Search results for "cellularity"

showing 9 items of 9 documents

Hypocellularity in the murine model for Down Syndrome Ts65Dn is not affected by adult neurogenesis

2016

Down syndrome (DS) is caused by the presence of an extra copy of the chromosome 21 and it is the most common aneuploidy producing intellectual disability. Neural mechanisms underlying this alteration may include defects in the formation of neuronal networks, information processing and brain plasticity. The murine model for DS, Ts65Dn, presents reduced adult neurogenesis. This reduction has been suggested to underlie the hypocellularity of the hippocampus as well as the deficit in olfactory learning in the Ts65Dn mice. Similar alterations have also been observed in individuals with DS. To determine whether the impairment in adult neurogenesis is, in fact, responsible for the hypocellularity …

0301 basic medicineanimal diseasesHippocampusSubventricular zoneBiotecnologiaHippocampusSubgranular zonelcsh:RC321-57103 medical and health sciences0302 clinical medicinedoublecortinNeuroplasticitymental disordersmedicineBrdUlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal ResearchbiologyGeneral NeuroscienceNeurogenesisOlfactory BulbOlfactory bulbDoublecortinCell biologyadult neurogenesisTs65Dn mice030104 developmental biologymedicine.anatomical_structureHypocellularityPsicobiologianervous systembiology.proteinDown SyndromeKi67Neuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Neuroscience
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Renal disease associated with myeloproliferative neoplasms and myelodysplastic syndrome/myeloproliferative neoplasms

2020

Aims Renal changes in patients with myeloproliferative neoplasms (MPNs) or myelodysplastic syndrome (MDS)/MPNs have been addressed by few, respectively no, reports. The aim of this study was to focus on a systematic evaluation of renal biopsies in patients with MPNs or MDS/MPNs. Methods and results The cohort comprised 29 patients (23 men) aged 67 ± 11 years (mean ± standard deviation), diagnosed with chronic myeloid leukaemia (n = 5), polycythaemia vera (n = 9), primary myelofibrosis (n = 5), essential thrombocythaemia (n = 2), or chronic myelomonocytic leukaemia (n = 4), as well as MPNs or MDS/MPNs not otherwise specified (n = 4). Patients manifested with proteinuria (93%), partially in t…

AdultMale0301 basic medicinemedicine.medical_specialtyPolycythaemiaHistologyThrombotic microangiopathy610 MedizinRenal functionMesangial hypercellularityGastroenterologyPathology and Forensic MedicineNephropathyCohort Studies03 medical and health sciencesGlomerulonephritis0302 clinical medicineRisk FactorsNeoplasmshemic and lymphatic diseasesInternal medicine610 Medical sciencesmedicineHumansddc:610MyelofibrosisAgedAged 80 and overMyeloproliferative DisordersProteinuriaThrombotic Microangiopathiesbusiness.industryGlomerulonephritisGeneral MedicineMiddle Agedmedicine.diseaseMyelodysplastic-Myeloproliferative Diseases030104 developmental biologyMyelodysplastic Syndromes030220 oncology & carcinogenesisFemaleKidney Diseasesmedicine.symptombusiness
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Morphometric Study of the Bone Marrow in Polycythemia Vera Following Interferon-Alpha Therapy

1993

Bone marrow cellularity and extent of fibrotic change were determined in nineteen patients with polycythemia vera, treated with interferon-alpha (IFN) for 1 year. The cellularity was evaluated with an interactive semiautomatic method using Leitz TAS plus microscope: in particular, number and size of megakaryocytes were evaluated after immunostaining with Y2/51 (CD 61); reticulin content was studied by light microscope with a semiquantitative method. Before IFN therapy mean cellularity was 80.5% (+/- 13.7). After 6 and 12 months mean cellularity was 75.4% and 68.4% respectively. Six months after cessation of IFN therapy the cellularity was 69.1%. A decrease of the number, density and morphom…

