Search results for "Type I"

showing 10 items of 966 documents

Mesoglea Extracellular Matrix Reorganization during Regenerative Process in Anemonia viridis (Forskål, 1775)

2021

Given the anatomical simplicity and the extraordinary ability to regenerate missing parts of the body, Cnidaria represent an excellent model for the study of the mechanisms regulating regenerative processes. They possess the mesoglea, an amorphous and practically acellular extracellular matrix (ECM) located between the epidermis and the gastrodermis of the body and tentacles and consists of the same molecules present in the ECM of vertebrates, such as collagen, laminin, fibronectin and proteoglycans. This feature makes cnidarians anthozoans valid models for understanding the ECM role during regenerative processes. Indeed, it is now clear that its role in animal tissues is not just tissue su…

0301 basic medicinecollagenAnemonia viridis Collagen Enzymatic activity Histology Morphology Regeneration Animals Collagen Type I Extracellular Matrix Sea Anemones Regeneration Wound HealingTentacleQH301-705.5enzymatic activityContext (language use)Anemonia viridisMesogleaArticleCollagen Type ICatalysisInorganic ChemistryExtracellular matrixhistology03 medical and health sciences0302 clinical medicinemorphologyAnimalsPhysical and Theoretical ChemistryBiology (General)Molecular BiologyQD1-999SpectroscopyWound HealingbiologyRegeneration (biology)Organic ChemistryGeneral MedicineRegenerative processExtracellular MatrixComputer Science ApplicationsCell biologyFibronectinChemistrySea Anemones030104 developmental biologyregenerationbiology.proteinAnemonia viridis; collagen; enzymatic activity; histology; morphology; regenerationWound healing<i>Anemonia viridis</i>030217 neurology & neurosurgeryInternational Journal of Molecular Sciences
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Intravenous delivery of the toll-like receptor 7 agonist SC1 confers tumor control by inducing a CD8+ T cell response

2019

TLR7 agonists are considered promising drugs for cancer therapy. The currently available compounds are not well tolerated when administered intravenously and therefore are restricted to disease settings amenable for topical application. Here we present the preclinical characterization of SC1, a novel synthetic agonist with exquisite specificity for TLR7. We found that intravenously administered SC1 mediates systemic release of type I interferon, but not of proinflammatory cytokines such as TNFα and IL6, and results in activation of circulating immune cells. Tumors of SC1-treated mice have brisk immune cell infiltrates and are polarized towards a Th1 type signature. Intratumoral CD8(+) T cel…

0301 basic medicinelcsh:Immunologic diseases. Allergycd8+ t cellsImmunologytype i interferonlcsh:RC254-282Proinflammatory cytokinetlr7 ligand03 medical and health sciences0302 clinical medicineImmune systemInterferonmedicineImmunology and AllergyCytotoxic T cellOriginal ResearchToll-like receptorcancer immunotherapybusiness.industryTLR7Acquired immune systemlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens030104 developmental biologyOncology030220 oncology & carcinogenesisCancer researchbusinesslcsh:RC581-607CD8medicine.drugOncoImmunology
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Inactivation of the KSRP gene modifies collagen antibody induced arthritis.

2017

Abstract The KH type splicing regulatory protein (KSRP) is a nucleic acid binding protein, which negatively regulates the stability and/or translatability of many mRNA species encoding immune-relevant proteins. As KSRP is expressed in immune cells including T and B cells, neutrophils, macrophages and dendritic cells, we wanted to analyze its importance for the development of autoimmune diseases. We chose collagen antibody-induced arthritis (CAIA) as an appropriate autoimmune disease mouse model in which neutrophils and macrophages constitute the main effector cell populations. We compared arthritis induction in wild type (WT) and KSRP−/− mice and paws were taken for histological sections an…

0301 basic medicinemedicine.drug_classmedicine.medical_treatmentInflammatory arthritisChemokine CXCL1ImmunologyArthritisAntigens Differentiation MyelomonocyticNitric Oxide Synthase Type IISpleenBiologyMonoclonal antibodyPeripheral blood mononuclear cellAntibodiesFlow cytometry03 medical and health sciencesInterferon-gammaMiceImmune systemAntigens CDmedicineAnimalsAntigens LyCalgranulin ARNA MessengerMolecular BiologyInflammationmedicine.diagnostic_testTumor Necrosis Factor-alphaMacrophagesRNA-Binding Proteinsmedicine.diseaseMolecular biologyArthritis ExperimentalLymphocyte Function-Associated Antigen-1Mice Inbred C57BL030104 developmental biologyCytokinemedicine.anatomical_structureImmunologyTrans-ActivatorsCytokinesCollagenMolecular immunology
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Uncoupling of eNOS in Cardiovascular Disease

