Search results for "catechol"
showing 10 items of 230 documents
CCDC 257715: Experimental Crystal Structure Determination
2005
Related Article: S.Floquet, A.J.Simaan, E.Riviere, M.Nierlich, P.Thuery, J.Ensling, P.Gutlich, J.-J.Girerd, M.-L.Boillot|2005|Dalton Trans.||1734|doi:10.1039/b418294d
CCDC 257716: Experimental Crystal Structure Determination
2005
Related Article: S.Floquet, A.J.Simaan, E.Riviere, M.Nierlich, P.Thuery, J.Ensling, P.Gutlich, J.-J.Girerd, M.-L.Boillot|2005|Dalton Trans.||1734|doi:10.1039/b418294d
CCDC 257720: Experimental Crystal Structure Determination
2005
Related Article: S.Floquet, A.J.Simaan, E.Riviere, M.Nierlich, P.Thuery, J.Ensling, P.Gutlich, J.-J.Girerd, M.-L.Boillot|2005|Dalton Trans.||1734|doi:10.1039/b418294d
CCDC 620598: Experimental Crystal Structure Determination
2007
Related Article: M.Albrecht, M.Fiege, M.Baumert, M.de Groot, R.Frohlich, L.Russo, K.Rissanen|2007|Eur.J.Inorg.Chem.||609|doi:10.1002/ejic.200600852
Catecholamine Response Curves of Male Hypertensives Identified by Lehmacher's Two Sample Configural Frequency Analysis
1997
A new application of LEHMACHER'S (1980) marginal homogeneity sign tests is given by analysis of bivariate response curves (or response surfaces) in two unpaired samples of hypertensive versus normotensive patients. Rationale and computations are illustrated by empirical data from sympathomedullary stress research.
Hydroxylation and conjugation of phenol by the frog Rana temporaria.
1985
1. Frogs injected with phenol excrete 67–95% of dose in 15h; 32–87% of dose are metabolites.2. Metabolites identified were phenyl sulphate (15–44% of dose), phenyl glucuronide (10–25% of dose), catechol sulphate (up to 7% of dose), quinol sulphate (1–25% of dose), resorcinol and catechol (traces).
Catecholato complexes of o-phenylenebis(salicylideneiminato)iron(III) and meso-tetra(parasulphonatephenyl)porphyrinatoferrate(III). A comment on the …
1986
Abstract Formation of catecholato complexes of Fe(saloph) + and Fe(TPPS) 3− in solution is studied. Fe(saloph)(cat) − contains a cat 2− bidentate ligand. Its formation in solution competes efficiently with the hydrolysis and dimerization of Fe(saloph) + to give Fe 2 (saloph) 2 O. This behaviour shows that the planar saloph 2− ligand, as the analogous salen 2− , is easily distorted, and is not as rigid as generally considered. Iron(III) porphyrin Fe(TPPS) 3− with catechol gives the complex [Fe(TPPS)(Hcat)] 4− . Deprotonation of the unidentate Hcat − ligand cannot be studied because the smaller stability of the complex, and the dimerization of the metalloporphyrin dominates in basic medium. T…
Mechanism of Sinoatrial Node Dysfunction in a RyR 2 R420Q Mouse Model Ofcatecholaminergic Polymorphic Ventricular Tachycardia
2017
Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a genetic disease characterized by stress-induced syncope and/or sudden death in young individuals with structurally normal heart. More than 150 mutations located in the cardiac Ca2+ release channel (type-2 ryanodine receptor, RyR2) gene are related to CPVT. Besides ventricular tachycardia (VT) under stress, sinoatrial node (SAN) dysfunction is frequently observed in CPVT patients. However, the cellular mechanisms remain underexplored. We created a KI mice model bearing a mutation in the N-terminal portion of the RyR2 found in a CPVT family, RyR2(R420Q). ECGs were recorded in KI and WT littermates in resting condition and after…
Vulnerability of peripheral catecholaminergic neurons to MPTP is not regulated by alpha-synuclein.
2010
Although generally considered a prototypical movement disorder, Parkinson's disease is commonly associated with a broad-spectrum of non-motor symptoms, including autonomic dysfunctions caused by significant alterations in catecholaminergic neurons of the peripheral sympathetic nervous system. Here we present evidence that alpha-synuclein is highly expressed by sympathetic ganglion neurons throughout embryonic and postnatal life and that it is found in tyrosine hydroxylase-positive sympathetic fibers innervating the heart of adult mice. However, mice deficient in alpha-synuclein do not exhibit any apparent alterations in sympathetic development. Sympathetic neurons isolated from mouse embryo…
RELEASE BY SYMPATHETIC STIMULATION OF ALPHA-METHYLNORADRENALINE STORED IN THE HEART AFTER ADMINISTRATION OF ALPHA-METHYLDOPA.
1963
Dans les cœurs de lapins traites par l'α-methyldopa, la depletion de la noradrenaline s'accompagne d'une fixation importante d'α-methylnoradrenaline. Sous l'effet de stimulation sympathique ou d'iodure de dimethylphenyl-piperazinium, ces cœurs liberent conjointement de la noradrenaline et de l'α-methylnoradrenaline.