Search results for "Transmitter"

showing 10 items of 348 documents

Alzheimer's disease: amino acid levels and brain metabolic status.

2013

Abstract To study brain free amino acids and their relation with dementia we measured, by high-performance liquid chromatography (HPLC), the concentration of eight free amino acids, amines and related compounds. We used temporal cortex (TC) samples obtained from 13 Alzheimer’s disease (AD) patients and an equal number of agematched controls (AC). The patterns of free amino acids, amines and related compounds showed significant quantitative changes in AD conditions with respect to healthy ones. In Alzheimer patients, lower levels of GABA were found in the TC (-57 %). Amino acids glutamate (Glu), and aspartate (Asp) concentrations, also appeared significantly reduced in the TC of AD patients …

Malemedicine.medical_specialtyDermatologyBiologychemistry.chemical_compoundBrain: Temporal cortexAlzheimer DiseaseSettore BIO/10 - BiochimicaInternal medicinemedicineHumansNeurotransmitterAmino AcidsChromatography High Pressure Liquidgamma-Aminobutyric AcidAgedTemporal cortexchemistry.chemical_classificationMethionineGlutamate receptorBrainGeneral Medicinemedicine.diseaseCystathionine beta synthaseAmino acidAmino acidGlutaminePsychiatry and Mental healthEndocrinologychemistryBiochemistryTemporal cortex; Amino acids; Neurotransmitters; [Keywords Alzheimer’s disease; Brain]biology.proteinFemaleKeywords Alzheimer’s diseaseNeurology (clinical)Alzheimer's diseaseTransmethylationNeurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology
researchProduct

Antagonist discrimination between subtypes of tachykinin receptors in the guinea-pig ileum.

1986

1. The effects of substance P and eledoisin on spontaneous and electrically-evoked release of [3H]acetylcholine, and on smooth muscle were studied in the guineapig myenteric plexus-longitudinal muscle preparation preloaded with [3H]choline. Substance P and eledoisin caused transient increases in spontaneous release of [3H]acetylcholine and in longitudinal muscle tone. Both tachykinins were equipotent in contracting the muscle, but eledoisin was more potent than substance P in eliciting [3H]acetylcholine release. The release caused by substance P was enhanced in the presence of naloxone and scopolamine which suggests that the release is modulated through opioid and muscarinic receptors. 2. S…

Malemedicine.medical_specialtyEledoisinGuinea PigsScopolamineSubstance P(+)-NaloxoneSubstance Pcomplex mixtureschemistry.chemical_compoundEledoisinIleumInternal medicineMuscarinic acetylcholine receptormedicineAnimalsNeurotransmitterReceptorReceptors TachykininPharmacologyNaloxoneGeneral MedicineAcetylcholineReceptors NeurotransmitterEndocrinologychemistryFemaleTachykinin receptorAcetylcholinemedicine.drugMuscle ContractionNaunyn-Schmiedeberg's archives of pharmacology
researchProduct

Electrical responses of pineal cells to melatonin and putative transmitters

1981

The effects of microelectrophoretically applied melatonin (N-acetyl-5-methoxy-tryptamine), noradrenaline (NOR) or acetylcholine (ACH) on the electrical activity of pineal cells during both the day- and night-time were studied in urethane anesthetised guinea-pigs. A total of 288 cells were tested with melatonin, and in addition with either NOR (120 cells) or ACH (138 cells). Of the 206 cells responding to melatonin application, 139 were excited and 67 inhibited. A total of 85 cells responded to the application of NOR, 45 being excited and 40 inhibited. Responses to ACH application were observed in 75 pineal cells, 49 units being inhibited and 26 excited. It was possible to observe excitation…

Malemedicine.medical_specialtyGuinea PigsNeural InhibitionStimulationPineal GlandMelatoninNorepinephrineNorepinephrinePineal glandInternal medicinemedicineAnimalsCircadian rhythmEvoked PotentialsMelatoninNeurotransmitter AgentsChemistryGeneral NeuroscienceNeural InhibitionAcetylcholineElectric StimulationCircadian RhythmEndocrinologymedicine.anatomical_structureCholinergicAcetylcholinemedicine.drugExperimental Brain Research
researchProduct

Changes of neurohumoral parameters and endothelin-1 in response to exercise in patients with mild to moderate congestive heart failure

1998

Plasma endothelin levels are increased in patients with moderate and severe CHF. Conflicting data exist about the endothelin-1 (ET) level in patients with mild to moderate CHF and the effect of maximal exercise on plasma ET levels.We determined the plasma levels of ET and various neurohumoral parameters in 93 patients with CHF in functional class II and III of the NYHA classification at rest and after maximal bicycle exercise. Baseline ET level was increased compared to an age-matched healthy volunteer group (6.95+/-0.31 vs 3.29+/-0.17 pg/ml, mean+/-S.E.M., P0.05), without significant differences between NYHA class II and III patients. Maximal exercise did not increase the ET level. In cont…

