Search results for "Viral replication"

showing 10 items of 157 documents

Identification and characterization of amphiphysin II as a novel cellular interaction partner of the hepatitis C virus NS5A protein.

2003

The hepatitis C virus (HCV) NS5A protein is highly phosphorylated by cellular protein kinases. To study how NS5A might be integrated in cellular kinase signalling, we isolated phosphoproteins from HuH-7 hepatoma cells that specifically interacted with recombinant NS5A protein. Subsequent mass spectrometry identified the adaptor protein amphiphysin II as a novel interaction partner of NS5A. Mutational analysis revealed that complex formation is primarily mediated by a proline-rich region in the C-terminal part of NS5A, which interacts with the amphiphysin II Src homology 3 domain. Importantly, we could further demonstrate specific co-precipitation and cellular co-localization of endogenous a…

CytoplasmProlinevirusesImmunoblottingNerve Tissue ProteinsHepacivirusBiologyProtein Serine-Threonine KinasesViral Nonstructural ProteinsVirus ReplicationSH3 domainVirologyTumor Cells CulturedHumansRepliconNS5AFluorescent Antibody Technique IndirectSubgenomic mRNALeucine ZippersKinasevirus diseasesSignal transducing adaptor proteinbiochemical phenomena metabolism and nutritionMAP Kinase Kinase KinasesRNA-Dependent RNA PolymeraseVirologyMolecular biologydigestive system diseasesRecombinant ProteinsViral replicationMutationPhosphorylationRepliconProtein BindingThe Journal of general virology
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Type I Interferon Protects Antiviral CD8+ T Cells from NK Cell Cytotoxicity

2014

Summary Despite development of new antiviral drugs, viral infections are still a major health problem. The most potent antiviral defense mechanism is the innate production of type I interferon (IFN-I), which not only limits virus replication but also promotes antiviral T cell immunity through mechanisms, which remain insufficiently studied. Using the murine lymphocytic choriomeningitis virus model system, we show here that IFN-I signaling on T cells prevented their rapid elimination in vivo. Microarray analyses uncovered that IFN-I triggered the expression of selected inhibitory NK-cell-receptor ligands. Consequently, T cell immunity of IFN-I receptor (IFNAR)-deficient T cells could be rest…

Cytotoxicity ImmunologicImmunologyMedizinReceptor Interferon alpha-betaCD8-Positive T-LymphocytesLymphocytic ChoriomeningitisVirus ReplicationLymphocytic choriomeningitisVirusMiceImmunityInterferonmedicineAnimalsLymphocytic choriomeningitis virusImmunology and AllergyCytotoxic T cellCells CulturedMice KnockoutbiologyPerforinNFIL3medicine.diseaseVirologyImmunity InnateKiller Cells NaturalMice Inbred C57BLBasic-Leucine Zipper Transcription FactorsInfectious DiseasesViral replicationPerforinInterferon Type IImmunologybiology.proteinSignal Transductionmedicine.drugImmunity
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Synergism between the components of the bipartite major immediate-early transcriptional enhancer of murine cytomegalovirus does not accelerate virus …

2009

Major immediate-early (MIE) transcriptional enhancers of cytomegaloviruses are key regulators that are regarded as determinants of virus replicative fitness and pathogenicity. The MIE locus of murine cytomegalovirus (mCMV) shows bidirectional gene-pair architecture, with a bipartite enhancer flanked by divergent core promoters. Here, we have constructed recombinant viruses mCMV-ΔEnh1 and mCMV-ΔEnh2 to study the impact of either enhancer component on bidirectional MIE gene transcription and on virus replication in cell culture and various host tissues that are relevant to CMV disease. The data revealed that the two unipartite enhancers can operate independently, but synergize in enhancing MI…

DNA ReplicationGene Expression Regulation ViralTranscription GeneticvirusesEnhancer RNAsBiologyVirus ReplicationVirusImmediate-Early ProteinsImmunocompromised HostMiceTranscription (biology)VirologyGene expressionAnimalsEnhancerAntigens ViralCells CulturedGeneticsPromoterFibroblastsVirologyEnhancer Elements GeneticViral replicationCell cultureDNA ViralJournal of General Virology
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Inhibition of human T-cell leukemia virus type I replication in primary human T cells that express antisense RNA

