Search results for "Type 2"

showing 10 items of 1034 documents

Exploring the Role of Skeletal Muscle in Insulin Resistance: Lessons from Cultured Cells to Animal Models

2021

Skeletal muscle is essential to maintain vital functions such as movement, breathing, and thermogenesis, and it is now recognized as an endocrine organ. Muscles release factors named myokines, which can regulate several physiological processes. Moreover, skeletal muscle is particularly important in maintaining body homeostasis, since it is responsible for more than 75% of all insulin-mediated glucose disposal. Alterations of skeletal muscle differentiation and function, with subsequent dysfunctional expression and secretion of myokines, play a key role in the pathogenesis of obesity, type 2 diabetes, and other metabolic diseases, finally leading to cardiometabolic complications. Hence, a de…

QH301-705.5glucose metabolismAdipose tissueReviewType 2 diabetesCarbohydrate metabolismMuscle DevelopmentBioinformaticsCatalysisInorganic ChemistryInsulin resistanceMyokineAnimalsHumansMedicinePhysical and Theoretical ChemistryBiology (General)Muscle SkeletalMolecular BiologyQD1-999Spectroscopybusiness.industryOrganic ChemistrySkeletal musclefree fatty acidsGeneral Medicinemedicine.diseaseComputer Science Applicationsadipose tissueglycemiaDisease Models AnimalChemistrymedicine.anatomical_structureDiabetes Mellitus Type 2Adipose tissue Free fatty acids Glucose metabolism Glycemia Myofibers Animals Diabetes Mellitus Type 2 Humans Muscle Skeletal Disease Models Animal Insulin Resistance Muscle DevelopmentInsulin ResistancebusinessThermogenesismyofibersHomeostasisInternational Journal of Molecular Sciences
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Healthy lifestyle, metabolomics and incident type 2 diabetes in a population-based cohort from Spain

2022

This work was supported by the Strategic Action for Research in Health sciences [PI10/0082, PI13/01848, PI14/00874, PI16/01402, PI11/00726, PI16/609, PI16/1512, PI18/287, PI19/319 and PI20/00896], the GUTMOM Project (JPI-A Healthy Diet for a Healthy Life INTIMIC-085, State Secretary of R + D + I PCIN-2017-117), the Cátedra de Epidemiología y Control del Riesgo Cardiovascular at UAM (#820024), the State Agency for Research (PID2019-108973RB-C21 and C22), the Valencia Government (GRUPOS 03/101; PROMETEO/2009/029 and ACOMP/2013/039), the Castilla-Leon Government (GRS/279/A/08) and European Network of Excellence Ingenious Hypercare (EPSS- 037093) from the European Commission; CIBER Fisiopatolog…

RC620-627Nutrition and DieteticsResearchMedicine (miscellaneous)Healthy lifestylePhysical Therapy Sports Therapy and RehabilitationBayes TheoremType 2 diabetesDiabetes Mellitus Type 2Risk FactorsSpainHumansMetabolomicsPublic aspects of medicineRA1-1270Nutritional diseases. Deficiency diseasesCohort studySpanish population
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Incidence of Diabetes Following Ramipril or Rosiglitazone Withdrawal

2011

OBJECTIVE To examine the impact of withdrawing rosiglitazone and ramipril medication on diabetes incidence after closeout of the Diabetes REduction Assessment with ramipril and rosiglitazone Medication (DREAM) trial. RESEARCH DESIGN AND METHODS The 3,366 DREAM subjects at trial end who had not developed diabetes while taking double-blind study medication were transferred to single-blind placebo for 2 to 3 months before undergoing an oral glucose tolerance test. Glycemic status was analyzed for the trial plus washout period and for the washout period alone. RESULTS Following median (interquartile range) 71 (63–86) days drug withdrawal, overall glycemic status remained modestly improved in t…

