Search results for "blood-brain barrier"

showing 10 items of 141 documents

Unraveling In vivo brain transport of protein‐coated fluorescent nanodiamonds

2019

The blood–brain barrier is the biggest hurdle to overcome for the treatment of neurological disorders. Here, protein‐coated nanodiamonds are delivered to the brain and taken up by neurovascular unit cells after intravenous injection. Thus, for the first time, nanodiamonds with their unique properties and a flexible protein coating for the attachment of therapeutics emerge as a potential platform for nanotheranostics of neurological disorders.Nanotheranostics, combining diagnostics and therapy, has the potential to revolutionize treatment of neurological disorders. But one of the major obstacles for treating central nervous system diseases is the blood–brain barrier (BBB) preventing systemic…

Cell SurvivalCentral nervous systemnanotheranosticsTunneling (Physics)Serum Albumin Human02 engineering and technology010402 general chemistryBlood–brain barrier01 natural sciencesFluorescencePolyethylene GlycolsNanodiamondsBiomaterialstunneling nanotubesMiceIn vivoCell MovementmedicineAnimalsBlut-Hirn-SchrankeGeneral Materials Scienceddc:610Blood-brain barrierNeuronsNanotubesChemistryBrainEndothelial CellsBiological TransportGeneral ChemistryHospitals Drug distribution systems021001 nanoscience & nanotechnologyHuman serum albuminPhotobleachingIn vitroEndocytosis0104 chemical sciencesmedicine.anatomical_structureTranscytosisBlood-Brain BarrierNanoröhreAstrocytesDrug deliverydrug deliveryBiophysics0210 nano-technologyDDC 610 / Medicine & healthBiotechnologymedicine.drug
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Studying the Neurovascular Unit: An Improved Blood–Brain Barrier Model

2009

The blood–brain barrier (BBB) closely interacts with the neuronal parenchyma in vivo. To replicate this interdependence in vitro, we established a murine coculture model composed of brain endothelial cell (BEC) monolayers with cortical organotypic slice cultures. The morphology of cell types, expression of tight junctions, formation of reactive oxygen species, caspase-3 activity in BECs, and alterations of electrical resistance under physiologic and pathophysiological conditions were investigated. This new BBB model allows the application of techniques such as laser scanning confocal microscopy, immunohistochemistry, fluorescent live cell imaging, and electrical cell substrate impedance se…

Cell typeBlood–brain barrierCell LineTight JunctionsBrain ischemiaMiceIn vivoLive cell imagingParenchymaElectric ImpedancemedicineAnimalsTight junctionCaspase 3ChemistryBrainEndothelial CellsMembrane Proteinsmedicine.diseaseImmunohistochemistryCoculture TechniquesEndothelial stem cellmedicine.anatomical_structureNeurologyBlood-Brain BarrierBiophysicsNeurology (clinical)Reactive Oxygen SpeciesCardiology and Cardiovascular MedicineNeuroscienceJournal of Cerebral Blood Flow & Metabolism
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Possible Pathomechanisms Responsible for Injury to the Central Nervous System in the Settings of Chronic Cerebrospinal Venous Insufficiency

2012

The discovery of stenoses in the azygous and internal jugular veins, the so-called chronic cerebrospinal venous insufficiency that accompanies multiple sclerosis, has enabled the reinterpretation of knowledge about this neurologic dis- ease. Pathologic venous outflow from the central nervous system appears to lead to two main problems. Firstly, it disas- sembles the blood-brain barrier and may allow the penetration of nervous parenchyma by glutamate and leukocytes. Sec- ondly, it may result in significant hypoperfusion of the brain and spinal cord. These two overlapping pathologies are likely to trigger plaques through caspase-1-driven pyroptosis of oligodendrocytes and to evoke neurodegene…

Central Nervous SystemPathologymedicine.medical_specialtyCentral nervous systemExcitotoxicityglutamatemultiple sclerosismedicine.disease_causeAxonal injuryCentral Nervous System Diseasescaspase 1venous insufficiencymedicineHumansBrachiocephalic Veinsjugular veinsPharmacologybusiness.industryMultiple sclerosisazygous veinNeurodegenerationPyroptosisGlutamate receptorGeneral Medicineblood-brain barriermedicine.diseaseSpinal cordChronic cerebrospinal venous insufficiencymedicine.anatomical_structureSpinal CordbusinessReviews on Recent Clinical Trials
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Neuronal Activity Drives Localized Blood-Brain-Barrier Transport of Serum Insulin-like Growth Factor-I into the CNS

