Search results for "White"

showing 10 items of 1158 documents

Multiple sclerosis: prevalence of the ‘central vein’ sign in white matter lesions on gadolinium-enhanced susceptibility-weighted images

2021

Aims To evaluate prospectively whether an intravenous gadolinium injection could improve the detection of the central vein sign on susceptibility-weighted imaging sequences obtained with a 1.5 T magnetic resonance scanner in patients with multiple sclerosis compared to unenhanced susceptibility-weighted images. Materials and methods This prospective, institution review board-approved study included 19 patients affected by multiple sclerosis (six men; 13 women; mean age 40.8 years, range 20–74 years). Patients had the relapsing–remitting clinical subtype in 95% of cases, and only one (5%) patient had the primary progressive clinical subtype of multiple sclerosis. T2-weighted images, fluid-at…

AdultMalemedicine.medical_specialtyMultiple SclerosisGadoliniumContrast Mediachemistry.chemical_elementGadolinium030218 nuclear medicine & medical imagingYoung Adult03 medical and health sciences0302 clinical medicinePrevalencemedicineHumansRadiology Nuclear Medicine and imagingProspective StudiesVeinAgedbusiness.industryMultiple sclerosisOriginal ArticlesGeneral MedicineMiddle Agedmedicine.diseaseMagnetic Resonance ImagingWhite MatterHyperintensitymedicine.anatomical_structurechemistrySusceptibility weighted imagingFemaleNeurology (clinical)Radiologybusiness030217 neurology & neurosurgerySign (mathematics)The Neuroradiology Journal
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Correlation between fatigue and brain atrophy and lesion load in multiple sclerosis patients independent of disability.

2007

Abstract Background Fatigue is a major problem in multiple sclerosis (MS), and its association with MRI features is debated. Objective To study the correlation between fatigue and lesion load, white matter (WM), and grey matter (GM), in MS patients independent of disability. Methods We studied 222 relapsing remitting MS patients with low disability (scores ≤ 2 at the Kurtzke Expanded Disability Status Scale). Lesion load, WM and GM were measured by fully automated, operator-independent, multi-parametric segmentation method. T1 and T2 lesion volume were also measured by a semi-automated method. Fatigue was assessed by the Fatigue Severity Scale (FSS), and patients divided in high-fatigue (FS…

AdultMalemedicine.medical_specialtyMultiple SclerosisStatistics as TopicGrey matterLesionWhite matterCentral nervous system diseaseDisability EvaluationAtrophyInternal medicinemedicineImage Processing Computer-AssistedHumansRisk factorFatigueAnalysis of VarianceBrain Mappingbusiness.industryMultiple sclerosisBrainmedicine.diseaseMagnetic Resonance ImagingSurgeryOxygenMultiple Sclerosis fatiguemedicine.anatomical_structureNeurologymultiple sclerosiFemaleNeurology (clinical)Analysis of variancemedicine.symptomAtrophybusinessbrain atrophyMRI
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Disturbed structural connectivity is related to inattention and impulsivity in adult attention deficit hyperactivity disorder

2010

Inattention and impulsivity are the most prominent clinical features of attention deficit hyperactivity disorder (ADHD) in adulthood. Structural and functional neuroimaging studies of subjects with ADHD have demonstrated abnormalities in several brain areas, including fronto-striatal and fronto-cerebellar networks. Mostly, these studies were based on volumetric measurements and have been conducted in children. We investigated white matter (WM) integrity and correlation with measures of attention and impulsivity in adult patients with ADHD adopting diffusion tensor imaging (DTI). N = 37 (21 males) never-medicated adult patients with ADHD combined subtype and N = 34 (16 males) healthy control…

AdultMalemedicine.medical_specialtyNeuropsychological TestsAudiologyImpulsivitybehavioral disciplines and activitiesWhite matterFunctional neuroimagingRating scaleImage Interpretation Computer-AssistedNeural Pathwaysmental disordersFractional anisotropymedicineHumansAttention deficit hyperactivity disorderPsychiatrymedicine.diagnostic_testGeneral NeuroscienceBrainNeuropsychological testmedicine.diseaseDiffusion Magnetic Resonance Imagingmedicine.anatomical_structurenervous systemAttention Deficit Disorder with HyperactivityImpulsive BehaviorFemalemedicine.symptomPsychologyDiffusion MRIEuropean Journal of Neuroscience
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A Novel Loss of Function Mutation of PCSK9 Gene in White Subjects With Low-Plasma Low-Density Lipoprotein Cholesterol

2007

Objectives— The PCSK9 gene, encoding a pro-protein convertase involved in posttranslational degradation of low-density lipoprotein receptor, has emerged as a key regulator of plasma low-density lipoprotein cholesterol. In African-Americans two nonsense mutations resulting in loss of function of PCSK9 are associated with a 30% to 40% reduction of plasma low-density lipoprotein cholesterol. The aim of this study was to assess whether loss of function mutations of PCSK9 were a cause of familial hypobetalipoproteinemia and a determinant of low-plasma low-density lipoprotein cholesterol in whites. Methods and Results— We sequenced PCSK9 gene in 18 familial hypobetalipoproteinemia subjects and i…

