Search results for "antagonist"

showing 10 items of 1660 documents

The metabolic syndrome in hypertension: European society of hypertension position statement.

2008

The metabolic syndrome considerably increases the risk of cardiovascular and renal events in hypertension. It has been associated with a wide range of classical and new cardiovascular risk factors as well as with early signs of subclinical cardiovascular and renal damage. Obesity and insulin resistance, beside a constellation of independent factors, which include molecules of hepatic, vascular, and immunologic origin with proinflammatory properties, have been implicated in the pathogenesis. The close relationships among the different components of the syndrome and their associated disturbances make it difficult to understand what the underlying causes and consequences are. At each of these …

medicine.medical_specialtyPhysiologySodium Chloride Symporter InhibitorsAdrenergic beta-AntagonistsPhysical exerciseAngiotensin-Converting Enzyme InhibitorsType 2 diabetesBioinformaticsInsulin resistanceWeight lossInternal medicineInternal MedicinemedicineHumansThiazideAntihypertensive AgentsMetabolic Syndromebusiness.industrymedicine.diseaseCalcium Channel BlockersObesityExercise TherapyEndocrinologyBlood pressureHypertensionmedicine.symptomMetabolic syndromeCardiology and Cardiovascular MedicinebusinessAngiotensin II Type 1 Receptor BlockersRisk Reduction Behaviormedicine.drugDiet TherapyJournal of hypertension
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Release of acetylcholine from murine embryonic stem cells: Effect of nicotinic and muscarinic receptors and blockade of organic cation transporter

2012

The non-neuronal cholinergic system is widely expressed in nature. The present experiments were performed to characterize the non-neuronal cholinergic system in murine embryonic stem cells (CGR8 cell line).CGR8 cells were cultured in gelatinized flasks with Glasgow's buffered minimal essential medium (Gibco, Germany). Acetylcholine was measured by HPLC combined with bioreactor and electrochemical detection.CGR8 cells contained 1.08±0.12 pmol acetylcholine/10(6) cells (n=7) which was reduced to 0.50±0.06 pmol/10(6) cells (n=6; p0.05) in the presence (4h) of 30μM bromoacetylcholine to block choline acetyltransferase. A time-dependent release of acetylcholine into the incubation medium was dem…

medicine.medical_specialtyPhysostigmineMuscarinic AntagonistsNicotinic AntagonistsMuscarinic AgonistsReceptors NicotinicGeneral Biochemistry Genetics and Molecular BiologyCell LineMicechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineMuscarinic acetylcholine receptor M4AnimalsCholinesterasesGeneral Pharmacology Toxicology and PharmaceuticsCation Transport ProteinsEmbryonic Stem CellsOrganic cation transport proteinsMuscarineQuininebiologyOxotremorineMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2General MedicineReceptors MuscarinicAcetylcholineCell biologyEndocrinologyNicotinic agonistchemistrybiology.proteinCholinesterase InhibitorsAcetylcholinemedicine.drugLife Sciences
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Isoprenaline and forskolin increase evoked vasopressin release from rat pituitary

1982

Isolated neurointermediate lobes of rat pituitaries were incubated in Krebs solution. The vasopressin release evoked by electrical stimulation (0.2 ms, 80 V, 15 Hz, 10 s trains at 10 s intervals for a total of 10 min) was completely inhibited by tetrodotoxin. Isoprenaline increased the evoked vasopressin release to a maximum of 60% (EC50 10 nM) and this effect was antagonized surmountably by propranolol. Forskolin increased the vasopressin release by 98%. These results suggest the presence within the neurohypophysis of a beta-adrenoceptor-linked adenylate cyclase facilitating vasopressin secretion.

medicine.medical_specialtyPituitary glandVasopressinVasopressinsStimulationPropranololchemistry.chemical_compoundPituitary Gland PosteriorInternal medicineIsoprenalinemedicineAnimalsPharmacologyForskolinColforsinIsoproterenolRats Inbred StrainsElectric StimulationStimulation ChemicalRatsEndocrinologymedicine.anatomical_structureVasopressin secretionchemistryPituitary GlandTetrodotoxinFemaleDiterpeneshormones hormone substitutes and hormone antagonistsmedicine.drugEuropean Journal of Pharmacology
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Agouti-related peptide-expressing neurons are mandatory for feeding.

