Search results for "basement membrane"

showing 10 items of 80 documents

Coexpression of extracellular matrix glycoproteins undulin and tenascin in human autosomal dominant polycystic kidney disease.

1993

Autosomal dominant polycystic kidney disease (ADPKD) is the most common entity of cystic diseases of the kidney leading to end-stage renal insufficiency. Changes in extracellular matrix (ECM) are regarded to be an important pathogenic factor connected with the genes assumed to be responsible for human ADPKD. In order to assess the biological significance of altered expression and deposition of ECM glycoproteins for human ADPKD at molecular levels fresh-frozen tissue from ADPKD kidneys, fetal kidneys and normal adult kidneys were comparatively tested by immunohistochemistry for the presence of multifunctional ECM glycoproteins undulin, tenascin and fibronectin, interstitial collagen types I,…

Pathologymedicine.medical_specialtyCell Adhesion Molecules NeuronalAutosomal dominant polycystic kidney diseaseTenascinGene ExpressionKidneyExtracellular matrixFetusLamininTransforming Growth Factor betamedicineHumansRNA MessengerCells CulturedGlycoproteinsBasement membraneKidneyExtracellular Matrix Proteinsbiologyurogenital systemTenascinmedicine.diseasePolycystic Kidney Autosomal DominantImmunohistochemistryEpitheliumCell biologyFibronectinsFibronectinmedicine.anatomical_structurebiology.proteinCollagenLamininNephron
researchProduct

Alport's syndrome: specificity and pathogenesis of glomerular basement membrane alterations.

1987

In Alport's syndrome (AS) thinning and splitting of the glomerular basement membrane (GBM) are assumed to be characteristic ultrastructural alterations. Both lesions are, however, non-specific because they can occur in other glomerulopathies. In addition, splitting may be found in non-glomerular structures. It should be emphasized that the characteristic lesion in AS is a result of the widespread combination of thin and split GBM in the same biopsy specimen. In our opinion the basic lesion is the thin GBM, which is characterized by a lamina densa (measuring 50-150 nm in thickness) which may begin to split as a result of focal detachment of podocyte pedicles (spacing) and repeated subepithel…

Pathologymedicine.medical_specialtyKidney GlomerulusNephritis HereditaryBiologyurologic and male genital diseasesBasement MembranePodocyteLesionPathogenesisBiopsymedicineHumansmedicine.diagnostic_testurogenital systemGlomerular basement membraneAnatomymedicine.diseasefemale genital diseases and pregnancy complicationsmedicine.anatomical_structureNephrologyPediatrics Perinatology and Child HealthUltrastructureLamina densamedicine.symptomNephritisPediatric nephrology (Berlin, Germany)
researchProduct

Neonatal form of nemaline myopathy, muscle immaturity, and a microvascular injury.

1990

An infant with a neonatal form of nemaline myopathy showed ultrastructural features of muscle immaturity. Immaturity was characterized by an abnormal presence of myotubes, as well as cells in clusters within a common basement membrane and a great number of satellite cells adhering to very small muscle fibers. In addition, degenerative changes and a severe microvascular lesion were observed. The pathologic findings in the muscle of this patient were those of neonatal nemaline myopathy complicating severe microvascular injury, possibly induced by an unknown toxic agent. ( J Child Neurol 1990;5:122-126).

Pathologymedicine.medical_specialtyMuscle HypotoniaBiopsyIschemiaBiologyMuscle Smooth VascularLesion03 medical and health sciences0302 clinical medicineNemaline myopathyMyofibrilsIschemia030225 pediatricsBiopsymedicineHumansBasement membraneInclusion BodiesAsphyxia NeonatorumRespiratory Distress Syndrome Newbornmedicine.diagnostic_testMyogenesisMicrocirculationMusclesInfant NewbornDisseminated Intravascular Coagulationmedicine.diseaseMicroscopy ElectronMuscular Atrophymedicine.anatomical_structurePediatrics Perinatology and Child HealthMuscle HypotoniaFemaleNeurology (clinical)medicine.symptomMyofibril030217 neurology & neurosurgeryJournal of child neurology
researchProduct

Methotrexate as a treatment in ocular cicatricial moderate pemphigoid.

2013

Abstract Clinical case A 73 year-old woman presented with a history of non-specific symptoms and photophobia in both eyes of 1 year progression. The examination revealed a bilateral symblepharon and fornix shortening. Immunohistochemical analysis confirmed the presence of linear deposits of IgG, IgM and C3 along the conjunctival basement membrane. With the diagnosis of ocular cicatricial pemphigoid, systemic treatment with subcutaneous methotrexate was prescribed. Discussion We consider such treatment a very effective initial immunosuppressive alternative in patients with moderate conjunctival inflammation and in cases of rapid progression.