RamiprilAdultMalemedicine.medical_specialtyEndocrinology Diabetes and Metabolism030209 endocrinology & metabolism030204 cardiovascular system & hematologyPlaceboRosiglitazone03 medical and health sciencesDrug withdrawal0302 clinical medicineRamiprilInterquartile rangeInternal medicineDiabetes mellitusInternal MedicinemedicineHumansHypoglycemic AgentsGlycemicOriginal ResearchAdvanced and Specialized Nursingbusiness.industryIncidence (epidemiology)IncidenceClinical Care/Education/Nutrition/Psychosocial ResearchMiddle Agedmedicine.disease3. Good healthSurgerySubstance Withdrawal SyndromeTreatment OutcomeDiabetes Mellitus Type 2FemaleThiazolidinedionesRosiglitazonebusinessmedicine.drugFollow-Up StudiesDiabetes Care
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Progressive right ventricular dysfunction and exercise impairment in patients with heart failure and diabetes mellitus: insights from the T.O.S.CA. R…

2022

Abstract Background Findings from the T.O.S.CA. Registry recently reported that patients with concomitant chronic heart failure (CHF) and impairment of insulin axis (either insulin resistance—IR or diabetes mellitus—T2D) display increased morbidity and mortality. However, little information is available on the relative impact of IR and T2D on cardiac structure and function, cardiopulmonary performance, and their longitudinal changes in CHF. Methods Patients enrolled in the T.O.S.CA. Registry performed echocardiography and cardiopulmonary exercise test at baseline and at a patient-average follow-up of 36 months. Patients were divided into three groups based on the degree of insulin impairmen…

RegistrieHeart FailureEndocrinology Diabetes and MetabolismVentricular Dysfunction RightDiabetesInsulinsSocio-culturaleStroke VolumeInsulin resistanceDiabeteCardiopulmonary exercise test Chronic heart failure Diabetes Insulin resistance Right ventricle TOSCA Registry..Chronic heart failureDiabetes Mellitus Type 2TOSCA RegistryExercise TestVentricular Function RightHumansInsulinRight ventricleRegistriesCardiology and Cardiovascular MedicineCardiopulmonary exercise testTOSCAHumanLS4_7
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Mboat7 down-regulation by hyper-insulinemia induces fat accumulation in hepatocytes.

2020

Background: Naturally occurring variation in Membrane-bound O-acyltransferase domain-containing 7 (MBOAT7), encoding for an enzyme involved in phosphatidylinositol acyl-chain remodelling, has been associated with fatty liver and hepatic disorders. Here, we examined the relationship between hepatic Mboat7 down-regulation and fat accumulation. Methods: Hepatic MBOAT7 expression was surveyed in 119 obese individuals and in experimental models. MBOAT7 was acutely silenced by antisense oligonucleotides in C57Bl/6 mice, and by CRISPR/Cas9 in HepG2 hepatocytes. Findings: In obese individuals, hepatic MBOAT7 mRNA decreased from normal liver to steatohepatitis, independently of diabetes, inflammatio…

