Search results for "cancer therapy"

showing 10 items of 101 documents

Small molecule DNA-PK inhibitors as potential cancer therapy: a patent review (2010–present)

2021

Introduction: DNA-dependent protein kinase (DNA-PK) plays a crucial role in the repair of DSBs via non-homologous end joining (NHEJ). Several DNA-PK inhibitors are being investigated for potential anticancer treatment in clinical trials.Area covered: This review aims to give an overview of patents published since 2010 by analyzing the patent space and structure features of scaffolds used in those patents. It also discusses the recent clinical developments and provides perspectives on future challenges and directions in this field.Expert opinion: As a key component of the DNA damage response (DDR) pathway, DNA-PK appears to be a viable drug target for anticancer therapy. The clinical investi…

DNA damageCancer therapyDNA-Activated Protein Kinase01 natural sciencesPatents as Topic03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDrug DevelopmentNeoplasmsAntineoplastic Combined Chemotherapy ProtocolsDrug DiscoveryCombination strategyAnimalsHumansMedicineProtein kinase AProtein Kinase InhibitorsPharmacologybusiness.industryGeneral MedicineSmall molecule0104 chemical sciences010404 medicinal & biomolecular chemistrychemistryAnticancer treatment030220 oncology & carcinogenesisPARP inhibitorCancer researchbusinessDNADNA DamageExpert Opinion on Therapeutic Patents
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Principal component analysis on molecular descriptors as an alternative point of view in the search of new Hsp90 inhibitors

2009

Inhibiting a protein that regulates multiple signal transduction pathways in cancer cells is an attractive goal for cancer therapy. Heat shock protein 90 (Hsp90) is one of the most promising molecular targets for such an approach. In fact, Hsp90 is a ubiquitous molecular chaperone protein that is involved in folding, activating and assembling of many key mediators of signal transduction, cellular growth, differentiation, stress-response and apoptothic pathways. With the aim to analyze which molecular descriptors have the higher importance in the binding interactions of these classes, we first performed molecular docking experiments on the 187 Hsp90 inhibitors included in the BindingDB, a pu…

Databases FactualProtein ConformationDrug Evaluation PreclinicalCancer therapyPrincipal component analysiNaphtholsBiochemistryBinding databaseMolecular descriptorsStructure-Activity RelationshipStructural BiologyMolecular descriptorHeat shock proteinComputer SimulationHSP90 Heat-Shock ProteinsPrincipal Component AnalysisBinding SitesbiologyHeat shock proteinOrganic ChemistryComputational BiologyIsoxazolesHsp90Settore CHIM/08 - Chimica FarmaceuticaComputational MathematicsBiochemistryPurinesDocking (molecular)Principal component analysisMolecular dockingbiology.proteinPyrazolesBindingDBSignal transduction
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Hypoxia: Importance in tumor biology, noninvasive measurement by imaging, and value of its measurement in the management of cancer therapy

2006

The Cancer Imaging Program of the National Cancer Institute convened a workshop to assess the current status of hypoxia imaging, to assess what is known about the biology of hypoxia as it relates to cancer and cancer therapy, and to define clinical scenarios in which in vivo hypoxia imaging could prove valuable.Hypoxia, or low oxygenation, has emerged as an important factor in tumor biology and response to cancer treatment. It has been correlated with angiogenesis, tumor aggressiveness, local recurrence, and metastasis, and it appears to be a prognostic factor for several cancers, including those of the cervix, head and neck, prostate, pancreas, and brain. The relationship between tumor oxy…

Diagnostic ImagingOncologymedicine.medical_specialtyCancer therapyBiologyAntigens NeoplasmNeoplasmsInternal medicineAntineoplastic Combined Chemotherapy ProtocolsBiomarkers TumormedicineHumansRadiology Nuclear Medicine and imagingMedical physicsCarbonic Anhydrase IXHypoxiaCarbonic AnhydrasesCancer Imaging ProgramRadiological and Ultrasound TechnologyTumor biologyReproducibility of ResultsHypoxia (medical)PrognosisUnited StatesCancer treatmentIsoenzymesOxygenRadiographyNational Institutes of Health (U.S.)Hypoxia-Inducible Factor 1medicine.symptomInternational Journal of Radiation Biology
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Gefitinib in lung cancer therapy. Clinical results, predictive markers of response and future perspectives.

