Search results for "chromosome"

showing 10 items of 1175 documents

Linkage analysis in Usher syndrome type I (USH1) families from Spain.

1998

Usher syndrome (USH) is an autosomal recessive hereditary disorder characterised by congenital sensorineural hearing loss and gradual visual impairment secondary to retinitis pigmentosa (RP). The disorder is clinically and genetically heterogeneous. With regard to Usher type I (USH1), several subtypes have been described, the most frequent being USH1B located on chromosome 11q13.5. Of 18 USH1 families studied by linkage analysis, 12 (67%) showed significant lod score values for locus D11S527 (Zmax=14.032, theta=0.000) situated on chromosome 11q. Our findings suggest considerable genetic heterogeneity in the Spanish USH1 population. It is important to note that one of our families linked to …

Genetic MarkersMaleGenetic LinkageHearing Loss SensorineuralUsher syndromePopulationLocus (genetics)BiologyGenetic HeterogeneityGene mappingGenetic linkageRetinitis pigmentosaotorhinolaryngologic diseasesGeneticsmedicineHumanseducationGenetics (clinical)Geneticseducation.field_of_studyGenetic heterogeneityChromosomes Human Pair 11HaplotypeSyndromemedicine.diseaseeye diseasesPedigreeHaplotypesSpainFemaleRetinitis PigmentosaResearch ArticleJournal of Medical Genetics
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The copy number variant involving part of the α7 nicotinic receptor gene contains a polymorphic inversion.

2008

The alpha7 nicotinic acetylcholine receptor gene (CHRNA7) is located at 15q13-q14 in a region that is strongly linked to the P50 sensory gating deficit, an endophenotype of schizophrenia and bipolar disorder. Part of the gene is a copy number variant, due to a duplication of exons 5-10 and 3' sequence in CHRFAM7A, which is present in many but not all humans. Maps of this region show that the two genes are in opposite orientation in the individual mainly represented in the public access human DNA sequence database (Build 36), suggesting that an inversion had occurred since the duplication. We have used fluorescent in situ hybridization to investigate this putative inversion. Analysis of inte…

Genetic MarkersMaleLinkage disequilibriumBipolar DisorderPan troglodytesalpha7 Nicotinic Acetylcholine ReceptorReceptors NicotinicLinkage DisequilibriumExonGene duplicationGeneticsSettore MED/48 -Scienze Infermierist. e Tecn. Neuro-Psichiatriche e Riabilitat.AnimalsHumansCopy-number variationGeneSettore MED/25 - PsichiatriaGenetics (clinical)Sequence DeletionSegmental duplicationChromosomal inversionGeneticsChromosomes Human Pair 15Polymorphism GeneticBase SequencebiologyCHRNA7Chromosome Mappinginversion schizophrenia bipolar disorder 15q13–q14 CHRNA7 segmental duplicationChromosome InversionSchizophreniabiology.proteinFemale
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Family studies in scleroderma (systemic sclerosis) demonstrating an HLA-linked increased chromosomal breakage rate in cultured lymphocytes

1988

An increased chromosomal breakage rate (ICBR) was found in 27 of 28 patients with scleroderma (systemic sclerosis, SS) - 5 with the syndrome including calcinosis cutis, Raynaud phenomenon, esophagus hypomotility, sclerodactyly and telangiectasia (CREST), 4 incomplete CREST, 1 overlapping syndrome, 18 progressive systemic sclerosis (PSS). Not only the patients, but also about half of their first-degree relatives showed an increased chromosomal breakage rate (more than 5 breaks per 100 metaphases). This character segregated as a dominant marker in nine families of scleroderma patients. In the six informative of the nine families, the ICBR trait showed close linkage with the HLA region on chro…

Genetic MarkersMaleSystemic diseaseGenetic LinkageHuman leukocyte antigenBiologySclerodermaCalcinosis cutisHLA AntigensGeneticsmedicineHumansLymphocytesCells CulturedGenetics (clinical)Chromosome AberrationsAutoimmune diseaseScleroderma SystemicSclerodactylyChromosome Fragilitymedicine.diseaseConnective tissue diseasePedigreeHaplotypesImmunologyFemalemedicine.symptomHuman Genetics
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The Genetic Legacy of Religious Diversity and Intolerance: Paternal Lineages of Christians, Jews, and Muslims in the Iberian Peninsula

2008

12 páginas, 6 figuras.-- et al.