Research paperTGFβ Transforming Growth Factor BetaIntracellular SpaceCRISPR Clustered Regularly Interspaced Short Palindromic RepeatshHEPS Human HepatocytesMice0302 clinical medicineLPIAT1DAG Diacylglyceroli.p. Intraperitonealmedia_commonFatty AcidsGeneral Medicine3. Good health030220 oncology & carcinogenesisHOMA-IR homeostasis Model Assessment of Insulin ResistanceMPO morpholinolcsh:Medicine (General)medicine.medical_specialtyPE Phosphatidyl-EthanolamineNashGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesTNFα tumor Necrosis Factor AlphaLDL Low Density LipoproteinsHyperinsulinismNAFLDSD Standard Dietmedia_common.cataloged_instanceHumansCPT1 Carnitine Palmitoyltransferase IPhosphatidylinositolGene SilencingEuropean unionVLDL Very Low Density Lipoproteinlcsh:RhHSC Human Hepatic Stellate Cellsmedicine.diseaseLipid MetabolismOA Oleic AcidCI Confidence IntervalMboat7 Membrane bound O-acyltransferase domain containing 7MCD methionine choline deficient diet030104 developmental biologyEndocrinologychemistryCDP Cytidine-DiphosphateFOXO1 Forkhead Box protein O1NAFLD nonalcoholic fatty liver diseaseSteatohepatitisBMI Body Mass IndexCL CardiolipinAcyltransferases0301 basic medicineAlcoholic liver diseaseCXCL10 C-X-C Motif Chemokine 10lcsh:Medicinechemistry.chemical_compoundNon-alcoholic Fatty Liver DiseaseIFG Impaired Fasting GlucoseAPOB Apolipoprotein BNonalcoholic fatty liver diseasePIP Phosphatidyl-Inositol-PhosphateSteatohepatitisqRT-PCR quantitative Real Time Polymerase Chain ReactionMice Knockoutlcsh:R5-920ORO Oil Red O StainingPI PhosphatidylinositolFatty liverTM6SF2 Transmembrane 6 Superfamily Member 2PhospholipidTAG TriglyceridesNASH Nonalcoholic SteatohepatitisLipogenesisLPA Lyso-Phosphatidic AcidPhosphatidylinositolSignal TransductionPS Phosphatidyl-SerinePA Palmitic AcidALD alcoholic liver diseasePC Phosphatidylcholinei.v. IntravenousFATP1 Fatty Acid Transport Protein 1Models BiologicalInternal medicinemedicineAnimalsNonalcoholic fatty liver diseasePPARα Peroxisome Proliferator-Activated Receptor alphaObesityG3P Glyceraldehyde-3-PhosphateSREBP1c Sterol Regulatory Element-Binding Protein 1HDL High Density Lipoproteinsbusiness.industryPI3K Phosphatidylinositol 3 KinaseMembrane ProteinsNHEJ Non-Homologues End JoiningPNPLA3 Patatin-like Phospholipase Domain-containing-3MTTP Microsomal Triglyceride Transfer ProteinLPIAT1 Lysophosphatidylinositol Acyltransferase 1TMC4 Transmembrane Channel-Like 4Disease Models AnimalGene Expression RegulationHepatocytesFOXA2 Forkhead Box A2mTOR mammalian target of RapamycinSteatosisInsulin ResistancebusinessPG Phosphatidyl-GlycerolFABP1 Fatty Acid-Binding Protein 1 FAS Fatty Acid SynthaseT2DM Type 2 Diabetes MellitusEBioMedicine
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La diabetes mellitus y el riesgo cardiovascular. ¿Es necesario el tratamiento integral de la diabetes mellitus tipo 2 y los factores de riesgo cardio…

2010

ResumenLas enfermedades cardiovasculares (CV) son la primera causa de muerte en la población española, tanto en personas diabéticas como no diabéticas. En la diabetes, la enfermedad CV es entre 2 y 4 veces más frecuente, más precoz y más agresiva. Con las medidas actuales se puede llegar a una prevención del ∼50% de la enfermedad CV. Los factores de riesgo CV en la diabetes son la hipertensión arterial (HTA), la dislipemia, el tabaquismo, la obesidad y el sedentarismo. Más del 80% de pacientes con diabetes tipo 2 presenta HTA y dislipemia, y aproximadamente un 15% sigue fumando. Pero menos de un 10% tiene todos estos factores controlados. Aunque la dislipemia es el factor más condicionante …

Riskmedicine.medical_specialtymedicine.medical_treatmentEnfermedad cardiovascularDiseaseType 2 diabetesRiesgoDiabetes tipo 2Internal medicineDiabetes mellitusmedicineCause of deathMedicine(all)business.industryPreventionType 2 diabetesGeneral MedicineAtención primariaCardiovascular diseasePrimary caremedicine.diseaseObesityPrevenciónBlood pressureSmoking cessationbusinessFamily PracticeDyslipidemiaAtención Primaria
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An Update on the Current and Emerging Use of Thiazolidinediones for Type 2 Diabetes.

