Search results for "Transmitter"

showing 10 items of 348 documents

In vitro effects of putative neurotransmitters on synaptic ribbon numbers and N-acetyltransferase activity in the rat pineal gland

1992

The pineal contains a large number of classical transmitters and neuropeptides. Some of these neurochemicals are involved in the regulation of serotonin N-acetyltransferase (NAT) activity and hence in melatonin synthesis. Synaptic ribbons present in the pineal gland also exhibit a numerical day/night rhythm parallel to that of NAT activity. There is scarcity of information regarding the regulation of synaptic ribbon (SR) numbers. In the present study, we have investigated in vitro effects of a number of classical neurotransmitters and neuropeptides. NAT activity was used to monitor melatonin synthesis under the experimental conditions used. Norepinephrine (NE), Delta sleep-inducing peptide …

Maleendocrine systemmedicine.medical_specialtyArylamine N-AcetyltransferaseVasoactive intestinal peptideNeuropeptideBiologyPineal Glandgamma-Aminobutyric acidRats Sprague-DawleyPineal glandOrgan Culture TechniquesInternal medicinemedicineAnimalsBiological PsychiatrySynaptic ribbonNeurotransmitter AgentsfungiCircadian RhythmRatsbody regionsMicroscopy ElectronPsychiatry and Mental healthmedicine.anatomical_structureEndocrinologySomatostatinNeurologySynaptic VesiclesNeurology (clinical)Serotoninhormones hormone substitutes and hormone antagonistsmedicine.drugEndocrine glandJournal of Neural Transmission
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Melatonin binding sites in brain of the 2-day-old chicken: An autoradiographic localisation

1990

Melatonin, released in a circadian manner from the avian pineal gland into the circulation, is concentrated in distinct brain areas, possibly by receptor-mediated uptake mechanisms. For anatomical localisation of putative melatonin receptors in birds, cryostat-cut, coronal 15 microns brain sections of 2-day-old chicken were investigated by means of in vitro receptorautoradiography, using 125I-melatonin as a ligand. Binding occurred in brain structures receiving or mediating visual or auditory sensory input. Binding characteristics were investigated in homogenates of a brain region labelled by 125I-melatonin and showed to be specific and saturable (Kd: 87.2 pM; Bmax: 16.1 fmol/mg protein). R…

Maleendocrine systemmedicine.medical_specialtyCentral nervous systemReceptors MelatoninBiologyMelatoninPineal glandInternal medicinemedicineAnimalsCircadian rhythmReceptorBiological PsychiatryMelatoninSuprachiasmatic nucleusBrainLigand (biochemistry)Receptors NeurotransmitterPsychiatry and Mental healthEndocrinologymedicine.anatomical_structureAnimals NewbornNeurologyMelatonin bindingAutoradiographyNeurology (clinical)Chickenshormones hormone substitutes and hormone antagonistsmedicine.drugJournal of Neural Transmission
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Neurochemistry of olivocochlear neurons in the hamster.

2009

The present study was conducted to characterize the superior olivary complex (SOC) of the lower brain stem in the pigmented Djungarian hamster Phodopus sungorus. Using Nissl-stained serial cryostat sections from fresh-frozen brains, we determined the borders of the SOC nuclei. We also identified olivocochlear (OC) neurons by retrograde neuronal tracing upon injection of Fluoro-Gold into the scala tympani. To evaluate the SOC as a putative source of neuronal nitric oxide synthase (nNOS), arginine-vasopressin (AVP), oxytocin (OT), vasoactive intestinal polypeptide (VIP), or pituitary adenylate cyclase-activating polypeptide (PACAP) that were all found in the cochlea, we conducted immunohistoc…

