Search results for "Unity"

showing 10 items of 3852 documents

Stimulation of synovial fluid mononuclear cells with the human 65-kD heat shock protein or with live enterobacteria leads to preferential expansion o…

1992

SUMMARY T lymphocyte responses to heterologous or self 65-kD heat shock protein (hsp) have been implicated in the pathogenesis of various forms of arthritis. To delineate the relationship of 65-kD hsp to different synovial fluid (SF) T cell subsets, we stimulated synovial fluid (SFMC) and peripheral blood mononuclear cells (PBMC) from patients with different inflammatory rheumatic diseases and from healthy controls with human or mycobacterial 65-kD hsp, tetanus toxoid (TT), heat-killed or live Yersinia enterocotitica. Phenotyping of the resulting T cell lines revealed an increase of up to 97% TCR-γδ+ lymphocytes in the 65-kD hsp-stimulatcd SF-derived lines. This expansion of TCR-γδ+ cells w…

Cellular immunityT cellReceptors Antigen T-Cell alpha-betaT-LymphocytesImmunologyBiologyYersiniaLymphocyte ActivationPeripheral blood mononuclear cellMonocytesCell LineAntigenEnterobacteriaceaeHeat shock proteinSynovial FluidmedicineImmunology and AllergySynovial fluidHumansHeat-Shock ProteinsReceptors Antigen T-Cell gamma-deltaT lymphocytebiology.organism_classificationClone Cellsmedicine.anatomical_structurePhenotypeImmunologyResearch Article
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The Human T Cell Response to Mitogenic Microbial Exotoxins

1991

Nearly every infectious pathogen has to cope with the host’s adaptive immune response. Common evasion mechanisms in this complex interaction are antigenic variations, the escape to immunologically privileged sites, or the use of immunosuppressive mechanisms. Many bacteria and other microorganisms elaborate soluble factors or toxins that act suppressively on cells of the immune system, such as pore-forming molecules or proteins that interfere with the function of G proteins. Gram-positive cocci and a mycoplasma have developed an extremely potent mechanism of T cell stimulation by closely mimicking recognition of specific antigen. From the functional similarity to antigen recognition and the …

Cellular immunityT cellT lymphocyteBiologymedicine.disease_causeAcquired immune systemMicrobiologyImmune systemmedicine.anatomical_structureAntigenImmunologymedicineSuperantigenExotoxin
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Research in practice: Different dendritic cell types in skin with various functions - important implications for intradermal vaccines

2011

Summary It was long believed that epidermal Langerhans cells (LC) are responsible for the initiation of cellular immunity. Only recently it has been shown that in skin alone 5 different subtypes of dendritic cells (DC) can be identified. Among these, LC, but also two Langerin-expressing dermal DC populations and two more Langerin-negative DC subtypes exist. Novel findings in the model disease leishmaniasis, as well as evidence from research in contact hypersensitivity, have revealed that activation of LC in skin leads to induction of regulatory, immunosuppressive T cells, whereas the other skin DC subtypes stimulate effector T cells. Thus, when producing vaccines designed for intradermal us…

Cellular immunityintegumentary systembusiness.industryEffectorIntradermal useContact hypersensitivityLeishmaniasisDermatologyDendritic cellmedicine.diseaseModel diseaseImmunologymedicinebusinessJDDG: Journal der Deutschen Dermatologischen Gesellschaft
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Cellular immunity to the Her-2/neu protooncogene

2002

Her-2/neu (HER-2) is a 185-kDa receptor-like glycoprotein that is overexpressed by a variety of tumors such as breast, ovarian, gastric, and colorectal carcinomas. Overexpression of this oncogene is directly associated with malignant transformation of epithelial cells. The frequency of HER-2 overexpression varies among the different types of cancers, but universally represents a marker of poor prognosis. The critical role of HER-2 in epithelial oncogenesis as well as its selective overexpression on malignant tissues makes it an ideal target for immunotherapy. Antibodies and T cells reactive to HER-2 are known to naturally occur in patients with HER-2 positive tumors, confirming the immunoge…

Cellular immunitymedicine.drug_classbusiness.industryT cellmedicine.medical_treatmentImmunotherapyMonoclonal antibodymedicine.anatomical_structureImmune systemAntigenTrastuzumabImmunologyMonoclonalmedicinebusinessmedicine.drug
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Remission of experimental autoimmune hepatitis is associated with antigen-specific and non-specific immunosuppression.

