Search results for "Transforming growth factor"

showing 10 items of 294 documents

MicroRNAs play a central role in molecular dysfunctions linking inflammation with cancer.

2013

It is now largely admitted that a pro-inflammatory environment may curtail anti-tumor immunity and favor cancer initiation and progression. The discovery that small non-coding regulatory RNAs, namely microRNAs (miRNAs), regulate all aspects of cell proliferation, differentiation, and function has shed a new light on regulatory mechanisms linking inflammation and cancer. Thus, miRNAs such as miR-21, miR-125b, miR-155, miR-196, and miR-210 that are critical for the immune response or hypoxia are often overexpressed in cancers and leukemias. Given the high number of their target transcripts, their deregulation may have a number of deleterious consequences, depending on the cellular context. In…

ImmunologyBiologyBioinformaticsmedicine.disease_causemiR-155Transforming Growth Factor betaNeoplasmsmicroRNAmedicineImmunology and AllergyAnimalsHumansTranscription factorCell ProliferationRegulation of gene expressionInflammationImmunityBiological TherapyGene Expression Regulation NeoplasticMicroRNAsCell Transformation NeoplasticTumor EscapeCancer researchTumor EscapeSignal transductionCarcinogenesisTransforming growth factorSignal TransductionTranscription FactorsImmunological reviews
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Inhibition of human allergic T-helper type 2 immune responses by induced regulatory T cells requires the combination of interleukin-10-treated dendri…

2006

Summary Background In grass pollen-allergic individuals, T cell anergy can be induced by IL-10-treated dendritic cells (IL-10-DC) resulting in the suppression of T helper type 1 (Th1) as well as Th2 cells. This study was performed to analyse whether such IL-10-DC-treated T cells are able to act as regulatory T cells (Treg) suppressing the function of other T cells in the periphery. As transforming growth factor (TGF)-β is also a potential inducer of Treg, we additionally analysed the inhibitory capacity of TGF-β-treated T cells in this system. Materials and Methods Freshly isolated CD4+ or CD4+CD25− T cells from grass pollen-allergic donors were stimulated with autologous mature monocyte-de…

ImmunologyEnzyme-Linked Immunosorbent AssayCell CommunicationBiologyPoaceaeT-Lymphocytes RegulatoryInterleukin 21Interferon-gammaTh2 CellsAntigens CDTransforming Growth Factor betaHypersensitivityImmunology and AllergyCytotoxic T cellHumansCTLA-4 AntigenIL-2 receptorAntigen-presenting cellCells CulturedInterleukin 3Cell ProliferationDendritic cellDendritic CellsAllergensNatural killer T cellFlow CytometryAntigens DifferentiationCell biologyInterleukin-10ImmunologyInterleukin 12PollenImmunizationInterleukin-4Interleukin-5Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology
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Reciprocal IFN-gamma and TGF-beta responses regulate the occurrence of mucosal inflammation.

1997

The above new findings concerning the immunological mechanisms governing mucosa, immune responses and oral tolerance in TCR-transgenic mice, as well as those operative in mice with experimental colitis, greatly expand our understanding of the processes that normally control mucosal inflammation and possibly other types of inflammation as well (Fig. 1). They indicate that, in the nondiseased mouse, ingested proteins evoke a Th1-cell (IFN-gamma) response in the mucosal follicles that is quickly counter-regulated by induction of T-cell anergy/deletion, if this Th1-cell response is inhibited (experimentally by anti-IL-12), TGF beta-producing cells appear, and these are capable of active immune …

ImmunologyMucosal inflammationGastrointestinal inflammationExperimental colitisBiologyColitisProinflammatory cytokineInterferon-gammaInterferon γTransforming Growth Factor betaImmunologyAnimalsHumansIntestinal MucosaOral toleranceTransforming growth factorImmunology today
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Total RNA-isolation of abdominal hernia of rats for quantitative real-time reverse transcription (RT) PCR assays.

2007

Abstract Increasing complications in incisional hernia surgery call for novel treatments. A gene expression analysis of injured tissues displays important parameters for tissue regeneration. Until today, no reliable method has been described for a quantitative gene expression analysis of hernia tissues. In this work, a protocol is described for the isolation of DNA‐free total RNA of incisional hernias for the first time. Moreover, real‐time RT PCR assays for collagen type I and III and TGF‐β1 are demonstrated for relative gene expression analyses. Both methods enable relative gene expression analyses of hernia tissues for the first time.