Pemphigoidmedicine.medical_specialtyPhotophobiaEtiologyMetotrexatePemphigoid Benign Mucous MembraneComplications scarHyperemiaBasement MembraneDiagnosis ocular cicatricial pemphigoidmedicineUso terapeuticoHumansOcular cicatricial pemphigoidAgedBasement membraneKeratitisbusiness.industrySymblepharonFornixTherapeutic useDiagnostico penfigoide ocular cicatricialGeneral MedicineComplement C3medicine.diseaseDermatologyeye diseasesAgentes inmunosupresoresmedicine.anatomical_structureMethotrexateImmunoglobulin MEtiologiaImmunoglobulin GEtiologyImmunohistochemistryMethotrexateComplicaciones cicatrizFemalesense organsPenfigoide ocular cicatricialmedicine.symptomImmunosuppressive agentsbusinessConjunctivaImmunosuppressive Agentsmedicine.drugArchivos de la Sociedad Espanola de Oftalmologia
researchProduct

Inhibition of glycosaminoglycan modification of perlecan domain I by site-directed mutagenesis changes protease sensitivity and laminin-1 binding act…

1998

AbstractGlycosaminoglycan attachment to perlecan domain I (173 residues) was completely prevented by site-directed mutagenesis of Ser-65, Ser-71 and Ser-76 as shown by recombinant production in mammalian cells. This did not interfere with the proper folding of the domain's SEA module but enhanced its sensitivity to neutral proteases. Lack of substitution also abolished binding to the two major heparin binding sites of laminin-1.

ProteasesBasement membraneRecombinant proteinmedicine.medical_treatmentMolecular Sequence DataBiophysicsPerlecanBiochemistrySubstrate SpecificityStructural BiologyLamininEndopeptidasesGeneticsmedicineAnimalsAmino Acid SequenceBinding siteSite-directed mutagenesisMolecular BiologyGlycosaminoglycansSite-directed mutagenesisBinding SitesProteasebiologyChemistryMutagenesisCell BiologyRecombinant ProteinsBiochemistryProteoglycanProteoglycanProteolysisMutagenesis Site-Directedbiology.proteinProteoglycansHeparitin SulfateLamininHeparan Sulfate ProteoglycansProtein BindingFEBS Letters
researchProduct

Endurance training damages small airway epithelium in mice.

2007

RATIONALE: In athletes, airway inflammatory cells were found to be increased in induced sputum or bronchial biopsies. Most data were obtained after exposure to cold and dry air at rest or during exercise. Whether training affects epithelial and inflammatory cells in small airways is unknown. OBJECTIVES: To test whether endurance training under standard environmental conditions causes epithelial damage and inflammation in the small airways of mice. METHODS AND MEASUREMENTS: Formalin-fixed, paraffin-embedded lung sections were obtained in sedentary (n = 14) and endurance-trained (n = 16) Swiss mice at baseline and after 15, 30, and 45 days of training. The following variables were assessed (m…

Pulmonary and Respiratory MedicineMalePathologymedicine.medical_specialtyInflammationApoptosisCritical Care and Intensive Care MedicineSettore BIO/09 - FisiologiaEpitheliumEpithelial DamageLeukocyte CountMiceEndurance trainingIntensive carePhysical Conditioning AnimalProliferating Cell Nuclear AntigenmedicineLeukocytesAnimalsBronchitisCell ProliferationBasement membraneLungAerobic exercise bronchial responsivenes methacholine deep inspiration leukotrienesbusiness.industryNF-kappa Brespiratory systemImmunohistochemistryEpitheliumrespiratory tract diseasesDisease Models Animalmedicine.anatomical_structureRespiratory epitheliummedicine.symptombusinessAmerican journal of respiratory and critical care medicine
researchProduct

Preparation and characterization of a biologic scaffold from esophageal mucosa

2013

Biologic scaffolds composed of extracellular matrix (ECM) are commonly used to facilitate a constructive remodeling response in several types of tissue, including the esophagus. Surgical manipulation of the esophagus is often complicated by stricture, but preclinical and clinical studies have shown that the use of an ECM scaffold can mitigate stricture and promote a constructive outcome after resection of full circumference esophageal mucosa. Recognizing the potential benefits of ECM derived from homologous tissue (i.e., site-specific ECM), the objective of the present study was to prepare, characterize, and assess the in-vivo remodeling properties of ECM from porcine esophageal mucosa. The…

ScaffoldPathologymedicine.medical_specialtySwineBiophysicsBioengineeringArticleBiomaterialsExtracellular matrixECM (extracellular matrix) Decellularization Scaffold Immune responseEsophagusTissue engineeringMaterials TestingmedicineAnimalsEsophagusCells CulturedBasement membraneDecellularizationMucous MembraneTissue EngineeringTissue Scaffoldsbusiness.industrySoft tissueExtracellular MatrixRatsmedicine.anatomical_structureMechanics of MaterialsCeramics and CompositesMicroscopy Electron ScanningStem cellbusiness
researchProduct