2010

Upon entry into the central nervous system (CNS), serum insulin-like growth factor-1 (IGF-I) modulates neuronal growth, survival, and excitability. Yet mechanisms that trigger IGF-I entry across the blood-brain barrier remain unclear. We show that neuronal activity elicited by electrical, sensory, or behavioral stimulation increases IGF-I input in activated regions. Entrance of serum IGF-I is triggered by diffusible messengers (i.e., ATP, arachidonic acid derivatives) released during neurovascular coupling. These messengers stimulate matrix metalloproteinase-9, leading to cleavage of the IGF binding protein-3 (IGFBP-3). Cleavage of IGFBP-3 allows the passage of serum IGF-I into the CNS thro…

Central Nervous SystemTime FactorsMicrodialysismedicine.medical_treatmentAction PotentialsStimulationFunctional LateralityBody TemperatureReceptor IGF Type 1chemistry.chemical_compoundNeural PathwaysPremovement neuronal activityDrug InteractionsInsulin-Like Growth Factor IMicroscopy ImmunoelectronReceptorCells CulturedNeuronsGeneral NeuroscienceSysneuro//purl.org/becyt/ford/3.1 [https]Protein TransportMedicina Básicamedicine.anatomical_structureMatrix Metalloproteinase 9Blood-Brain BarrierSIGNALING//purl.org/becyt/ford/3 [https]Arachidonic acidNeurogliaLow Density Lipoprotein Receptor-Related Protein-1CIENCIAS MÉDICAS Y DE LA SALUDNeuroscience(all)Central nervous systemNeurocienciasBiophysicsGlutamic AcidEnzyme-Linked Immunosorbent AssayNerve Tissue ProteinsBiologyBlood–brain barrierMOLNEUROmedicineAnimalsHumansImmunoprecipitationRats WistarAnalysis of VarianceGrowth factorEndothelial CellsTransporterCoculture TechniquesElectric StimulationSignalingRatsMolneurochemistryRegional Blood FlowVibrissaeSYSNEURODigoxigeninExcitatory Amino Acid AntagonistsNeuroscience
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Peritumoral Edema in Meningiomas

1994

Although generally benign tumors, meningiomas may be associated with extensive peritumoral brain edema as seen on computed tomographic scans. Fifty-two patients with intracranial meningiomas were studied, and the hypodense areas on computed tomographic scans were related to the intraoperative microsurgical findings and to the sizes of the tumors. We have identified three kinds of tumor-brain interfaces characterized by different difficulties in microsurgical dissection: smooth type, transitional type, and invasive type. These different microsurgical interfaces seem to correlate very precisely with computed tomographic images of halo-like and finger-like hypodense areas, allowing prediction …

Cerebral CortexMaleMicrosurgerySettore MED/27 - NeurochirurgiaIntracranial meningiomas brain tumo interfacesBrain EdemaBlood-Brain BarrierBrain edema Computed tomography Meningiomas MicrosurgeryMeningeal NeoplasmsHumansFemaleSurgeryNeurology (clinical)MeningiomaTomography X-Ray ComputedNeurosurgery
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The action of TH17 cells on blood brain barrier in multiple sclerosis and experimental autoimmune encephalomyelitis.

2019

Th17 cells, known as a highly pro-inflammatory subtype of Th cells, are involved very early in numerous aspects of multiple sclerosis (MS) and experimental autoimmune encephalomyelitis (EAE) neuropathology. A crucial event for the formation and accumulation of MS lesions is represented by the disruption of the blood brain barrier (BBB) in relapsing-remitting MS. Th17 cells also contribute to the progression of MS/EAE. These events will allow for the passage of inflammatory cells into the brain. Secondary to this, increased recruitment of neutrophils occurs, followed by increased protease activity that will continue to attract macrophages and monocytes, leading to brain inflammation with sus…

ChemokineEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisImmunologyInflammationBlood–brain barrierTight JunctionsMyelinCell MovementmedicineImmunology and AllergyAnimalsHumansAxonbiologybusiness.industryMultiple sclerosisNeurodegenerationExperimental autoimmune encephalomyelitisEndothelial CellsGeneral MedicineTh1 Cellsmedicine.diseaseCell biologymedicine.anatomical_structureBlood-Brain Barrierbiology.proteinCytokinesTh17 Cellsmedicine.symptombusinessHuman immunology
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Permeability and toxicological profile estimation of organochlorine compounds by biopartitioning micellar chromatography

2008

This paper points out the usefulness of biopartitioning micellar chromatography (BMC) as a high-throughput primary screening tool providing key information about the oral absorption, skin permeability (Kp), brain–blood distribution coefficient (BB) and ecotoxicological parameters such as median lethal concentration (LC50) and bioconcentration factors of 15 organochloride compounds. The retention data of compounds in BMC conditions were interpolated in previously developed quantitative–retention activity relationships by our research group. Results show that the compounds studied readily cross the intestinal barrier (oral absorption >ercnt;) and the blood–brain barrier (log BB >p;0.4). In ad…

