Search results for "defects"

showing 10 items of 339 documents

Esophageal atresia in newborns: a wide spectrum from the isolated forms to a full VACTERL phenotype?

2013

Background: VATER association was first described in 1972 by Quan and Smith as an acronym which identifies a non-random co-occurrence of Vertebral anomalies, Anal atresia, Tracheoesophageal fistula and/or Esophageal atresia, Radial dysplasia. It is even possible to find out Cardiovascular, Renal and Limb anomalies and the acronym VACTERL was adopted, also, embodying Vascular, as single umbilical artery, and external genitalia anomalies. Methods: Data on patients with esophageal atresia (EA) with or without tracheoesophageal fistula (TEF) admitted in the Neonatal Intensive Care Unit (NICU) between January 2003 and January 2013 were evaluated for the contingent occurrence of typical VACTERL a…

Heart Defects CongenitalMalemedicine.medical_specialtyPediatricsVATERLimb Deformities CongenitalAnal CanalTracheoesophageal fistulaKidneyNervous System MalformationsUmbilical ArteriesAssociationAnus ImperforateEsophagusSettore MED/38 - Pediatria Generale E SpecialisticaIntensive Care Units NeonatalVACTERLmedicinePrevalenceHumansEsophagusEsophageal AtresiaSicilyRetrospective StudiesCongenital malformationsSingle umbilical arterybusiness.industryTracheo-esophageal fistulaResearchSettore MED/20 - Chirurgia Pediatrica E InfantileRadial dysplasiaInfant NewbornAnomaliesSyndromeAnal canalToesmedicine.diseaseVACTERL associationSpineSurgeryTracheaEsophageal atresia; Tracheo-esophageal fistula; VATER; VACTERL; Association; Congenital malformations; Anomaliesmedicine.anatomical_structureAnal atresiaPhenotypeAtresiaCongenital malformationFemalebusiness
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THE NONCOMPACTION OF THE LEFT VENTRICULAR MYOCARDIUM: OUR PEDIATRIC EXPERIENCE

2007

OBJECTIVES: The noncompaction of the left ventricular myocardium is a rare congenital heart disease, characterized by an excessive prominence of trabecular meshwork, spaced out by deep intertrabecular recesses, consequent to the arrest of the normal myocardium embryogenesis. Although there are numerous descriptions, the physiopathological effects of the structural alterations, just like the clinical spectrum and the evolution of the disease, are not totally clarified. In the present study, we have evaluated the natural history of the disease, the familial incidence and the alterations of the systolic and diastolic function. METHODS: We collected a series of 21 young patients who were affect…

Heart Defects CongenitalMalemedicine.medical_specialtygenetic structuresHeart diseaseCardiomyopathyInternal medicineHumansMedicinecardiovascular diseasesNoncompactionChildVentricular dysfunctionUltrasonographybusiness.industryHeart Septal DefectsMyocardiumGeneral Medicinemedicine.diseaseeye diseasesmedicine.anatomical_structureChild Preschoolcardiovascular systemCardiologyLeft ventricular myocardiumFemalesense organsTrabecular meshworkTissue DopplerUltrasonographyCardiology and Cardiovascular Medicinebusiness
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Mutations in KIAA0586 Cause Lethal Ciliopathies Ranging from a Hydrolethalus Phenotype to Short-Rib Polydactyly Syndrome

2015

KIAA0586, the human ortholog of chicken TALPID3, is a centrosomal protein that is essential for primary ciliogenesis. Its disruption in animal models causes defects attributed to abnormal hedgehog signaling; these defects include polydactyly and abnormal dorsoventral patterning of the neural tube. Here, we report homozygous mutations of KIAA0586 in four families affected by lethal ciliopathies ranging from a hydrolethalus phenotype to short-rib polydactyly. We show defective ciliogenesis, as well as abnormal response to SHH-signaling activation in cells derived from affected individuals, consistent with a role of KIAA0586 in primary cilia biogenesis. Whereas centriolar maturation seemed una…

Heart Defects CongenitalMolecular Sequence DataCell Cycle ProteinsBiologyShort Rib-Polydactyly SyndromeCiliopathies03 medical and health sciencesFatal OutcomeCiliogenesisReportGLI3GeneticsmedicineHumansGenetics(clinical)Europe EasternGenetics (clinical)030304 developmental biologyGenetics0303 health sciencesLikelihood FunctionsShort rib – polydactyly syndromePolydactylyBase SequenceCilium030305 genetics & hereditySequence Analysis DNAmedicine.diseasePhenotypeHuman geneticsHedgehog signaling pathwayFounder EffectPedigreePhenotypeCodon NonsenseCentriolar satelliteErratumHand Deformities CongenitalCiliary Motility DisordersHydrocephalus
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Effects of cardiopulmonary rehabilitation on the muscle function of children with Congenital Heart Disease : a prospective cohort study

