Search results for "celiac"

showing 10 items of 255 documents

Quality of Life in People with Coeliac Disease: Psychological and Socio- Economic Aspects.

2019

Background and Objective: Coeliac disease (CD) is a chronic autoimmune intestinal disorder characterized by intolerance to gluten, a protein contained in certain cereals. The main physiopathological basis of CD is the progressive destruction of intestinal villi caused by gluten ingestion by genetically-susceptible individuals. Patients who receive a diagnosis of CD must make significant changes to their daily habits and this can affect their quality of life. The objective of this review is to summarize the evidence regarding the economic, physical and social limitations which can affect the quality of life in patients with CD. Results: Different factors such as physical changes, psychologi…

GerontologyMaleEndocrinology Diabetes and MetabolismAffect (psychology)Coeliac disease03 medical and health sciencesInterpersonal relationshipDiet Gluten-Free0302 clinical medicineQuality of lifeCost of IllnessmedicineImmunology and AllergyHumans030212 general & internal medicineDepression (differential diagnoses)business.industrymedicine.diseaseCeliac DiseaseSocioeconomic FactorsQuality of LifeAnxiety030211 gastroenterology & hepatologyHealth educationFemaleIntestinal Disordermedicine.symptombusinessEndocrine, metabolicimmune disorders drug targets
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Treatment of giardiasis reverses "active" coeliac disease to "latent" coeliac disease.

2001

In patients with coeliac disease, a regression of intestinal damage without a gluten-free diet is a very rare event. We describe a young child with diarrhoea, intestinal mucosa atrophy and positive serum anti-endomysial and anti-tissue transglutaminase (anti-tTG) antibodies during intestinal giardiasis infection. He showed normal intestinal mucosa architecture and negative anti-endomysial and anti-tTG antibodies after his giardiasis was cured, although he continued to assume a normal diet. Re-evaluations on a 6-monthly basis showed that he was symptom free, and all haemato-chemical parameters were within normal limits. Three years after the initial diagnosis, a third intestinal biopsy showe…

GiardiasisMalemedicine.medical_specialtyNormal dietTissue transglutaminaseAntiprotozoal AgentsEnzyme-Linked Immunosorbent AssaySensitivity and SpecificityCoeliac diseaseAtrophyIntestinal mucosaImmunopathologyBiopsyMedicineHumansIntestinal MucosaAutoantibodiesTransglutaminasesHepatologybiologymedicine.diagnostic_testbusiness.industryBiopsy NeedleGastroenterologyInfantmedicine.diseaseImmunohistochemistryImmunoglobulin ACeliac DiseaseImmunologybiology.proteinHistopathologybusinessFollow-Up StudiesEuropean journal of gastroenterologyhepatology
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Wheat amylase trypsin inhibitors drive intestinal inflammation via activation of toll-like receptor 4

2012

Ingestion of wheat, barley, or rye triggers small intestinal inflammation in patients with celiac disease. Specifically, the storage proteins of these cereals (gluten) elicit an adaptive Th1-mediated immune response in individuals carrying HLA-DQ2 or HLA-DQ8 as major genetic predisposition. This well-defined role of adaptive immunity contrasts with an ill-defined component of innate immunity in celiac disease. We identify the α-amylase/trypsin inhibitors (ATIs) CM3 and 0.19, pest resistance molecules in wheat, as strong activators of innate immune responses in monocytes, macrophages, and dendritic cells. ATIs engage the TLR4–MD2–CD14 complex and lead to up-regulation of maturation markers a…

GliadinMice0302 clinical medicineHEK293 CellImmunology and AllergyTriticumPlant Proteins2. Zero hungerMice Knockout0303 health sciencesToll-like receptorMice Inbred C3Hfood and beveragesPlant ProteinU937 CellsAcquired immune system3. Good health030211 gastroenterology & hepatologymedicine.symptomTrypsin InhibitorsHumanSignal TransductionImmunologyMolecular Sequence DataInflammationBiologyProinflammatory cytokineCell Line03 medical and health sciencesImmune systemImmunitymedicineAnimalsHumansAmino Acid Sequence030304 developmental biologyInnate immune systemSequence Homology Amino AcidAnimalBIO/13 - BIOLOGIA APPLICATAnutritional and metabolic diseasesHordeumImmunity InnateToll-Like Receptor 4Mice Inbred C57BLCeliac DiseaseHEK293 CellsImmunologyMyeloid Differentiation Factor 88TLR4Trypsin Inhibitor
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Effect of The Gluten-Free Diet on Quality of Life, Gastrointestinal Symptoms and Immune System in Patients with Fibromyalgia and Non-Celiac Wheat Sen…

