Search results for "contributes"

showing 7 items of 7 documents

Virtualization of work in global supply chains

2016

ABSTRACT. Background: The paper is devoted to the notion and benefits of implementing virtual work in global supply chains. Virtual work must be understood as an intentional activity of a human being, aimed at rendering services (tangible and intangible), by means of ITC tools, performed in a distance from the traditional place of work, in a mobile manner. The empirical research were conducted on the basis of 4 case studies of global leaders of supply chains, which in accordance with M. Fisher's classification, represent two types. The case studies confirmed the positive influence of virtual work both in effective and flexible supply chains. Favourable market and technological conditions an…

supply chain managementbut it also creates possibilities of better usage of own resourcenew methods of work. INTRODUCTION Global economic crisis significantly increases the pressure on reduction of cost and risk connected with the functioning of global supply chains. Among many ways to reduce the cost and the uncertainty of demand for the products of supply chain the essential one is the increase of information transparency due to modern ICT solutions. The popularisation of SCM class business apps not only contributes to the increase of the scope of logistic and production outsourcingvirtual workeffective supply chainsupply chainflexible supply chainLogForum
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VEGF-B-induced vascular growth leads to metabolic reprogramming and ischemia resistance in the heart

2014

Abstract Angiogenic growth factors have recently been linked to tissue metabolism. We have used genetic gain‐ and loss‐of function models to elucidate the effects and mechanisms of action of vascular endothelial growth factor‐B (VEGF‐B) in the heart. A cardiomyocyte‐specific VEGF‐B transgene induced an expanded coronary arterial tree and reprogramming of cardiomyocyte metabolism. This was associated with protection against myocardial infarction and preservation of mitochondrial complex I function upon ischemia‐reperfusion. VEGF‐B increased VEGF signals via VEGF receptor‐2 to activate Erk1/2, which resulted in vascular growth. Akt and mTORC1 pathways were upregulated and AMPK downregulated, …

VEGF‐Bmedicine.medical_specialtyMedicine (General)AngiogenesiseducationMOUSE HEARTIschemiaVEGF-B610 Medicine & healthmTORC1ischemiaBiologyQH426-470CONTRIBUTESchemistry.chemical_compoundangiogenesisR5-920CARDIAC-FUNCTIONInternal medicinemedicineGeneticsFAILUREta318Myocardial infarctionFATTY-ACID UPTAKEREPERFUSION INJURY610 Medicine & healthProtein kinase BMYOCARDIAL HYPERTROPHYAMPKta3121medicine.diseaseCell biologyARTERIOGENESISVascular endothelial growth factorMICEEndocrinologychemistry3121 General medicine internal medicine and other clinical medicineendothelial cellMolecular Medicine3111 BiomedicineReperfusion injurymetabolism
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Encuentro de saberes: Huellas de memoria pluriversa y descolonización de la Universidad contemporánea

2018

This paper studies the Encounter of Knowledge as a project that, by welcoming mestres and mestras of ancestral knowledge into universities, contributes to decolonize higher education, broadening its field of memory. Based on a systematization exercise, we established a theoretical dialogue with Bajtin and Bejamin. Based on the participation of four mestres -Lucely Pío, raicera del Cerrado, Mãe Lu and Tata Mutá Imé, religious leaders of Candomblé and the political leader and shaman Álvaro Tukano of the Tukano nation- we show how the project promotes in the contemporary Brazilian university a space of mnemic pluriversity, capable of activating diverse narrative planes (biographical, political…

