Search results for "complex"

showing 10 items of 5889 documents

In vitro generation of CD4+CD25+ regulatory cells from murine naive T cells

2007

CD4+ CD25+ regulatory T cells (Tregs) are crucial for the maintenance of immunological tolerance. Recent data indicate that Tregs not only develop in the thymus during ontogeny but can also differentiate from naive T cells in the periphery. The following protocol describes a method by which Tregs are generated in vitro by stimulation of naive T cells in the presence of transforming growth factor beta (Ti-Tregs). In vitro-induced regulatory T cells express markers of conventional Treg such as CD25 and the genetic program committing transcription factor FoxP3. Functionally the in vitro-generated Ti-Tregs suppress T-cell activation and proliferation while in vivo these cells have been proven t…

CD4-Positive T-LymphocytesCD3 ComplexT-Lymphocytesmedicine.medical_treatmentchemical and pharmacologic phenomenaBiologyBioinformaticsT-Lymphocytes RegulatoryGeneral Biochemistry Genetics and Molecular BiologyMiceInterleukin 21CD28 AntigensmedicineAnimalsCytotoxic T cellIL-2 receptorInterleukin 3Mice Inbred BALB CInterleukin-2 Receptor alpha SubunitFOXP3hemic and immune systemsTransfectionImmunotherapyTransforming growth factor betaCell biologyCD4 Antigensbiology.proteinBiomarkersNature Protocols
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Analysis of parathyroid graft rejection suggests alloantigen-specific production of nitric oxide by iNOS-positive intragraft macrophages

2009

Abstract Background During acute rejection of organ or tissue allografts T cells and macrophages are dominant infiltrating cells. CD4-positive T cells are important for the induction of allograft rejection and macrophages are important effector cells mediating cytotoxicity via production of nitric oxide (NO) by the inducible NO-synthase (iNOS). In the present study we analysed whether the destruction of primarily nonvascularised parathyroid allografts is also mediated by iNOS-positive macrophages. Methods Hypocalcaemic Lewis rats received parathyroid isografts (from Lewis donors) and allografts (from Wistar Furth donors), respectively, under the kidney capsule. Levels of serum calcium above…

CD4-Positive T-LymphocytesGraft RejectionMaleImmunologyThyroid GlandNitric Oxide Synthase Type IIRats Inbred WFInflammationCell CommunicationLymphocyte ActivationMajor histocompatibility complexNitric oxidechemistry.chemical_compoundAntigenCell MovementHistocompatibility AntigensmedicineAnimalsTransplantation HomologousImmunology and AllergyCytotoxic T cellMacrophageTransplantationbiologyChemistryMacrophage ActivationAntigens DifferentiationPeptide FragmentsRatsEnzyme ActivationTransplantationMononuclear cell infiltrationGene Expression RegulationRats Inbred LewImmunologyDisease ProgressionMacrophages Peritonealbiology.proteinCalciumImmunizationmedicine.symptomTransplant Immunology
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MHC class II tetramer guided detection of Mycobacterium tuberculosis-specific CD4+ T cells in peripheral blood from patients with pulmonary tuberculo…

2007

Novel diagnostic tools are needed to diagnose latent infection and to provide biologically meaningful surrogate markers to define cellular immune responses against Mycobacterium tuberculosis (MTB). Interferon gamma-based assays have recently been developed in addition to the more than 100-year-old tuberculin skin test (TST) for the immune diagnosis of MTB in blood. The advent of soluble MHC/peptide tetramer molecules allows to objectively enumerate antigen-specific T cells. We identified novel MHC class II-restricted MTB epitopes and used HLA-DR4 tetrameric complexes to visualize ex vivo CD4(+) T cells directed against the antigens Ag85B and the 19-kDa lipoprotein, shared between MTB and ot…

CD4-Positive T-LymphocytesImmunologyMolecular Sequence DataEpitopes T-Lymphocytechemical and pharmacologic phenomenaMajor histocompatibility complexEpitopeImmune systemAntigenMHC class IHumansAmino Acid SequenceTuberculosis PulmonaryMHC class IIAntigen PresentationAntigens BacterialbiologyHistocompatibility Antigens Class IICD28General MedicineMycobacterium tuberculosisrespiratory systembacterial infections and mycosesVirologyImmunologybiology.proteinCD8Scandinavian journal of immunology
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Ci8 short, a novel LPS-induced peptide from the ascidian Ciona intestinalis,modulates responses of the human immune system

2017

The selective modulation of immunity is an emerging concept driven by the vast advances in our understanding of this crucial host defense system. Invertebrates have raised researchers’ interest as potential sources of new bioactive molecules owing to their antibacterial, anticancer and immunomodulatory activities. A LipoPolySaccharide (LPS) challenge in the ascidian Ciona intestinalis generates the transcript, Ci8 short, with cisregulatory elements in the 3′ UTR region that are essential for shaping innate immune responses. The derived amino acidic sequence in silico analysis showed specific binding to human Major Histocompatibility Complex (MHC) Class I and Class II alleles. The role of Ci…

