Search results for "TRES"

showing 10 items of 6927 documents

DHA protects PC12 cells against oxidative stress and apoptotic signals through the activation of the NFE2L2/HO-1 axis

2019

Docosahexaenoic acid (DHA) is an omega‑3 polyunsaturated fatty acid, derived mainly from fish oil. It is well known that DHA is present in high concentrations in nervous tissue and plays an important role in brain development and neuroprotection. However, the molecular mechanisms underlying its role remain to be fully elucidated. In this study, to enhance our understanding of the pathophysiological role of DHA, we investigated the possible neuroprotective mechanisms of action of DHA against hydrogen peroxide (H2O2)‑induced oxidative damage in a rat pheochromocytoma cell line (PC12). Specifically, we evaluated the viability, oxidation potential, and the expression and production of antioxida…

0301 basic medicineAnimals; Apoptosis; Docosahexaenoic Acids; Glutathione Peroxidase; Heme Oxygenase-1; Hydrogen Peroxide; NF-E2-Related Factor 2; Neuroprotective Agents; Oxidative Stress; PC12 Cells; Rats; Superoxide DismutaseAntioxidantDocosahexaenoic AcidsSettore BIO/14 - FARMACOLOGIADHA neuroprotection PV12 cellsNF-E2-Related Factor 2medicine.medical_treatmentApoptosismedicine.disease_causePC12 CellsNeuroprotectionSuperoxide dismutase03 medical and health scienceschemistry.chemical_compound0302 clinical medicinedecosahexaenoic acidGeneticsmedicineAnimalschemistry.chemical_classificationGlutathione PeroxidasebiologySuperoxide DismutaseChemistryGlutathione peroxidasenuclear factorHydrogen PeroxideGeneral MedicineAscorbic acidMalondialdehydeNFE2L2RatsCell biologyOxidative StressNeuroprotective Agents030104 developmental biology030220 oncology & carcinogenesisbiology.proteinHeme Oxygenase-1Oxidative stressInternational Journal of Molecular Medicine
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Polyphenolic Extract from Tarocco (Citrus sinensis L. Osbeck) Clone "Lempso" Exerts Anti-Inflammatory and Antioxidant Effects via NF-kB and Nrf-2 Act…

2018

Citrus fruits are often employed as ingredients for functional drinks. Among Citrus, the variety, “Lempso„, a typical hybrid of the Calabria region (Southern Italy), has been reported to possess superior antioxidant activity when compared to other common Citrus varieties. For these reasons, the aim of this study is to investigate in vitro the nutraceutical value of the Tarocco clone, “Lempso„, highlighting its anti-inflammatory and antioxidant potential. A post-column 2,2′-diphenyl-1-picrylhydrazyl (DPPH•) radical scavenging assay for the screening of antioxidant compounds in these complex matrices was developed. Subsequently, polyphenolic extract was tes…

0301 basic medicineAnthocyaninAntioxidantDPPHMacrophagemedicine.medical_treatmentAnti-Inflammatory AgentsPharmacologyAntioxidantschemistry.chemical_compoundMiceoxidative stressCitrus sinensiNutrition and DieteticsbiologyNF-kappa Bon-line HPLC-DPPHanthocyaninsNitric oxide synthaseBiphenyl compoundAnti-Inflammatory AgentCytokinesAntioxidantmacrophages J774A.1lcsh:Nutrition. Foods and food supplyCitrus × sinensisCitrus sinensisLPSmedicine.drug_classCell SurvivalNF-E2-Related Factor 2lcsh:TX341-641Anti-inflammatoryArticleNitric oxidePlant ExtractCell LineAnthocyanins; LPS; Macrophages J774A.1; On-line HPLC-DPPH; Oxidative stress; Food Science; Nutrition and Dietetics03 medical and health sciencesNutraceuticalPicratesmedicineAnimalsCytokineanthocyanins; LPS; macrophages J774A.1; on-line HPLC-DPPH; oxidative stressoxidative streAnimalPlant ExtractsMacrophagesBiphenyl CompoundsPolyphenolsSettore CHIM/08 - Chimica Farmaceutica030104 developmental biologychemistrybiology.proteinBiphenyl CompoundPicrateFood ScienceNutrients
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In vitro effects of vitamins C and E, n-3 and n-6 PUFA and n-9 MUFA on placental cell function and redox status in type 1 diabetic pregnant women.

