Search results for "choline"

showing 10 items of 1138 documents

Failure of opioids to affect excitation and contraction in isolated ventricular heart muscle

1989

The opioid agonists morphine (selective for mu-receptors) and ethylketocyclazocine (selective for kappa-receptors), at concentrations evoking strong effects in neuronal structures, did not significantly affect the configuration of the intracellularly recorded action potential and the force of contraction in ventricular heart muscle isolated from guinea pigs, rabbits and man. These results suggest that any changes of heart functions in vivo in response to opioid-like drugs are probably not mediated postsynaptically at the myocardial cell membrane but rather presynaptically, influencing the release of noradrenaline and/or acetylcholine from the nerve terminals.

medicine.medical_specialtyContraction (grammar)EthylketocyclazocineGuinea PigsAction PotentialsEthylketocyclazocineBiologyGuinea pigNorepinephrineCellular and Molecular NeuroscienceInternal medicineHeart ratemedicineAnimalsCyclazocineHumansOpioid peptideMolecular BiologyPharmacologyMorphineNaloxoneCell BiologyPapillary MusclesMyocardial ContractionAcetylcholineEndocrinologyOpioidSynapsesCirculatory systemMolecular MedicineRabbitsAcetylcholinemedicine.drugExperientia
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Different muscarine receptors mediate the prejunctional inhibition of [3H]-noradrenaline release in rat or guinea-pig iris and the contraction of the…

1989

To investigate the muscarine receptor type mediating inhibition of [3H]-noradrenaline release from the isolated rat and guinea-pig iris we have determined the potency of antimuscarinic drugs to antagonize the methacholine-induced inhibition of [3H]-noradrenaline overflow evoked by field stimulation (3 Hz, 2 min). The prejunctional apparent affinities were compared with those obtained for postjunctional muscarine receptors mediating the methacholine-induced contraction of the isolated rabbit iris sphincter muscle. Prejunctional apparent affinity constants of pirenzepine (6.67), himbacine (8.51), methoctramine (7.92), 4-diphenylacetoxy-N-methylpiperidine methiodide (4-DAMP, 8.00), hexahydro-d…

medicine.medical_specialtyContraction (grammar)Iris sphincter muscleIrisIn Vitro TechniquesBiologyGuinea pigNorepinephrinechemistry.chemical_compoundPiperidinesInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsNeurotransmitterReceptorPharmacologyMuscarineMuscle SmoothRats Inbred StrainsGeneral MedicineReceptors MuscarinicElectric StimulationRatsKineticsmedicine.anatomical_structureEndocrinologychemistrySphincterRabbitsMuscle ContractionNaunyn-Schmiedebergs Archives of Pharmacology
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Effects of inhibitors of cGMP-dependent protein kinase in atrial heart and aortic smooth muscle from rats

1995

Several activators of cGMP-dependent protein kinase (protein kinase G) such as 8-Br-cGMP reduced force of contraction in rat left atria. Inhibitors of protein kinase G antagonized the negative inotropic effect of 8-Br-cGMP but not of acetylcholine in atria. However, the acetylcholine-induced relaxation in aortic rings was significantly inhibited by protein kinase G inhibition. It is concluded that the reduction by 8-Br-cGMP of force of contraction in atria is related to activation of protein kinase G. In response to acetylcholine, activation of protein kinase G is probably a major step in smooth muscle relaxation but is not involved in the reduction of force of contraction in atria.

medicine.medical_specialtyContraction (grammar)Muscle RelaxationAorta ThoracicIn Vitro TechniquesMuscle Smooth VascularIsometric ContractionInternal medicineCyclic GMP-Dependent Protein KinasesmedicineAnimalsHeart AtriaProtein kinase ACyclic GMPRho-associated protein kinasePharmacologybiologyHeartMyocardial ContractionAcetylcholineRatsEnzyme ActivationEndocrinologyEnzyme inhibitorSecond messenger systemcardiovascular systembiology.proteinmedicine.symptomcGMP-dependent protein kinaseAcetylcholineMuscle Contractionmedicine.drugMuscle contractionEuropean Journal of Pharmacology
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The Sources of Ca2+ for Muscarinic Receptor-induced Contraction in the Rat Ileum