Clinical BiochemistryBioconcentrationAbsorption (skin)Models BiologicalBiochemistryPermeabilityAnalytical Chemistrychemistry.chemical_compoundDrug DiscoveryHydrocarbons ChlorinatedAnimalsHumansDicofolPesticidesMolecular BiologySkinPharmacologyChromatographyChromatographyGeneral MedicineHexachlorobenzeneOrganochlorideBioavailabilityPartition coefficientchemistryBlood-Brain BarrierChlorobenzeneEnvironmental chemistrySoftwareBiomedical Chromatography
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Functional feature of a novel model of blood brain barrier: Studies on permeation of test compounds

2001

Drug delivery to the central nervous system (CNS) is subject to the permeability limitations imposed by the blood-brain barrier (BBB). Several systems in vitro have been described to reproduce the physical and biochemical behavior of intact BBB, most of which lack the feature of the in vivo barrier. We developed a fully formed monolayer of RBE4.B immortalized rat brain microvessel endothelial cells (ECs), grown on top of polycarbonate filter inserts with cortical neuronal cells grown on the outside. Neurons induce ECs to synthesize and sort occludin to the cell periphery. Occludin localization is regulated by both compositions of the substratum and soluble signals released by cortical co-cu…

DopamineL-DOPAPharmaceutical ScienceBrain capillaries endothelial cells (ECs)OccludinBlood–brain barrierDopamine agonistPermeabilityLevodopaRats Sprague-DawleyDopamineIn vivoSettore BIO/10 - BiochimicamedicineAnimalsCells CulturedChemistryTryptophanPermeationRatsEndothelial stem cellBlood-brain barrier (BBB)medicine.anatomical_structureBiochemistryPermeability (electromagnetism)Blood-Brain BarrierSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoBiophysicsSettore MED/26 - Neurologiamedicine.drugL-Tryptophan
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Studies on a new potential dopaminergic agent: in vitro BBB permeability, in vivo behavioural effects and molecular docking evaluation.

2015

2-Amino-N-[2-(3,4-dihydroxy-phenyl)-ethyl]-3-phenyl-propionamide (DA-PHEN) has been previously synthesized to obtain a potential prodrug capable of release dopamine (DA) into CNS. However, DA-PHEN could act per se as a dopaminergic drug. In this study, the permeability transport (Pe), obtained by parallel artificial permeability assay (PAMPA), indicated a low passive transcellular transport (Pe = 0.32 ± 0.01 × 10(-6 )cm/s). Using the Caco-2 cell system, the Papp AP-BL in absorptive direction (3.36 ± 0.02 × 10(-5 )cm/s) was significantly higher than the Papp BL-AP in secretive direction (1.75 ± 0.07 × 10(-5 )cm/s), suggesting a polarized transport. The efflux ratio (Papp AP-BL/Papp BL-AP = 0…

DopaminePhenylalanineDopamine AgentsPharmaceutical ScienceMorris water navigation taskPharmacologyBiologyCognitive flexibilityPermeabilityIn vivoDopamineSettore BIO/10 - BiochimicaPAMPA-BBBmedicineHumansIn vivo behavioural effectDopaminergicProdrugSettore CHIM/08 - Chimica FarmaceuticaMolecular Docking SimulationSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoBlood-Brain BarrierParacellular transportMolecular docking D1-receptorSettore BIO/14 - FarmacologiaEffluxCaco-2 bidirectional assayCaco-2 CellsTranscytosisBehavioural despair testmedicine.drugJournal of drug targeting
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Amphiphilic HPMA-LMA copolymers increase the transport of Rhodamine 123 across a BBB model without harming its barrier integrity.

2012

Abstract The successful non-invasive treatment of diseases associated with the central nervous system (CNS) is generally limited by poor brain permeability of various developed drugs. The blood–brain barrier (BBB) prevents the passage of therapeutics to their site of action. Polymeric drug delivery systems are promising solutions to effectively transport drugs into the brain. We recently showed that amphiphilic random copolymers based on the hydrophilic p(N-(2-hydroxypropyl)-methacrylamide), pHPMA, possessing randomly distributed hydrophobic p(laurylmethacrylate), pLMA, are able to mediate delivery of domperidone into the brain of mice in vivo. To gain further insight into structure–propert…

Drug CarriersPharmaceutical SciencePolymer architectureBiological TransportPharmacologyBlood–brain barrierRhodamine 123Models BiologicalPermeabilityCell Linechemistry.chemical_compoundmedicine.anatomical_structurechemistryTranscytosisIn vivoBlood-Brain BarrierNanoparticles for drug delivery to the brainAmphiphilemedicineHumansMethacrylatesRhodamine 123Barrier functionFluorescent DyesJournal of controlled release : official journal of the Controlled Release Society
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