1989

The human T-cell leukemia virus type I is associated with adult T-cell leukemia-lymphoma in humans, a disease which is induced by a malignant transformation of T lymphocytes. Retrovirus vectors carrying human T-cell leukemia virus type I-derived sequences in reversed transcriptional orientation were used to express antisense RNA transcripts in primary human leukocytes. Human T-cell leukemia virus type I replication and virus-mediated immortalization were inhibited in cells harboring antisense constructs. This study suggests that retrovirus-mediated antisense RNA inhibition can be used to protect primary human T-lymphocytes from human T-cell leukemia virus type I-mediated cell transformation.

DNA ReplicationT-LymphocytesvirusesGenetic VectorsImmunologyViral transformationVirus ReplicationMicrobiologyVirusCell LineRetrovirushemic and lymphatic diseasesVirologymedicineHumansRNA AntisenseHuman T-lymphotropic virus 1biologyRNAbiology.organism_classificationmedicine.diseaseVirologyMolecular biologyAntisense RNALeukemiaGene Expression RegulationViral replicationInsect ScienceHuman T-lymphotropic virus 1RNACell DivisionResearch ArticleJournal of Virology
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Replication of the hepatitis C virus

2000

Infection with the hepatitis C virus (HCV) is a major cause of chronic liver disease. HCV is an enveloped plus-strand RNA virus closely related to flavi- and pestiviruses. The first cloning of the HCV genome, about 10 years ago, initiated research efforts leading to the elucidation of the genomic organization and the definition of the functions of most viral proteins. Despite this progress the lack of convenient animal models and appropriate in vitro propagation systems have hampered a full understanding of the way the virus multiplies. This review summarizes our current knowledge about HCV replication and describes attempts pursued in the last few years to establish efficient and reliable …

DNA ReplicationbiologyHepatitis C virusGastroenterologyRNA virusGenome ViralHepacivirusHepatitis CVirus Replicationbiology.organism_classificationmedicine.diseasemedicine.disease_causeHepatitis CVirologyVirusFlaviviridaeViral replicationHepatocytesmedicineAnimalsHumansRNA ViralViral diseaseOncovirusBest Practice & Research Clinical Gastroenterology
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The biological activity of bacteriophage DNA, prepared by the cationic detergent dilution technique

1975

Abstract The preparation of phage lambda DNA infecting E. coli K 12 with cationic detergent is described. This DNA infects E. coli spheroblasts with the same efficiency as DNA prepared by phenol methods.

DetergentsViral Plaque AssayBiologyVirus Replicationmedicine.disease_causeColiphagesBacteriophagechemistry.chemical_compoundEscherichia coliGeneticsmedicinePhenolEscherichia coliVirus quantificationDNA VirusesBiological activityLambda phageChromatography Ion Exchangebiology.organism_classificationMolecular biologyQuaternary Ammonium CompoundschemistryBiochemistryViral replicationDNA ViralDNANucleic Acids Research
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Intracerebral Borna Disease Virus Infection of Bank Voles Leading to Peripheral Spread and Reverse Transcription of Viral RNA

2011

Bornaviruses, which chronically infect many species, can cause severe neurological diseases in some animal species; their association with human neuropsychiatric disorders is, however, debatable. The epidemiology of Borna disease virus (BDV), as for other members of the family Bornaviridae, is largely unknown, although evidence exists for a reservoir in small mammals, for example bank voles (Myodes glareolus). In addition to the current exogenous infections and despite the fact that bornaviruses have an RNA genome, bornavirus sequences integrated into the genomes of several vertebrates millions of years ago. Our hypothesis is that the bank vole, a common wild rodent species in traditional B…