2017

Abstract Under physiological conditions, nitric oxide (NO) produced by the endothelial NO synthase (eNOS) represents a key vasoprotective factor. Under conditions of cardiovascular diseases, such as hypertension, diabetes, and atherosclerosis, eNOS may become uncoupled. Uncoupled eNOS generates superoxide at the expense of NO and contributes significantly to endothelial dysfunction and atherogenesis. Major mechanisms of eNOS uncoupling include depletion of tetrahydrobiopterin, an essential cofactor for the eNOS enzyme, and deficiency of l -arginine, the eNOS substrate, and/or eNOS S-glutathionylation. Reversal of eNOS uncoupling may represent a novel therapeutic strategy for the prevention …

0301 basic medicinemedicine.medical_specialtyArgininebiologySuperoxidebusiness.industryNitric Oxide Synthase Type IIITetrahydrobiopterin030204 cardiovascular system & hematologybiology.organism_classificationmedicine.diseaseVasoprotectiveNitric oxide03 medical and health scienceschemistry.chemical_compound030104 developmental biology0302 clinical medicineEndocrinologychemistryEnosInternal medicinemedicineEndothelial dysfunctionbusinessmedicine.drug
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Consensus Statement on the Use of Bone Turnover Markers for Short-Term Monitoring of Osteoporosis Treatment in the Asia-Pacific Region

2019

Osteoporosis is a major health issue. By 2050, a greater than 2-fold increase in patients number with hip fractures will occur in Asia representing 50% of all hip fractures worldwide. For the Asia-Pacific (AP) region, more efforts on controlling osteoporosis and the subsequent fractures are crucial. Bone mineral density (BMD) by dual energy X-ray absorptiometry (DXA) is commonly used to diagnose osteoporosis and monitor osteoporosis treatment. However, the inconvenience, cost, limited availability of DXA and the delay in detection of BMD changes after treatment initiation support an important role for bone turnover markers (BTMs), as short-term tools to monitor therapy. With regards to low …

0301 basic medicinemedicine.medical_specialtyConsensusEndocrinology Diabetes and MetabolismOsteoporosis030209 endocrinology & metabolismBone resorption makerAsia pacific regionCollagen Type IBone remodeling03 medical and health sciences0302 clinical medicineN-terminal telopeptideBone DensityOsteoporosis treatmentHealth caremedicineHumansRadiology Nuclear Medicine and imagingOrthopedics and Sports MedicineIntensive care medicineReimbursementAnabolicsBone mineralddc:616Hip Fracturesbusiness.industryAnti-resorptivesmedicine.diseasePeptide FragmentsBone formation makerOsteoporosisBone Remodeling030101 anatomy & morphologybusinessBiomarkersProcollagen
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Effects of resveratrol on eNOS in the endothelium and the perivascular adipose tissue

2017

Under physiological conditions, nitric oxide (NO) is produced in the vasculature mainly by the endothelial NO synthase (eNOS). Experiments using gene-disrupted mice have demonstrated that eNOS has antihypertensive, antithrombotic, and antiatherosclerotic effects. Recent studies show that eNOS is expressed not only in the endothelium but also in the perivascular adipose tissue (PVAT). Resveratrol prevents eNOS uncoupling and upregulates eNOS expression and activity. These effects of resveratrol are well established for the eNOS enzyme in the endothelium. Interestingly, resveratrol also improves PVAT function. However, a causal role for eNOS in the effects of resveratrol on PVAT function has …

0301 basic medicinemedicine.medical_specialtyEndotheliumAdipose tissue030204 cardiovascular system & hematologyResveratrolGeneral Biochemistry Genetics and Molecular BiologyNitric oxide03 medical and health scienceschemistry.chemical_compound0302 clinical medicineHistory and Philosophy of ScienceEnosInternal medicineAntithromboticmedicinebiologyChemistryGeneral NeuroscienceNitric Oxide Synthase Type IIIbiology.organism_classificationNitric oxide synthase030104 developmental biologyEndocrinologymedicine.anatomical_structurebiology.proteinAnnals of the New York Academy of Sciences
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International working group identifies need for newborn screening for mucopolysaccharidosis type I but states that existing hurdles must be overcome

2018

Abstract Aim Mucopolysaccharidosis type I is a lysosomal storage disorder that can result in significant disease burden, disability and premature death, if left untreated. The aim of this review was to elaborate on the diagnosis of mucopolysaccharidosis type I and the pros and cons of newborn screening. Methods An international working group was established to discuss ways to improve the early diagnosis of mucopolysaccharidosis type I. It consisted of 13 experts in paediatrics, rare diseases and inherited metabolic diseases from Europe and the Middle East. Results It is becoming increasingly clearer that the delay between symptom onset and clinical diagnosis is considerable for mucopolysacc…

0301 basic medicinemedicine.medical_specialtyHaematopoietic stem cell transplantLysosomal storage disorderMucopolysaccharidosis ILysosomal storage disordersReview ArticleDisease03 medical and health sciencesMucopolysaccharidosis type INeonatal Screening0302 clinical medicinemedicineHumansLaronidasePediatrics Perinatology and Child HealthIntensive care medicineReview ArticlesDisease burdenNewborn screeningbusiness.industryMucopolysaccharidosis type IInfant NewbornGeneral MedicineEnzyme replacement therapyInternational working group030104 developmental biologyEnzyme replacement therapyClinical diagnosisPediatrics Perinatology and Child Healthbusiness030217 neurology & neurosurgeryActa Paediatrica
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Genetic inactivation of the sigma-1 chaperone protein results in decreased expression of the R2 subunit of the GABA-B receptor and increased suscepti…

