Search results for "cerebral"

showing 10 items of 1357 documents

Update on mechanism and therapeutic implications of spinal cord stimulation and cerebral hemodynamics: A narrative review

2017

Spinal cord stimulation (SCS) is well known for its early role in the management of chronic pain, mainly failed back surgery syndrome (FBSS), spasticity, and bowel and bladder dysfunction. In more recent years, SCS has been proposed for patients suffering from refractory angina or peripheral vasculopathies in order to gain symptom relief, thus indicating some hemodynamic effect on the peripheral circulation. Taking into account this scientific observation, since the late1980s, researchers have started to investigate the potential effect of SCS on cerebral blood flow (CBF) regulation and its possible application in certain pathological settings dealing with vascular pattern dysfunction, such…

Subarachnoid hemorrhage030204 cardiovascular system & hematologyCerebral autoregulationBrain IschemiaBrain ischemia03 medical and health sciences0302 clinical medicineAnimalsHomeostasisHumansVasospasm IntracranialMedicineSpasticitySympathectomyBrain injuryStrokeBrain Neoplasmsbusiness.industrySettore MED/27 - NeurochirurgiaBrain injury; Cerebral autoregulation; Cerebral blood flow; Functional sympathectomy; Spinal cord stimulation; Stroke; Vasospasm; Surgery; Neurology (clinical)HemodynamicsChronic painVasospasmFunctional sympathectomySubarachnoid HemorrhageCerebral blood flowmedicine.diseaseVasospasmCerebral autoregulationStrokenervous systemCerebral blood flowSpinal cord stimulationBrain InjuriesCerebrovascular CirculationAnesthesiaSurgeryNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgery
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Changes in the cerebrovascular effects of endothelin-1 and nicardipine after experimental subarachnoid hemorrhage.

1993

The role of endothelium-related factors in the pathogenesis of cerebral vasospasm after subarachnoid hemorrhage (SAH) has gained interest since the discovery of endothelin-1 (ET-1). We have examined, before and after SAH, the responsiveness of the cerebrovascular bed of the goat to ET-1, the sources of Ca2+ in ET-1-induced responses, and the ability of the Ca2+ entry blocker nicardipine to counteract them. Before SAH, injection of ET-1 into the cerebral circulation increased cerebrovascular resistance, thereby producing dose-dependent reductions in cerebral blood flow (CBF), which were prevented by nicardipine. In isolated middle cerebral arteries, ET-1 induced concentration-dependent contr…

Subarachnoid hemorrhageCerebral arteriesNicardipineBlood PressureCerebral circulationNicardipineCerebral vasospasmHeart RatemedicineAnimalscardiovascular diseasesDose-Response Relationship Drugbusiness.industryEndothelinsGoatsHemodynamicsBrainVasospasmSubarachnoid Hemorrhagemedicine.diseasenervous system diseasesmedicine.anatomical_structureCerebral blood flowVasoconstrictionAnesthesiaSurgeryFemaleVascular ResistanceNeurology (clinical)businessBlood Flow VelocityBlood vesselmedicine.drugNeurosurgery
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Effect of Intra-Arterial Infusion of Papaverine Hydrochloride on Brain Tissue Oxygen Pressure in the Management of Severe Vasospasm Following Aneurys…

2001

Despite considerable advances in diagnostic, surgical and interventional techniques, the overall outcome for patients with aneurysmal subarachnoid hemorrhage remains poor. Concerning perioperative course, cerebral ischaemia is one of the major factors causing secondary brain demage. The aim of all therapeutic interventions is the stabilization and maintance of cerebral blood flow and tissue oxygenation in order to prevent cerebral ischaemia and consecutively brain infarction [1].