Maleendocrine systemmedicine.medical_specialtyHistologyAuditory PathwaysPhodopusStilbamidinesVasoactive intestinal peptideHamsterNitric Oxide Synthase Type IOlivary NucleusNitric OxideOxytocinEfferent Pathwayschemistry.chemical_compoundInternal medicineCricetinaeNitrergic NeuronsPonsotorhinolaryngologic diseasesmedicineAnimalsNeurotransmitterEcology Evolution Behavior and SystematicsbiologyStaining and LabelingOlivocochlear systembiology.organism_classificationRetrograde tracingImmunohistochemistryCochleaNeuronal tracingPhodopusArginine VasopressinEndocrinologynervous systemchemistrySuperior olivary complexPituitary Adenylate Cyclase-Activating Polypeptidesense organsAnatomyhormones hormone substitutes and hormone antagonistsBiotechnologyAnatomical record (Hoboken, N.J. : 2007)
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Candidate genetic pathways for attention-deficit/hyperactivity disorder (ADHD) show association to hyperactive/impulsive symptoms in children with AD…

2013

Item does not contain fulltext OBJECTIVE: Because multiple genes with small effect sizes are assumed to play a role in attention-deficit/hyperactivity disorder (ADHD) etiology, considering multiple variants within the same analysis likely increases the total explained phenotypic variance, thereby boosting the power of genetic studies. This study investigated whether pathway-based analysis could bring scientists closer to unraveling the biology of ADHD. METHOD: The pathway was described as a predefined gene selection based on a well-established database or literature data. Common genetic variants in pathways involved in dopamine/norepinephrine and serotonin neurotransmission and genes involv…

Malegenetic pathwaysMedizinSocial SciencesGenome-wide association studyDCN PAC - Perception action and controlSeverity of Illness Index/dk/atira/pure/sustainabledevelopmentgoals/clean_water_and_sanitationneuritic outgrowth2738 Psychiatry and Mental HealthDOPAMINE0302 clinical medicineDevelopmental and Educational PsychologyIsraelChildQUANTITATIVE TRAITS10058 Department of Child and Adolescent PsychiatryGenomic disorders and inherited multi-system disorders [DCN PAC - Perception action and control IGMD 3]EuropePsychiatry and Mental healthattention-deficit/hyperactivity disorder symptomsPhenotypeChild PreschoolFemalemedicine.symptomPsychologySDG 6 - Clean Water and Sanitationmedicine.drugClinical psychologymedicine.medical_specialtyDIAGNOSTIC-APPROACHAdolescentGenotypeLARGE MULTICENTER ADHDIDENTIFIES ASSOCIATIONDEFICIT HYPERACTIVITY DISORDERDCN MP - Plasticity and memoryneurotransmitter systems610 Medicine & healthQuantitative trait locusHyperkinesisImpulsivityMental health [NCEBP 9]Genomic disorders and inherited multi-system disorders DCN MP - Plasticity and memory [IGMD 3]03 medical and health sciencesDopamineRating scalemedicineCriterion validityddc:61Attention deficit hyperactivity disorderHumansddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersDCN PAC - Perception action and control NCEBP 9 - Mental healthGENOME-WIDE ASSOCIATIONNITRIC-OXIDE SYNTHASEPsychiatryGenetic Association StudiesMETAANALYSISPsychiatric Status Rating Scales3204 Developmental and Educational Psychologymedicine.disease030227 psychiatryAttention Deficit Disorder with HyperactivityImpulsive BehaviorEtiologyCRITERION VALIDITY030217 neurology & neurosurgery
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Day- and night-time contents of monoamines and their metabolites in the medial preoptic area of the rat hypothalamus.

