsFemalebusinessCohort studymedicine.drugGinekologia polska
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Age-related changes in linoleic acid bioconversion by isolated hepatocytes from spontaneously hypertensive and normotensive rats

1994

This study points out the hepatocyte interconversion of the linoleic acid family during hypertension. Hepatocyte delta 6 desaturase activity was higher in 1 month-old spontaneously hypertensive rats than in normotensive controls. A similar tendency was observed in 6 month-old SHR. delta 5 desaturase activity was higher only in 1 month-old spontaneously hypertensive rats as compared to controls. Desaturase activities were particularly high at the age of 6 months. The hepatocyte fatty acid composition showed an impairment of n-6 polyunsaturated fatty acid metabolism in spontaneously hypertensive animals. Changes were greater in the young prehypertensive rats than in adults. A storage of n-3 l…

Agingmedicine.medical_specialtyClinical chemistryLinoleic acidClinical BiochemistryProstaglandinRats Inbred WKYLinoleic Acidchemistry.chemical_compoundRats Inbred SHRInternal medicinemedicineAnimalsMolecular Biologychemistry.chemical_classificationKidneyFatty AcidsCell BiologyGeneral MedicineMetabolismPeroxisomeRatsEndocrinologymedicine.anatomical_structureLinoleic AcidsLiverchemistryHepatocyteHypertensionPolyunsaturated fatty acidMolecular and Cellular Biochemistry
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Prostacyclin receptor desensitization is a reversible phenomenon in human platelets.

1997

Background Long-term exposure of platelets to endogenous or exogenous prostacyclin or its analogues might result in desensitization of the platelet prostacyclin receptor in vitro and in vivo accompanied by a loss in receptor density on the platelet surface and a reduced sensitivity toward the inhibitory effects of prostacyclins. However, the reversibility of this process in platelets has not yet been investigated. Methods and Results Human platelets desensitized by the chemically stable prostacyclin analogue iloprost showed a significant reduction in [ 3 H]-iloprost binding sites that was reversed by saponin permeabilization. This indicates functionally active internalized prostacyclin rec…

AgonistBlood PlateletsMalemedicine.medical_specialtyCell Membrane Permeabilitymedicine.drug_classReceptors ProstaglandinProstaglandinProstacyclinReceptors EpoprostenolProstacyclin receptor bindingchemistry.chemical_compoundReference ValuesPhysiology (medical)Internal medicinemedicineCyclic AMPHumansPlateletIloprostProstacyclin receptorbusiness.industryEndocrinologychemistrycardiovascular systemPlatelet aggregation inhibitorlipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicinebusinessPlatelet Aggregation Inhibitorsmedicine.drugIloprostCirculation
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Endothelin Antagonism: Effects of FP Receptor Agonists Prostaglandin F2αand Fluprostenol on Trabecular Meshwork Contractility

2006

Purpose This study analyzes additional mechanisms behind the ocular hypotensive effect of prostaglandin F (PGF) receptor (FP receptor) agonists PGF2alpha and fluprostenol (fluprostenol-isopropyl ester [travoprost]), which reduce intraocular pressure (IOP) in patients with glaucoma probably by enhancing uveoscleral flow. The trabecular meshwork (TM) is actively involved in IOP regulation through contractile mechanisms. Contractility of TM is induced by endothelin (ET)-1, a possible pathogenic factor in glaucoma. The involvement of FP receptor agonists in the ET-1 effects on TM function was studied. Methods The effects of FP receptor agonists on contractility of bovine TM (BTM) were investiga…

Agonistmedicine.medical_specialtyCarbacholmedicine.drug_classBlotting WesternReceptors ProstaglandinDinoprostContractilityTravoprostTrabecular MeshworkInternal medicinemedicineAnimalsReceptorCells CulturedEndothelin-1ChemistryCloprostenolMuscle SmoothEndothelin 1medicine.anatomical_structureProstaglandin F2alphaEndocrinologyCalciumCattleTrabecular meshworkFura-2Endothelin receptorMuscle Contractionmedicine.drugInvestigative Opthalmology & Visual Science
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