2016

IF 2.972; International audience; The aim of this investigation was to determine the in vitro effects of vitamin C and E, n-3 and n-6 PUFA and n-9 MUFA on placental cell proliferation and function in type 1 diabetes. Placenta tissues were collected from 30 control healthy and 30 type 1 diabetic women at delivery. Placental cells were isolated and were cultured in RPMI medium supplemented with vitamin C (50 μM), vitamin E (50 μM), n-3 PUFA (100 μM), n-6 PUFA (100 μM) or n-9 MUFA (100 μM). Cell proliferation, cell glucose uptake and intracellular oxidative status were investigated. Our results showed that basal placental cell proliferation, glucose uptake, malondialdehyde (MDA) and carbonyl p…

0301 basic medicineAntioxidantGlucose uptakemedicine.medical_treatmentPlacentaProliferationPregnancy in DiabeticsAscorbic Acidmedicine.disease_causeAntioxidantsFatty Acids Monounsaturatedchemistry.chemical_compound0302 clinical medicinePregnancyMalondialdehydeVitamin EVitamin C[ SDV.MHEP.GEO ] Life Sciences [q-bio]/Human health and pathology/Gynecology and obstetrics030219 obstetrics & reproductive medicineTrophoblastObstetrics and Gynecologyfood and beveragesCatalasemedicine.anatomical_structureType 1 diabetes[ SDV.BDLR ] Life Sciences [q-bio]/Reproductive BiologyHypertensionFemalelipids (amino acids peptides and proteins)Oxidant/antioxidant statusOxidation-ReductionIntracellularPolyunsaturated fatty-acidsVitaminAdultRiskmedicine.medical_specialtyPlacental cellsBiology03 medical and health sciencesYoung AdultInternal medicinePlacentaFatty Acids Omega-6Fatty Acids Omega-3medicineHumans[ SDV.BDD ] Life Sciences [q-bio]/Development BiologyCell ProliferationVitamin CSuperoxide DismutaseVitamin EMellitusPreeclampsiaDiet030104 developmental biologyEndocrinologyDiabetes Mellitus Type 1MetabolismReproductive MedicinechemistryOxidative stressOxidative stressPUFADevelopmental Biology
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Pinolenic acid ameliorates oleic acid-induced lipogenesis and oxidative stress via AMPK/SIRT1 signaling pathway in HepG2 cells

2019

Pinolenic acid (PLA), a natural compound isolated from pine nut oil, has been reported to exert bioactivity against lipid anabolism. Nonetheless, the underlying mechanisms still poorly elucidated. The aim of this study is to comprehensively demonstrate the effects of PLA on oleic acid (OA)-induced non-alcoholic fatty liver disease (NAFLD) and their relationship with the lipid metabolic regulation. The results demonstrated that treatment with PLA dramatically inhibited lipid accumulation, oxidative stress as well as inflammatory responses induced by oleic acid in HepG2 cells. PLA also obviously decreased the levels of cellular triglyceride (TG), total cholesterol (TC), malondialdehyde (MDA),…

0301 basic medicineAntioxidantLinolenic Acidsmedicine.medical_treatmentPinolenic acidAMP-Activated Protein KinasesNitric Oxidemedicine.disease_causePine nut oil03 medical and health scienceschemistry.chemical_compound0302 clinical medicineSirtuin 1medicineHumansPPAR alphaPharmacologybiologyLipogenesisAMPKHep G2 CellsOxidative StressFatty acid synthaseOleic acid030104 developmental biologyGene Expression RegulationchemistryBiochemistryLipogenesisbiology.proteinlipids (amino acids peptides and proteins)030217 neurology & neurosurgeryOxidative stressOleic AcidSignal TransductionEuropean Journal of Pharmacology
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Dietary phytochemicals in the protection against oxysterol-induced damage.

