Search results for "TOXICITY"

showing 10 items of 2261 documents

Guanosine Protects Glial Cells Against 6-Hydroxydopamine Toxicity

2014

Increasing body of evidence indicates that neuron-neuroglia interaction may play a key role in determining the progression of neurodegenerative diseases including Parkinson’s disease (PD), a chronic pathological condition characterized by selective loss of dopaminergic (DA) neurons in the substantia nigra. We have previously reported that guanosine (GUO) antagonizes MPP+-induced cytotoxicity in neuroblastoma cells and exerts neuroprotective effects against 6-hydroxydopamine (6-OHDA) and beta-amyloid-induced apoptosis of SH-SY5Y cells. In the present study we demonstrate that GUO protected C6 glioma cells, taken as a model system for astrocytes, from 6-OHDA-induced neurotoxicity. We show tha…

HydroxydopaminebiologyChemistryNeurodegenerationNeurotoxicitySubstantia nigraNucleoside transporterPharmacologymedicine.diseaseNeuroprotectionNeurotrophic factorsbiology.proteinmedicinePI3K/AKT/mTOR pathway
researchProduct

The effect of methionine and folic acid administered in ovo on the hematological parameters of chickens (Gallus gallus domesticus)

2020

Abstract Methionine (Met), an essential amino acid in poultry diets, when overdosed may cause hyperhomocysteinemia, which is mainly a trigger for cardiovascular diseases in humans. Homocysteine is neutralized (remethylated) in the presence of folic acid (FA), which also plays an important role in hematopoiesis and participates in the synthesis of DNA, and its deficiencies may result in the development of neural tube defects. One of the basic tools in studying the impact of both xenobiotics and nutrients on the animal organism is hematological analysis. Thus, the aim of this study was to determine the effect of in ovo supplementation with Met and FA on the hematological parameters of broiler…

Hyperhomocysteinemiamedicine.medical_specialtyHomocysteineZygotePhysiology and ReproductionChick EmbryoBiologyIn ovochemistry.chemical_compoundFolic AcidMethioninebloodInternal medicinemedicineAnimalsIncubationEssential amino acidlcsh:SF1-1100chemistry.chemical_classificationMethionineBroilertoxicityGeneral Medicinemedicine.diseaseEndocrinologychemistryToxicityAnimal Science and Zoologyegglcsh:Animal cultureChickensamino acidPoultry Science
researchProduct

Tetrachlorodecaoxide Improves the Oxygenation Status of Multicellular Tumor Spheroids

1986

Insufficient O2 supply to solid tumors has been recognized since many years to be a crucial problem in cancer therapy. Hypoxia and anoxia may increase the resistance of tumor cells to certain antiproliferative drugs or to ionizing radiation. Numerous approaches to overcoming this problem have been reported in the literature. Efforts have been made in mainly two basic directions: (1) the development of therapeutic means which are particularly efficient in hypoxic and nutrient-deprived tissue areas or which are at least independent of the tissue oxygenation, and (2) the development of methods for improving the oxygenation status of solid tumors. Among the former approaches, the cytotoxicity o…

HyperthermiaCellular respirationbusiness.industryBlood flowOxygenationHypoxia (medical)medicine.diseaseTetrachlorodecaoxidemedicineCancer researchArterial bloodmedicine.symptomCytotoxicitybusinessmedicine.drug
researchProduct

Hyperthermia Enhances CD95-Ligand Gene Expression in T Lymphocytes

2004

Abstract Hyperthermia represents an interesting therapeutic strategy for the treatment of tumors. Moreover, it is able to regulate several aspects of the immune response. Fas (APO-1/CD95) and its ligand (FasL) are cell surface proteins whose interaction activates apoptosis of Fas-expressing targets. In T cells, the Fas-Fas-L system regulates activation-induced cell death, is implicated in diseases in which lymphocyte homeostasis is compromised, and plays an important role during cytotoxic and regulatory actions mediated by these cells. In this study we describe the effect of hyperthermia on activation of the fas-L gene in T lymphocytes. We show that hyperthermic treatment enhances Fas-L-med…