Incisional herniaAbdominal HerniaBiologyBiochemistryCollagen Type ITransforming Growth Factor beta1Gene expressionmedicineAnimalsHerniaGeneBase SequenceReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingGeneral Medicinemedicine.diseaseMolecular biologyReverse transcriptaseHernia AbdominalRatssurgical procedures operativeReal-time polymerase chain reactionCollagen Type IIIRNABiological AssayRNA extractionBiotechnologyPreparative biochemistrybiotechnology
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Geldanamycin and its derivatives as Hsp90 inhibitors

2012

The Hsp90 molecule, one of the most abundant heat shock proteins in mammalian cells, maintains homeostasis and prevents stress-induced cellular damage. Hsp90 is expressed under normal conditions at a level of about 1-2 Percent of total proteins, while its expression increases 2-10 fold in cancer cells. The two main constitutively expressed isoforms of Hsp90 are known as Hsp90-alpha and Hsp90-beta, and their upregulation is associated with tumor progression, invasion and formation of metastases, as well as development of drug resistance. The Hsp90 is a key target for many newly established, potent anticancer agents containing Hsp90 N-terminal ATP binding inhibitors, such as geldanamycin, and…

IndolesLactams MacrocyclicCyclin-Dependent KinaseAntineoplastic AgentsTanespimycinBenzoquinoneModels BiologicalAntineoplastic Agentchemistry.chemical_compoundDownregulation and upregulationTransforming Growth Factor betaCyclin-dependent kinaseHeat shock proteinBenzoquinonespolycyclic compoundsAnimalsHumansHSP90 Heat-Shock ProteinsbiologyAnimalTriazolesGeldanamycinHsp90Cyclin-Dependent KinasesProto-Oncogene Proteins c-rafHSP90 Heat-Shock Proteinsrc-Family KinaseschemistryTumor progressionMutationCancer cellbiology.proteinCancer researchMacrolidesMacrolideTriazoleTumor Suppressor Protein p53Animals; Antineoplastic Agents; Benzoquinones; Cyclin-Dependent Kinases; HSP90 Heat-Shock Proteins; Humans; Lactams Macrocyclic; Macrolides; Models Biological; Mutation; Novobiocin; Proto-Oncogene Proteins c-raf; Transforming Growth Factor beta; Triazoles; Tumor Suppressor Protein p53; src-Family KinasesNovobiocinHumanFrontiers in Bioscience
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The inhibitor of differentiation-1 (Id1) enables lung cancer liver colonization through activation of an EMT program in tumor cells and establishment…

2017

Abstract: Id1 promotes carcinogenesis and metastasis, and predicts prognosis of non-small cell lung cancer (NSCLC)-adenocarcionoma patients. We hypothesized that Id1 may play a critical role in lung cancer colonization of the liver by affecting both tumor cells and the microenvironment. Depleted levels of Id1 in LLC (Lewis lung carcinoma cells, LLC shId1) significantly reduced cell proliferation and migration in vitro. Genetic loss of Id1 in the host tissue (Id1(-/-) mice) impaired liver colonization and increased survival of Id1 animals. Histologically, the presence of Idl in tumor cells of liver metastasis was responsible for liver colonization. Microarray analysis comparing liver tumor n…

Inhibitor of Differentiation Protein 10301 basic medicineCancer ResearchPathologyLung NeoplasmsTime Factors10255 Clinic for Thoracic SurgeryVimentinmedicine.disease_causeMetastasisCarcinoma Lewis Lung0302 clinical medicineCell MovementCarcinoma Non-Small-Cell LungTumor Microenvironment1306 Cancer ResearchMice KnockoutTissue microarrayIntegrin beta1Liver NeoplasmsTumor BurdenGene Expression Regulation NeoplasticOncology030220 oncology & carcinogenesis2730 OncologySignal Transductionmedicine.medical_specialtyEpithelial-Mesenchymal TransitionLiver tumor610 Medicine & healthBiologyTransforming Growth Factor beta103 medical and health sciencesCell Line Tumor10049 Institute of Pathology and Molecular PathologymedicineAnimalsHumansVimentinEpithelial–mesenchymal transitionLung cancerCell ProliferationLewis lung carcinomamedicine.diseaseMice Inbred C57BL030104 developmental biologyCancer researchbiology.proteinHuman medicineSnail Family Transcription FactorsCarcinogenesisCancer Letters
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Prestress in the extracellular matrix sensitizes latent TGF-β1 for activation

2014

A mild strain induced by matrix remodeling mechanically primes latent TGF-β1 for its subsequent activation and release in response to contractile forces.