AdultMalemedicine.medical_specialtyNonsense mutationBiologymedicine.disease_causePolymorphism Single NucleotideRisk AssessmentSensitivity and SpecificityStatistics NonparametricWhite Peopleloss of function mutationHypobetalipoproteinemiaschemistry.chemical_compoundPCSK9 GeneGene FrequencyInternal medicinemedicineHumansGenetic Predisposition to DiseaseMutationhypocholesterolemiaCholesterolIncidencePCSK9Serine EndopeptidasesCholesterol LDLmedicine.diseaseHypocholesterolemiaEndocrinologyfamilial hypobetalipoproteinemiachemistryCodon NonsensePCSK9 geneCase-Control Studiesfamilial hypobetalipoproteinemia hypocholesterolemia loss of function mutation PCSK9 genefamilial hypobetalipoproteinemia; hypocholesterolemia; loss of function mutation; PCSK9 gene.FemaleProprotein ConvertasesHypobetalipoproteinemiaProprotein Convertase 9Cardiology and Cardiovascular MedicineLipoprotein
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Acute extrahepatic infectious or inflammatory diseases are a cause of transient mosaic pattern on CT and MR imaging related to sinusoidal dilatation …

2015

To report the association of a mosaic enhancement pattern on contrast-enhanced CT or MR imaging and hepatic sinusoidal dilatation (SD) with acute inflammatory conditions affecting extrahepatic organs. From 2007 to 2012, patients with acute inflammatory diseases who underwent contrast-enhanced CT and/or MRI of the liver with a mosaic enhancement pattern were selected. Clinico-biological and other imaging features were collected at diagnosis and during follow-up. Sixteen patients were included (15 women, median age 27 years; range 18–68). Five women (33 %) were receiving oral contraceptives. Acute inflammatory diseases included pyelonephritis (n = 10), pancreatitis (n = 2), pneumonia (n = 1),…

AdultMalemedicine.medical_specialtyPathologyAdolescentBiopsyContrast Media030218 nuclear medicine & medical imagingYoung Adult03 medical and health sciences0302 clinical medicineWhite blood cellBiopsymedicineRadiology Nuclear Medicine and imagingComputed tomographyAgedNeuroradiologyInflammationmedicine.diagnostic_testPortal Veinbusiness.industryMosaic enhancement patternSinusoidal dilatationMagnetic resonance imagingInterventional radiologyGeneral MedicineMiddle Agedmedicine.diseaseMagnetic Resonance ImagingInflammation and infectionPneumoniamedicine.anatomical_structureLiver NeoplasmLiver biopsyAcute DiseaseHepatic VeinPancreatitisFemale030211 gastroenterology & hepatologyRadiologyInfectionTomography X-Ray ComputedbusinessDilatation PathologicHumanEuropean Radiology
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Genetic variants associated with cardiac structure and function: a meta-analysis and replication of genome-wide association data.

2009

Context Echocardiographic measures of left ventricular (LV) structure and function are heritable phenotypes of cardiovascular disease. Objective To identify common genetic variants associated with cardiac structure and function by conducting a meta-analysis of genome-wide association data in 5 population-based cohort studies (stage 1) with replication (stage 2) in 2 other community-based samples. Design, Setting, and Participants Within each of 5 community-based cohorts comprising the EchoGen consortium (stage 1; n = 12 612 individuals of European ancestry; 55% women, aged 26-95 years; examinations between 1978-2008), we estimated the association between approximately 2.5 million single-nuc…

AdultMalemedicine.medical_specialtyPathologyGenotypeHeart VentriclesPopulationLocus (genetics)Genome-wide association studySingle-nucleotide polymorphismPolymorphism Single NucleotideVentricular Function LeftWhite PeopleArticleVentricular Dysfunction LeftSDG 3 - Good Health and Well-beingRisk FactorsInternal medicineMedicineHumansHeart AtriaInternational HapMap ProjecteducationAortaAgedAged 80 and overeducation.field_of_studybusiness.industryGeneral MedicineOrgan SizeMiddle AgedPhenotypeCardiovascular DiseasesEchocardiographyMeta-analysisCohortCardiologyFemalebusinessCohort studyGenome-Wide Association StudyJAMA
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Enzyme Replacement Therapy Stabilized White Matter Lesion Progression in Fabry Disease

2014

<b><i>Background:</i></b> The central nervous system manifestations in Fabry disease (FD) include progressive white matter lesions (WMLs) and stroke. Due to progressive microvascular involvement, men and women with FD over 35 years of age develop WMLs. Moreover, the prevalence of stroke has been estimated to be 12 times higher in FD compared with the general population. Enzyme replacement therapy (ERT) is available and has shown beneficial effects on renal, cardiac, and peripheral nerve function in FD, but the ERT effect on the progression of WMLs, or the reduction in cerebrovascular events, remains unknown. <b><i>Methods:</i></b> The WML burd…

AdultMalemedicine.medical_specialtyPopulationPlaceboLeft ventricular hypertrophyYoung AdultLeukoencephalopathiesInternal medicinemedicineHumansEnzyme Replacement TherapyeducationStrokeAgededucation.field_of_studyVascular diseasebusiness.industryBrainEnzyme replacement therapyMiddle Agedmedicine.diseaseMagnetic Resonance ImagingWhite MatterFabry diseaseHyperintensitySurgeryIsoenzymesNeurologyalpha-GalactosidaseDisease ProgressionCardiologyFabry DiseaseFemaleNeurology (clinical)Cardiology and Cardiovascular MedicinebusinessCerebrovascular Diseases
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Prevalence of refractive errors in the European adult population: the Gutenberg Health Study (GHS).