AdultMalePathologymedicine.medical_specialtyAlpha interferonCell CountPathology and Forensic MedicinePolycythemia veraBone MarrowFibrosishemic and lymphatic diseasesmedicineHumansMyelofibrosisPolycythemia VeraAgedAged 80 and overbusiness.industryInterferon-alphaCell BiologyMiddle Agedmedicine.diseaseBone marrow cellularitymedicine.anatomical_structurePrimary MyelofibrosisMarrow fibrosisFemaleBone marrowbusinessMegakaryocytesImmunostainingPathology - Research and Practice
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Nasal Cellularity in 183 Unselected Schoolchildren Aged 9 to 11 Years

2002

Objective. Although rhinitis is extremely frequent in children, methods for assessing the severity of nasal inflammation produce results with wide variability and hence weak clinical significance. We designed this epidemiologic investigation to define the clinical usefulness of assessing nasal cellularity in children. Methods. We studied 183 of 203 eligible unselected schoolchildren who were aged 9 to 11 years and whose parents gave informed consent and completed a questionnaire on the history of atopic and respiratory symptoms. In all children, nasal swabs were obtained from both nostrils and eluted in saline and slides were prepared from cytospin preparations for staining and white cell c…

Allergymedicine.medical_treatmentRespiratory Tract DiseasesAnti-Inflammatory AgentsMucous membrane of noseLeukocyte CountneutrophilsFlunisolideAnti-Inflammatory Agents; Reproducibility of Results; Fluocinolone Acetonide; Skin Tests; Humans; Rhinitis; Child; Biopsy Needle; Exudates and Transudates; Leukocyte Count; Base Sequence; Respiratory Tract Diseases; Neutrophils; Nitrogen Oxides; Administration Intranasal; Nasal Mucosa; Breath TestsChildRespiratory Tract DiseaseSalineRhinitiSkin Testnasal cellularityBiopsy NeedleNeutrophilschoolchildrenExudates and Transudatesrespiratory systemExudates and TransudateNasal decongestantAnti-Inflammatory AgentFluocinolone AcetonideBreath TestsNasal SwabNitrogen Oxidesmedicine.symptomHumanmedicine.drugmedicine.medical_specialtyReproducibility of ResultNitrogen OxiderhinitisInternal medicineotorhinolaryngologic diseasesmedicineHumansAdministration IntranasalSkin TestsBase Sequencebusiness.industrynasal cellularity; neutrophils; rhinitis; allergy; schoolchildrenReproducibility of Resultsallergymedicine.diseaseNeutrophiliaNasal MucosaPediatrics Perinatology and Child HealthImmunologyNasal administrationbusinessPediatrics
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Phylostratic Shift of Whole-Genome Duplications in Normal Mammalian Tissues towards Unicellularity Is Driven by Developmental Bivalent Genes and Reve…

2020

Tumours were recently revealed to undergo a phylostratic and phenotypic shift to unicellularity. As well, aggressive tumours are characterized by an increased proportion of polyploid cells. In order to investigate a possible shared causation of these two features, we performed a comparative phylostratigraphic analysis of ploidy-related genes, obtained from transcriptomic data for polyploid and diploid human and mouse tissues using pairwise cross-species transcriptome comparison and principal component analysis. Our results indicate that polyploidy shifts the evolutionary age balance of the expressed genes from the late metazoan phylostrata towards the upregulation of unicellular and early m…

CarcinogenesisCircadian clockAntineoplastic AgentsBiologyGenomeArticleCatalysisBivalent (genetics)Epigenesis Geneticlcsh:ChemistryProto-Oncogene Proteins c-mycInorganic ChemistryTranscriptomeMicePolyploidGene DuplicationNeoplasmsProtein Interaction MappingAnimalsHumanscancerEpigeneticsPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologyGenepolyploidybivalent genesSpectroscopyGeneticsGenomePloidiesCircadian Rhythm Signaling Peptides and ProteinsOrganic Chemistryearly multicellularityviral-origin oncogenesOncogenesGeneral MedicineembryonalityPhenotypeNeoplasm ProteinsunicellularityComputer Science ApplicationsGene Expression Regulation Neoplasticlcsh:Biology (General)lcsh:QD1-999Drug Resistance NeoplasmMetabolic Networks and PathwaysInternational Journal of Molecular Sciences
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On closures of discrete sets

2018

The depth of a topological space $X$ ($g(X)$) is defined as the supremum of the cardinalities of closures of discrete subsets of $X$. Solving a problem of Mart\'inez-Ruiz, Ram\'irez-P\'aramo and Romero-Morales, we prove that the cardinal inequality $|X| \leq g(X)^{L(X) \cdot F(X)}$ holds for every Hausdorff space $X$, where $L(X)$ is the Lindel\"of number of $X$ and $F(X)$ is the supremum of the cardinalities of the free sequences in $X$.