Malemedicine.medical_specialtyHeart diseaseneurohumoral activationheart failurePeptide hormonePLASMA ENDOTHELINInternal medicinemedicineHumanscardiovascular diseasesExercise physiologyExerciseAgedNeurotransmitter AgentsEndothelin-1BLOOD-FLOWbusiness.industryBlood flowMiddle Agedmedicine.diseaseEndothelin 1Pathophysiologyexercise capacityEndocrinologyHeart failureExercise TestCardiologycardiovascular systemFemaleCardiology and Cardiovascular MedicineEndothelin receptorbusinessendothelin
researchProduct

Nitric oxide and glutamate interaction in the control of cortical and hippocampal excitability.

1999

Summary: Purpose: We investigated the role of nitric oxide (NO) as a new neurotransmitter in the control of excitability of the hippocampus and the cerebral cortex, as well as the possible functional interaction between NO and the glutamate systems. Methods: The experiments were performed on anesthetized rats. The bioelectrical activities of the somatosensory cortex and the CA1 region of the hippocampus of these rats were recorded. Pharmacologic inhibition of NO synthase (NOS) through the nonselective and brain-selective inhibitors, N-nitro-L-arginine methyl ester (l-NAME) and 7-nitroindazole (7-NI), was performed. Results: The treatments caused the appearance of an interictal discharge act…

Malemedicine.medical_specialtyHippocampusGlutamic AcidHippocampal formationNeurotransmissionBiologyNitric OxideHippocampusReceptors N-Methyl-D-Aspartatechemistry.chemical_compoundInternal medicinemedicineAnimalsRats WistarNeurotransmitterCerebral CortexEpilepsyGlutamate receptorSomatosensory CortexRatsEndocrinologyNeurologychemistryCNQXExcitatory postsynaptic potentialNMDA receptorNeurology (clinical)Epilepsia
researchProduct

Nitric oxide-sensitive guanylyl cyclase inhibits acetylcholine release and excitatory motor transmission in the guinea-pig ileum

1997

Abstract This study examined the mechanism through which nitric oxide inhibits the release of acetylcholine and excitatory motor neurotransmission in the guinea-pig ileum. The selective inhibitor of nitric oxide-sensitive guanylyl cyclase, 1 H -[1,2,4]oxadiazolo[4,3- a ]quinoxalin-1-one (ODQ), concentration-dependently enhanced both basal release (−log EC 50 : 6.8) and electrically (10 Hz) -evoked release (−log EC 50 : 6.0) of [ 3 H]acetylcholine from longitudinal muscle-myenteric plexus preparations preincubated with [ 3 H]choline. The increase by ODQ of basal release appeared to be exocytotic since it was prevented by tetrodotoxin (300 nM) and absence of calcium from the superfusion mediu…

Malemedicine.medical_specialtyIndazolesGuinea PigsMyenteric PlexusNeurotransmissionNitric OxideNitroarginineSynaptic TransmissionNitric oxidechemistry.chemical_compoundIleumQuinoxalinesInternal medicinemedicineAnimalsEnzyme InhibitorsNeurotransmitterMyenteric plexusMotor NeuronsOxadiazolesbiologyGeneral NeuroscienceMuscle SmoothAcetylcholineElectric StimulationNitric oxide synthaseEndocrinologychemistryGuanylate CyclaseDepression Chemicalbiology.proteinCholinergicFemaleNitric Oxide SynthaseSoluble guanylyl cyclaseAcetylcholineMuscle Contractionmedicine.drugNeuroscience
researchProduct

Inhibition by interleukin-1 beta of noradrenaline release in rat spleen: involvement of lymphocytes, NO and opioid receptors.

1995

Effects of indomethacin, N omega-nitro-L-arginine (NNA) and naloxone, and of pretreatment with cyclophosphamide (CY), on the interleukin (IL)-1 beta induced inhibition of exocytotic noradrenaline release were investigated in the isolated, vascularly perfused spleen of the rat. Neurotransmitter release was evoked by perivascular electrical stimulation (4 Hz) and the overflow of endogenous noradrenaline was determined by HPLC with electrochemical detection. Perfusion of the spleen with Tyrode's solution containing IL-1 beta (100 pg/ml) for 90 min caused an inhibition of the stimulation-evoked noradrenaline overflow which persisted for at least 20 min after washout of the IL. The evoked overfl…