Subarachnoid hemorrhagePapaverine Hydrochloridebusiness.industryVasospasmIntra arterial infusionPerioperativemedicine.diseaseIntensive care unitlaw.inventionCerebral vasospasmCerebral blood flowlawAnesthesiamedicinecardiovascular diseasesbusiness
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Use of middle cerebral velocity and blood pressure for the analysis of cerebral autoregulation at various frequencies: The coherence index

1998

A common component in many protocols for the evaluation of cerebral autoregulation is the comparison of transcranial Doppler ultrasound (TCD) velocities with blood pressure recordings, in which correlations between these two signals correspond to impaired autoregulation. With long data sets and complicated paradigms, however, visual inspection alone cannot adequately distinguish random coincidence from consistent correlation in a statistically valid fashion. We suggest and illustrate the use of the coherence index for this purpose. To illustrate this technique, long-term recordings of TCD velocity and blood pressure were obtained from 6 normal subjects and using 23 data segments from 8 pati…

Subarachnoid hemorrhageUltrasonography Doppler TranscranialHemodynamicsBlood PressureCerebral autoregulationCorrelationNuclear magnetic resonanceReference ValuesmedicineTidal VolumeHomeostasisHumansAutoregulationTranscranial doppler cerebral autoregulationMathematicsFourier AnalysisSettore MED/27 - NeurochirurgiaGeneral MedicineCoherence (statistics)Cerebral ArteriesSubarachnoid Hemorrhagemedicine.diseaseTranscranial DopplerPlethysmographyBlood pressureNeurologyAnesthesiaNeurology (clinical)Blood Flow VelocitySoftware
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ERYTHROPOIETIN FOR THE TREATMENT OF SUBARACHNOID HEMORRAGE: A FEASIBLE INGREDIENT FOR A SUCCESS MEDICAL RECIPE

2015

Subarachnoid hemorrhage (SAH) following aneurysm bleeding accounts for 6% to 8% of all cerebrovascular accidents. Although an aneurysm can be effectively managed by surgery or endovascular therapy, delayed cerebral ischemia is diagnosed in a high percentage of patients resulting in significant morbidity and mortality. Cerebral vasospasm occurs in more than half of all patients after aneurysm rupture and is recognized as the leading cause of delayed cerebral ischemia after SAH. Hemodynamic strategies and endovascular procedures may be considered for the treatment of cerebral vasospasm. In recent years, the mechanisms contributing to the development of vasospasm, abnormal reactivity of cerebr…

Subarachnoid hemorrhagebusiness.industrySettore MED/27 - NeurochirurgiaCerebral arteriesIschemiaHemodynamicsVasospasmReview Articlemedicine.diseasenervous system diseasesCerebral vasospasmAneurysmErythropoietinAnesthesiaRECOMBINANT-HUMAN-ERYTHROPOIETIN NITRIC-OXIDE SYNTHASE ENDOTHELIAL NO SYNTHASE BLOOD-BRAIN-BARRIER IN-VIVO EVIDENCE CEREBRAL-ISCHEMIA DARBEPOETIN-ALPHA TISSUE PROTECTION DOUBLE-BLIND RECEPTORGeneticscardiovascular systemMolecular MedicineMedicinecardiovascular diseasesbusinessMolecular BiologyGenetics (clinical)medicine.drugsubarachnoid hemorrage erytropoietin
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The zona incerta system: Involvement in attention and movement

2021

The zona incerta (ZI) is a large structure made of four neurochemically defined regions (at least, in rodents). It is globally involved in complex connections with telencephalic and brainstem centers. In this work, we focus on some of the anatomical links this structure develops with the cerebral cortex and the tectum. We also point to its integration within a larger basal ganglia network. The functions of this region are still mysterious, even if recent works suggest its participation in behavioral expression. Studies about the functional organization of the vibrissal system have provided the first integrated model, illustrating the ZI's role in sensory-motor programing. In addition, ZI co…

Subthalamic nucleusmedicine.anatomical_structureCerebral cortexSuperior colliculusBasal gangliamedicineZona incertaCognitionBrainstemBiologyTectumNeuroscience
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Triiodothyronine accelerates the synthesis of synapsin I in developing neurons from fetal rat brain cultured in a synthetic medium.

1990

The effect of Triiodothyronine (T3) on Synapsin I appearance in rat cortical neurons has been investigated in vitro. Neuronal cultures from 16-day-old fetal rat brain grown in the absence of T3, express immunohystochemically detectable Synapsin I at the 14th day in vitro. The addition of the hormone to the culture medium determines an early (at the 7th day in vitro) appearance of fluorescent dots specific for Synapsin I. © 1990 Plenum Publishing Corporation.