Disease reservoirviruksetEpidemiologyanimal diseasesvirusesVeterinary MicrobiologyUrineVirus ReplicationMOUSE413 Veterinary sciencePolymerase Chain ReactionFecesInfectious Diseases of the Nervous SystemZoonosesBRAINBorna disease virusAntigens Viralbornavirus0303 health sciencesBorna diseaseMultidisciplinarybiologyArvicolinaeZoonotic DiseasesQR3. Good healthBank voleInfectious DiseasesBorna Virus InfectionVeterinary DiseasesArvicolinaeMedical MicrobiologyWILD RODENTSRNA ViralMedicineViral VectorsVeterinary PathologyResearch ArticleEXPRESSIONNeurovirulenceScienceUrinary BladdereducationANTIGENMicrobiologyVector BiologyInfectious Disease EpidemiologyVirusRATSPERSISTENT03 medical and health sciencesVirologyPeripheral Nervous SystemAnimalsHumansViral Nucleic AcidViral sheddingBiologyDisease Reservoirs030304 developmental biology030306 microbiologySTRAINSCENTRAL-NERVOUS-SYSTEMReproducibility of ResultsReverse TranscriptionVeterinary Virologybiology.organism_classificationVirologyViral ReplicationReverse transcriptaseMODELAnimals NewbornViral replicationBorna DiseaseAntibody FormationDNA ViralVeterinary ScienceViral Transmission and InfectionPLoS ONE
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Heterogeneity in the Response of Different Subtypes of Drosophila melanogaster Midgut Cells to Viral Infections

2021

This article belongs to the Section Viral Immunology, Vaccines, and Antivirals.

Drosophila virusesSingle-cell genomicsvirusesVirus-host interactionMicrobiologyViruscell-type-specific gene expressionTranscriptomeVirologyMelanogasterHeat shockGeneSingle-cell RNA-seqsingle-cell RNA-seqvirus-host interactionbiologydual RNA-seqsingle-cell genomicsRNAbiology.organism_classificationVirologyQR1-502Infectious DiseasesViral replicationantiviral heat shock responseCell-type-specific gene expression<i>Drosophila</i> virusesDrosophila melanogasterDual RNA-seqViruses
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Increasing growth temperature alters the within-host competition of viral strains and influences virus genetic variation

2020

AbstractThe emergence of viral diseases in plant crops hamper the sustainability of food production, and this may be boosted by global warming. Concurrently, mixed viral infections are becoming common in plants, of which epidemiology are unpredictable due to within-host virus-virus interactions. However, the extent in which the combined effect of variations in the abiotic components of the plant ecological niche (e.g., temperature) and the prevalence of mixed infections (i.e., within-host interactions among viruses) affect the evolutionary dynamics of viral populations is not well understood. Here, we explore the interplay between ecological and evolutionary factors during viral infections,…

Ecological nicheGeneticsGenetic diversityViral replicationHost (biology)virusesmedia_common.quotation_subjectGenetic variationBiologyEvolutionary dynamicsCompetition (biology)Virusmedia_common
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Improvement in Nuclear Entry and Transgene Expression of Baculoviruses by Disintegration of Microtubules in Human Hepatocytes

2005

ABSTRACT Autographa californica multicapsid nucleopolyhedrovirus (AcMNPV), a potent virus for mammalian cell gene delivery, possesses an ability to transduce mammalian cells without viral replication. We examined the role of the cellular cytoskeleton in the cytoplasmic trafficking of viral particles toward the nucleus in human hepatic cells. Microscopic studies showed that capsids were found in the nucleus after either viral inoculation or cytoplasmic microinjection of nucleocapsids. The presence of microtubule (MT) depolymerizing agents caused the amount of nuclear capsids to increase. Overexpression of p50/dynamitin, an inhibitor of dynein-dependent endocytic trafficking from peripheral e…

EndosomeMicrotubule-associated proteinvirusesImmunologyEndocytic cycleGenetic VectorsActive Transport Cell NucleusGene ExpressionBiologyGene deliveryMicrobiologyMicrotubulesCell Linechemistry.chemical_compoundTransduction GeneticVirologyHumansNucleocapsidCytoskeletonDynactin Complexbeta-GalactosidaseMolecular biologyNucleopolyhedrovirusesRecombinant ProteinsVirus-Cell InteractionsNocodazoleMicroscopy ElectronViral replicationchemistryLac OperonCell cultureCytoplasmInsect ScienceHepatocytesMicrotubule-Associated Proteins
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