1993

SUMMARY Experimental autoimmune hepatitis (EAH) is an animal model for autoimmune hepatitis. The disease is T cell-mediated and runs a subacute course, with maximal disease activity around week four after disease induction and a slow ensuing recovery. The aim of the present study was to investigate the immunoregulatory mechanisms that may account for recovery in EAH. It was found that T cell reactivity to liver antigens preceded histological disease, but at the peak of disease activity this T cell response was already suppressed. Active and antigen-specific suppression could be demonstrated, as irradiated splenocytes from animals at the beginning of recovery from EAH were able to suppress i…

Cellular immunitymedicine.medical_treatmentT-LymphocytesImmunologyAutoimmune hepatitisBiologyHepatitis AnimalAutoimmune DiseasesMiceImmune systemAntigenmedicineImmune ToleranceTetanus ToxoidImmunology and AllergyAnimalsAntigensAutoimmune diseaseHepatitisMice Inbred BALB CImmunosuppressionT lymphocytemedicine.diseaseMice Inbred C57BLImmunologyFemaleResearch Article
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Recombinant virus-like particles of a norovirus (genogroup II strain) administered intranasally and orally with mucosal adjuvants LT and LT(R192G) in…

2003

We investigated the immune response induced by mucosal immunization of BALB/c mice with virus-like particles (VLPs) of a genogroup II norovirus, Dijon171/96 virus, produced in the baculovirus system. VLPs administered alone by the intranasal route induced a high serum antibody response as well as fecal IgA, which were enhanced when the heat-labile Escherichia coli toxin or its non toxic mutant LT(R192G) was coadministered. In these conditions, the oral route was also efficient. Cytokine production by cells from different lymphoid tissues was then assessed after in vitro restimulation. A Th1/Th2-like response was observed in cervical lymph node and Peyer's patch (PP) cell cultures from mice …

Cellular immunityvirusesmedicine.medical_treatmentAdministration OralEnzyme-Linked Immunosorbent AssayAntibodies ViralBALB/cMicrobiologyFecesMiceTh2 CellsImmune systemAdjuvants ImmunologicVirus-like particlemedicineAnimalsAdministration IntranasalCells CulturedImmunity CellularMice Inbred BALB CVaccines SyntheticGeneral VeterinaryGeneral Immunology and MicrobiologybiologyNorovirusPublic Health Environmental and Occupational HealthViral VaccinesTh1 Cellsbiology.organism_classificationVirologyInfectious DiseasesCytokineAntibody FormationHumoral immunitybiology.proteinMolecular MedicineFemaleLymph NodesAntibodyAdjuvantSpleenVaccine
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Intrinsic TNFR2 signaling in T regulatory cells provides protection in CNS autoimmunity

2018

Significance In spite of TNF involvement in the pathogenesis of multiple sclerosis (MS), systemic TNF neutralization in MS patients was not successful. One of the possible reasons is that TNF possesses both pathogenic and protective features that may be related to TNFR1 versus TNFR2 receptor engagement. This study uncovers one of such protective functions of TNF mediated by intrinsic TNFR2 signaling in Treg cells. In mice bearing humanized TNF and TNFR2 genetic loci, TNFR2 ablation restricted to Treg cells led to reduced capacity to control Th17 cell responses, exacerbated experimental autoimmune encephalomyelitis (EAE) development, and affected the maintenance of Treg cells. These findings…

Central Nervous System0301 basic medicineEncephalomyelitis Autoimmune ExperimentalT regulatory cellsmedicine.medical_treatmentAutoimmunitychemical and pharmacologic phenomenaBiologymedicine.disease_causeT-Lymphocytes RegulatoryneuroinflammationAutoimmunityMice03 medical and health sciencesImmunology and Inflammation0302 clinical medicineImmune systemmedicineAnimalsHumansReceptors Tumor Necrosis Factor Type IIIL-2 receptorCells CulturedNeuroinflammationMice KnockoutAutoimmune diseaseMultidisciplinaryEAETumor Necrosis Factor-alphaExperimental autoimmune encephalomyelitisFOXP3hemic and immune systemsBiological Sciencesmedicine.diseaseTNF/TNFR2Mice Inbred C57BLDisease Models Animalhumanized mice030104 developmental biologyCytokineGene Expression RegulationImmunology030215 immunologyProceedings of the National Academy of Sciences
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Myeloid Cells in the Central Nervous System