2021

Fibromyalgia (FM) is a clinical syndrome characterized by chronic pain. FM patients complain hyperalgesia and allodynia and they are frequently affected by Non Celiac Wheat Sensitivity (NCWS), a condition where gastrointestinal and extraintestinal symptoms are triggered by gluten and/or wheat ingestion. The gluten-free diet (GFD) impact was evaluated on fibromyalgia-related and gastrointestinal symptoms, health-related quality of life and immune response of patients with both FM and NCWS in order to detect a possible pathogenetic role of wheat/gluten in the triggering of the inflammatory process. Peripheral blood from 8 FM patients, 10 FM and NCWS patients (FM+NCWS patients), 13 NCWS patien…

Gluten Free DietFibromyalgiaSettore MED/09 - Medicina InternaPro-inflammatory CytokinesAdaptative immunityNon Celiac Wheat Sensitivity
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Co-factors, Microbes, and Immunogenetics in Celiac Disease to Guide Novel Approaches for Diagnosis and Treatment.

2021

Celiac disease (CeD) is a frequent immune-mediated disease that affects not only the small intestine but also many extraintestinal sites. The role of gluten proteins as dietary triggers, HLA-DQ2 or -DQ8 as major necessary genetic predisposition, and tissue transglutaminase (TG2) as mechanistically involved autoantigen, are unique features of CeD. Recent research implicates many cofactors working in synergism with these key triggers, including the intestinal microbiota and their metabolites, nongluten dietary triggers, intestinal barrier defects, novel immune cell phenotypes, and mediators and cytokines. In addition, apart from HLA-DQ2 and -DQ8, multiple and complex predisposing genetic fact…

GlutensTissue transglutaminaseHuman leukocyte antigenDiseaseGut floraImmunologic Testsmedicine.disease_causeBioinformaticsAutoimmunityImmune systemPredictive Value of TestsRisk FactorsGenetic predispositionMedicineAnimalsHumansGenetic Predisposition to DiseaseImmunogenetic PhenomenaIrritable bowel syndromeHepatologybiologyBacteriabusiness.industryfungiGastroenterologynutritional and metabolic diseasesmedicine.diseasebiology.organism_classificationPrognosisGastrointestinal MicrobiomeIntestinesCeliac DiseaseDisease Models AnimalPhenotypeHost-Pathogen Interactionsbiology.proteinbusinessGastroenterology
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Intestinal Microbiota and Celiac Disease: Cause, Consequence or Co-Evolution?

2015

It is widely recognized that the intestinal microbiota plays a role in the initiation and perpetuation of intestinal inflammation in numerous chronic conditions. Most studies report intestinal dysbiosis in celiac disease (CD) patients, untreated and treated with a gluten-free diet (GFD), compared to healthy controls. CD patients with gastrointestinal symptoms are also known to have a different microbiota compared to patients with dermatitis herpetiformis and controls, suggesting that the microbiota is involved in disease manifestation. Furthermore, a dysbiotic microbiota seems to be associated with persistent gastrointestinal symptoms in treated CD patients, suggesting its pathogenic implic…

Glutensmedicine.drug_classAntibioticslcsh:TX341-641ReviewDiseaseBiologyGut floradigestive systemEpigenesis GeneticPathogenesisDiet Gluten-FreeRisk FactorsDermatitis herpetiformismedicineHumansCeliac diseaseEpigeneticsNutrition and DieteticsProbioticsMicrobiotaGastrointestinal Microbiomemedicine.diseasebiology.organism_classificationBiological EvolutionGastrointestinal MicrobiomeIntestinesHost-Pathogen InteractionsImmunologyGluten-free dietDysbiosislcsh:Nutrition. Foods and food supplyDysbiosisFood ScienceNutrients
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Sourdough Fermentation Degrades Wheat Alpha-Amylase/Trypsin Inhibitor (ATI) and Reduces Pro-Inflammatory Activity

2020

The ingestion of gluten-containing foods can cause wheat-related disorders in up to 15% of wheat consuming populations. Besides the role of gluten, &alpha