Genetic MarkersMalemedia_common.quotation_subjectPopulationIslamHaplogroupArticleChristianityReligious intoleranceGenetic driftPopulation GroupsReligious conversionPeninsulaGeneticsEthnicityHumansGenetics(clinical)educationGenetics (clinical)Phylogenymedia_commonDemographyGeneticsgeographyeducation.field_of_studyGenetic diversitygeography.geographical_feature_categoryChromosomes Human YPortugalEmigration and ImmigrationGenealogyhumanitiesHaplotypesSpainJewsDiversity (politics)
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Tetrasomy 18p de novo: Identification by FISH with conventional and microdissection probes and analysis of parental origin and formation by short seq…

1996

We report a de novo supernumerary isochromosome 18p in a child with tetrasomy 18p, analyzed by a straightforward combination of cytogenetic and molecular cytogenetic methods. The diagnostic procedure consisted of standard banding techniques and fluorescence in situ hybridization (FISH) with centromere and library DNA probes for chromosome 18, and 18p-specific FISH probes prepared by chromosome microdissection and in vitro amplification. The maternal origin as well as the most probable cell stages of formation of the supernumerary isochromosome were determined by typing of short sequence repeats (SSRs). The pattern of allelic distribution suggests a nondisjunction during meiosis followed by …

Genetic MarkersMalemedicine.medical_specialtyMarker chromosomeCentromereIsochromosomeMothersBiologyFathersTetrasomy 18pChromosome 18GeneticsmedicineHumansAllelesIn Situ Hybridization FluorescenceGenetics (clinical)Repetitive Sequences Nucleic AcidGeneticsmedicine.diagnostic_testCytogeneticsChromosome MappingInfantAneuploidymedicine.diseaseChromosome microdissectionMolecular biologyChild PreschoolTetrasomyFemaleChromosomes Human Pair 18DNA ProbesFluorescence in situ hybridizationHuman Genetics
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Autosomal dominant polycystic kidney disease—in vitro culture of cyst-lining epithelial cells

1992

The major form of autosomal dominant polycystic kidney disease (ADPKD) in humans is linked to the PKD1 gene on chromosome 16p. The identity of the gene and the underlying pathogenetic mechanisms are not yet defined. Cyst-lining epithelial cells derived from a polycystic kidney were successfully grown in culture and designated MZ-PKD-1 cells. By linkage analysis, the related pedigree of the nephrectomized patient could be linked to the PKD1 gene on chromosome 16p. Thus, these cells exhibit the genotype of a mutated PKD1 gene and represent an in vitro culture model for ADPKD involving chromosome 16p. The antigenic phenotype was characterized immunohistologically by epithelial differentiation …

Genetic MarkersPathologymedicine.medical_specialtyAutosomal dominant polycystic kidney diseaseHLA-C AntigensBiologyEpitheliumGenetic linkagemedicineHumansNorthern blotGeneCells CulturedHLA-A AntigensPKD1urogenital systemAntibodies MonoclonalGeneral MedicineMiddle AgedBlotting NorthernPolycystic Kidney Autosomal Dominantmedicine.diseaseImmunohistochemistryMolecular biologyPhenotypePedigreeBlotMicroscopy ElectronPhenotypeHLA-B AntigensCell cultureFemaleChromosomes Human Pair 16Virchows Archiv B Cell Pathology Including Molecular Pathology
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Clinical relevance of polymorphic markers of arterial thrombosis.

1997

Case-control and cross-sectional studies show that some common molecular variations (polymorphisms) of genes coding for proteins involved in atherosclerosis and thrombosis are often present in subjects who have experienced cerebrovascular or cardiovascular events. The clinical impact of the majority of polymorphic markers is disputed by prospective reports. In contrast, their pathophysiological implications and their role in monitoring parameters that are difficult to be checked by alternative means, are documented by the large majority of the reports. From the evidence available, there may be suggestion for further impact of polymorphic markers in vascular medicine. To substantiate this, n…

Genetic MarkersPathologymedicine.medical_specialtyPolymorphism GeneticGenetic inheritanceArterial diseaseVascular diseasebusiness.industryChromosome MappingThrombosisHematologyPeptidyl-Dipeptidase Amedicine.diseaseBioinformaticsThrombosisIschemiaRisk FactorsGenetic markermedicineHumansClinical significanceProspective cohort studybusinessVascular Medicine
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Karyotype analysis and marker chromosomes of renal cell carcinoma.