2014

Objective To study the distribution of refractive errors among adults of European descent. Design Population-based eye study in Germany with15 010 participants aged 35–74 years. Methods The study participants underwent a detailed ophthalmic examination according to a standardised protocol. Refractive error was determined by an automatic refraction device (Humphrey HARK 599) without cycloplegia. Definitions for the analysis were myopia +0.5 D, astigmatism >0.5 cylinder D and anisometropia >1.0 D difference in the spherical equivalent between the eyes. Exclusion criterion was previous cataract or refractive surgery. Results 13 959 subjects were eligible. Refractive errors ranged from −21.5 to…

AdultMalemedicine.medical_specialtyRefractive errorgenetic structuresmedicine.medical_treatmentPopulationVisual AcuityAstigmatismWhite PeopleCellular and Molecular NeuroscienceAge DistributionRefractive surgeryOphthalmologyGermanymedicinePrevalenceHumansVision testProspective StudiesSex DistributioneducationRetinoscopyAnisometropiaAgededucation.field_of_studymedicine.diagnostic_testbusiness.industryVision TestsCycloplegiaMiddle Agedmedicine.diseaseRefractive ErrorsHealth Surveyseye diseasesSensory SystemsOphthalmologyOptometryFemalemedicine.symptombusinessRetinoscopyThe British journal of ophthalmology
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Evoked potential study in facio-scapulo-humeral muscular dystrophy.

1997

Nerve conduction velocities (NCVs), somatosensory (SEPs) and auditory evoked potentials (BAEPs) were recorded in 9 patients with facio-scapulo-humeral dystrophy (FSHD) and in 20 age-matched controls. In FSHD patients a significant increase of the nerve distal sensory latencies and of the absolute SEP latencies revealed a subclinical involvement of the afferent sensory pathways, as well as the abnormal slowing of the later components of the BAEPs, pointed to a central auditory dysfunction. Moreover all patients underwent brain MRI that showed the presence of white matter hyperintense lesions in 4 of them (44%). No correlations were found between individual or total number of SEP and BAEP abn…

AdultMalemedicine.medical_specialtyShoulderNeural ConductionSensory systemSomatosensory systemSeverity of Illness IndexNerve conduction velocityMuscular DystrophiesWhite matterInternal medicineEvoked Potentials SomatosensorymedicineEvoked Potentials Auditory Brain StemHumansMuscular dystrophyAuditory Diseases CentralSubclinical infectionChi-Square DistributionDystrophyBrainGenetic VariationGeneral MedicineMiddle Agedmedicine.diseaseMagnetic Resonance ImagingMedian Nervemedicine.anatomical_structureNeurologySomatosensory evoked potentialCase-Control StudiesFaceCardiologyArmFemaleNeurology (clinical)Tibial NervePsychologyNeuroscienceActa neurologica Scandinavica
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The PTPN22gain-of-function+1858T(+) genotypes correlate with low IL-2 expression in thymomas and predispose to myasthenia gravis

2009

Protein tyrosine phosphatase, non-receptor type 22 (PTPN22) inhibits T-cell activation and interleukin-2 (IL-2) production. The PTPN22(gain-of-function)+1858T(+) genotypes predispose to multiple autoimmune diseases, including early-onset (non-thymomatous) myasthenia gravis (MG). The disease association and the requirement of IL-2/IL-2 receptor signaling for intrathymic, negative T-cell selection have suggested that these genotypes may weaken T-cell receptor (TCR) signaling and impair the deletion of autoreactive T cells. Evidence for this hypothesis is missing. Thymoma-associated MG, which depends on intratumorous generation and export of mature autoreactive CD4(+) T cells, is a model of au…

AdultMalemedicine.medical_specialtyThymomaAdolescentGenotypeThymomaImmunologyBiologymedicine.disease_causePolymorphism Single NucleotideWhite PeopleAutoimmunityPTPN22Young AdultAntigens CDInternal medicineMyasthenia GravisCentral tolerance inductionGeneticsmedicineHumansCTLA-4 AntigenGenetic Predisposition to DiseaseReceptorGenetics (clinical)AgedAged 80 and overT-cell receptorProtein Tyrosine Phosphatase Non-Receptor Type 22Thymus NeoplasmsMiddle Agedmedicine.diseaseMyasthenia gravisEndocrinologyImmunologyInterleukin-2FemaleCentral toleranceGenes & Immunity
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