CD4-Positive T-LymphocytesLipopolysaccharides0301 basic medicineUntranslated regionImmunologyReceptors Antigen T-CellLymphocyte ActivationMajor histocompatibility complexInterferon-gamma03 medical and health sciences0302 clinical medicineImmune systemAnimalsHumansImmunology and AllergyCiona intestinalisClonal Selection Antigen-Mediated3' Untranslated RegionsCells CulturedCell ProliferationGeneticsZAP-70 Protein-Tyrosine KinaseInnate immune systembiologyThree prime untranslated regionT-cell receptorHematologyAcquired immune systembiology.organism_classificationHuman PBMCs Adaptive immunityT cellsImmunity InnateCiona intestinalisCell biology030104 developmental biologyLeukocytes Mononuclearbiology.proteinAntimicrobial Cationic Peptides030215 immunology
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Glycoprotein 96-activated dendritic cells induce a CD8-biased T cell response.

2005

Heat shock proteins (Hsps) are able to induce protective immune responses against pathogens and tumors after injection into immunocompetent hosts. The activation of components of the adaptive immune system, including cytotoxic T lymphocytes specific for pathogen- or tumor-derived peptides, is crucial for the establishment of immuno- protection. Hsps acquire these peptides during intracellular protein degradation and when released during necrotic cell death, facilitate their uptake and Minor Histocompatibility Complex (MHC)-restricted representation by professional antigen-presenting cells (APCs). In addition, the interaction of Hsps with APCs, including the Endoplasmatic Reticulum (ER)-resi…

CD4-Positive T-LymphocytesLipopolysaccharidesAntigen-Presenting CellsBone Marrow CellsMice TransgenicReceptors Cell SurfaceBiologyCD8-Positive T-LymphocytesMajor histocompatibility complexLymphocyte ActivationBiochemistryMiceImmune systemHeat shock proteinCytotoxic T cellAnimalsHumansAntigen-presenting cellCells CulturedMembrane GlycoproteinsToll-Like ReceptorsCell DifferentiationCell BiologyDendritic cellDendritic CellsOriginal ArticlesAcquired immune systemLymphocyte SubsetsCell biologyMice Inbred C57BLToll-Like Receptor 4biology.proteinInflammation MediatorsCD8Signal TransductionCell stresschaperones
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Co-activation of naive CD4+ T cells and bone marrow-derived mast cells results in the development of Th2 cells

1995

Activation of naive dense CD4+ T cells by plate-bound anti-CD3 antibodies favors the development of Th1 cells which, upon re-stimulation, produce significant amounts of IFN-gamma but no IL-4. However, co-activation of such naive T cells in the presence of IgE [anti-dinitrophenyl (DNP)]-loaded bone marrow-derived mast cells (BMMC) on plates coated with anti-CD3 antibodies and DNP-BSA led to the development of IL-4-producing Th2 cells. The same result could be observed if irradiated (800 rad) BMMC were applied as co-stimulators. Moreover, BMMC could be replaced by the supernatant of IgE-activated BMMC suggesting that a soluble mediator, presumably IL-4, was responsible for this effect. This a…

CD4-Positive T-LymphocytesMaleCD3 ComplexT cellImmunologyBone Marrow CellsLymphocyte ActivationMiceInterleukin 21Th2 CellsmedicineAnimalsImmunology and AllergyCytotoxic T cellMast CellsIL-2 receptorCells CulturedInterleukin 3Mice Inbred BALB CReceptors IgEChemistryIonomycinDegranulationGeneral MedicineMolecular biologyInterleukin 33medicine.anatomical_structureImmunologyInterleukin 12CytokinesFemaleInterleukin-4International Immunology
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PD-1 signalling in CD4+T cells restrains their clonal expansion to an immunogenic stimulus, but is not critically required for peptide-induced tolera…

2010

Summary The ultimate outcome of T-cell recognition of peptide–major histocompatibility complex (MHC) complexes is determined by the molecular context in which antigen presentation is provided. The paradigm is that, after exposure to peptides presented by steady-state dendritic cells (DCs), inhibitory signals dominate, leading to the deletion and/or functional inactivation of antigen-reactive T cells. This has been utilized in a variety of models providing peptide antigen in soluble form in the absence of adjuvant. A co-inhibitory molecule of considerable current interest is PD-1. Here we show that there is the opportunity for the PD-1/PD-L1 interaction to function in inhibiting the T-cell r…