raicera del Cerradopluriversidad mnémicacapable of activating diverse narrative planes (biographicalreligious leaders of Candomblé and the political leader and shaman Álvaro Tukano of the Tukano nation- we show how the project promotes in the contemporary Brazilian university a space of mnemic pluriversityspiritualbroadening its field of memory. Based on a systematization exerciseuniversityMeeting of knowledgeUNESCO::SOCIOLOGÍAJosé JorgeMãe Lu and Tata Mutá IméUniversidadBrasilBrazil 143 150narraciónHuellas de memoria pluriversa y descolonización de la Universidad contemporánea de Carvalho [1137-7038 8537 Arxius de sociologia 514142 2018 39 6874492 Encuentro de saberes]:SOCIOLOGÍA [UNESCO]we established a theoretical dialogue with Bajtin and Bejamin. Based on the participation of four mestres -Lucely Píoinclusiónmnemic diversityFlóreznarrationJuliana This paper studies the Encounter of Knowledge as a project thatinclusioncolonialidadcolonialitypoliticalby welcoming mestres and mestras of ancestral knowledge into universitiescontributes to decolonize higher education1137-7038 8537 Arxius de sociologia 514142 2018 39 6874492 Encuentro de saberes: Huellas de memoria pluriversa y descolonización de la Universidad contemporánea de Carvalhoancestral and mythical) and widening its epistemic horizon. Encuentro de saberes
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Prognostic value of the interaction between galectin-3 and antigen carbohydrate 125 in acute heart failure

2015

AIM:Galectin-3 (Gal-3) and carbohydrate antigen 125 (CA125) have emerged as robust prognostic biomarkers in heart failure. Experimental data have also suggested a potential molecular interaction between CA125 and Gal-3; however, the biological and clinical relevance of this interaction is still uncertain. We sought to evaluate, in patients admitted for acute heart failure, the association between plasma Gal-3 with all-cause mortality and the risk for rehospitalizations among high and low levels of CA125. METHODS AND RESULTS: We included 264 consecutive patients admitted for acute heart failure to the Cardiology Department in a third-level center. Both biomarkers were measured on admission. …

Maleendocrine system diseasesGalectin 3lcsh:MedicineRisk FactorsBlood plasmaANTIGEN CARBOHYDRATE 125lcsh:ScienceAged 80 and overMultidisciplinaryca125Mortality rateBlood Proteins//purl.org/becyt/ford/3.1 [https]Middle AgedPrognosisMedicina BásicaGalectin-3Female//purl.org/becyt/ford/3 [https]Research Articlemedicine.medical_specialtyCIENCIAS MÉDICAS Y DE LA SALUDcontributesGalectinsInmunologíaPatient ReadmissionAntigenInternal medicinemedicineHumanssurfaceClinical significanceIn patientIntensive care medicineAgedProportional Hazards ModelsHeart FailureGALECTIN-3business.industryProportional hazards modellcsh:Rassociationmedicine.diseaseACUTE HEART FAILUREstatin therapyinflammationCA-125 AntigenHeart failurecellslcsh:QbusinessBiomarkersFollow-Up Studies
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DNA Methylation and Non-Coding RNAs during Tissue-Injury Associated Pain.

2022

While about half of the population experience persistent pain associated with tissue damages during their lifetime, current symptom-based approaches often fail to reduce such pain to a satisfactory level. To provide better patient care, mechanism-based analgesic approaches must be developed, which necessitates a comprehensive understanding of the nociceptive mechanism leading to tissue injury-associated persistent pain. Epigenetic events leading the altered transcription in the nervous system are pivotal in the maintenance of pain in tissue injury. However, the mechanisms through which those events contribute to the persistence of pain are not fully understood. This review provides a summar…

INFLAMMATORY PAINRNA UntranslatedChemistry MultidisciplinaryAdaptation BiologicalReviewUP-REGULATIONEpigenesis GeneticCpG islandsTranscripció genèticalncRNANeurociènciesnociceptionBiology (General)SpectroscopyGENE-EXPRESSIONGeneral MedicineComputer Science ApplicationsChemistryPhysical SciencesDisease SusceptibilityChronic PainLife Sciences & BiomedicineepigeneticALLEVIATES NEUROPATHIC PAINBiochemistry & Molecular Biologydorsal root ganglionQH301-705.50699 Other Biological SciencesCatalysisCONTRIBUTESInorganic ChemistryDiagnosis DifferentialCENTRAL SENSITIZATION0399 Other Chemical SciencesHumansPhysical and Theoretical ChemistryQD1-999Molecular Biologyspinal dorsal hornmiRNACHRONIC CONSTRICTION INJURYneuropathic pain0604 GeneticsScience & TechnologyChemical PhysicsNERVE INJURYMICRORNAGene Expression ProfilingOrganic ChemistryDNA MethylationCPG-BINDING PROTEIN-2gene transcriptionGene Expression RegulationsiRNARNAWounds and InjuriesBiomarkersInternational journal of molecular sciences
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Loss of histone macroH2A1 in hepatocellular carcinoma cells promotes paracrine-mediated chemoresistance and CD4+CD25+FoxP3+ regulatory T cells activa…