1999

The present study was conducted to investigate whether monoamines and their metabolites in the medial preoptic area (mPOA) of the rat hypothalamus exhibit differences in their contents between day and night. We therefore sampled the mPOA from adult animals of either sex at the middle of the light or dark period, respectively, and analyzed the tissue by means of high performance liquid chromatography with electrochemical detection. We found that, in female animals at mid-night, dopamine and 3,4-dihydroxyphenyl acetic acid (DOPAC) was reduced to 43 and 30%, respectively, of daytime levels, while the norepinephrine content was doubled. No significant differences were observed in male animals. …

Malemedicine.medical_specialty34-Dihydroxyphenylacetic acidTyrosine 3-MonooxygenaseDopamine beta-HydroxylaseBiologyRats Sprague-Dawleychemistry.chemical_compoundNorepinephrineDopamineInternal medicinemedicineAnimalsBiogenic MonoaminesNeurotransmitterSex CharacteristicsTyrosine hydroxylaseGeneral NeuroscienceImmunohistochemistryPreoptic AreaCircadian RhythmRatsPreoptic areaPerfusionMonoamine neurotransmitterEndocrinologychemistryHypothalamusCatecholamine34-Dihydroxyphenylacetic AcidFemalemedicine.drugNeuroscience letters
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Are the effects of the antidepressants amitriptyline, maprotiline, and fluoxetine on inhibitory avoidance state-dependent?

2005

Abstract State-dependent learning (SDL) is a phenomenon in which the retrieval of newly acquired information is possible if the subject is in the same physiological state as during the encoding phase. SDL makes it possible to separate the effects of drugs per se on learning from the effects due to changes in drug state during the task. The present work was designed to investigate whether the antidepressants amitriptyline (30 mg/kg), maprotiline (25 mg/kg), and fluoxetine (15 mg/kg) produce SDL of the inhibitory avoidance conditioning in male and female CD1 mice. In three separate experiments, independent groups were used for each pharmacological treatment and for each sex using a 2 × 2 expe…

Malemedicine.medical_specialtyAmitriptylinePharmacologyMiceBehavioral Neurosciencechemistry.chemical_compoundSex FactorsFluoxetineAvoidance LearningmedicineAnimalsAmitriptylineNeurotransmitterPsychiatryMaprotilineFluoxetineBehavior AnimalAntidepressive AgentsInhibition PsychologicalMaprotilinechemistryFacilitationConditioningFemaleSerotoninReuptake inhibitorPsychologymedicine.drugBehavioural Brain Research
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Prenatal cocaine alters later responses to morphine in adult male mice.

2006

Mice prenatally exposed to cocaine (25 mg/kg), physiological saline or non-treated during the last 6 days of pregnancy were evaluated as adults for the rewarding properties of 2 mg/kg of morphine, using the conditioned place preference (CPP) procedure. Likewise, isolated animals underwent a social interaction test with conspecifics after receiving the same morphine dose. Unlike control or animals pre-treated with saline, subjects prenatally treated with cocaine did not develop CPP with this dose of morphine. Only cocaine-exposed animals showed increased threat, avoidance and fleeing during the social encounter. No differences in motor effects of morphine were observed. Analysis of monoamine…

Malemedicine.medical_specialtyBiogenic AminesOffspringmedicine.medical_treatmentPharmacologyMotor ActivityMiceCocaineRewardPregnancyInternal medicinemedicineAnimalsInterpersonal RelationsSalineBiological PsychiatryPharmacologyBrain ChemistryPregnancyBehavior AnimalMorphinePrenatal cocaine exposuremedicine.diseaseConditioned place preferenceAggressionAnalgesics OpioidMonoamine neurotransmitterEndocrinologyPrenatal Exposure Delayed EffectsToxicityMorphineExploratory BehaviorConditioning OperantFemalePsychologymedicine.drugProgress in neuro-psychopharmacologybiological psychiatry
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Acute ammonia toxicity is mediated by the NMDA type of glutamate receptors

1992

AbstractPrevious experiments in our laboratory suggested that ammonium toxicity could be mediated by the NMDA type of glutamate receptors. To assess this hypothesis we tested if MK-801, a specific antagonist of the NMDA receptor, is able to prevent ammonium toxicity. Mice and rats were injected i.p. with 12 and 7 mmol/kg of ammonium acetate, respectively, 73% of the mice and 70% of the rats died. However, when the animals were injected i.p. with 2 mg/kg of MK-801, 15 min before ammonium injection, only 5% of the mice and 15% of the rats died. The remarkable protection afforded by MK-801 indicates that ammonia toxicity is mediated by the NMDA receptor.

