Search results for "Time Factor"

showing 10 items of 3219 documents

Preservation of Multiple Mammalian Tissues to Maximize Science Return from Ground Based and Spaceflight Experiments.

2016

Background Even with recent scientific advancements, challenges posed by limited resources and capabilities at the time of sample dissection continue to limit the collection of high quality tissues from experiments that can be conducted only infrequently and at high cost, such as in space. The resources and time it takes to harvest tissues post-euthanasia, and the methods and duration of long duration storage, potentially have negative impacts on sample quantity and quality, thereby limiting the scientific outcome that can be achieved. Objectives The goals of this study were to optimize methods for both sample recovery and science return from rodent experiments, with possible relevance to b…

0301 basic medicineTime FactorsPhysiologyMolecular biologyRNA Stabilitylcsh:MedicineBiochemistrylaw.inventionMice0302 clinical medicinelawSpecimen StorageBone MarrowImmune PhysiologyGene expressionFreezingMedicine and Health Scienceslcsh:ScienceMammalsMultidisciplinaryPreservation methodsLimitingEye MusclesGlutathioneEnzymesRNA isolation030220 oncology & carcinogenesisTissue and Organ HarvestingSmall IntestineSample collectionAnatomyResearch ArticleOcular AnatomyImmunologyRNA integrity numberBiologySpaceflightResearch and Analysis MethodsBiomolecular isolationSpecimen HandlingAndrology03 medical and health sciencesOcular SystemAnimalsHumansTime pointCryopreservationlcsh:RRNABiology and Life SciencesProteinsSpace FlightGastrointestinal Tract030104 developmental biologyMolecular biology techniquesStorage and HandlingImmune SystemEnzymologylcsh:QPeptidesDigestive SystemSpleenCatalasesPloS one
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Human Upcyte Hepatocytes: Characterization of the Hepatic Phenotype and Evaluation for Acute and Long-Term Hepatotoxicity Routine Testing

2016

The capacity of human hepatic cell-based models to predict hepatotoxicity depends on the functional performance of cells. The major limitations of human hepatocytes include the scarce availability and rapid loss of the hepatic phenotype. Hepatoma cells are readily available and easy to handle, but are metabolically poor compared with hepatocytes. Recently developed human upcyte hepatocytes offer the advantage of combining many features of primary hepatocytes with the unlimited availability of hepatoma cells. We analyzed the phenotype of upcyte hepatocytes comparatively with HepG2 cells and adult primary human hepatocytes to characterize their functional features as a differentiated hepatic …

0301 basic medicineTime FactorsPrimary Cell CultureTransfectionToxicologyRisk AssessmentTranscriptome03 medical and health sciences0302 clinical medicineMetabolomicsCytochrome P-450 Enzyme SystemIn vivoToxicity TestsmedicineHumansChildGlycogen synthaseDose-Response Relationship DrugbiologyInfant NewbornCytochrome P450Hep G2 CellsMiddle Agedmedicine.diseasePhenotypeHigh-Throughput Screening AssaysIsoenzymesOxidative StressPhenotype030104 developmental biologyGene Expression RegulationLiver030220 oncology & carcinogenesisHepatocytesbiology.proteinHepatic stellate cellCancer researchChemical and Drug Induced Liver InjurySteatosisTranscriptomeToxicological Sciences
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The influence of meal frequency and timing on health in humans: The role of fasting

2019

The influence of meal frequency and timing on health and disease has been a topic of interest for many years. While epidemiological evidence indicates an association between higher meal frequencies and lower disease risk, experimental trials have shown conflicting results. Furthermore, recent prospective research has demonstrated a significant increase in disease risk with a high meal frequency (≥6 meals/day) as compared to a low meal frequency (1–2 meals/day). Apart from meal frequency and timing we also have to consider breakfast consumption and the distribution of daily energy intake, caloric restriction, and night-time eating. A central role in this complex scenario is playe…

0301 basic medicineTime FactorsTime FactorCircadian clockPhysiology030209 endocrinology & metabolismlcsh:TX341-641ReviewDiseaseGut floraDiabete03 medical and health sciences0302 clinical medicineDiabetes mellitusTime-restricted feedingmedicineHumanscardiovascular health; diabetes; fasting; meal frequency; meal timing; obesity; time-restricted feedingCircadian rhythmObesityMealsMeal030109 nutrition & dieteticsNutrition and Dieteticsbiologydiabetesbusiness.industryMeal timingdigestive oral and skin physiologyFastingFeeding Behaviorbiology.organism_classificationmedicine.diseaseObesityCardiovascular healthMeal frequencyProspective researchbusinesslcsh:Nutrition. Foods and food supplyFood ScienceHuman
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BAG2 Interferes with CHIP-Mediated Ubiquitination of HSP72