2022

Guidelines have increasingly stressed the concept that adequate glycemic control is required to prevent or decrease the macro- and microvascular complications of type 2 diabetes mellitus (T2DM). PPAR-gamma agonists (“glitazones”) are no longer prioritized due to their effects on heart failure. However, the association between these drugs and innovative therapies could be a valuable tool to attenuate the risk factors of the metabolic syndrome. Glitazones are used for the treatment of diabetes and associated comorbidities. There is substantial scientific evidence demonstrating the effect of glitazones at a cardiometabolic level, as well as on hematological and neurological pathologies that po…

RosiglitazoneDiabetes Mellitus Type 2PioglitazonePeroxisome Proliferator-Activated ReceptorsHumansHypoglycemic AgentsThiazolidinedionescardiovascular risk metabolic syndrome pioglitazone type 2 diabetes mellitusGeneral MedicineMedicina (Kaunas, Lithuania)
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The effect of physical activity counseling on diabetics' mobility : results of a secondary analysis

2013

The preservation of mobility is essential for maintaining an independent and active lifestyle in old age. However, diabetes greatly increases older people’s susceptibility to mobility loss, leading to an increased risk of dependence. Given the increased prevalence of diabetes with age, and the increasing incidence of the disease worldwide, public health initiatives for preventing mobility loss with age must also prove effective for diabetics. The screening and counseling for physical activity among older people (SCAMOB) study examines the effectiveness of physical activity counseling for the prevention of mobility loss in an older population. Although the cohort as a whole has shown positiv…

SCAMOBolder peopleikääntyminenliikkuvuusdiabetesmobility lossliikuntaneuvontatype 2 diabetesaikuistyypin diabetesikääntyneetfyysinen aktiivisuusphysical activity counseling
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Magnesium homeostasis and aging.

2010

Aging is very often associated with magnesium (Mg) deficit. Total plasma magnesium concentrations are remarkably constant in healthy subjects throughout life, while total body Mg and Mg in the intracellular compartment tend to decrease with age. Dietary Mg deficiencies are common in the elderly population. Other frequent causes of Mg deficits in the elderly include reduced Mg intestinal absorption, reduced Mg bone stores, and excess urinary loss. Secondary Mg deficit in aging may result from different conditions and diseases often observed in the elderly (i.e. insulin resistance and/or type 2 diabetes mellitus) and drugs (i.e. use of hypermagnesuric diuretics). Chronic Mg deficits have been…

SenescenceAdultmedicine.medical_specialtyAgingClinical BiochemistryType 2 diabetesmedicine.disease_causeBiochemistryIntestinal absorptionYoung AdultInsulin resistanceRisk FactorsDiabetes mellitusInternal medicinemedicineHomeostasisHumansMagnesiumMolecular BiologyAgedAged 80 and overInflammationbusiness.industryType 2 Diabetes MellitusMiddle Agedmedicine.diseaseDietOxidative StressEndocrinologySarcopeniabusinessMagnesium DeficiencyOxidative stressDNA DamageMagnesium research
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Endotoxaemia resulting from decreased serotonin tranporter (5-HTT) function: A reciprocal risk factor for depression and insulin resistance?

2015

International audience; Depression and diabetes are serious diseases with an increasing global prevalence. Intriguingly, recent meta-analyses have highlighted an asymmetrical relationship between the two conditions as depressed patients were found to display a higher risk of developing type 2 diabetes than those individuals suffering from diabetes are to become depressed. Based on recent findings, we favor a hypothesis where by decreased peripheral serotonin (5-HT) transporter (5-HTT) function is a reciprocal risk factor for the comorbidity of depression and diabetes, as it can trigger inflammatory pathogenetic mechanisms of both conditions. Higher intestinal levels of 5-HT and 5-HT3 recept…

Serotoninmedicine.medical_specialty5-HTAntidepressantComorbidityType 2 diabetesBehavioral NeuroscienceInsulin resistanceDiabetes mellitusInternal medicineAnimal models of depressionmedicineAnimalsHumansDepression (differential diagnoses)Serotonin Plasma Membrane Transport ProteinsInflammationIntestinal permeabilitybiologybusiness.industryDepressionInsulin resistancemedicine.diseaseAntidepressive AgentsReceptor InsulinEndotoxemia3. Good healthInsulin receptorEndocrinologyDiabetes Mellitus Type 2Immunologybiology.proteinAntidepressant[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Receptors Serotonin 5-HT3businessSignal Transduction
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