Search results for "cell migration"

showing 10 items of 128 documents

Extracellular HSP27 mediates angiogenesis through Toll-like receptor 3.

2013

The heat-shock protein 27 (HSP27) is up-regulated in tumor cells and released in their microenvironment. Here, we show that extracellular HSP27 has a proangiogenic effect evidenced on chick chorioallantoic membrane. To explore this effect, we test the recombinant human protein (rhHSP27) at physiopathological doses (0.1-10 μg/ml) onto human microvascular endothelial cells (HMECs) grown as monolayers or spheroids. When added onto HMECs, rhHSP27 dose-dependently accelerates cell migration (with a peak at 5 μg/ml) and favors spheroid sprouting within 12-24 h. rhHSP27 increases VEGF gene transcription and promotes secretion of VEGF-activating VEGF receptor type 2. Increased VEGF transcription is…

Vascular Endothelial Growth Factor AImmunoprecipitationAngiogenesisHSP27 Heat-Shock ProteinsNeovascularization PhysiologicBiochemistry03 medical and health sciences0302 clinical medicineHsp27GeneticsExtracellularAnimalsSecretionReceptorMolecular BiologyCells Cultured030304 developmental biology0303 health sciencesbiologyNF-kappa BEndothelial CellsCell migrationMolecular biologyToll-Like Receptor 3Chorioallantoic membraneGene Expression Regulation030220 oncology & carcinogenesisbiology.proteinCalciumBiotechnologyFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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Integrin-dependent and -independent functions of astrocytic fibronectin in retinal angiogenesis

2011

Fibronectin (FN) is a major component of the extracellular matrix and functions in cell adhesion, cell spreading and cell migration. In the retina, FN is transiently expressed and assembled on astrocytes (ACs), which guide sprouting tip cells and deposit a provisional matrix for sprouting angiogenesis. The precise function of FN in retinal angiogenesis is largely unknown. Using genetic tools, we show that astrocytes are the major source of cellular FN during angiogenesis in the mouse retina. Deletion of astrocytic FN reduces radial endothelial migration during vascular plexus formation in a gene dose-dependent manner. This effect correlates with reduced VEGF receptor 2 and PI3K/AKT signalli…

Vascular Endothelial Growth Factor AIntegrinsAngiogenesisIntegrinNeovascularization PhysiologicMice TransgenicExtracellular matrixMicePhosphatidylinositol 3-KinasesCell MovementAnimalsProtein Interaction Domains and MotifsMolecular BiologyResearch ArticlesMice KnockoutSprouting angiogenesisbiologyRetinal VesselsCell migrationKinase insert domain receptorVascular Endothelial Growth Factor Receptor-2Extracellular MatrixFibronectinsCell biologyMice Inbred C57BLFibronectinVascular endothelial growth factor AAstrocytesbiology.proteinHeparitin SulfateOligopeptidesProto-Oncogene Proteins c-aktIntegrin alpha5beta1Signal TransductionDevelopmental BiologyDevelopment
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Notch and Numb are required for normal migration of peripheral glia in Drosophila

2006

Abstract A prominent feature of glial cells is their ability to migrate along axons to finally wrap and insulate them. In the embryonic Drosophila PNS, most glial cells are born in the CNS and have to migrate to reach their final destinations. To understand how migration of the peripheral glia is regulated, we have conducted a genetic screen looking for mutants that disrupt the normal glial pattern. Here we present an analysis of two of these mutants: Notch and numb. Complete loss of Notch function leads to an increase in the number of glial cells. Embryos hemizygous for the weak NotchB-8X allele display an irregular migration phenotype and mutant glial cells show an increased formation of …

animal structuresMutantNotch signaling pathwayBiologyCell MovementGlial cell migrationAnimalsDrosophila ProteinsMolecular BiologyGeneticsReceptors NotchCell migrationCell BiologyImmunohistochemistryEmbryonic stem cellPhenotypeCell biologyJuvenile Hormonesnervous systemMutagenesisNUMBDrosophilaNeurogliaSignal TransductionGenetic screenDevelopmental BiologyDevelopmental Biology
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Cellul�re Immunreaktionen gegen�ber homologen leberspezifischen Antigenen (HLP) bei chronischen Leberentz�ndungen