1996

Abstract The contractile responses obtained by activation of different muscarinic receptor subtypes in the longitudinal muscle of the rat ileum and especially the responses of this muscle to acetylcholine in a Ca2+-free medium have been investigated. In Ca2+-containing solution, acetylcholine elicited similar concentration-dependent contractile responses in the duodenum, jejunum and ileum strips of the rat intestine. The response to a maximal concentration of the agonist (1 μM) consisted of a rapid phasic response followed by a slower tonic one. Nifedipine completely relaxes or inhibits the sustained response and only partially diminishes the phasic one, which suggests that the phasic contr…

medicine.medical_specialtyContraction (grammar)NifedipinePharmaceutical ScienceIn Vitro TechniquesMuscarinic AgonistsBiologyTonic (physiology)chemistry.chemical_compoundIleumInternal medicineMuscarinic acetylcholine receptormedicineMethoctramineAnimalsRats WistarPharmacologyMuscle SmoothPirenzepineCalcium Channel BlockersReceptors MuscarinicPirenzepineAcetylcholineRatsAtropineEndocrinologychemistryCalciumFemalemedicine.symptomAcetylcholineMuscle Contractionmedicine.drugMuscle contractionJournal of Pharmacy and Pharmacology
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Effect of inhibition of the electrogenic Na+/K+ pump on the mechanical activity in the rat uterus.

1996

The effects of ouabain and K(+)-free solution were studied in estrogen-primed rat uterine strips under resting tone or repeatedly stimulated with KCl, acetylcholine or oxytocin applied for 20 minutes at 60 minute intervals. These effects were compared with those of the K+ channel opener cromakalim. In preparations under resting tone, ouabain (0.1 mM and 0.3 mM) induced rhythmic contractions which disappeared after 20-30 minutes whereas at a higher concentration (1 mM) it evoked a rapid, phasic response followed by a small tonic contraction. Exposure of the strip to a K(+)-free solution induced either rhythmic waves, which ceased after 8-10 minutes, or a single phasic contraction which was f…

medicine.medical_specialtyCromakalimPotassium ChannelsIn Vitro TechniquesOxytocinOuabainchemistry.chemical_compoundUterine ContractionInternal medicinemedicineAnimalsPharmacology (medical)BenzopyransPyrrolesNa+/K+-ATPaseEnzyme InhibitorsRats WistarOuabainPharmacologyTetraethylammoniumVoltage-dependent calcium channelUterusMyometriumPotassium channel blockerAcetylcholineRatsEndocrinologychemistryCalciumFemalemedicine.symptomSodium-Potassium-Exchanging ATPaseCromakalimmedicine.drugMuscle contractionFundamentalclinical pharmacology
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Cromakalim inhibits electrically-evoked [3H]acetylcholine release from a tube-preparation of the rat isolated trachea by an epithelium-dependent mech…

1993

Rat isolated tracheae were labelled by incubation with [3H]choline to measure the tritium efflux elicited by electrical stimulation of the extrinsic parasympathetic nerves in vitro. Stimulated tritium efflux reflects the neuronal release of newly synthesized acetylcholine; the effects of potassium channel openers on the stimulated tritium efflux were investigated. In tracheae opened longitudinally neither cromakalim nor its 3S,4R-enantiomer, BRL 38227, reduced the stimulated tritium efflux, whereas in intact tube-preparations cromakalim (0.01-1 mumol/l) mediated a concentration-dependent inhibition. The inhibitory effect of 1 mumol/l cromakalim was prevented by 0.1 mumol/l glibenclamide. Li…

medicine.medical_specialtyCromakalimPotassium ChannelsStimulationIn Vitro TechniquesEpitheliumGlibenclamidechemistry.chemical_compoundInternal medicinemedicineAnimalsBenzopyransPyrrolesPharmacologyStereoisomerismGeneral MedicinePotassium channelAcetylcholineRatsTracheaEndocrinologyMechanism of actionchemistrycardiovascular systemBiophysicsLiberationEffluxmedicine.symptomCromakalimAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Facilitatory and inhibitory muscarine receptors on the rat phrenic nerve: effects of pirenzepine and dicyclomine

1988

Neuronal transmitter stores of the rat phrenic nerve were labelled by an incubation with [3H]choline. Release of [3H]acetylcholine was elicited either by a short (100 pulses, 5 Hz) or by a long (1500 pulses, 5 or 25 Hz) period of electrical nerve stimulation. Pirenzepine and dicyclomine enhanced transmitter release evoked by the short stimulation period. Both antagonists reduced transmitter release evoked by the long stimulation period. Pirenzepine reduced transmitter release at low concentrations (1 nmol/l) whereas a higher concentration was necessary for the enhancing effect; the opposite pattern was found for dicyclomine. A low concentration of oxotremorine (10 nmol/l) enhanced and a hig…

medicine.medical_specialtyCyclohexanecarboxylic AcidsNeuromuscular JunctionDicyclomineStimulationIn Vitro TechniquesSynaptic TransmissionDicyclominechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineAnimalsEvoked PotentialsPharmacologyMuscarineChemistryRats Inbred StrainsMuscarinic acetylcholine receptor M2PirenzepineGeneral MedicineMuscarinic acetylcholine receptor M1Receptors MuscarinicPirenzepineAcetylcholineRatsPhrenic NerveEndocrinologyAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Effects of nicotine receptor agonists on acetylcholine release from the isolated motor nerve, small intestine and trachea of rats and guinea-pigs