2009

Over the past few years, epidermal growth factor receptor has emerged as one of the most important targets in tumorgenesis and several drugs targeting signal transduction pathways have been developed. The first among these agents to be approved for the treatment of NSCLC was gefitinib, a potent, selective and reversible inhibitor of HER1/EGFR tyrosine kinase activity. The review summarizes its clinical development and the new therapeutic options, with particular focus on predictive markers of susceptibility to this drug.

DrugOncologyCancer Researchmedicine.medical_specialtyLung Neoplasmsmolecular markersmedia_common.quotation_subjectgefitinibAntineoplastic AgentsGefitinibcancer therapyGefitinibCarcinoma Non-Small-Cell LungInternal medicinetyrosine kinase inhibitorsmedicineAnimalsHumansgefitinib; non-small cell lung cancer (NSCLC); epidermal growth factor receptor (HER1/EGFR); tyrosine kinase inhibitors; target therapy; molecular markers; EGFR mutationsEpidermal growth factor receptorLung cancermedia_commonPharmacologyClinical Trials as Topicbiologybusiness.industrytarget therapymedicine.diseaseEGFR mutationsepidermal growth factor receptor (HER1/EGFR)ErbB Receptorsnon-small cell lung cancer (NSCLC)OncologyQuinazolinesbiology.proteinMolecular MedicineSignal transductionbusinessBiomarkersEgfr tyrosine kinaseSignal Transductionmedicine.drug
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Identification of chemosensitizers by drug repurposing to enhance the efficacy of cancer therapy

2020

Abstract The progressively rising drug resistance has driven the development of chemosensitizers, which aim to enhance the current chemotherapeutic efficacy through either quantitatively increasing intracellular drug concentration by inhibiting drug efflux, vesicle sequestration, and metabolic inactivation or qualitatively exerting concomitant effects on cellular regulation to promote apoptosis. Given the high failure rates and costs during standard drug development, drug repurposing represents an effective and economic approach for chemosensitizer identification. By applying drug repurposing, a wide range of existing clinic drugs including anticardiovascular agents, immunosuppressants, ant…

Drugbusiness.industrymedia_common.quotation_subjectCellular RegulationCancer therapyChemosensitizerDrug resistancePharmacologyDrug repositioningDrug concentrationDrug developmentMedicinebusinessmedia_common
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Relevance of Multidrug Resistance Proteins on the Clinical Efficacy of Cancer Therapy

2005

Variations in drug uptake and efflux, as well as changes in intracellular drug entrapment and distribution may represent important resistance mechanisms to cancer therapy. A variety of ATP binding cassette transporters (ABC) localised in multiple cell membranes is implied in those phenomena, representing a mechanism of protection of cells against xenobiotics. Many cancer cell lines over express some ABC transporters, especially p-glycoprotein, MRP1 and BCRP. This over expression is related to worse cancer treatment outcome and, in some cases, reduced overall survival of cancer patients. This paper reviews the location and physiological role of the three transporters mentioned and also descr…

Drugmedia_common.quotation_subjectCellCancer therapyPharmaceutical ScienceAntineoplastic AgentsATP-binding cassette transporterTransporterPharmacologyBiologyTreatment Outcomemedicine.anatomical_structureDrug Resistance NeoplasmNeoplasmsmedicineAnimalsHumansEffluxMultidrug Resistance-Associated ProteinsIntracellularMultidrug Resistance-Associated Proteinsmedia_commonCurrent Drug Delivery
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Management of chemotherapy extravasation: ESMO–EONS Clinical Practice Guidelines

2012

Extravasation is the process by which any liquid (fluid or drug) accidentally leaks into the surrounding tissue. In terms of cancer therapy, extravasation refers to the inadvertent infiltration of chemotherapy into the subcutaneous or subdermal tissues surrounding the intravenous or intra-arterial administration site. Extravasated drugs are classified according to their potential for causing damage as ‘vesicant’, ‘irritant’ and ‘nonvesicant’ (Table 1). Some vesicant drugs are further classified into two groups: DNA binding and non-DNA binding. Allwood et al. (2002) divided the drugs into vesicants, exfoliants, irritants, inflammitants and neutrals.