2021

There is a growing body of evidence demonstrating the significant involvement of the sigma-1 chaperone protein in the modulation of seizures. Several sigma-1 receptor (Sig1R) ligands have been demonstrated to regulate the seizure threshold in acute and chronic seizure models. However, the mechanism by which Sig1R modulates the excitatory and inhibitory pathways in the brain has not been elucidated. The aim of this study was to compare the susceptibility to seizures of wild type (WT) and Sig1R knockout (Sig1R&minus;/&minus;) mice in intravenous pentylenetetrazol (PTZ) and (+)-bicuculline (BIC) infusion-induced acute seizure and Sig1R antagonist NE-100-induced seizure models. To determine pos…

0301 basic medicinemedicine.medical_specialtyKnockoutGene ExpressionNitric Oxide Synthase Type IISigma-1 receptorConvulsantsAnisolesSigma-1 receptor Knockout GABA-B receptor Seizures Medial habenula NE-100BicucullineHippocampuslcsh:RC321-571Mice03 medical and health sciences0302 clinical medicineDownregulation and upregulationSeizuresInternal medicineGene expressionmedicineAnimalsReceptors sigmaGABA-B receptorGenetic Predisposition to DiseasePentylenetetrazolReceptorlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMice KnockoutHabenulaSigma-1 receptorPropylaminesSeizure thresholdChemistryMedial habenulaWild typeAntagonistReceptors GABA-A030104 developmental biologyEndocrinologyReceptors GABA-BNeurologyNE-100Pentylenetetrazole030217 neurology & neurosurgerymedicine.drugNeurobiology of Disease
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Participation of Heme Oxygenase-1 in a Model of Acute Inflammation

2003

In this study, the role of heme oxygenase-1 (HO-1) in the inflammatory response elicited by zymosan in the mouse air pouch model has been examined. This response is characterized by a time-dependent increase in HO-1 expression in the leukocytes migrating into the exudates. At 24–48 h maximal HO-1 expression was accompanied by reduced cyclooxygenase-2 (COX-2) and nitric oxide synthase-2 (NOS-2) expression as well as low levels of inflammatory mediators. Hemin administration into the air pouch caused an elevation of HO-1 protein and bilirubin levels induced by zymosan with inhibition of COX-2 expression. In mouse peritoneal macrophages from hemin-injected mice, we also observed an increased …

0301 basic medicinemedicine.medical_specialtyNitric Oxide Synthase Type IIInflammationGeneral Biochemistry Genetics and Molecular BiologyNitric oxideMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicineAnimalsProstaglandin E2HemeInflammationbiologyZymosanZymosanMembrane ProteinsIsoenzymesHeme oxygenaseDisease Models Animal030104 developmental biologyEndocrinologychemistryCyclooxygenase 2Prostaglandin-Endoperoxide Synthases030220 oncology & carcinogenesisHeme Oxygenase (Decyclizing)ImmunologyMacrophages Peritonealbiology.proteinHeminFemaleCyclooxygenaseNitric Oxide Synthasemedicine.symptomHeme Oxygenase-1medicine.drugHeminExperimental Biology and Medicine
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Roles of Vascular Oxidative Stress and Nitric Oxide in the Pathogenesis of Atherosclerosis.

2017

Major reactive oxygen species (ROS)–producing systems in vascular wall include NADPH (reduced form of nicotinamide adenine dinucleotide phosphate) oxidase, xanthine oxidase, the mitochondrial electron transport chain, and uncoupled endothelial nitric oxide (NO) synthase. ROS at moderate concentrations have important signaling roles under physiological conditions. Excessive or sustained ROS production, however, when exceeding the available antioxidant defense systems, leads to oxidative stress. Animal studies have provided compelling evidence demonstrating the roles of vascular oxidative stress and NO in atherosclerosis. All established cardiovascular risk factors such as hypercholesterolemi…

0301 basic medicinemedicine.medical_specialtyNitric Oxide Synthase Type IIIPhysiologyOxidative phosphorylationBiologymedicine.disease_causeNitric OxideNitric oxide03 medical and health scienceschemistry.chemical_compoundSpecies SpecificityInternal medicinemedicineAnimalsHumansXanthine oxidasechemistry.chemical_classificationReactive oxygen speciesNitric Oxide Synthase Type IIIAtherosclerosisEndothelial stem cellOxidative Stress030104 developmental biologyEndocrinologychemistryEndothelium VascularCardiology and Cardiovascular MedicineReactive Oxygen SpeciesOxidative stressNicotinamide adenine dinucleotide phosphateCirculation research
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