1974

Es wird eine Zweiphasentechnik des Leukocyten-Migrations-Inhibitionstestes beschrieben und zum Nachweis einer cellularen Immunreaktion gegenuber Leberantigenen bei chronisch-entzundlichen Lebererkrankungen eingesetzt. Mit dieser Technik wurde bei 85% der Patienten mit hypergammaglobulinamischer Verlaufsform einer chronisch-aktiven Hepatitis und 42% der Patienten mit aktiv fortschreitenden kryptogenen Lebercirrhosen eine Migrationshemmung nachgewiesen. Lebergesunde Kontrollen zeigten keine, Patienten mit dem Zustand nach Hepatitis und chronisch-persistierender Hepatitis selten eine Leukocytenmigrationsinhibition gegenuber leberspezifischen Antigenen. 17% der Patienten mit chronisch-aktiver H…

business.industryCell Migration InhibitionGeneral MedicineMolecular medicineMolecular biologyChronic diseaseAntigenImmunityDrug DiscoveryHomologous chromosomeMolecular MedicineMedicineImmune reactionbusinessGenetics (clinical)Liver immunologyKlinische Wochenschrift
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Bisphosphonates affect migration ability and cell viability of HUVEC, fibroblasts and osteoblasts in vitro

2010

Oral Diseases (2011) 17, 194–199 Objectives:  Bisphosphonate-associated osteonecrosis of the jaw (BP-ONJ) is a side effect in patients being treated with bisphosphonates. The bisphosphonates most often associated with BP-ONJ are the highly potent nitrogen-containing bisphosphonates, e.g. pamidronate or zoledronate. In terms of BP-ONJ aetiology, several theories are being discussed: inhibition of bone remodelling, effect on soft tissues, and antiangiogenic effect of bisphosphonates. The aim of this in vitro study was to investigate the effect of different potent bisphosphonates on osteoblasts, fibroblasts and human umbilicord vein endothelial cells (HUVEC). Materials and methods:  Three nitr…

business.industrymedicine.medical_treatmentOsteoblastCell migrationBisphosphonatemedicine.diseaseIbandronic acidZoledronic acidmedicine.anatomical_structureOtorhinolaryngologyImmunologymedicineCancer researchViability assaybusinessWound healingOsteonecrosis of the jawGeneral Dentistrymedicine.drugOral Diseases
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The Status of EGFR Modulates the Effect of miRNA-200c on ZEB1 Expression and Cell Migration in Glioblastoma Cells

2020

Migration of glioblastoma cells into surrounding tissue is one of the main features that makes this tumor incurable. We evaluated whole-genome miRNA expression profiling associated with different EGFR amplification patterns in 30 cases of primary glioblastoma. From the 64 miRNAs that showed differential expression between tumors with a high level of EGFR amplification and tumors without EGFR amplification, 40% were related with cell migration, being miR-200c the most differentially expressed between these two groups. We investigated the effect of miR-200c on ZEB1 expression and cell migration in an in vitro transfection model with a miR-200c mimic, a miR-200c inhibitor and siRNA targeting E…

cell migrationEGFR AmplificationApoptosisBiologyArticleCatalysismiR-200clcsh:ChemistryInorganic ChemistryDownregulation and upregulationCell MovementmicroRNABiomarkers TumorTumor Cells CulturedmedicineZEB1HumansGene silencingEGFR amplificationPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologySpectroscopyCell ProliferationOrganic ChemistryglioblastomaGene AmplificationZinc Finger E-box-Binding Homeobox 1Cell migrationGeneral MedicineTransfectionPrognosismedicine.diseaseComputer Science ApplicationsErbB ReceptorsGene Expression Regulation NeoplasticMicroRNAslcsh:Biology (General)lcsh:QD1-999Cell cultureMutationCancer researchGlioblastomaInternational Journal of Molecular Sciences
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In vivo imaging and quantification of the continuous keratin filament network turnover

2008

Keratin polypeptides are major components of the epithelial cytoskeleton forming a filamentous 3D-network. Like intermediate filament polypeptides of other cell types, keratins make up a stable, but elastic network that is responsible for mechanical stress resilience. At the same time the keratin network is able to change its shape during development, cell division, metastasis and cell migration.

chemistry.chemical_classificationCell typeKeratin Filamentintegumentary systemCell divisionCell migrationmacromolecular substancesBiologyCell biologychemistryKeratinCytoskeletonIntermediate filamentPreclinical imaging
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Biological Effects of New Hydraulic Materials on Human Periodontal Ligament Stem Cells.

