Search results for "VASODILATION"

showing 10 items of 228 documents

Hypertension and Impotence

1991

In a group of 472 impotent patients who were evaluated with pharmacologic duplex sonography, 117 patient (24.8%) had a history of hypertension, 26 of them (22.2%) for more than 10 years. Objective data about the changes in pulsation, diameter and blood flow velocity of the penile arteries after papaverine injection and the resulting erectile response allowed indirect assessment of the penile venous function. Varying degrees of impaired arterial function were diagnosed in 85% of the patients. The duration of hypertension had less deteriorating effects on the penile arterial system than second risk factors such as diabetes mellitus (n = 31) or smoking (n = 26). Patients on antihypertensive me…

MaleVenous leakmedicine.medical_specialtyUrologyErectile tissueVasodilationDiabetes ComplicationsErectile DysfunctionRisk FactorsPapaverineInternal medicineDiabetes mellitusmedicineHumansAntihypertensive AgentsPapaverinebusiness.industryPenile ErectionSmokingBlood flowMiddle Agedmedicine.diseaseSurgeryCompliance (physiology)medicine.anatomical_structureBlood pressureRegional Blood FlowHypertensionCardiologybusinessPenismedicine.drugEuropean Urology
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The Janus face of chlorogenic acid on vascular reactivity: A study on rat isolated vessels

2016

Abstract Background Chlorogenic acid (CGA), the main polyphenol contained in coffee, is a major contributor to dietary polyphenol intake. Few studies reported its anti-hypertensive properties but the mechanisms are still indefinite. Purpose The present study assessed the direct effect of CGA in endothelium denuded or intact aortic rings from male Wistar rats and the mechanisms involved. Methods/Results CGA induced a direct endothelium-dependent relaxation that was significantly reduced by L-NAME (10 −4  M), indomethacin (10 −5  M) and combination of apamin (10 −7  M) and charybdotoxin (10 −7  M). Incubation of rings with CGA induced a dual effect on agonist-induced vasorelaxation. At 10 −6 …

Maleendocrine systemCharybdotoxinEndotheliumPharmaceutical ScienceVasodilation030204 cardiovascular system & hematologyPharmacologyApaminMuscle Smooth Vascular03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDrug DiscoverymedicineAnimals[CHIM]Chemical SciencesRats WistarPhenylephrineAntihypertensive AgentsComputingMilieux_MISCELLANEOUSPharmacology[SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciencesRatsVasodilationmedicine.anatomical_structureComplementary and alternative medicineBiochemistrychemistry030220 oncology & carcinogenesisHypertensionMolecular MedicineSodium nitroprussideEndothelium Vascularmedicine.symptomChlorogenic AcidVasoconstrictionAcetylcholinemedicine.drug
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Plasma concentrations of nitric oxide and asymmetric dimethylarginine in human alcoholic cirrhosis.

2004

The liver plays a prominent role in the metabolism of asymmetric dimethyl-l-arginine (ADMA), an endogenous inhibitor of nitric oxide (NO) synthase. This study was designed to determine whether plasma levels of ADMA and NO production are altered in patients with compensated and decompensated alcoholic cirrhosis.Plasma levels of l-arginine, ADMA, symmetric dimethylarginine (SDMA) and NO (nitrite plus nitrate, NOx) were measured in nine patients with compensated alcoholic cirrhosis (Child-Pugh A) and 11 patients with advanced cirrhosis (Child-Pugh B-C). Seven healthy volunteers served as controls.ADMA and NOx concentrations in decompensated cirrhosis were higher than in the compensated group a…

Malemedicine.medical_specialtyAlcoholic liver diseaseCirrhosisVasodilationArginineNitric OxideNitric oxidechemistry.chemical_compoundLiver Cirrhosis AlcoholicInternal medicineBlood plasmamedicineHumansNitriteNitritesNitratesHepatologybusiness.industryMetabolismMiddle Agedmedicine.diseaseVasodilationEndocrinologychemistryFemaleEndothelium VascularbusinessAsymmetric dimethylarginineJournal of hepatology
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Aerobic training and angiogenesis activation in patients with stable chronic heart failure: a preliminary report.