HyperthermiaFas Ligand ProteinFeverT-LymphocytesT cellBlotting WesternImmunologyBiologyLymphocyte ActivationTransfectionFas ligandJurkat CellsTransactivationImmune systemHeat Shock Transcription FactorsLymphocyte homeostasismedicineAnimalsHumansImmunology and AllergyCytotoxic T cellRNA MessengerPromoter Regions GeneticTranscription factorProtein Kinase CMembrane GlycoproteinsNF-kappa BBlotting NorthernCytotoxicity Tests Immunologicmedicine.diseaseMolecular biologyCell biologyDNA-Binding ProteinsTranscription Factor AP-1medicine.anatomical_structureGene Expression RegulationMutationTranscription FactorsThe Journal of Immunology
researchProduct

Microwave-Induced Hyperthermia and Cytostatic Agent for Superficial Bladder Cancer

2005

The efficacy and local toxicity of the intravesical combination of microwave-induced hyperthermia with a cytostatic agent in patients affected by superficial bladder cancer is presented. Between 1994 and 2002, two studies were performed on ablative and adjuvant intravesical thermo-chemotherapy. The ablative study demonstrated complete tumor eradication in 88% of patients who should have undergone cystectomy due to diffuse multiple papillary tumors non-responsive TUR and intravescical adjuvant therapy. In a randomized study, comparing adjuvant thermo-chemotherapy vs. standard chemotherapy with mitomycin C, recurrence-free survival analysis at 24 months revealed a significant difference in f…

Hyperthermiabusiness.industryToxicitySuperficial bladder cancerCancer researchMedicineThermo chemotherapyIn patientGeneral Medicinebusinessmedicine.diseaseUrologia Journal
researchProduct

Microcirculatory and pH Alterations in Isotransplanted Rat and Xenotransplanted Human Tumors Associated with Hyperthermia

1988

The rationale for considering the use of hyperthermia as an antitumor agent is based on three different mechanisms of action depending on the hyperthermia levels chosen: At moderate hyperthermia levels (40°–42.5° C) heat can increase the radiosensitivity and/or the chemosensitivity. At higher tissue temperatures ( > 42.5° C) hyperthermia acts as a cytotoxic agent since mammalian cells die after heating in a temperature-, time-, and cell cycle-dependent manner. Besides direct effects on the cell membranes, on the cytoskeleton, on metabolic processes, on DNA replication, and on RNA and protein synthesis, indirect effects distinctly modulating the anticancer action of heat have to be considere…

Hyperthermiamedicine.medical_specialtyChemistryCellHypoxia (medical)medicine.diseaseIn vitroMicrocirculationEndocrinologymedicine.anatomical_structureIn vivoInternal medicinemedicineCancer researchRadiosensitivitymedicine.symptomCytotoxicity
researchProduct

Detection of DNA effects in human cells with the comet assay and their relevance for mutagenesis

1996

The single cell gel test (SCG-test or comet assay) is a rapid and sensitive method for measuring DNA damage and repair in individual cells. A wide variety of mutagens have been shown to cause DNA alterations detectable with the comet assay, but it is not yet clear whether a relationship exists between the DNA effects and the induction of mutations. We are therefore investigating in a cell culture system with human cells (MRC5CV1) the induction of DNA damage by environmental mutagens and the formation of mutations at the HPRT gene. In the present study we investigated benzo[a]pyrene (BP), an environmental mutagenic and carcinogenic polycyclic aromatic hydrocarbon, and its reactive metabolite…

Hypoxanthine PhosphoribosyltransferaseDNA repairDNA damageCytological TechniquesMutagenGene mutationToxicologymedicine.disease_causechemistry.chemical_compoundBenzo(a)pyrenemedicineHumansCell Line TransformedElectrophoresis Agar GelGeneticsCell DeathMutagenesisfood and beveragesGeneral MedicineMolecular biologyComet assaychemistryMutagenesisEnvironmental PollutantsDNAGenotoxicityDNA DamageToxicology Letters
researchProduct

Protection by beverages, fruits, vegetables, herbs, and flavonoids against genotoxicity of 2-acetylaminofluorene and 2-amino-1-methyl-6-phenylimidazo…

