Search results for "cytoskeleton"

showing 10 items of 272 documents

Regulatory T cell-derived adenosine induces dendritic cell migration through the Epac-Rap1 pathway.

2014

Abstract Dendritic cells (DC) are one target for immune suppression by regulatory T cells (Treg), because their interaction results in reduced T cell stimulatory capacity and secretion of inhibitory cytokines in DC. We show that DC in the presence of Treg are more mobile as compared with cocultures with conventional CD4+ T cells and form DC–Treg aggregates within 2 h of culture. The migration of DC was specifically directed toward Treg, as Treg, but not CD4+ T cells, attracted DC in Boyden chambers. Treg deficient for the ectonucleotidase CD39 were unable to attract DC. Likewise, addition of antagonists for A2A adenosine receptors abolished the formation of DC–Treg clusters, indicating a ro…

AdenosineRegulatory T cellT cellImmunologyMedizinchemical and pharmacologic phenomenaCell CommunicationBiologyT-Lymphocytes RegulatoryMiceAdenosine TriphosphateAntigens CDCell MovementmedicineImmunology and AllergyAnimalsGuanine Nucleotide Exchange FactorsDendritic cell migrationReceptors Adenosine A2Apyraserap1 GTP-Binding Proteinshemic and immune systemsDendritic CellsActin cytoskeletonAdenosineAdenosine receptorCell biologyActin Cytoskeletonmedicine.anatomical_structureRap1Signal transductionmedicine.drugSignal TransductionJournal of immunology (Baltimore, Md. : 1950)
researchProduct

Potential effects of age-associated oxidative stress on mammalian oocytes/embryos

1996

This bioessay aims to explain the different effects of maternal ageing and postovulatory oocyte ageing on mammalian oocytes/embryos under the scope of 'the oxygen radical-mitochondrial injury hypothesis of ageing'. This hypothesis assumes a key role in the senescent process of oxygen radical damage to mitochondrial DNA, proteins and lipids. It is proposed that a decrease in intracellular ATP concentrations and glutathione (GSH)/glutathione disulphide (GSSG) ratio together with a concomitant increase in cytosolic Ca2+ are major factors causing the observed detrimental effects of ageing on cytoskeletal fibres, fertilization and embryo development.

AdultFetal ProteinsEmbryologyBiologymedicine.disease_causeDNA MitochondrialCongenital AbnormalitiesMicechemistry.chemical_compoundAdenosine TriphosphateNeoplasmsGeneticsmedicineAnimalsHumansMolecular BiologyCellular SenescenceCytoskeletonMammalsEgg ProteinsEmbryogenesisObstetrics and GynecologyEmbryoCell BiologyGlutathioneEmbryo MammalianOocyteGlutathioneCell biologyOxidative StressCytosolFertilitymedicine.anatomical_structureReproductive MedicineBiochemistrychemistryAgeingFertilizationOocytesReactive Oxygen SpeciesOxidation-ReductionIntracellularOxidative stressMaternal AgeDevelopmental BiologyMolecular Human Reproduction
researchProduct

Hereditary motor sensory neuropathy type II with neurofilament accumulation: new finding or new disorder?

1985

Peroneal muscular atrophy is now known to be heterogeneous and to be due to various underlying genetic mechanisms. Exploring this heterogeneity further, we report on a German kinship with the clinical, genetic, and nerve conduction features of hereditary motor and sensory neuropathy type II (HMSN type II) but whose sural nerves on biopsy were found to show infrequent axonal swellings with neurofilament accumulations not previously described. The dominant inheritance and absence of kinky hair set this disorder apart from giant axonal neuropathy. There was no history of toxic exposure to industrial chemicals. We conclude that the disorder either is a new type of HMSN or is HMSN type II with p…

AdultMaleNeurofilamentAdolescentBiopsyCardiomyopathyNeural ConductionCardiovascular System03 medical and health sciences0302 clinical medicineSural NerveEvoked Potentials SomatosensoryBiopsymedicineHumansAxonHereditary Sensory and Autonomic NeuropathiesChildCytoskeleton030304 developmental biologyGiant axonal neuropathyAged0303 health sciencesmedicine.diagnostic_testbusiness.industryElectromyographyPeroneal muscular atrophyMiddle Agedmedicine.diseaseAxonsPedigreeMicroscopy ElectronMuscular Atrophymedicine.anatomical_structureNeurologyEvoked Potentials VisualFemaleNeurology (clinical)AbnormalityHereditary motor and sensory neuropathybusinessNeuroscience030217 neurology & neurosurgeryAnnals of neurology
researchProduct

Characterization of target antigens from anti-neutrophil cytoplasmic antibodies in autoimmune hepatitis type-I.