Malemedicine.medical_specialtyIndomethacinSpleenEndogenyStimulation(+)-NaloxoneIn Vitro TechniquesArginineNitric OxideNitroargininechemistry.chemical_compoundNorepinephrineInternal medicinemedicineAnimalsLymphocytesRats WistarNeurotransmitterReceptorCyclophosphamidePharmacologyNaloxoneBody WeightInterleukinGeneral MedicineOrgan SizeElectric StimulationRatsmedicine.anatomical_structureEndocrinologychemistryOpioidReceptors OpioidSpleenmedicine.drugInterleukin-1Naunyn-Schmiedeberg's archives of pharmacology
researchProduct

Chronic Fluoxetine Treatment Increases the Expression of PSA-NCAM in the Medial Prefrontal Cortex

2006

Recent hypotheses suggest that changes in neuronal structure and connectivity may underlie the etiology of depression. The medial prefrontal cortex (mPFC) is affected by depression and shows neuronal remodeling during adulthood. This plasticity may be mediated by the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), which is intensely expressed in the adult mPFC. As the expression of PSA-NCAM is increased by serotonin in other cerebral regions, antidepressants acting on serotonin reuptake may influence PSA-NCAM expression and thus counteract the effects of depression by modulating neuronal structural plasticity. Using immunohistochemistry, we have studied the relationship…

Malemedicine.medical_specialtyInterneuronFluorescent Antibody TechniquePrefrontal CortexCell CountNeural Cell Adhesion Molecule L1urologic and male genital diseasesSerotonergicRats Sprague-Dawleychemistry.chemical_compoundFluoxetineInternal medicinemedicineNeuropilAnimalsPrefrontal cortexNeurotransmitter5-HT receptorNeuronsPharmacologyAnalysis of VarianceRatsPsychiatry and Mental healthmedicine.anatomical_structureEndocrinologyGene Expression Regulationnervous systemchemistryReceptors SerotoninSialic AcidsAntidepressive Agents Second-GenerationNeural cell adhesion moleculeSerotoninPsychologyNeuroscienceNeuropsychopharmacology
researchProduct

Opposite effects of γ1- and γ2-melanocyte stimulating hormone on regulation of the dopaminergic mesolimbic system in rats

2004

By use of the brain microdialysis technique we show that administration of gamma(1)-melanocyte stimulating hormone (gamma(1)-MSH) into the ventral tegmental area of anaesthetized rats causes an increase in the release of extracellular dopamine and its metabolite 3,4-dihydroxyphenylacetic acid in the nucleus accumbens, while gamma(2)-MSH causes the opposite effect. Moreover, gamma(2)-MSH pre-treatment considerably reduced the gamma(1)-MSH-induced effects. Our findings suggest an opposing action of two gamma-MSH-activated pathways on the mesolimbic dopaminergic system, which could be important in the maintenance of a balanced psychoactivation state.

Malemedicine.medical_specialtyMelanocyte-stimulating hormoneDopamineNucleus accumbensBiologyNucleus AccumbensRats Sprague-Dawleygamma-MSHchemistry.chemical_compoundDopamineInternal medicineNeural PathwaysLimbic SystemmedicineAnimalsNeurotransmitterBrain ChemistryNeuronsGeneral NeuroscienceVentral Tegmental AreaDopaminergicRatsUp-RegulationVentral tegmental areaEndocrinologymedicine.anatomical_structurechemistryCatecholamine34-Dihydroxyphenylacetic Acidmedicine.drugHormoneNeuroscience Letters
researchProduct

Mesolimbic dopaminergic system activity as a function of food reward: A microdialysis study

1996

The mesolimbic dopaminergic system (MDS) has been shown to be implicated in feeding behaviors. The present experiment was conducted to examine the effects of the sensory properties of food ingested on MDS activity. Microdialysis coupled to high-performance liquid chromatography with electrochemical detection was employed to measure the extracellular levels of dopamine (DA) and its main metabolites (DOPAC and HVA) in the nucleus accumbens of freely moving rats. During microdialysis sessions rats had access or not to powdered foods varying in palatability: short cakes as highly palatable (HP) food and regular chow as low palatable (LP) food. In the absence of food, there were no alterations i…

Malemedicine.medical_specialtyMicrodialysisDopamineMicrodialysisClinical BiochemistryNucleus accumbensToxicologyBiochemistryNucleus AccumbensRats Sprague-DawleyBehavioral Neurosciencechemistry.chemical_compoundRewardDopamineInternal medicineLimbic SystemmedicineAnimalsIngestionPalatabilityNeurotransmitterBiological PsychiatryPharmacologydigestive oral and skin physiologyDopaminergicHomovanillic AcidRatsEndocrinologychemistryFoodCatecholamine34-Dihydroxyphenylacetic Acidmedicine.drugPharmacology Biochemistry and Behavior
researchProduct