2016

The maintenance of cellular proteostasis is dependent on molecular chaperones and protein degradation pathways. Chaperones facilitate protein folding, maturation, and degradation, and the particular fate of a misfolded protein is determined by the interaction of chaperones with co-chaperones. The co-factor CHIP (C-terminus of HSP70-inteacting protein, STUB1) ubiquitinates chaperone substrates and directs proteins to the cellular degradation systems. The activity of CHIP is regulated by two co-chaperones, BAG2 and HSPBP1, which are potent inhibitors of the E3 ubiquitin ligase activity. Here, we examined the functional correlation of HSP72, CHIP, and BAG2, employing human primary fibroblasts.…

0301 basic medicineTime FactorsUbiquitin-Protein LigasesImmunoblottingHSP72 Heat-Shock ProteinsUbiquitin-conjugating enzymeProtein degradationArticleCatalysisCell Linelcsh:ChemistryInorganic Chemistry03 medical and health sciencesUbiquitinddc:570Humansaging; BAG2; CHIP; HSP72; proteostasis; ubiquitinationPhysical and Theoretical ChemistryHSP72lcsh:QH301-705.5Molecular BiologyCellular SenescenceSpectroscopySTUB1proteostasisBAG2biologyCHIPagingOrganic ChemistryUbiquitinationGeneral MedicineComputer Science ApplicationsUbiquitin ligaseCell biology030104 developmental biologyProteostasislcsh:Biology (General)lcsh:QD1-999Chaperone (protein)biology.proteinRNA InterferenceProtein foldingMolecular ChaperonesInternational Journal of Molecular Sciences
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Characterizing microstructural tissue properties in multiple sclerosis with diffusion MRI at 7 T and 3 T: The impact of the experimental design

2019

The recent introduction of advanced magnetic resonance (MR) imaging techniques to characterize focal and global degeneration in multiple sclerosis (MS), like the Composite Hindered and Restricted Model of Diffusion, or CHARMED, diffusional kurtosis imaging (DKI) and Neurite Orientation Dispersion and Density Imaging (NODDI) made available new tools to image axonal pathology non-invasively in vivo. These methods already showed greater sensitivity and specificity compared to conventional diffusion tensor-based metrics (e.g., fractional anisotropy), overcoming some of its limitations. While previous studies uncovered global and focal axonal degeneration in MS patients compared to healthy contr…

0301 basic medicineTime FactorsUltra-high field MRIAxonal pathologyCohort Studies0302 clinical medicineNuclear magnetic resonancemethods [Diffusion Magnetic Resonance Imaging]MicrostructureNODDImedicine.diagnostic_testGeneral NeuroscienceWATER DIFFUSIONmedicine.anatomical_structureResearch DesignKurtosisMulti-shell diffusion MRIAxonal degenerationWHITE-MATTERTENSORAdultMaterials sciencetherapy [Multiple Sclerosis]Sensitivity and SpecificityWhite matterMultiple sclerosis03 medical and health sciencesFractional anisotropyImage Interpretation Computer-Assistedmedicinediagnostic imaging [Nerve Degeneration]Journal ArticleHumansddc:610OPTIMIZATIONMultiple sclerosisinstrumentation [Diffusion Magnetic Resonance Imaging]diagnostic imaging [Multiple Sclerosis]Magnetic resonance imagingQUANTIFICATIONmedicine.diseaseMODELPATHOLOGYDiffusion Magnetic Resonance Imaging030104 developmental biologyRESOLUTIONDENSITYNerve Degeneration030217 neurology & neurosurgeryDiffusion MRI
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Dibutyl Phthalate (DBP)-Induced Apoptosis and Neurotoxicity are Mediated via the Aryl Hydrocarbon Receptor (AhR) but not by Estrogen Receptor Alpha (…

2016

Dibutyl phthalate (di-n-butyl phthalate, DBP) is one of the most commonly used phthalate esters. DBP is widely used as a plasticizer in a variety of household industries and consumer products. Because phthalates are not chemically bound to products, they can easily leak out to enter the environment. DBP can pass through the placental and blood–brain barriers due to its chemical structure, but little is known about its mechanism of action in neuronal cells. This study demonstrated the toxic and apoptotic effects of DBP in mouse neocortical neurons in primary cultures. DBP stimulated caspase-3 and LDH activities as well as ROS formation in a concentration (10 nM–100 µM) and time-dependent (3–…