1997

The occurrence of anti-neutrophil cytoplasmic antibodies (ANCA) has been described in sera of patients with autoimmune hepatitis (AIH). The significance of this finding remains uncertain and the nature of the target antigen(s) has not yet been defined. We studied 32 sera from patients with AIH type-I and prepared extracts of human neutrophils to identify the target antigen(s). A 43 kDa dominant immunoreactive protein was found and identified as the cytoskeletal component actin. Initial studies to define the antigenic determinants identified three different actin domains.

AdultMaleNeutrophilsClinical BiochemistryBlotting WesternMolecular Sequence DataFluorescent Antibody TechniqueAutoimmune hepatitisBiologyBiochemistryAnalytical ChemistryAntibodies Antineutrophil CytoplasmicAutoimmune DiseasesHepatitisEpitopesAntigenimmune system diseasesmedicineTumor Cells CulturedAnimalsHumansAmino Acid SequenceAntigensCytoskeletonAnti-neutrophil cytoplasmic antibodyAgedAged 80 and overMiddle Agedmedicine.diseaseRatsBlotEpitope mappingCytoplasmImmunologybiology.proteinFemaleAntibodyElectrophoresis
researchProduct

Correlation of anti-cytoskeleton antibody activities in synovial fluid with interleukin-6 in patients with osteoarthritis and inflammatory joint dise…

1990

Synovial fluids and sera from patients with rheumatoid arthritis, psoriatic arthritis, yersinia arthritis, Behçet's syndrome, Crohn's disease, and osteoarthritis were tested for antinuclear antibodies and antibodies to five cytoskeletal components in sensitive enzyme-linked immunosorbent assay (ELISA) systems and for IL-6 concentrations in a proliferation assay (IL-6 dependent hybridoma cell line B13.29, subclone B9). Statistically significant correlations between antibody activities and IL-6 levels were found for vimentin antibodies (r = 0.56; p less than 0.05) and actin antibodies (r = 0.44; p less than 0.05). In patients with chronic and active disease like rheumatoid arthritis and psori…

AdultMalePathologymedicine.medical_specialtyAnti-nuclear antibodyArthritisEnzyme-Linked Immunosorbent AssayVimentinOsteoarthritisPsoriatic arthritisOsteoarthritisSynovial FluidDrug DiscoveryHumansVimentinMedicineSynovial fluidInterleukin 6Cells CulturedCytoskeletonGenetics (clinical)AgedInflammationbiologyInterleukin-6business.industrySynovial MembraneGeneral MedicineMiddle Agedmedicine.diseaseActinsAntibodies AntinuclearRheumatoid arthritisImmunologybiology.proteinMolecular MedicineFemaleJoint DiseasesbusinessKlinische Wochenschrift
researchProduct

Intermediate filaments and desmosomal plaque proteins in testicular seminomas and non-seminomatous germ cell tumours as revealed by immunohistochemis…

1987

Seminomas and non-seminomatous testicular germ cell tumours were studied for the presence of cytokeratin and vimentin filaments and desmosomes using immunohistochemical methods. In the majority of the classical seminomas and in seminomatous areas of mixed tumours most tumour cells appeared to lack cytokeratin filaments. Some seminomas contained a focally variable proportion of cells exhibiting cytokeratin-positive structures while other cases contained only few seminoma cells with a well developed fibrillar cytokeratin network. Gel electrophoresis of cytoskeletal proteins from microdissected regions revealed cytokeratin polypeptides nos. 8 and 18 typical of simple epithelia. In one seminoma…

AdultMalePathologymedicine.medical_specialtyendocrine system diseasesAdolescentIntermediate FilamentsFluorescent Antibody TechniqueVimentinmacromolecular substancesAntigen-Antibody ComplexDysgerminomaBiologyurologic and male genital diseasesAntibodiesPathology and Forensic MedicineEmbryonal carcinomaCytokeratinTesticular NeoplasmsmedicineCarcinomaHumansVimentinIntermediate filamentMolecular BiologyCytoskeletonDesmoplakinMembrane ProteinsCell BiologyGeneral MedicineSeminomaDesmosomesmedicine.diseaseCytoskeletal ProteinsMicroscopy Electronmedicine.anatomical_structureDesmoplakinsbiology.proteinKeratinsGerm cellVirchows Archiv. A, Pathological anatomy and histopathology
researchProduct

Desmin-related myopathies

1997

Desmin-related myopathies are marked by accumulation of desmin, which is often familial and associated with cardiomyopathy. When multifocal this excess is characterized by inclusions such as cytoplasmic or spheroid bodies, when disseminated the excess is called granulofilamentous material. Excess of desmin might represent an abnormal type of protein metabolism.