1992

The effects of nicotine receptor agonists on the release of [3H]acetylcholine from the phrenic nerve, the small intestine and the trachea were investigated to characterize neuronal nicotine receptors within the peripheral nervous system. Contraction of the indirectly-stimulated hemidiaphragm was recorded to investigate desensitization of the postsynaptic muscular nicotine receptors. Nicotine, cytisine, 1,1-dimethyl-4-phenylpiperazinium and 2-(4-aminophenyl)-ethyl-trimethyl-ammoniumiodide caused a concentration-dependent (0.1-30 microM) increase in evoked [3H]acetylcholine release from the phrenic nerve, whereby bell-shaped concentration-response curves were obtained. The rank order of decre…

medicine.medical_specialtyDiaphragmGuinea PigsMyenteric PlexusMotor nerveReceptors NicotinicMotor EndplateNicotineCytisinechemistry.chemical_compoundPostsynaptic potentialInternal medicineIntestine SmallDrug DiscoverymedicineAnimalsGenetics (clinical)Phrenic nerveDose-Response Relationship DrugMuscle SmoothGeneral MedicineAcetylcholineStimulation ChemicalRatsPhrenic NerveTracheaEndocrinologymedicine.anatomical_structureParasympathomimeticschemistryPeripheral nervous systemMolecular Medicinemedicine.symptomSecretory RateAcetylcholineMuscle Contractionmedicine.drugMuscle contractionThe Clinical Investigator
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Antihistaminic and anticholinergic activities of mequitazine in comparison with clemizole

1988

Abstract The antihistamine and anticholinergic properties of mequitazine have been investigated and compared with those of clemizole. Both mequitazine and clemizole antagonized the effect of histamine in guinea-pig ileum competitively, the pA2 values calculated by Schild plot were 9.95 ± 0.44 for mequitazine and 10.54 ± 0.44 for clemizole. Mequitazine at 10−7 M produced a parallel shift of the dose-response curve to acetylcholine in the rat duodenum, clemizole and the lower doses of mequitazine failed to modify the effect of acetylcholine. The potency of mequitazine and clemizole as H1-histamine blockers is similar, but only mequitazine at highest concentration used showed anticholinergic a…

medicine.medical_specialtyDuodenummedicine.drug_classmedicine.medical_treatmentGuinea PigsPharmaceutical ScienceIn Vitro TechniquesBiologyParasympatholyticchemistry.chemical_compoundPhenothiazinesInternal medicinemedicineAnticholinergicAnimalsPotencyMequitazinePharmacologyParasympatholyticsMuscle SmoothRats Inbred StrainsAcetylcholineRatsClemizoleSchild regressionEndocrinologychemistryHistamine H1 AntagonistsBenzimidazolesAntihistamineHistamineHistamineMuscle Contractionmedicine.drugJournal of Pharmacy and Pharmacology
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Toxicity effects of the organic UV-filter 4-Methylbenzylidene camphor in zebrafish embryos

2019

Abstract Ultraviolet (UV) filters are widely used in personal care products and due to their lipophilicity these chemicals tend to bioaccumulate in the aquatic biota. 4-Methylbenzylidene camphor (4-MBC) is one of the most used UV-filters, and it is commonly detected in freshwater fish tissues. This substance is suspected to be an endocrine disruptor due to its interaction with Hypothalamus-Pituitary-Gonadal (HPG) and HP-Thyroid (HPT)-axis. The main objective of this study was to evaluate the effects of 4-MBC on apical endpoints, biochemical markers and on genes involved in endocrine pathways in Danio rerio. Zebrafish embryos were exposed to 4-MBC (0.083–0.77 mg/l) from 0 to 96 h post-fertil…

medicine.medical_specialtyEmbryo Nonmammaliananimal structuresEnvironmental EngineeringHealth Toxicology and Mutagenesis0208 environmental biotechnologyDanioEndocrine System02 engineering and technologyEndocrine Disruptors010501 environmental sciencesmedicine.disease_causeSynaptic Transmission01 natural scienceschemistry.chemical_compoundInternal medicinemedicineAnimalsEnvironmental ChemistryYolk sacZebrafishZebrafishGlutathione Transferase0105 earth and related environmental sciencesbiologyEmbryogenesisPublic Health Environmental and Occupational HealthGeneral MedicineGeneral Chemistrybiology.organism_classificationPollutionAcetylcholinesteraseCamphor020801 environmental engineeringOxidative Stressmedicine.anatomical_structureEndocrinologychemistryEndocrine disruptorEnzyme InductionToxicityAcetylcholinesteraseFiltrationOxidative stressChemosphere
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