2002

Abstract Chinese hamster lung fibroblasts, genetically engineered for the expression of rat cytochrome P450 dependent monooxygenase 1A2 and rat sulfotransferase 1C1 (V79-rCYP1A2-rSULT1C1 cells), were utilized to check for possible protective effects of beverages of plant origin, fruits, vegetables, and spices against genotoxicity induced by 2-acetylaminofluorene (AAF) or 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP). Antigenotoxic activities of juices from spinach and red beets against AAF could be monitored with similar effectivity by the HPRT-mutagenicity test (IC50=0.64%; 2.57%) and alkaline single cell gel electrophoresis (comet assay; IC50=0.12%; 0.89%) which detects DNA stran…

Hypoxanthine PhosphoribosyltransferaseHealth Toxicology and Mutagenesismedicine.disease_causeCell LineBeverageschemistry.chemical_compoundCricetulusCytochrome P-450 CYP1A2CricetinaeVegetablesGeneticsmedicineAnimalsWineFlavonoids2-Amino-1-methyl-6-phenylimidazo(45-b)pyridinePlants MedicinalbiologyMutagenicity TestsImidazolesfood and beveragesAntimutagenic AgentsMonooxygenase2-AcetylaminofluoreneFibroblastsbiology.organism_classificationRecombinant ProteinsRatsComet assayBiochemistrychemistryWhite WineFruitFlavanonesSpinachQuercetin2-AcetylaminofluoreneComet AssaySulfotransferasesGenotoxicityMutagensMutation research
researchProduct

Influence of DNA Repair on Nonlinear Dose-Responses for Mutation

2013

Recent evidence has challenged the default assumption that all DNA-reactive alkylating agents exhibit a linear dose-response. Emerging evidence suggests that the model alkylating agents methyl- and ethylmethanesulfonate and methylnitrosourea (MNU) and ethylnitrosourea observe a nonlinear dose-response with a no observed genotoxic effect level (NOGEL). Follow-up mechanistic studies are essential to understand the mechanism of cellular tolerance and biological relevance of such NOGELs. MNU is one of the most mutagenic simple alkylators. Therefore, understanding the mechanism of mutation induction, following low-dose MNU treatment, sets precedence for weaker mutagenic alkylating agents. Here, …

Hypoxanthine PhosphoribosyltransferaseMethyltransferaseDNA RepairDNA repairBiologyToxicologymedicine.disease_causePolymerase Chain ReactionCell Linechemistry.chemical_compoundalkylating agentsmedicineHumansnon-linearDNA Modification Methylasesgenetic toxicologyHypoxanthineDNA Primersdose-responsemutagenBase SequenceDose-Response Relationship DrugTumor Suppressor ProteinsgenotoxicityMutagenesisrisk assessmentDNA adductsO-6-methylguanine-DNA methyltransferaseMolecular biologyDNA Repair EnzymeschemistryMutationNOGELGenotoxicityMutagensResearch ArticleHypoxanthine PhosphoribosyltransferaseEthylnitrosoureaToxicological Sciences
researchProduct

Cathepsin-B Induced Controlled Release from Peptide-Capped Mesoporous Silica Nanoparticles

2014

New capped silica mesoporous nanoparticles for intracellular controlled cargo release within cathepsin B expressing cells are described. Nanometric mesoporous MCM-41 supports loaded with safranin O (S1-P) or doxorubicin (S2-P) containing a molecular gate based on a cathepsin B target peptidic sequence were synthesized. Solids were designed to show "zero delivery" and to display cargo release in the presence of cathepsin B enzyme, which selectively hydrolyzed in vitro the capping peptide sequence. Controlled delivery in HeLa, MEFs WT, and MEFs lacking cathepsin B cell lines were also tested. Release of safranin O and doxorubicin in these cells took place when cathepsin B was active or presen…

INGENIERIA DE LA CONSTRUCCIONCell Survivalgated mesoporous materialsPeptideAntineoplastic AgentsCatalysisCathepsin BCell LineCathepsin BHeLaQUIMICA ORGANICAHumansCytotoxicityPeptide sequencechemistry.chemical_classificationDrug CarriersbiologyOrganic ChemistryQUIMICA INORGANICAGeneral ChemistryMesoporous silicabiology.organism_classificationSilicon DioxideControlled releasechemistryBiochemistryDoxorubicinBiophysicspeptidesnanoparticlescontrolled releasePorosityIntracellularHeLa Cells
researchProduct