2017

The intake of fruits and vegetables is associated with reduced incidence of many chronic diseases. These foods contain phytochemicals that often possess antioxidant and free radical scavenging capacity and show anti-inflammatory action, which are also the basis of other bioactivities and health benefits, such as anticancer, anti-aging, and protective action for cardiovascular diseases, diabetes mellitus, obesity and neurodegenerative disorders. Many factors can be included in the etiopathogenesis of all of these multifactorial diseases that involve oxidative stress, inflammation and/or cell death processes, oxysterols, i.e. cholesterol oxidation products (COPs) as well as phytosterol oxidat…

0301 basic medicineAntioxidantOxysterolmedicine.medical_treatmentPhytochemicalsContext (language use)PhytochemicalPharmacologymedicine.disease_causeBiochemistryAntioxidants03 medical and health scienceschemistry.chemical_compoundOxysterol0302 clinical medicineDiabetes mellitusmedicinePhytosterol oxidation productAnimalsHumansCholesterol oxidation productMolecular BiologyCell DeathAnimalCholesterolPhytosterolOrganic ChemistryHuman chronic diseaseCell BiologyOxysterolsmedicine.diseaseObesity030104 developmental biologyBiochemistrychemistry030220 oncology & carcinogenesisChronic DiseaseAntioxidantOxidative stressHumanChemistry and physics of lipids
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The Antioxidant Therapy: New Insights in the Treatment of Hypertension

2018

Reactive oxygen species (ROS) and reactive nitrogen species (RNS) play a key role in the regulation of the physiological and pathological signaling within the vasculature. In physiological conditions, a delicate balance between oxidants and antioxidants protects cells from the detrimental effects of ROS/RNS. Indeed, the imbalance between ROS/RNS production and antioxidant defense mechanisms leads to oxidative and nitrosative stress within the cell. These processes promote the vascular damage observed in chronic conditions, such as hypertension. The strong implication of ROS/RNS in the etiology of hypertension suggest that antioxidants could be effective in the treatment of this pathology. I…

0301 basic medicineAntioxidantPhysiologymedicine.medical_treatmentReview030204 cardiovascular system & hematologymedicine.disease_causeEssential hypertensionBioinformaticslcsh:Physiologyvitamin D deficiencyNitrosative stre03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePhysiology (medical)medicineVitamin D and neurologyoxidative stressReactive nitrogen specieslcsh:QP1-981business.industryROSmedicine.diseasenitrosative stressNebivololantioxidants030104 developmental biologyBlood pressurechemistryHypertensionOxidative streAntioxidantbusinessOxidative stressmedicine.drugFrontiers in Physiology
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Physiological Levels of Nitric Oxide Diminish Mitochondrial Superoxide. Potential Role of Mitochondrial Dinitrosyl Iron Complexes and Nitrosothiols.

2017

Mitochondria are the major source of superoxide radicals and superoxide overproduction contributes to cardiovascular diseases and metabolic disorders. Endothelial dysfunction and diminished nitric oxide levels are early steps in the development of these pathological conditions. It is known that physiological production of nitric oxide reduces oxidative stress and inflammation, however, the precise mechanism of “antioxidant” effect of nitric oxide is not clear. In this work we tested the hypothesis that physiological levels of nitric oxide diminish mitochondrial superoxide production without inhibition of mitochondrial respiration. In order to test this hypothesis we analyzed effect of low p…

0301 basic medicineAntioxidantPhysiologymedicine.medical_treatmentdinitrosyl iron complexesMitochondrionmedicine.disease_causelcsh:PhysiologyNitric oxide03 medical and health scienceschemistry.chemical_compoundnitric oxidePhysiology (medical)medicineHydrogen peroxideOriginal Researchchemistry.chemical_classificationReactive oxygen specieslcsh:QP1-981SuperoxideNitrosylationelectron spin resonancenitrosothiolsmitochondria030104 developmental biologychemistryBiophysicssuperoxideOxidative stressFrontiers in physiology
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Peripheral artery disease, redox signaling, oxidative stress – Basic and clinical aspects

2017

Reactive oxygen and nitrogen species (ROS and RNS, e.g. H2O2, nitric oxide) confer redox regulation of essential cellular signaling pathways such as cell differentiation, proliferation, migration and apoptosis. At higher concentrations, ROS and RNS lead to oxidative stress and oxidative damage of biomolecules (e.g. via formation of peroxynitrite, fenton chemistry). Peripheral artery disease (PAD) is characterized by severe ischemic conditions in the periphery leading to intermittent claudication and critical limb ischemia (end stage). It is well known that redox biology and oxidative stress play an important role in this setting. We here discuss the major pathways of oxidative stress and re…