2013

The pathophysiology of chronic heart failure (CHF) involves multiple hystologic and molecular alterations. To determine the effects of physical training on circulating endothelial progenitor cells (EPCs), angiogenesis (angiogenin, angiopoietin-1 and -2, VEGF, Tie-2, SDF-1α) and inflammation (IL-6, CRP), we compared data obtained from 11 CHF pts before and after 3 months aerobic exercise training, to those from 10 non trained CHF pts (CHF-C group, age 64 + 2 years, NYHA 2). At the end of the study, EPCs count and AP-2 serum levels significantly increased in the CHF-TR group. These preliminary data suggest a significant effect of even a short program of physical training on angiogenic activat…

Malemedicine.medical_specialtyAngiogeninBrachial ArteryAngiogenesisHealth Toxicology and MutagenesisClinical BiochemistryNeovascularization PhysiologicCardiovascular disease growth factors/cytokines/inflammatory mediators sports science/exerciseInflammationPilot ProjectsBiochemistryNeovascularizationInternal medicinemedicineAerobic exerciseHumanscardiovascular diseasesProspective StudiesEndothelial dysfunctionAngiogenic ProteinsExerciseAgedHeart Failurebusiness.industryStem CellsEndothelial CellsStroke VolumeStroke volumeMiddle Agedmedicine.diseaseSettore MED/11 - Malattie Dell'Apparato CardiovascolareExercise TherapyVasodilationTreatment OutcomeHeart failureImmunologyChronic Diseasecardiovascular systemCardiologymedicine.symptombusinessBiomarkerscirculatory and respiratory physiologyBiomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals
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AT1-receptor blockade with irbesartan improves peripheral but not coronary endothelial dysfunction in patients with stable coronary artery disease

2007

Activation of the renin-angiotensin-aldosterone system plays an important role in the pathogenesis of endothelial dysfunction and atherosclerosis. Studies evaluating the effect of AT1-receptor blockers on endothelial dysfunction in patients with coronary artery disease (CAD) revealed mixed results. Studies addressing the effects of AT1-receptor blockers on the coronary and peripheral function in the same study population, are still lacking. We therefore aimed to test the effects of long-term therapy with the AT1-receptor blocker irbesartan (IRB) on both, the coronary and peripheral endothelial function in patients with CAD. Seventy-two patients with CAD were randomly assigned to double-blin…

Malemedicine.medical_specialtyBrachial ArteryEndotheliumTetrazolesCoronary Artery DiseaseCoronary artery diseaseIrbesartanDouble-Blind MethodInternal medicinemedicine.arterymedicineHumanscardiovascular diseasesEndothelial dysfunctionBrachial arteryUltrasonographybusiness.industryVascular diseaseBiphenyl CompoundsIrbesartanMiddle Agedmedicine.diseaseCoronary VesselsAngiotensin IIPeripheralVasodilationmedicine.anatomical_structureEndocrinologyRegional Blood FlowCardiologyFemaleEndothelium VascularCardiology and Cardiovascular MedicinebusinessAngiotensin II Type 1 Receptor Blockersmedicine.drugAtherosclerosis
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Common Genetic Variation in Relation to Brachial Vascular Dimensions and Flow-Mediated Vasodilation

2019

Common Genetic Variation in Relation to Brachial Vascular Dimensions and Flow-Mediated Vasodilation

Malemedicine.medical_specialtyBrachial ArteryVasodilationGenome-wide association studyPolymorphism Single NucleotideArticleInternal medicinemedicine.arteryGenetic variationmedicineHumanscardiovascular diseasesBrachial arteryCation Transport ProteinsUltrasonographybusiness.industryGenetic VariationGeneral MedicineMethyltransferasesta3121Middle Agedbody regionsVasodilationInsulin-Like Growth Factor Binding Protein 3PhenotypeCardiovascular DiseasesGenetic Locicardiovascular systemCardiologyMedical geneticsDilation (morphology)Femalebusinesscirculatory and respiratory physiologyFlow-Mediated Vasodilation
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Role of K+ and Ca2+ fluxes in the cerebroarterial vasoactive effects of sildenafil

2007

The aim of this study was to assess the role of K(+) and Ca(2+) fluxes in the cerebroarterial vasoactive effects of the phosphodiesterase-5 inhibitor sildenafil. We used isolated rabbit basilar arteries to assess the effects of extracellular K(+) raising on sildenafil-induced vasodilatation, and studied the pharmacological interaction of sildenafil with selective modulators of membrane K(+) and Ca(2+) channels. Expression of Kv1 subunits of K(+) channels was assessed at messenger and protein levels. Parallel experiments were carried out with zaprinast for comparison. Sildenafil (10 nM-0.1 mM) induced concentration-dependent relaxation of endothelin-1 (10 nM)-precontracted arteries, which wa…