CombinatoricsMathematics (miscellaneous)Cardinal invariants Lindelof space Discrete set Elementary submodel CellularityGeneral Topology (math.GN)FOS: MathematicsHausdorff spaceMathematics::General TopologySettore MAT/03 - GeometriaTopological spaceDiscrete setInfimum and supremumMathematics - General TopologyMathematics
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European consensus on grading bone marrow fibrosis and assessment of cellularity.

2005

Quantification of characteristic bone marrow biopsy features includes basic parameters such as cellularity and fiber content. These are important to assess the dynamics of disease processes with a significant impact on risk stratification, survival patterns and, especially, therapy-related changes. A panel of experienced European pathologists and a foreign expert evaluated, at a multi-headed microscope, a large number of representative slides of trephine biopsies from patients with myelofibrosis in an attempt to reach a consensus on how to grade cellularity and fibrosis. This included a critical evaluation of previously described scoring systems. During the microscopic analysis and subseque…

EuropediagnosisBone MarrowPrimary MyelofibrosisMyelofibrosisHumansReproducibility of ResultsMyelofibrosis; diagnosisbone marrow cellularity grading of myelofibrosis standardization trephine biopsies
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A common extension of Arhangel'skii's Theorem and the Hajnal-Juhasz inequality

2019

AbstractWe present a result about $G_{\unicode[STIX]{x1D6FF}}$ covers of a Hausdorff space that implies various known cardinal inequalities, including the following two fundamental results in the theory of cardinal invariants in topology: $|X|\leqslant 2^{L(X)\unicode[STIX]{x1D712}(X)}$ (Arhangel’skiĭ) and $|X|\leqslant 2^{c(X)\unicode[STIX]{x1D712}(X)}$ (Hajnal–Juhász). This solves a question that goes back to Bell, Ginsburg and Woods’s 1978 paper (M. Bell, J.N. Ginsburg and R.G. Woods, Cardinal inequalities for topological spaces involving the weak Lindelöf number, Pacific J. Math. 79(1978), 37–45) and is mentioned in Hodel’s survey on Arhangel’skiĭ’s Theorem (R. Hodel, Arhangel’skii’s so…

Inequalitycardinal invariantsLindelofGeneral Mathematicsmedia_common.quotation_subject010102 general mathematicsGeneral Topology (math.GN)Hausdorff spaceContrast (statistics)Mathematics::General TopologyExtension (predicate logic)01 natural sciencesSeparation axiom010101 applied mathematicsCombinatoricsMathematics::LogiccellularityCardinality boundsFOS: MathematicsSettore MAT/03 - Geometria0101 mathematicsTopology (chemistry)media_commonMathematicsMathematics - General Topology
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Pathobiology of Hodgkin Lymphoma

2010

Despite its well-known histological and clinical features, Hodgkin's lymphoma (HL) has recently been the object of intense research activity, leading to a better understanding of its phenotype, molecular characteristics, histogenesis, and possible mechanisms of lymphomagenesis. There is complete consensus on the B-cell derivation of the tumor in most cases, and on the relevance of Epstein-Barr virus infection and defective cytokinesis in at least a proportion of patients. The REAL/WHO classification recognizes a basic distinction between lymphocyte predominance HL (LP-HL) and classic HL (cHL), reflecting the differences in clinical presentation and behavior, morphology, phenotype, and molec…

Pathologymedicine.medical_specialtybusiness.industryMixed cellularityLymphocyteHematologyReview ArticleHistogenesismedicine.diseasePhenotypeVirusLymphomaPathobiologymedicine.anatomical_structurehemic and lymphatic diseasesmedicineHodgkin lymphomaDiseases of the blood and blood-forming organsRC633-647.5businessWho classificationHodgkin lymphoma; microenvironment.Hodgkin lymphomamicroenvironment.Advances in Hematology
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