2020

Rationale: Loss of histone macroH2A1 induces appearance of cancer stem cells (CSCs)-like cells in hepatocellular carcinoma (HCC). How CSCs interact with the tumor microenvironment and the adaptive immune system is unclear. Methods: We screened aggressive human HCC for macroH2A1 and CD44 CSC marker expression. We also knocked down (KD) macroH2A1 in HCC cells, and performed integrated transcriptomic and secretomic analyses. Results: Human HCC showed low macroH2A1 and high CD44 expression compared to control tissues. MacroH2A1 KD CSC-like cells transferred paracrinally their chemoresistant properties to parental HCC cells. MacroH2A1 KD conditioned media transcriptionally reprogrammed parental …

EXPRESSION0301 basic medicineLIVERAdaptive immune systemPOSTTRANSCRIPTIONAL CONTROLHepatocellular carcinomaMedicine (miscellaneous)PROGRESSIONHistone macroH2A1Research & Experimental MedicineCONTRIBUTES03 medical and health sciencesParacrine signalling0302 clinical medicineadaptive immune systemCancer stem cellCANCER STEM-CELLSmedicinechemoresistance.TRANSCRIPTIONIL-2 receptorneoplasmsPharmacology Toxicology and Pharmaceutics (miscellaneous)Tumor microenvironmentScience & Technologybiologyhistone macroH2A1CD44PROLIFERATIONchemoresistanceCancerFOXP3hepatocellular carcinomamedicine.diseaseAcquired immune systemdigestive system diseases3. Good healthCYTOKINE030104 developmental biologyMedicine Research & ExperimentalSENESCENCE030220 oncology & carcinogenesisCancer researchbiology.proteinLife Sciences & BiomedicineChemoresistance
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The antifibrotic potential of a sustained release formulation of a PDGF beta-receptor targeted rho kinase inhibitor

2019

Rho kinase activity in hepatic stellate cells (HSCs) is associated with activation, transformation and contraction of these cells, leading to extracellular matrix production and portal hypertension in liver cirrhosis. Inhibition of rho kinase activity can reduce these activities, but may also lead to side effects, for instance systemic hypotension. This can be circumvented by liver-specific delivery of a rho kinase inhibitor to effector cells. Therefore, we targeted the rho kinase inhibitor Y27632 to the key pathogenic cells in liver fibrosis, i.e. myofibroblasts including activated HSCs that highly express the PDGF beta-receptor, using the drug carrier pPB-MSA. This carrier consists of mou…

Liver CirrhosisDrug targetingPyridinesPolymeric microspheresPharmaceutical Science02 engineering and technologyPharmacologychemistry.chemical_compoundY-27632FibrosisControlled releaseRho-associated protein kinaseMice Knockout0303 health sciencesDrug Carriersrho-Associated KinasesChemistryCIRRHOTIC RATS021001 nanoscience & nanotechnologyMicrospheresY-27632Drug deliveryFemale0210 nano-technologyDrug carrierATP Binding Cassette Transporter Subfamily BSIGNALING CONTRIBUTESLiver fibrosisBiologicalsHEPATIC STELLATE CELLSCell LineMECHANISMSReceptor Platelet-Derived Growth Factor beta03 medical and health sciencesDELIVERYROCK INHIBITORmedicineAnimalsHumansProtein Kinase Inhibitors030304 developmental biologyProtein deliveryPORTAL PRESSUREmedicine.diseaseAmidesTargeted drug deliveryRho kinase inhibitorDelayed-Action PreparationsHepatic stellate cellVASODILATIONJournal of Controlled Release
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