Drugmedicine.medical_specialtymedicine.medical_treatmentmedia_common.quotation_subjectAntidotesMEDLINECancer therapyAntineoplastic AgentsDiagnosis DifferentialRisk FactorsNeoplasmsHumansMedicineIntensive care medicinemedia_commonChemotherapyOncology (nursing)business.industryGeneral MedicineHematologymedicine.diseaseChemotherapy regimenExtravasationSurgeryClinical PracticeOncologybusinessInfiltration (medical)Extravasation of Diagnostic and Therapeutic MaterialsAnnals of Oncology
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Targeting epigenetics in cancer: therapeutic potential of flavonoids

2021

Irrespective of sex and age, cancer is the leading cause of mortality around the globe. Therapeutic incompliance, unwanted effects, and economic burdens imparted by cancer treatments, are primary health challenges. The heritable features in gene expression that are propagated through cell division and contribute to cellular identity without a change in DNA sequence are considered epigenetic characteristics and agents that could interfere with these features and are regarded as potential therapeutic targets. The genetic modification accounts for the recurrence and uncontrolled changes in the physiology of cancer cells. This review focuses on plant-derived flavonoids as a therapeutic tool for…

FlavonolsAnticancer; cancer therapy; epigenetic; flavonoids; Diet; Epigenesis Genetic; Flavonols; Humans; Flavonoids; NeoplasmsBioinformaticsFlavonesIndustrial and Manufacturing EngineeringEpigenesis Geneticchemistry.chemical_compoundFlavonolsGeneticNeoplasmsmedicineHumansEpigeneticsFlavonoidschemistry.chemical_classificationbusiness.industryfood and beveragesCancerGeneral MedicineIsoflavonesmedicine.diseaseDietClinical trialAnticancerchemistryCancer cellcancer therapyDelphinidinbusinessAnticancer; cancer therapy; epigenetic; flavonoidsepigeneticEpigenesisFood ScienceCritical Reviews in Food Science and Nutrition
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Highly Homogeneous Biotinylated Carbon Nanodots: Red-Emitting Nanoheaters as Theranostic Agents toward Precision Cancer Medicine

2019

Very recent red-emissive carbon nanodots (CDs) have shown potential as near-infrared converting tools to produce local heat useful in cancer theranostics. Besides, CDs seem very appealing for clinical applications combining hyperthermia, imaging, and drug delivery in a single platform capable of selectively targeting cancer cells. However, CDs still suffer from dramatic dot-to-dot variability issues such that a rational design of their structural, optical, and chemical characteristics for medical applications has been impossible so far. Herein, we report for the first time a simple and highly controllable layer-by-layer synthesis of biotin-decorated CDs with monodisperse size distribution, …

Fluorescence-lifetime imaging microscopyphotothermal therapyMaterials scienceCell SurvivalAntineoplastic AgentsNanotechnology02 engineering and technology010402 general chemistrytargeted cancer therapy01 natural sciencesDrug Delivery Systemsbiotincarbon nanodotCell Line TumorCarbon nanodotsHumansGeneral Materials SciencePrecision MedicineRational designimagingPhotothermal therapy021001 nanoscience & nanotechnologyCarbonNanostructures0104 chemical sciencesbiotin; carbon nanodots; imaging; photothermal therapy; targeted cancer therapy.Settore CHIM/09 - Farmaceutico Tecnologico ApplicativoBiotinylationDrug deliveryCancer cellMCF-7 CellsSurface modification0210 nano-technologyACS Applied Materials & Interfaces
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NUTRITION AND CANCER

1969

The last decades have seen the identification of a number of drugs targeting oncogene pathways, therefore contributing to moving the cancer therapy field toward precision medicine. However, existing and acquired resistance to targeted therapies represent major obstacles against their long-term effectiveness. In fact, the initial efficacy of targeted therapies is often limited to a portion of the patient population and is frequently followed by the acquisition of drug-resistant disease.

Gerontologymedicine.medical_specialtyLung Neoplasmsbusiness.industryCancer therapyCancerGeneral MedicineDiseasePrecision medicinemedicine.diseasePatient populationAcquired resistanceNeoplasmsCarcinoma Squamous CellmedicineNutritional Physiological PhenomenaIntensive care medicinebusinessDiet TherapyThe Lancet
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