Health (social science)wheat sensitivity030309 nutrition & dieteticsPlant Sciencelcsh:Chemical technologyHealth Professions (miscellaneous)ACTIVATION0302 clinical medicineGLUTATHIONElcsh:TP1-1185Amylaseinnate immunityfermentation2. Zero hungerchemistry.chemical_classificationAMYLASE-TRYPSIN-INHIBITORS0303 health sciencesbiologydigestive oral and skin physiologyC100LACTOBACILLIfood and beveragesC500C700TrypsinBiochemistry030211 gastroenterology & hepatology3143 NutritionAlpha-amylaseCELIAC GLUTEN SENSITIVITYmedicine.drugProteasesINTESTINAL INFLAMMATIONPROTEINSTrypsin inhibitordigestive systemMicrobiologyArticle03 medical and health sciencesmedicineYEASTnutritional and metabolic diseasesGlutendigestive system diseasesYeastlactic acid bacteriaDOUGH416 Food Sciencechemistrybioactivitybiology.proteinFermentationPROLAMIN HYDROLYSISFood ScienceFoods
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Hyaluronic acid based hydrogels for antibiotics and enzymes release: from academic research to industry

2016

Conclusion: HA-g-PDLLA hydrogel is suitable to coat a prosthesis remaining adherent to it and distributing itself in the bone cavity. The hydrogel has been proven effective in a rabbit model of highly contaminated implant. In vivo studies have demonstrated how the intraoperative coating of implants with DAC is safe and feasible. HA-EDA-MA hydrogel is able to protect PEP from alteration during storage and it seems a good candidate for a potential once-daily oral formulation in the treatment of celiac disease.

Hyaluronic acid drug delivery celiac diseaseSettore CHIM/09 - Farmaceutico Tecnologico Applicativo
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Correlation between IgA tissue transglutaminase antibody ratio and histological finding in celiac disease.

2011

OBJECTIVES: Positivity of both immunoglobulin A anti-tissue transglutaminase (TTG) and anti-endomysium antibodies (EMA) has a positive predictive value of nearly 100% for celiac disease (CD). The objective of the present study was to evaluate whether patients of any age, with high pretest probability of CD and high titre of anti-TTG and EMA positivity, have a high probability of intestinal damage and may not require the biopsy for final diagnosis. METHODS: A retrospective analysis of 412 consecutively referred patients, age range 10 months to 72 years, who underwent small-bowel biopsy for suspicion of CD and positivity to both anti-TTG and EMA, was performed at 4 Italian centers. Biopsies w…

Immunoglobulin AAdultMalemedicine.medical_specialtyPathologyAdolescentTissue transglutaminaseDuodenumBiopsySettore MED/08 - Anatomia PatologicaGastroenterologySeverity of Illness IndexCoeliac diseaseStatistics NonparametricYoung AdultCoeliac disease Transglutaminase antibodyGTP-Binding ProteinsPredictive Value of TestsInternal medicineSeverity of illnessBiopsymedicineHumansProtein Glutamine gamma Glutamyltransferase 2ChildAgedRetrospective StudiesSettore MED/12 - GastroenterologiaTransglutaminasesbiologymedicine.diagnostic_testbusiness.industryMusclesGastroenterologyInfantRetrospective cohort studyMiddle Agedmedicine.diseaseImmunoglobulin APre- and post-test probabilityCeliac DiseaseLogistic ModelsPredictive value of testsChild PreschoolPediatrics Perinatology and Child Healthbiology.proteinFemalebusinessJournal of pediatric gastroenterology and nutrition
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Orally based diagnosis of celiac disease:current perspective

2008

Celiac disease (CD) is a lifelong immune-mediated disorder caused by the ingestion of wheat gluten in genetically susceptible persons. Most cases of CD are atypical and remain undiagnosed, which exposes the individuals to the risk of life-threatening complications. Serologic endomysial and tissue transglutaminase antibody tests are used to screen at-risk individuals, although a firm diagnosis requires demonstration of characteristic histopathologic findings in the small-intestinal mucosa. A gluten challenge, with a repeat biopsy to demonstrate recurrence of histopathologic changes in the intestinal mucosa after the re-introduction of gluten, is considered for those persons in whom diagnosi…

Immunoglobulin Aceliac disease diagnosis oral mucosa saliva screeningmedicine.medical_specialtySalivaGlutensDiseaseGastroenterologyAntibodiesSerologyIntestinal mucosaInternal medicinemedicineHumansMass ScreeningOral mucosaSalivaGeneral Dentistrychemistry.chemical_classificationbiologybusiness.industryMouth MucosaGlutenCeliac Diseasemedicine.anatomical_structurechemistryImmunoglobulin A SecretoryImmunologybiology.proteinAntibodybusiness
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