Matrix metalloproteinases 2 and 9, and extracellular matrix in Kaposi's sarcoma

2010

Matrix metalloproteinases (MMPs) are associated with Kaposi's sarcoma (KS) tumorigenesis and may contribute to the mechanism of KS invasive growth. To date, only a few MMPs have been studied in KS lesions, and exactly which MMPs are involved in KS development and progression remains unanswered. However, MMPs 2 and 9 have been associated with different phases of angiogenesis, but their role in the proteolytic modification of the extracellular matrix has not been investigated. The results of this study confirm that MMPs, specifically MMP-2 and MMP-9, can contribute to angiogenesis by disrupting the vessel basement membrane and other extracellular matrix barriers, and enabling endothelial cell…

Skin NeoplasmsAngiogenesisDermatologyMatrix metalloproteinasemedicine.disease_causeBasement MembraneExtracellular matrixCell Movementmatrix metalloproteinase-9Settore MED/35 - Malattie Cutanee E VenereemedicineHumansSarcoma KaposiKaposi's sarcomaBasement membraneNeovascularization Pathologicbusiness.industryGeneral Medicinemedicine.diseaseVirologyExtracellular Matrixmedicine.anatomical_structureMatrix Metalloproteinase 9Invasive growthsarcoma of KaposiCancer researchMatrix Metalloproteinase 2SarcomaCarcinogenesisbusinessmatrix metalloproteinase- 2Dermatologic Therapy
researchProduct

The effect of detergents on the basement membrane complex of a biologic scaffold material

2013

The basement membrane complex (BMC) is a critical component of the extracellular matrix (ECM) that supports and facilitates the growth of cells. This study investigates four detergents commonly used in the process of tissue decellularization and their effect upon the BMC. The BMC of porcine urinary bladder was subjected to 3% Triton-X 100, 8 mM 3-[(3-cholamidopropyl) dimethylammonio]-1-propanesulfonate (CHAPS), 4% sodium deoxycholate or 1% sodium dodecyl sulfate (SDS) for 24 h. The BMC structure for each treatment group was assessed by immunolabeling, scanning electron microscopy (SEM) and second harmonic generation (SHG) imaging of the fiber network. The composition was assessed by quantif…

Sus scrofaFluorescent Antibody TechniqueBiochemistryBasement MembraneGlycosaminoglycanExtracellular matrixImmunolabelingchemistry.chemical_compoundTissue ScaffoldChapsSodium dodecyl sulfateDecellularizationGlycosaminoglycansMicrovesselEndothelial CellDecellularizationTissue ScaffoldsIntegrin beta1Extracellular matrixGeneral Medicinemedicine.anatomical_structureCollagenHumanBiotechnologyDetergentMaterials scienceDetergentsBiomedical EngineeringArticleBiomaterialsImaging Three-DimensionalRe-endothelizationIn Situ Nick-End LabelingmedicineAnimalsHumansMolecular BiologyOrgan engineeringBasement membraneStaining and LabelingAnimalBiologic scaffoldAntigens CD29Endothelial CellsDNABiomaterialMolecular biologyKi-67 AntigenGlycosaminoglycanchemistryTissue DecellularizationMicrovesselsActa Biomaterialia
researchProduct

Differential expression of collagen types I, III, and IV by fat-storing (Ito) cells in vitro

1992

It has been observed that Ito cells in vitro undergo phenotypical changes ("activation") similar to those noted in vivo during the development of liver fibrosis. Because conflicting data have been published on the amount and different types of collagens synthesized by Ito cells in vitro, collagen biosynthesis was studied at different "activation" stages on both the protein and RNA levels. Immunoprecipitation of endogenously labeled collagen showed that freshly isolated ("resting") Ito cells synthesize mainly collagen type IV. Collagen type I was hardly detectable in the earlier stage of primary culture, but it clearly increased starting 5 days after isolation. Compared with the basal rates …

Transcription GeneticMolecular Sequence DataCell SeparationBiologychemistry.chemical_compoundBiosynthesisIn vivomedicineAnimalsCells CulturedBasement membraneBase SequenceHepatologyGastroenterologyRNARats Inbred StrainsImmunohistochemistryPrecipitin TestsMolecular biologyIn vitroRatsmedicine.anatomical_structureAdipose TissueBiochemistrychemistryCell cultureHepatic stellate cellRNAImmunohistochemistryElectrophoresis Polyacrylamide GelFemaleCollagenGastroenterology
researchProduct