2005

Multiple hormones controlling energy homeostasis regulate the expression of neuropeptide Y (NPY) and agouti-related peptide (AgRP) in the arcuate nucleus of the hypothalamus. Nevertheless, inactivation of the genes encoding NPY and/or AgRP has no impact on food intake in mice. Here we demonstrate that induced selective ablation of AgRP-expressing neurons in adult mice results in acute reduction of feeding, demonstrating direct evidence for a critical role of these neurons in the regulation of energy homeostasis.

medicine.medical_specialtyPro-OpiomelanocortinTime FactorsPeptideCell CountBiologyEnergy homeostasisEatingMiceArcuate nucleusInternal medicinemedicineAnimalsAgouti-Related ProteinDiphtheria ToxinNeuropeptide YRegulation of gene expressionchemistry.chemical_classificationMice KnockoutNeuronsGeneral Neurosciencedigestive oral and skin physiologyBody WeightArcuate Nucleus of HypothalamusProteinsFeeding BehaviorNeuropeptide Y receptorbeta-GalactosidaseAnorexiaEndocrinologynervous systemchemistryGene Expression RegulationHypothalamusIntercellular Signaling Peptides and ProteinsAgouti-related peptidehormones hormone substitutes and hormone antagonistsHormoneNature neuroscience
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Midregion parathyroid hormone-related protein inhibits growth and invasion in vitro and tumorigenesis in vivo of human breast cancer cells.

2001

Parathyroid hormone-related protein (PTHrP) is critical for normal mammary development and is overexpressed by breast cancers. PTHrP is a peptide hormone that undergoes extensive post-translational processing, and PTHrP(38–94)-amide is one of the mature secretory forms of the peptide. In this study, we explored the effect of PTHrP(38–94)-amide in a panel of six breast cancer cell lines “in vitro” and in MDA-MB231 cells “in vivo” specifically examining cell viability, proliferation, invasiveness, and growth in nude mice. PTHrP(38–94)-amide markedly inhibited proliferation and also caused striking toxicity and accelerated cell death in breast cancer cells. In addition, direct injection of PTH…

medicine.medical_specialtyProgrammed cell deathEndocrinology Diabetes and MetabolismMammary glandMice NudeAntineoplastic AgentsBreast NeoplasmsCell CountBiologymedicine.disease_causeMiceInternal medicinemedicineTumor Cells CulturedAnimalsHumansOrthopedics and Sports MedicineNeoplasm Invasivenessskin and connective tissue diseasesParathyroid hormone-related proteinCell growthParathyroid Hormone-Related ProteinCancerProteinsNeoplasms Experimentalmedicine.diseasePeptide FragmentsDisease Models AnimalEndocrinologymedicine.anatomical_structureCell cultureCancer cellCancer researchFemaleCarcinogenesishormones hormone substitutes and hormone antagonistsCell DivisionJournal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research
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Estradiol, acting through estrogen receptor alpha, restores dimethylarginine dimethylaminohydrolase activity and nitric oxide production in oxLDL-tre…

2011

Asymmetric dimethylarginine (ADMA) is an endogenous inhibitor of nitric oxide (NO) synthase. ADMA accumulation, mainly due to a decreased dimethylarginine dimethylaminohydrolase (DDAH) activity, has been related to the development of cardiovascular diseases. We investigate whether estradiol prevents the changes induced by oxidized low density lipoprotein (oxLDL) on the DDAH/ADMA/NO pathway in human umbilical artery endothelial cells (HUAEC). HUAEC were exposed to estradiol, native LDL (nLDL), oxLDL and their combinations for 24 h. In some experiments, cells were also exposed to the unspecific estrogen receptor (ER) antagonist ICI 182780, the specific ERα antagonist MPP or specific agonists …

medicine.medical_specialtyProtein-Arginine N-MethyltransferasesEndotheliumNitric Oxide Synthase Type IIImedicine.drug_classBlotting WesternArginineNitric OxideBiochemistryUmbilical ArteriesNitric oxideAmidohydrolasesReceptors G-Protein-Coupledchemistry.chemical_compoundEndocrinologyEnosInternal medicinemedicineEstrogen Receptor betaHumansEstrogens Non-SteroidalMolecular BiologyCells CulturedbiologyEstradiolArtèriesProtein StabilityEstrogen AntagonistsEstrogen Receptor alphaEndoteli vascularbiology.organism_classificationNitric oxide synthaseIsoenzymesLipoproteins LDLRepressor Proteinsmedicine.anatomical_structureEndocrinologychemistryReceptors EstrogenEstrogenbiology.proteinlipids (amino acids peptides and proteins)Endothelium VascularAsymmetric dimethylarginineEstrogen receptor alphaGPER
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Mechanism of New Antipsychotic Medications