Malemedicine.medical_specialtyCalcium Channels L-Typemedicine.drug_mechanism_of_actionPhosphodiesterase InhibitorsVasodilationIn Vitro TechniquesPharmacologyPiperazinesSildenafil Citratechemistry.chemical_compoundInternal medicinemedicineAnimalsChannel blockerRNA MessengerSulfonesPharmacologyTetraethylammoniumDose-Response Relationship DrugChemistryDepolarization3-Pyridinecarboxylic acid 14-dihydro-26-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)- Methyl esterIberiotoxinEndocrinologyPurinesBasilar ArterycGMP-specific phosphodiesterase type 5PotassiumShaker Superfamily of Potassium ChannelsCalciumRabbitsZaprinastPhosphodiesterase 5 inhibitorEuropean Journal of Pharmacology
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Hepatopulmonary Syndrome in a Patient With Adenocarcinoma of the Colon Metastatic to the Liver and No Apparent Chronic Liver Disease

2006

Hepatopulmonary syndrome consists of a clinical triad: arterial blood deoxygenation, intrapulmonary vasodilation, and liver disease. Both acute and chronic cases of this syndrome have been reported, and the most common cause is cirrhosis. The principle disease mechanism is dilation of the pulmonary blood vessels causing alterations in gas exchange. Increased pulmonary production of nitric acid has been implicated as the primary pathogenic mechanism of vasodilation although it has also been associated with imbalance between vasodilators and vasoconstrictors. We describe the case of a patient with hepatopulmonary syndrome and adenocarcinoma of the colon with metastases to a previously healthy…

Malemedicine.medical_specialtyCirrhosisColonVasodilationDiseaseAdenocarcinomaChronic liver diseaseGastroenterologyLiver diseaseLiver Function TestsInternal medicineHumansMedicineHepatopulmonary syndromebusiness.industryLiver NeoplasmsColonoscopyGeneral MedicineMiddle Agedmedicine.diseaseLiverColonic NeoplasmsArterial bloodAdenocarcinomaTomography X-Ray ComputedbusinessHepatopulmonary SyndromeArchivos de Bronconeumología ((English Edition))
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Influence of nitric oxide on neurogenic contraction and relaxation of the human gastroepiploic artery.

2003

Abstract Background The objective of this study was to characterize the neurogenic contraction and relaxation of the human gastroepiploic artery and to determine whether the responses are mediated by nitric oxide (NO) from neural or endothelial origin. Methods Rings of human gastroepiploic artery were obtained from 18 patients (12 men, 6 women) undergoing gastrectomy. The rings were suspended in organ baths for isometric recording of tension. We studied the contractile and relaxant responses to electrical field stimulation. Results In arteries under resting conditions, electrical field stimulation (2 to 8 Hz) caused frequency-dependent contractions that were of greater magnitude in arteries…

Malemedicine.medical_specialtyContraction (grammar)EndotheliumGastroepiploic ArteryTetrodotoxinIn Vitro TechniquesArginineAutonomic Nervous SystemNitric OxideNitric oxidechemistry.chemical_compoundInternal medicineInternal MedicinemedicineHumansAnesthetics LocalEnzyme InhibitorsGuanethidineAgedAged 80 and overomega-N-Methylargininebusiness.industryMiddle AgedVasodilationmedicine.anatomical_structureEndocrinologychemistryVasoconstrictionAnesthesiaCirculatory systemTetrodotoxinFemaleEndothelium Vascularmedicine.symptomNitric Oxide SynthasebusinessGastroepiploic ArteryMuscle contractionmedicine.drugAmerican journal of hypertension
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Contribution of endothelin receptors and cyclooxygenase-derivatives to the altered response of the rabbit renal artery to endothelin-1 in diabetes

2006

Abstract The influence of diabetes on regulatory mechanisms and specific receptors implicated in the response of isolated rabbit renal artery to endothelin-1 was examined. Endothelin-1 induced a concentration-dependent contraction that was less potent in arteries from diabetic rabbits than in arteries from control rabbits. Endothelium removal or NG-nitro- l -arginine (L-NOARG) enhanced contractions to endothelin-1 either in control and diabetic arteries. Indomethacin inhibited endothelin-1-induced response in control arteries, but enhanced it in diabetic arteries. In contrast to that observed in rubbed and in L-NOARG treated arteries, in the presence of indomethacin the contractile action o…

Malemedicine.medical_specialtyEndotheliumIndomethacinVasodilationNitroargininePeptides CyclicDiabetes Mellitus ExperimentalRenal ArteryInternal medicinemedicine.arteryAlloxanmedicineAnimalsCyclooxygenase InhibitorsRenal arteryReceptorAntihypertensive AgentsPharmacologyDose-Response Relationship DrugEndothelin-1biologyChemistryReceptor Endothelin AEndothelin 1Endocrinologymedicine.anatomical_structureProstaglandin-Endoperoxide SynthasesVasoconstrictioncardiovascular systembiology.proteinEndothelium VascularRabbitsCyclooxygenaseNitric Oxide Synthasemedicine.symptomEndothelin receptorVasoconstrictionEuropean Journal of Pharmacology
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