AdultPathologymedicine.medical_specialtyGranulofilamentous materialCardiomyopathyChromosome DisordersGenes Recessivemacromolecular substancesBiologyDesminMuscular DiseasesmedicineHumansChildMuscle SkeletalGenotype-Phenotype CorrelationsGenes DominantChromosome AberrationsInclusion BodiesDESMIN-RELATED MYOPATHYMyocardiumMolecular pathogenesismusculoskeletal systemmedicine.diseaseActin CytoskeletonNeurologyCytoplasmDesminNeurology (clinical)CardiomyopathiesCurrent Opinion in Neurology
researchProduct

Cytoskeletal Heterogeneity of an Epithelioid Sarcoma with Expression of Vimentin, Cytokeratins, and Neurofilaments

1990

We studied an unusual sarcoma with morphologic features diagnostic of epithelioid sarcoma by conventional light microscopy, transmission electron microscopy, and immunohistochemistry. The primary tumor, which was located in the deep soft tissues of the buttock of a 32-year-old woman, and its metastases to lymph nodes, liver, and lung were available for investigation. The histomorphological and ultrastructural appearance of the primary tumor and its metastatic deposits were typical of epithelioid sarcoma. Immunohistochemistry revealed a strong and uniform reactivity for vimentin in both the primary tumor and its metastases. In contrast, a marked cytoskeletal heterogeneity became evident for …

AdultPathologymedicine.medical_specialtyLung NeoplasmsNeurofilamentEpithelioid sarcomaIntermediate FilamentsVimentinBiologyEndoplasmic ReticulumPathology and Forensic MedicinemedicineHumansVimentinIntermediate filamentCytoskeletonLiver NeoplasmsSarcomamedicine.diseaseImmunohistochemistryPrimary tumorMicroscopy ElectronLymphatic Metastasisbiology.proteinButtocksKeratinsImmunohistochemistryFemaleSurgeryLymph NodesSarcomaLymphAnatomyThe American Journal of Surgical Pathology
researchProduct

Fetal akinesia caused by a novel actin filament aggregate myopathy skeletal muscle actin gene (ACTA1) mutation.

2010

We report a female newborn, diagnosed with fetal akinesia in utero, who died one hour after birth. Post-mortem muscle biopsy demonstrated actin-filament myopathy based on immunolabelling for sarcomeric actin, and large areas of filaments, without rod formation, ultrastructurally. Analysis of DNA extracted from the muscle disclosed a novel de novo heterozygous c.44G>A, GGC>GAC, 'p.Gly15Asp' mutation in the ACTA1 gene. Analysis of the location of the mutated amino-acid in the actin molecule suggests the mutation most likely causes abnormal nucleotide binding, and consequent pathological actin polymerization. This case emphasizes the association of fetal akinesia with actin-filament myopathy.

AdultSarcomeresmacromolecular substancesBiologymedicine.disease_causeSarcomereNemaline myopathyPregnancymedicineHumansMyopathyMuscle SkeletalGenetics (clinical)ActinMutationMuscle biopsymedicine.diagnostic_testMicrofilament ProteinsInfant NewbornSkeletal muscleDNANeuromuscular DiseasesActin cytoskeletonmedicine.diseaseMolecular biologyActin CytoskeletonFetal Diseasesmedicine.anatomical_structureNeurologyBiochemistryPediatrics Perinatology and Child HealthMutationFemaleNeurology (clinical)medicine.symptomNeuromuscular disorders : NMD
researchProduct

Effects of Parietaria judaica pollen extract on human microvascular endothelial cells

2008

Abstract Pollinosis from Parietaria judaica is one of the main causes of allergy in the Mediterranean area. The present study is designed to assess if P. judaica pollens contain bioactive compounds able to elicit a functional response in endothelial cells. We have demonstrated that addition of pollen extract to human lung microvascular endothelial cells (HMVEC-L) induces a modification of cell morphology, actin cytoskeletal rearrangements and an increase in endothelial cell permeability. We further showed that the treatment of endothelial cells with pollen extract causes an increase of E-selectin and VCAM-1 protein levels as well as an increase of IL-8 production. The stimulation of cell–ce…

AllergyNeutrophilsBiophysicsVascular Cell Adhesion Molecule-1parietaria judaicaBiologymedicine.disease_causeCell morphologyBiochemistryPermeabilitycell adhesion moleculesPollenCell AdhesionmedicineHumansProtease InhibitorsCytoskeletonLungMolecular BiologyCells CulturedActinPlant ExtractsCell adhesion moleculeInterleukin-8Cell BiologyAdhesionbiology.organism_classificationCapillariesCell biologyEndothelial stem cellParietariaImmunologyParietaria judaicaendothelial cellPollenEndothelium VascularE-Selectin
researchProduct