0301 basic medicineTime Factorsgenetic structuresPPARγPeroxisome proliferator-activated receptorApoptosis010501 environmental sciencesToxicology01 natural sciencesDBPMicechemistry.chemical_compoundERβReceptorCells CulturedERαCerebral CortexNeuronschemistry.chemical_classificationbiologyCaspase 3General NeurosciencePhthalateDibutyl PhthalatePhthalateOriginal ArticleSignal transductioncirculatory and respiratory physiologymedicine.medical_specialtyCell SurvivalDibutyl phthalateNeuroscience(all)03 medical and health sciencesInternal medicinemedicineAnimalsEstrogen Receptor betaRNA Messengercardiovascular diseasesEstrogen receptor beta0105 earth and related environmental sciencesDose-Response Relationship DrugAhREstrogen Receptor alphaNeuronAryl hydrocarbon receptorPPAR gamma030104 developmental biologyEndocrinologyReceptors Aryl Hydrocarbonchemistrybiology.proteinReactive Oxygen SpeciesEstrogen receptor alphaNeurotoxicity Research
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Phenolic Compounds in Extra Virgin Olive Oil Stimulate Human Osteoblastic Cell Proliferation.

2016

In this study, we aimed to clarify the effects of phenolic compounds and extracts from different extra virgin olive oil (EVOO) varieties obtained from fruits of different ripening stages on osteoblast cells (MG-63) proliferation. Cell proliferation was increased by hydroxytyrosol, luteolin, apigenin, p-coumaric, caffeic, and ferulic acids by approximately 11-16%, as compared with controls that were treated with one vehicle alone, while (+)-pinoresinol, oleuropein, sinapic, vanillic acid and derivative (vanillin) did not affect cell proliferation. All phenolic extracts stimulated MG-63 cell growth, and they induced higher cell proliferation rates than individual compounds. The most effective…

0301 basic medicineTime Factorslcsh:MedicineBiochemistryMass SpectrometryTreeschemistry.chemical_compoundAnimal CellsPlant ProductsMedicine and Health SciencesCaffeic acidApigeninlcsh:ScienceLuteolinChromatography High Pressure LiquidConnective Tissue CellsCultured Tumor CellsPrincipal Component AnalysisMultidisciplinaryAgricultureCell DifferentiationRipeningPlantsPhenylethyl AlcoholLipidsOsteoblast DifferentiationChemistryBiochemistryCell ProcessesConnective TissuePhysical SciencesApigeninBiological CulturesCellular TypesAnatomyResearch ArticleOlive TreesCoumaric AcidsResearch and Analysis MethodsVegetable Oils03 medical and health sciencesCaffeic AcidsPhenolsOleuropeinCell Line TumorOleaVanillic acidHumansPhenolsOlive OilCell ProliferationAnalysis of Variance030109 nutrition & dieteticsOsteoblastsDose-Response Relationship Druglcsh:RChemical CompoundsOrganismsBiology and Life SciencesCell BiologyCell CulturesOsteosarcoma CellsAgronomyOlive treesBiological Tissue030104 developmental biologychemistryFruitHydroxytyrosollcsh:QOilsCrop ScienceDevelopmental Biology
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Utilizing the nanosecond pulse technique to improve antigen intracellular delivery and presentation to treat tongue squamous cell carcinoma

2017

Background Tongue squamous cell carcinoma is the most common squamous cell carcinoma of the head and neck. Immunotherapy has great potential in the treatment of tongue squamous cell carcinoma because of its unique advantages. However, the efficacy of immunotherapy is limited by the efficiency of antigen phagocytosis by immune cells. Material and Methods We extracted dendritic cells (DCs) from human peripheral blood. Utilizing a nanosecond pulsed electric field (nsPEF), we deliver the tumour lysate protein into DCs and then incubate the DCs with PBMCs to obtain specific T cells to kill tumour cells. The biosafety of nsPEF was evaluated by the ANNEXIN V-FITC/PI kit. The efficacy of lysate pro…