CD4-Positive T-LymphocytesOvalbuminTransgeneProgrammed Cell Death 1 ReceptorImmunologyAntigen presentationMice TransgenicCell SeparationCD8-Positive T-LymphocytesBiologyLymphocyte ActivationMajor histocompatibility complexMiceImmune systemBlocking antibodyImmune ToleranceAnimalsImmunology and AllergyT-cell receptorOriginal ArticlesFlow CytometryAntigens DifferentiationPeptide FragmentsCell biologyMice Inbred C57BLTolerance inductionPhenotypeImmunologybiology.proteinCD8Signal TransductionImmunology
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Tcgfiii/p40 is produced by naive murine cd4+ t cells but is not a general t cell growth factor*

1989

Several antigen-specific T cell lines were found to secrete a lymphokine upon activation by antigen or lectin that was provisionally termed T cell growth factor III (TCGF III) because it induced the proliferation of a CD4+ T cell clone independently from IL2 and IL4. Amino acid sequence analysis (and the functional properties of TCGF III) revealed that TCGF III was identical with a recently identified lymphokine termed P40. TCGF III/P40 was not only produced by long-term cultured T cell lines but also upon stimulation of freshly isolated Mlsa-reactive T cells. In addition, naive CD4+ T cells secreted TCGF III/P40 upon activation by lectin or allo-major histocompatibility complex structures.…

CD4-Positive T-LymphocytesT cellMolecular Sequence DataImmunologyMice Inbred StrainsBiologyMajor histocompatibility complexCell LineMiceAntigenmedicineAnimalsImmunology and AllergyCytotoxic T cellInterleukin 9Amino Acid SequenceGrowth SubstancesInterleukin 4GlycoproteinsLymphokinesInterleukin-9LymphokineT-Lymphocytes Helper-InducerT lymphocyteVirologyMolecular biologymedicine.anatomical_structurebiology.proteinInterleukin-2Interleukin-4Lymphocyte Culture Test MixedEuropean Journal of Immunology
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Highly focused T cell responses in latent human pulmonary Mycobacterium tuberculosis infection.

2005

Abstract The elucidation of the molecular and immunological mechanisms mediating maintenance of latency in human tuberculosis aids to develop more effective vaccines and to define biologically meaningful markers for immune protection. We analyzed granuloma-associated lymphocytes (GALs) from human lung biopsies of five patients with latent Mycobacterium tuberculosis (MTB) infection. MTB CD4+ and CD8+ T cell response was highly focused in the lung, distinct from PBL, as assessed by TCR-CDR3 spectratyping coupled with a quantitative analysis of TCR VB frequencies. GALs produced IFN-γ in response to autologous macrophages infected with MTB and to defined MTB-derived HLA-A2-presented peptides Ag…

CD4-Positive T-LymphocytesT cellReceptors Antigen T-Cell alpha-betaImmunologyAntigen presentationMolecular Sequence DataEpitopes T-Lymphocytechemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesEpitopeMycobacterium tuberculosisInterferon-gammaAntigenBacterial ProteinsMHC class IHLA-A2 AntigenmedicineImmunology and AllergyHumansAmino Acid SequenceTuberculosis PulmonaryAntigen PresentationAntigens BacterialGranulomaMacrophagesT-cell receptorMycobacterium tuberculosisTh1 Cellsbacterial infections and mycosesbiology.organism_classificationVirologyPeptide FragmentsClone Cellsmedicine.anatomical_structureReceptor-CD3 Complex Antigen T-CellImmunologybiology.proteinCytokinesCD8Protein BindingJournal of immunology (Baltimore, Md. : 1950)
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Increased antigen presentation efficiency by coupling antigens to MHC class I trafficking signals.

2007

Abstract Genetic modification of vaccines by linking the Ag to lysosomal or endosomal targeting signals has been used to route Ags into MHC class II processing compartments for improvement of CD4+ T cell responses. We report in this study that combining an N-terminal leader peptide with an MHC class I trafficking signal (MITD) attached to the C terminus of the Ag strongly improves the presentation of MHC class I and class II epitopes in human and murine dendritic cells (DCs). Such chimeric fusion proteins display a maturation state-dependent subcellular distribution pattern in immature and mature DCs, mimicking the dynamic trafficking properties of MHC molecules. T cell response analysis in…

CD4-Positive T-LymphocytesT cellRecombinant Fusion ProteinsImmunologyAntigen presentationMolecular Sequence DataMice Inbred StrainsCD8-Positive T-LymphocytesProtein Sorting SignalsMajor histocompatibility complexTransfectionViral Matrix ProteinsEpitopesMiceAntigens NeoplasmMHC class ImedicineImmunology and AllergyAnimalsHumansAmino Acid SequenceAntigensMHC class IIAntigen PresentationbiologyAntigen processingHistocompatibility Antigens Class IVaccinationMembrane ProteinsDendritic CellsMHC restrictionPhosphoproteinsCell biologyProtein Transportmedicine.anatomical_structurebiology.proteinCD8Journal of immunology (Baltimore, Md. : 1950)
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