2017

The central nervous system (CNS) and its meningeal coverings accommodate a diverse myeloid compartment that includes parenchymal microglia and perivascular macrophages, as well as choroid plexus and meningeal macrophages, dendritic cells, and granulocytes. These myeloid populations enjoy an intimate relationship with the CNS, where they play an essential role in both health and disease. Although the importance of these cells is clearly recognized, their exact function in the CNS continues to be explored. Here, we review the subsets of myeloid cells that inhabit the parenchyma, meninges, and choroid plexus and discuss their roles in CNS homeostasis. We also discuss the role of these cells in…

Central Nervous System0301 basic medicinePathologymedicine.medical_specialtyMyeloidNeuroimmunomodulationImmunologyCentral nervous systemBiologyInfectionsmedicine.disease_causeNeuroprotectionArticleAutoimmune DiseasesAutoimmunity03 medical and health sciencesMeningesmedicineAnimalsHumansImmunology and AllergyMyeloid CellsMicrogliaNeurodegenerationMeningesNeurodegenerative Diseasesmedicine.diseaseNeuroprotection030104 developmental biologyInfectious Diseasesmedicine.anatomical_structureChoroid PlexusImmunologyWounds and InjuriesChoroid plexusImmunity
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Role of Sortilin in Models of Autoimmune Neuroinflammation

2015

Abstract The proneurotrophin receptor sortilin is a protein with dual functions, being involved in intracellular protein transport, as well as cellular signal transduction. The relevance of the receptor for various neuronal disorders, such as dementia, seizures, and brain injury, is well established. In contrast, little is known about the role of sortilin in immune cells and inflammatory diseases. The aim of our study was to elucidate the distribution of sortilin in different immune cell types in mice and humans and to analyze its function in autoimmune CNS inflammation. Sortilin was expressed most profoundly in murine and human macrophages and dendritic cells and to a much lesser extent in…

Central Nervous SystemCell typeEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisT-LymphocytesEncephalomyelitisImmunologyAutoimmunityBiologyMiceImmune systemmedicineAnimalsHumansImmunology and AllergyReceptorNeuroinflammationMice KnockoutAutoimmune diseaseAntigen PresentationMacrophagesExperimental autoimmune encephalomyelitisDendritic Cellsmedicine.diseaseImmunity InnateMice Inbred C57BLAdaptor Proteins Vesicular TransportBrain InjuriesImmunologyNeurogenic InflammationSignal transductionSignal Transduction
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Cytosolic RIG-I–like helicases act as negative regulators of sterile inflammation in the CNS

2011

The action of cytosolic RIG-I-like helicases (RLHs) in the CNS during autoimmunity is largely unknown. Using a mouse model of multiple sclerosis, we found that mice lacking the RLH adaptor IPS-1 developed exacerbated disease that was accompanied by markedly higher inflammation, increased axonal damage and elevated demyelination with increased encephalitogenic immune responses. Furthermore, activation of RLH ligands such as 5'-triphosphate RNA oligonucleotides decreased CNS inflammation and improved clinical signs of disease. RLH stimulation repressed the maintenance and expansion of committed T(H)1 and T(H)17 cells, whereas T-cell differentiation was not altered. Notably, T(H)1 and T(H)17 s…

Central Nervous SystemEncephalomyelitis Autoimmune ExperimentalCell SurvivalT-LymphocytesAutoimmunityInflammationStimulationReceptor Interferon alpha-betamedicine.disease_causeAutoimmunityMiceCytosolImmune systemmedicineAnimalsbiologyMicrogliaRIG-IGeneral NeuroscienceMultiple sclerosisHelicaseCell DifferentiationDendritic Cellsmedicine.diseasemedicine.anatomical_structurebiology.proteinmedicine.symptomNeuroscienceRNA HelicasesNature Neuroscience
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