0301 basic medicineAntioxidantRedox signalingmedicine.medical_treatmentCellular differentiationClinical BiochemistryReview Article030204 cardiovascular system & hematologyPharmacologymedicine.disease_causeBiochemistrychemistry.chemical_compound0302 clinical medicineGene Regulatory Networks610 Medicine & healthlcsh:QH301-705.5chemistry.chemical_classificationlcsh:R5-920Anticholesteremic AgentsReactive Nitrogen Speciesmedicine.symptomlcsh:Medicine (General)Oxidation-ReductionPeroxynitriteSignal Transductionmedicine.medical_specialtyCell signalingAntioxidant therapy610 Medicine & healthNitric oxide03 medical and health sciencesPeripheral Arterial DiseasemedicineHumansExerciseReactive oxygen speciesbusiness.industryOrganic ChemistryClaudication and critical limb ischemiaWalking distanceIntermittent claudicationSurgeryOxidative Stress030104 developmental biologychemistrylcsh:Biology (General)Peripheral artery (occlusive) diseasebusinessReactive Oxygen SpeciesOxidative stressRedox Biology
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Protective Effect of Cactus Cladode Extracts on Peroxisomal Functions in Microglial BV-2 Cells Activated by Different Lipopolysaccharides

2017

International audience; In this study, we aimed to evaluate the antioxidant and anti-inflammatory properties of Opuntia ficus-indica cactus cladode extracts in microglia BV-2 cells. Inflammation associated with microglia activation in neuronal injury can be achieved by LPS exposure. Using four different structurally and biologically well-characterized LPS serotypes, we revealed a structure-related differential effect of LPS on fatty acid β-oxidation and antioxidant enzymes in peroxisomes: Escherichia coli-LPS decreased ACOX1 activity while Salmonella minnesota-LPS reduced only catalase activity. Different cactus cladode extracts showed an antioxidant effect through microglial catalase activ…

0301 basic medicineAntioxidant[SDV]Life Sciences [q-bio]medicine.medical_treatmentAnti-Inflammatory AgentsPharmaceutical Scienceacyl-CoA oxidase 1; catalase; β-oxidation; <i>Escherichia coli</i>; lipopolysaccharides; LPS; nitric oxide; Opuntia; peroxisomes; <i>Salmonella minnesota</i>AntioxidantsAnalytical ChemistryMicechemistry.chemical_compoundSalmonellaDrug Discoverychemistry.chemical_classificationbiologyMicrogliaFatty AcidscatalaseOpuntiaPeroxisome[SDV] Life Sciences [q-bio]Neuroprotective Agentsmedicine.anatomical_structureBiochemistryChemistry (miscellaneous)CatalaseMolecular MedicineACOX1Microgliamedicine.symptomOxidation-ReductionLPSInflammationArticleCell LineNitric oxideMicrobiologylcsh:QD241-44103 medical and health scienceslcsh:Organic chemistrynitric oxideEscherichia colimedicineAnimalsSalmonella minnesotaPhysical and Theoretical Chemistryacyl-CoA oxidase 1[ SDV ] Life Sciences [q-bio]Plant ExtractsOrganic ChemistryperoxisomeslipopolysaccharidesOxidative Stress030104 developmental biologyEnzymechemistrybiology.proteinβ-oxidationReactive Oxygen SpeciesMolecules
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MiRNome of epileptic children suggests the involvement of antioxidant pathways in the neuroprotective role of ketogenic diet

2018

Ketogenic diet (KD) has been used as treatment for refractory epilepsy in children since the 1920s. However, the neuroprotective mechanism of this diet is still unknown. Recent evidence points to a multifactorial effect of KD that involves a decrease of oxidative stress. Mononuclear cells from 14 children with refractory epilepsy, from the Paediatric Hospital “Nino Jesus” in Madrid, were obtained before and after treatment with a ketogenic diet for 6 months. A global analysis of miRNA expression (miRNome) indicates that there are 230 miRNAs which are expressed differently before and after the treatment with KD. If additionally, we restrict the miRNAs with a fold change > |1.3|we obtain 11 r…

0301 basic medicineAntioxidantbusiness.industrymedicine.medical_treatmentPharmacologymedicine.disease_causeBiochemistryPeripheral blood mononuclear cellNeuroprotectionFold change03 medical and health sciences030104 developmental biology0302 clinical medicineDownregulation and upregulationPhysiology (medical)microRNAmedicinebusiness030217 neurology & neurosurgeryOxidative stressKetogenic dietFree Radical Biology and Medicine
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