IntegrinsAnimals; Cell Differentiation; Cells Cultured; Extracellular Matrix/metabolism; HEK293 Cells; Humans; Integrins/metabolism; Integrins/physiology; Mechanotransduction Cellular; Myofibroblasts/cytology; Myofibroblasts/metabolism; Rats Wistar; Transforming Growth Factor beta1/metabolismmedicine.medical_treatmentCellular differentiationCèl·lulesIntegrinContext (language use)BiologyMechanotransduction CellularArticleExtracellular matrixTransforming Growth Factor beta1Membranes (Biologia)medicineAnimalsHumansRats WistarMyofibroblastsCells CulturedResearch ArticlesGrowth factorHEK 293 cellsCell DifferentiationCell BiologyExtracellular MatrixHEK293 Cellsbiology.proteinBiophysicsMyofibroblastTransforming growth factor
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Human CD25+ regulatory T cells: two subsets defined by the integrins alpha 4 beta 7 or alpha 4 beta 1 confer distinct suppressive properties upon CD4…

2004

Down-regulation of autoreactive T cell responses in vivo includes cell-contact-dependent as well as contact-independent mechanisms. Infectious tolerance is a contact-dependent mechanism used by naturally occurring CD25(+) T regulatory cells (Tregs) to confer suppressive activity upon conventional CD4(+) T cells thereby generating secondary T helper suppressor cells(Th(sup)), which inhibit T cell activation via soluble mediators. Here, we describe two distinct subsets of human Tregs, characterized by expression of either the alpha(4)beta(7) integrin or the alpha(4)beta(1) integrin. Upon activation, both subsets show an enhanced expression of FoxP3, recently described as a key transcription f…

IntegrinsbiologyT cellImmunologyIntegrinFOXP3Receptors Interleukin-2T lymphocyteT-Lymphocytes Helper-InducerCell biologyInterleukin-10Interleukin 21medicine.anatomical_structureT-Lymphocyte SubsetsTransforming Growth Factor betaImmunologymedicinebiology.proteinImmunology and AllergyCytotoxic T cellHumansIL-2 receptorBeta (finance)European journal of immunology
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Mast cells promote Th1 and Th17 responses by modulating dendritic cell maturation and function

2011

Mast cells (MCs) play an important role in the regulation of protective adaptive immune responses against pathogens. However, it is still unclear whether MCs promote such host defense responses via direct effects on T cells or rather by modifying the functions of antigen-presenting cells. To identify the underlying mechanisms of the immunoregulatory capacity of MCs, we investigated the impact of MCs on dendritic cell (DC) maturation and function. We found that murine peritoneal MCs underwent direct crosstalk with immature DCs that induced DC maturation as evidenced by enhanced expression of costimulatory molecules. Furthermore, the MC/DC interaction resulted in the release of the T-cell mod…

Interleukin 2Cell growthImmunologyDendritic cellTransforming growth factor betaBiologyhumanitiesCell biologyImmune systemInterleukin 12medicinebiology.proteinImmunology and AllergyInterferon gammaInterleukin 17medicine.drugEuropean Journal of Immunology
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Expansion of intestinal CD4+CD25highTreg cells in patients with ankylosing spondylitis: A putative role for interleukin-10 in preventing intestinal T…

2010

Objective Subclinical gut inflammation has been demonstrated in patients with ankylosing spondylitis (AS). This study was undertaken to determine the frequency of regulatory CD4+CD25high T cells (Treg cells) and to evaluate Treg cell–related cytokines (interleukin-2 [IL-2], transforming growth factor β [TGFβ], and IL-10) and transcription factors (FoxP3 and STAT-5) in the ileum of patients with AS. Methods Quantitative gene expression analysis, by reverse transcriptase–polymerase chain reaction, of Treg-related cytokines (IL-2, TGFβ, and IL-10) and transcription factors (STAT-5 and FoxP3) was performed on ileal biopsy specimens from 18 patients with AS, 15 patients with active Crohn's disea…

Interleukin 2medicine.diagnostic_testImmunologyFOXP3InflammationBiologyInterleukin 10RheumatologyIntestinal mucosaImmunologyBiopsymedicineInterleukin 23Immunology and AllergyPharmacology (medical)medicine.symptomTransforming growth factormedicine.drugArthritis & Rheumatism
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