1988

Karyotyping was performed in 46 human renal cell carcinomas of various histological grades. Controls included chromosome analysis of normal renal parenchymal cells from the same patients. Various numerical chromosome aberrations were found as well as marker chromosomes. They are specific of the individual tumor but no single marker was identified occurring in all tumors tested. Only trisomy 3 was found in different tumors (31%), suggesting a more general character of this aberration. Histological dedifferentiation and large tumor size correlate with focusing of the karyotype towards distinct chromosomal modes indicative of distinct tumor cell subclones within these tumors. Their rapid growt…

Genetic MarkersPathologymedicine.medical_specialtyPoor prognosisbusiness.industryUrologyCellChromosomeKaryotypeTrisomymedicine.diseaseKidneyKidney Neoplasmsmedicine.anatomical_structureChromosome 3Renal cell carcinomaKaryotypingParenchymamedicineHumansRing ChromosomesHistological gradesChromosomes Human Pair 3businessCarcinoma Renal CellEuropean urology
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Molecular Characterization of a Chromosomal Rearrangement Involved in the Adaptive Evolution of Yeast Strains

2002

Wine yeast strains show a high level of chromosome length polymorphism. This polymorphism is mainly generated by illegitimate recombination mediated by Ty transposons or subtelomeric repeated sequences. We have found, however, that the SSU1-R allele, which confers sulfite resistance to yeast cells, is the product of a reciprocal translocation between chromosomes VIII and XVI due to unequal crossing-over mediated by microhomology between very short sequences on the 5' upstream regions of the SSU1 and ECM34 genes. We also show that this translocation is only present in wine yeast strains, suggesting that the use for millennia of sulfite as a preservative in wine production could have favored …

Genetic MarkersSaccharomyces cerevisiae ProteinsLetterChromosomal rearrangementsAnion Transport ProteinsGenes FungalMolecular Sequence DataSaccharomyces cerevisiaeSaccharomyces cerevisiaeChromosomal rearrangementSaccharomycesGenètica molecularTranslocation GeneticEvolution MolecularSaccharomycesGene FrequencySpecies SpecificityGeneticsVinificationDNA FungalGeneGenetics (clinical)Wine yeastsGene RearrangementRecombination GeneticGeneticsBase SequencebiologyGene rearrangementbiology.organism_classificationYeastYeast in winemakingChromosomes FungalGenome FungalPloidyGenome Research
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Comparative architectural aspects of regions of conserved synteny on human chromosome 11p15.3 and mouse chromosome 7 (including genes WEE1 and LMO1)

2001

Human chromosome 11p15.3 is associated with chromosome aberrations in the Beckwith Wiedemann Syndrome and implicated in the pathogenesis of different tumor types including lung cancer and leukemias. To date, only single tumor-relevant genes with linkage to this region (e.g. LMO1) have been found suggesting that this region may harbor additional potential disease associated genes. Although this genomic area has been studied for years, the exact order of genes/chromosome markers between D11S572 and the WEE1 gene locus remained unclear. Using the FISH technique and PAC clones of the flanking markers we determined the order of the genomic markers. Based on these clones we established a PAC cont…

Genetic Markerscongenital hereditary and neonatal diseases and abnormalitiesBeckwith–Wiedemann syndromeCell Cycle ProteinsBiologyChromosomesEvolution MolecularContig MappingMiceChromosome regionsGene OrderMetalloproteinsGeneticsmedicineAnimalsHumansCloning MolecularMolecular BiologyGeneConserved SequenceIn Situ Hybridization FluorescenceGenetics (clinical)Repetitive Sequences Nucleic AcidSyntenyOncogene ProteinsGeneticsChromosome 7 (human)Base CompositionChromosomes Human Pair 11Nuclear ProteinsChromosomeSequence Analysis DNALIM Domain ProteinsProtein-Tyrosine Kinasesmedicine.diseaseAT Rich SequenceGC Rich SequenceDNA-Binding ProteinsChromosome 3CpG IslandsChromosome 21Transcription FactorsCytogenetic and Genome Research
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