Malemedicine.medical_specialtyBiophysicsGlutamic AcidReceptors N-Methyl-D-AspartateBiochemistryAmmonia toxicityMicechemistry.chemical_compoundGlutamatesAmmoniaStructural BiologyInternal medicineGeneticsmedicineAnimalsHyperammonemiaNeurotransmitter metabolismAmmoniumReceptorMolecular BiologyMK-801Glutamate receptorRats Inbred StrainsValineCell BiologyGlutamic acidNMDA receptorRatsReceptors NeurotransmitterEndocrinologyReceptors GlutamatechemistryToxicityNMDA receptorDizocilpine MaleateAmmonium acetateFEBS Letters
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Impaired cannabinoid receptor type 1 signaling interferes with stress-coping behavior in mice.

2007

Dysregulation of the endocannabinoid system is known to interfere with emotional processing of stressful events. Here, we studied the role of cannabinoid receptor type 1 (CB1) signaling in stress-coping behaviors using the forced swim test (FST) with repeated exposures. We compared effects of genetic inactivation with pharmacological blockade of CB1 receptors both in male and female mice. In addition, we investigated potential interactions of the endocannabinoid system with monoaminergic and neurotrophin systems of the brain. Naive CB1 receptor-deficient mice (CB1-/-) showed increased passive stress-coping behaviors as compared to wild-type littermates (CB1+/+) in the FST, independent of se…

Malemedicine.medical_specialtyCannabinoid receptormedicine.medical_treatmentBiologyPharmacologyHippocampusMicePiperidinesReceptor Cannabinoid CB1Internal medicineCannabinoid receptor type 1MonoaminergicAdaptation PsychologicalGeneticsmedicineAnimalsBiogenic MonoaminesRNA MessengerReceptorMonoamine OxidaseSwimmingPharmacologyBrain-derived neurotrophic factormusculoskeletal neural and ocular physiologyBrain-Derived Neurotrophic FactorDesipraminefood and beveragesEndocannabinoid systemMice Inbred C57BLMonoamine neurotransmitterEndocrinologynervous systemVesicular Glutamate Transport Protein 1Molecular MedicinePyrazoleslipids (amino acids peptides and proteins)FemaleCannabinoidRimonabantpsychological phenomena and processesStress PsychologicalSignal TransductionThe pharmacogenomics journal
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Neurotensin: dual effect on the motor activity of rat duodenum

1992

The effects of neurotensin on mechanical activity of rat duodenum were investigated using an isometric-isovolumic preparation. Neurotensin (1 pM to 10 nM) induced a concentration-dependent, tetrodotoxin (TTX)-insensitive fall in both endoluminal pressure and isometric tension. At higher concentrations of neurotensin (1 nM to 1 microM) the relaxation was followed by a concentration-dependent TTX-insensitive contraction, detected only by an increase in endoluminal pressure. Different concentrations of neurotensin were required to desensitize the relaxant and the contractile actions of the neuropeptide. The relaxation was antagonized by apamin, while the contractile response was blocked by nif…

Malemedicine.medical_specialtyContraction (grammar)NifedipineDuodenumMuscle RelaxationNeuropeptideIn Vitro TechniquesBiologyApamincomplex mixturesdigestive systemchemistry.chemical_compoundNifedipineInternal medicinemedicineAnimalsReceptors NeurotensinNeurotensinPharmacologymusculoskeletal neural and ocular physiologydigestive oral and skin physiologyRats Inbred StrainsElectric StimulationRatsReceptors NeurotransmitterMuscle relaxationEndocrinologyApaminnervous systemchemistryTetrodotoxinCalciummedicine.symptomGastrointestinal MotilityMuscle ContractionNeurotensinmedicine.drugMuscle contractionEuropean Journal of Pharmacology
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