0301 basic medicineTime Factorsmedicine.medical_treatmentAntigen presentationFlow cytometry03 medical and health sciences0302 clinical medicineImmune systemElectricityAntigenAntigens NeoplasmmedicineCarcinomaHumansGeneral DentistryAntigen Presentationmedicine.diagnostic_testbusiness.industryResearchDendritic CellsIntracellular MembranesImmunotherapyDendritic cellmedicine.disease:CIENCIAS MÉDICAS [UNESCO]Tongue Neoplasms030104 developmental biologyCell killingOtorhinolaryngology030220 oncology & carcinogenesisUNESCO::CIENCIAS MÉDICASCarcinoma Squamous CellCancer researchSurgeryImmunotherapyOral Surgerybusiness
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Anti-inflammatory and cognitive effects of interferon-β1a (IFNβ1a) in a rat model of Alzheimer’s disease

2018

Background: Aβ 1-42 peptide abnormal production is associated with the development and maintenance of neuroinflammation and oxidative stress in brains from Alzheimer disease (AD) patients. Suppression of neuroinflammation may then represent a suitable therapeutic target in AD. We evaluated the efficacy of IFNβ1a in attenuating cognitive impairment and inflammation in an animal model of AD. Methods: A rat model of AD was obtained by intra-hippocampal injection of Aβ 1-42 peptide (23 μg/2 μl). After 6 days, 3.6 μg of IFNβ1a was given subcutaneously (s.c.) for 12 days. Using the novel object recognition (NOR) test, we evaluated changes in cognitive function. Measurement of pro-inflammatory or …

0301 basic medicineTime Factorsmedicine.medical_treatmentHippocampusCell CountPharmacologymedicine.disease_causeHippocampuslcsh:RC346-429Superoxide Dismutase-10302 clinical medicineNeuroinflammationNF-kBMicrogliaGeneral NeuroscienceMicrofilament ProteinsROSPro-inflammatory cytokineIFNβ1amedicine.anatomical_structureCytokineNeurologyIL-10CytokinesFemalemedicine.symptomAlzheimer's diseaseInterferon beta-1aPro-inflammatory cytokinesImmunologyAβ 1-42InflammationProinflammatory cytokine03 medical and health sciencesCellular and Molecular NeuroscienceHippocampuAlzheimer DiseaseGlial Fibrillary Acidic ProteinmedicineAnimalsAβ1-42Rats WistarSODMaze Learninglcsh:Neurology. Diseases of the nervous systemNeuroinflammationInflammationAmyloid beta-PeptidesNeuroscience (all)Superoxide Dismutasebusiness.industryResearchCalcium-Binding ProteinsRecognition Psychologymedicine.diseasePeptide FragmentsRatsDisease Models Animal030104 developmental biologyLipid PeroxidationCognition DisordersReactive Oxygen Speciesbusiness030217 neurology & neurosurgeryOxidative stressJournal of Neuroinflammation
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Long-term genomic coevolution of host-parasite interaction in the natural environment

2017

Antagonistic coevolution of parasite infectivity and host resistance may alter the biological functionality of species, yet these dynamics in nature are still poorly understood. Here we show the molecular details of a long-term phage–bacterium arms race in the environment. Bacteria (Flavobacterium columnare) are generally resistant to phages from the past and susceptible to phages isolated in years after bacterial isolation. Bacterial resistance selects for increased phage infectivity and host range, which is also associated with expansion of phage genome size. We identified two CRISPR loci in the bacterial host: a type II-C locus and a type VI-B locus. While maintaining a core set of conse…

0301 basic medicineTime Factorsmedicine.medical_treatmentvirusesGeneral Physics and AstronomyGenomeCRISPR SpacersbakteeritBacteriophageEnvironmental MicrobiologyCRISPRBacteriophagesClustered Regularly Interspaced Short Palindromic RepeatsANTAGONISTIC COEVOLUTIONADAPTATIONbacteriaInfectivityGenetics0303 health scienceseducation.field_of_studyMultidisciplinaryQgenomiikkaBACTERIOPHAGE RESISTANCE MECHANISMSresistance (medicine)bacteriophagesPhage therapyScienceAntagonistic Coevolution030106 microbiologyPopulationevoluutioVirulencePHAGELocus (genetics)Genome ViralBiologyFlavobacteriumArticlebakteriofagitGeneral Biochemistry Genetics and Molecular BiologyHost-Parasite InteractionsEvolution Molecular03 medical and health sciencesCRISPR-CAS SYSTEMSFISHevolutionmedicinegenomicseducationGenome size1172 Environmental sciences030304 developmental biology030306 microbiologyGeneral Chemistrybiology.organism_classificationEVOLUTIONresistenssiPATHOGEN FLAVOBACTERIUM-COLUMNARE030104 developmental biologyMutationCRISPR LociVIRULENCEIMMUNE-SYSTEMGenome BacterialNature Communications
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