Synapsin Imedicine.medical_specialtyCNS developmentCentral nervous systemFluorescent Antibody TechniqueNerve Tissue ProteinsBiologySettore BIO/19 - Microbiologia GeneraleBiochemistryCellular and Molecular NeuroscienceFetusInternal medicinemedicineAnimalschemically defined mediumimmunofluorescenceCells CulturedNeuronsFetusTriiodothyronineNeuroscience (all)BrainGeneral MedicineSynapsinsIn vitroCulture MediaRatsChemically defined mediummedicine.anatomical_structureEndocrinologynervous systemneuronal cultureCerebral cortexCell cultureTriiodothyronineSettore MED/26 - NeurologiaSynapsin 1Neurochemical research
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Infarctus cérébral et plasticité : focus sur le BDNF

2011

Functional recovery after ischemic stroke largely involves brain plasticity and more accurately its ability to reorganize the neuronal networks spared by the infarct. Studies conducted on animals using different ischemic stroke models have demonstrated that plastic changes are induced by BDNF (Brain-Derived Neurotrophic Factor). Hence, increasing levels of BDNF in the brain is considered a promising therapeutic strategy to reduce post-stroke impairments. In this context, our work had 2 aims: 1) In a rat model, to identify cells involved in the over-production of BDNF and to evaluate the pertinence of the measurement of circulating BDNF levels to estimate brain BDNF levels; 2) In ischemic st…

SérumSerum[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyRécupération motricePlasticityMotor recoveryAccident vasculaire cérébralStrokePlasmaBDNFPlasticitéCerebral infarctFluoxetin[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyRatInfarctus cérébralFluoxétine
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Cortical neurons selectively inhibit MHC class II induction in astrocytes but not in microglial cells.

1993

Astrocytes have been shown to act as potent accessory cells for MHC class II-restricted T cell responses in vitro after treatment with interferon-gamma. In contrast, even under conditions of severe central nervous system (CNS) inflammation, they seem to express little, if any, class II molecules in vivo. Thus the role of astroglial cells as accessory cells in immune responses in the CNS remains to be determined. We have studied neuron--glia interactions with respect to induction of MHC class II molecules. Surprisingly, in a co-culture system, viable neurons inhibited the induction of class II restriction elements on astrocytes. This effect was only observed when neurons had contact to astro…

T cellT-LymphocytesImmunologyAntigen presentationAntigen-Presenting CellsDown-RegulationLymphocyte ActivationMHC class ImedicineImmunology and AllergyAnimalsCells CulturedCerebral CortexNeuronsMHC class IIbiologyMicrogliaHistocompatibility Antigens Class IIGeneral MedicineCell biologyRatsmedicine.anatomical_structurenervous systemAstrocytesImmunologybiology.proteinNeurogliaNeuronNeurogliaAstrocyteInternational immunology
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Differential evolution of PSA-NCAM expression during aging of the rat telencephalon

2007

Changes in the ability of neuronal networks to undergo structural remodeling may be involved in the age-associated cognitive decline. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) declines dramatically during postnatal development, but persists in several regions of the young-adult rat telencephalon, where it participates, through its anti-adhesive properties, in neuronal structural plasticity. However, PSA-NCAM expression during aging has only been studied in the dentate gyrus and the piriform cortex layer II, where it is strongly downregulated in adult (middle-aged) individuals. Using immunohistochemistry, we have observed that in most of the telencephalic areas …

TelencephalonAgingDendritic SpinesDown-RegulationHippocampusCell CountNeural Cell Adhesion Molecule L1BiologyPiriform cortexCell AdhesionLimbic SystemmedicineNeuropilAnimalsCognitive declineCerebral CortexNeuronsNeuronal PlasticityNeocortexGeneral NeuroscienceDentate gyrusAmygdalaImmunohistochemistryRats Inbred F344RatsDisease Models Animalmedicine.anatomical_structurenervous systemSialic AcidsFemaleNeural cell adhesion moleculeNeurology (clinical)Geriatrics and GerontologyNeuroscienceBiomarkersDevelopmental BiologyStratum lucidumNeurobiology of Aging
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