2003

Antagonism of D 2 -like dopamine receptors is the putative mechanism underlying the antipsychotic efficacy of psychotropic drugs. Positron emission tomographic studies suggest that the antipsychotic effect of dopamine receptor antagonists occurs within a therapeutic window between 60% and 80%(striatal) D 2 receptor occupancy. The incidence of extrapyramidal side effects increases above the 80% threshold. However, the novel atypical antipsychotic drug, aripiprazole, occupies up to 95% of striatal D 2 -like dopamine receptors at clinical doses, and the incidence of extrapyramidal side effects with aripiprazole is no higher than with placebo. The most likely explanation for this finding is ari…

medicine.medical_specialtyPsychosismedicine.drug_classmedicine.medical_treatmentAripiprazoleAtypical antipsychoticQuinolonesPharmacologyPartial agonistPiperazinesBasal Ganglia DiseasesArts and Humanities (miscellaneous)Dopamine receptor D2Internal medicinemedicineHumansAntipsychoticDose-Response Relationship DrugReceptors Dopamine D2Putamenmedicine.diseaseCorpus StriatumProlactinDopamine D2 Receptor AntagonistsPsychiatry and Mental healthEndocrinologyMechanism of actionDopamine receptorSchizophreniaAripiprazolemedicine.symptomPsychologyAntipsychotic AgentsTomography Emission-Computedmedicine.drugArchives of General Psychiatry
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SEXUALHORMONE-BINDING GLOBULINE (SHBG) AND FREE TESTOSTERONE (FTE) IN PATIENTS WITH ULLRICH TURNLR SYNDROME(UTS) RECEIVING OXANDROLONE

1993

Our investigation showed that the concentration of SHBG decreased in healthy girls and boys at increased FIE concentrations. It was therefore the aim of the present study to examine the effects of anabolic steroid oxandrolone in SHBG and FTE concentrations in patients with Turner's syndrome. The normal dose to promote growth in patients wilh UTS is 0,1 mg/kg/BW. Our patients received a very low dose of 1,25 mg oxandrolone/doy. We analysed 21 sera of UTS patients. The determination of SHBG concentrations was performed with the IRMA (Famos diagnostics, Oulunsala, Finnland), the concentration of FTE was determined with the RIA (Immunchem.Corporation, Carson, USA). In 7 untreated UIS patients w…

medicine.medical_specialtyPubic hair stagebiologyFree testosteronebusiness.industrymedicine.medical_treatmentLow doseOxandroloneSex hormone-binding globulinEndocrinologyInternal medicinePediatrics Perinatology and Child Healthbiology.proteinMedicineIn patientbusinesshormones hormone substitutes and hormone antagonistsTestosteroneAnabolic steroidmedicine.drugPediatric Research
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New strategies for medical management of overactive bladder in children.

2011

Purpose of review The medical treatment of children with non-neurogenic overactive bladder syndrome (OAB) is still limited to a small number of drugs approved for use in childhood according to the national regulations of each country. Recent findings Over the last few years, there were several studies on the use of antimuscarinics other than oxybutynin in children, as well as some on the use of extended release oxybutynin and tolterodine and transdermal oxybutynin. It was shown that the combination of two different anticholinergics might be a well tolerated and successful option in children with OAB refractory to monotherapy, as well as administration of a receptor-selective antimuscarinic …

medicine.medical_specialtyQuinuclidinesBotulinum ToxinsCombination therapyTolterodine TartrateNortropanesUrologyPhenylpropanolamineUrologyUrinationMuscarinic AntagonistsBenzilatesCresolsTetrahydroisoquinolinesmedicineHumansBenzhydryl CompoundsOxybutyninIntensive care medicineChildSolifenacinbusiness.industryUrinary Bladder OveractiveStandard treatmentSolifenacin Succinatemedicine.diseaseBotulinum toxinReceptors MuscarinicOveractive bladderMandelic AcidsPropiverineTolterodinebusinessmedicine.drugCurrent opinion in urology
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D2-receptor imaging with [123I]IBZM and Single Photon Emission Tomography in psychiatry: a survey of current status

1995

D2-dopamine receptors can be visualized in the human brain in vivo by Single Photon Emission Tomography (SPECT) and the radiolabeled benzamide [123I]IBZM. The present paper reviews the current status of this type of functional brain imaging with respect to basic methodological aspects, data analysis and quantification. The results from published clinical studies in different psychiatric patient populations and normal controls with [123I]IBZM are reviewed. [123I]IBZM-SPECT is a powerful tool for the investigation of D2-dopamine receptor status in psychiatric disorders, different types of drug treatment as well as therapeutic and side effects of pharmacologic agents. However, there still is a…

medicine.medical_specialtyReceptor StatusPyrrolidinesNeurologyContrast MediaDrug treatmentDopamine receptor D2Image Processing Computer-AssistedmedicineSingle Photon Emission TomographyHumansPsychiatryBiological PsychiatryBrain ChemistryTomography Emission-Computed Single-Photonmedicine.diagnostic_testReceptors Dopamine D2business.industryBrainHuman brainPsychiatry and Mental healthmedicine.anatomical_structureNeurology123i ibzmPositron emission tomographyBenzamidesDopamine AntagonistsNeurology (clinical)Nuclear medicinebusinessPsychologyJournal of Neural Transmission
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