2019

Background: The aim of this study was: to evaluate the biological properties of new hydraulic materials: Bio-C Repair and Bio-C Sealer. Methods: Periodontal ligament stem cells were exposed to several dilutions of Bio-C Repair and Bio-C Sealer. The ion release profile and pH were determined. Metabolic activity, cell migration and cell survival were assessed using the 3-(4, 5-dimethylthiazol-2-yl)-2, 5-diphenyltetrazolium bromide (MTT), wound-healing assays and Annexin assays, respectively. Cells were cultured in direct contact with the surface of each material. These were then analyzed via scanning electron microscopy (SEM) and energy dispersive X-ray (EDX). Statistical differences were ass…

hydraulic cementsSerial dilutionPeriodontal ligament stem cellsScanning electron microscopelcsh:Medicine02 engineering and technologyOdontologiaArticle03 medical and health sciences0302 clinical medicineAnnexinMedicineViability assayCell adhesionCytotoxicityendodontic cementsbusiness.industrylcsh:Rhuman periodontal ligament stem cellsCell migration030206 dentistryGeneral Medicine021001 nanoscience & nanotechnologyMolecular biologybioceramicscytotoxicity0210 nano-technologybusinessJournal of clinical medicine
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Macrophages as an Emerging Source of Wnt Ligands: Relevance in Mucosal Integrity

2019

The Wnt signaling pathway is a conserved pathway involved in important cellular processes such as the control of embryonic development, cellular polarity, cellular migration, and cell proliferation. In addition to playing a central role during embryogenesis, this pathway is also an essential part of adult homeostasis. Indeed, it controls the proliferation of epithelial cells in different organs such as intestine, lung, and kidney, and guarantees the maintenance of the mucosa in physiological conditions. The origin of this molecular pathway is the binding between Wnt ligands (belonging to a family of 19 different homologous secreted glycoproteins) and their specific membrane receptors, from …

lcsh:Immunologic diseases. Allergy0301 basic medicineFrizzledCellular polarityImmunologyReviewmacrophageBiologyLigandsProinflammatory cytokine03 medical and health sciencesParacrine signalling0302 clinical medicineNeoplasmsWnt ligandsmucosal homeostasisHumanscancerImmunology and AllergyAutocrine signallingWnt Signaling PathwayInflammationMucous MembraneInnate immune systemMacrophagesfibrosisWnt signaling pathwayCell migrationImmunity InnateCell biologyWnt Proteins030104 developmental biologyregenerationlcsh:RC581-607Protein Binding030215 immunologyFrontiers in Immunology
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EGF-Induced Acetylation of Heterogeneous Nuclear Ribonucleoproteins Is Dependent on KRAS Mutational Status in Colorectal Cancer Cells.

2015

KRAS mutational status is considered a negative predictive marker of the response to anti-EGFR therapies in colorectal cancer (CRC) patients. However, conflicting data exist regarding the variable response to EGFR-targeted therapy. The effects of oncogenic KRAS on downstream targets were studied in cell lines with different KRAS mutations. Cells harboring a single KRASG13D allele showed the most tumorigenic profile, with constitutive activation of the downstream pathway, rendering them EGF-unresponsive. Conversely, KRASA146T cells showed a full EGF-response in terms of signal transduction pathways, cell proliferation, migration or adhesion. Moreover, the global acetylome of CRC cells was al…

lcsh:MedicineBiologymedicine.disease_causeHeterogeneous-Nuclear RibonucleoproteinsProto-Oncogene Proteins p21(ras)Epidermal growth factorCell Line TumormedicineHumansCell adhesionlcsh:ScienceMutationMultidisciplinaryEpidermal Growth FactorCell growthlcsh:RAcetylationCell migrationHCT116 CellsGene Expression Regulation NeoplasticDrug Resistance NeoplasmAcetylationMutationCancer researchlcsh:QKRASSignal transductionColorectal NeoplasmsResearch ArticleSignal TransductionPLoS ONE
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