2021

Critical medical and surgical advances have led to a shift in the care and management of children with congenital heart disease (CHD). These patients present with muscle deconditioning, which negatively influences their response to exercise, functional capacities, and quality of life. This study evaluates the influence of a cardiopulmonary rehabilitation program (CPRP) on the function of peripheral musculature of children with CHD. A single-center prospective cohort study was designed. Fifteen CHD subjects, between 12 and 16 years of age, with reduced aerobic capacity on a cardiopulmonary exercise test, were included in a three-month, 24-session CPRP. Measurements of the subjects’ handgrip …

Heart Defects CongenitalMúsculos - Fuerza - Entrenamiento.medicine.medical_specialtyEjercicio físico - Uso terapéutico.Heart diseaseHealth Toxicology and MutagenesisHeart - Abnormalities - Treatment.030204 cardiovascular system & hematologyBicepsArticle03 medical and health sciences0302 clinical medicineDeconditioningQuality of lifeMuscles - Training.Corazón - Anomalías y malformaciones - Tratamiento.HumansMedicineProspective Studies030212 general & internal medicineChildMuscle SkeletalProspective cohort studyEducación física para niños.Aerobic capacityChildren - Abnormalities - Treatment.congenital abnormalitiesHand Strengthexercisebusiness.industryCardiopulmonary rehabilitationRPublic Health Environmental and Occupational Healthmedicine.diseaseAdditional researchPhysical education and training for children.cardiac rehabilitationpediatricQuality of LifePhysical therapymuscle strengthMedicineAnomalías y malformaciones en los niños - Tratamiento.resistance trainingbusinessExercise - Therapeutic use.
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Genetic syndrome suspicion: examples of clinical approach in the neonatal unit.

2010

Overgrowth syndromes: the practical clinical approach. Excessive growth can be present in a variety of medical conditions as result of abnormal fetal metabolism (i.e., maternal gestational diabetes) or of an overgrowth syndrome. Within this latter group of diseases, a LGA newborn requires a complex differential diagnosis encompassing several syndromes, such as Beckwith-Wiedemman, Sotos, Weaver, Simpson-Golabi-Behmel, Perlman, and Bannayan-Riley-Ruvalcaba. Partial or global overgrowth, other dysmorphisms, abdominal organs anomalies, as well as benign and malignant tumors are the common issues to examine for the diagnosis and the monitoring of all these disorders. The molecular bases of these…

Heart Defects CongenitalPatient Care Teamgenetic syndromesGenetic Diseases InbornInfant NewbornLimb Deformities CongenitalAnal CanalSyndromeKidneySpineCongenital AbnormalitiesFetal MacrosomiaTracheaEarly DiagnosisEsophagusSettore MED/38 - Pediatria Generale E SpecialisticaIntensive Care Units NeonatalBirth WeightHumansAbnormalities MultipleHedgehog ProteinsGenetic TestingNeonatologyEsophageal Atresiasyndrome genetic
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Cardiac pacing in premature infants and neonates: steroid eluting leads and automatic output adaptation

1999

Abstract Background . Appropriate generator and lead selection as well as techniques of implantation are most important aspects of cardiac pacing in the extremely young patient. Here we report the clinical results using a new technique with automatic output adaptation based on evoked response in combination with steroid-eluting epicardial leads in small children. Methods . One neonate and 2 premature infants underwent permanent pacemaker implantation because of congenital high-degree atrioventricular block or postoperative complete heart block, respectively. Steroid-eluting epicardial leads and a multiprogrammable pacemaker with automatic output adaptation were used. Results . Intermuscular…

Heart Defects CongenitalPulmonary and Respiratory MedicinePacemaker ArtificialCardiac pacingInfant Premature DiseasesPostoperative complete heart blockmedicineHumansLead (electronics)Bipolar leadbusiness.industryCardiac Pacing ArtificialInfant NewbornSmall childrenInfantmedicine.diseasePatient populationAnesthesiaFemaleSurgeryPermanent pacemakerCardiology and Cardiovascular MedicinebusinessAtrioventricular blockAlgorithmsInfant PrematureThe Annals of Thoracic Surgery
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Perlecan Maintains the Integrity of Cartilage and Some Basement Membranes

1999

Perlecan is a heparan sulfate proteoglycan that is expressed in all basement membranes (BMs), in cartilage, and several other mesenchymal tissues during development. Perlecan binds growth factors and interacts with various extracellular matrix proteins and cell adhesion molecules. Homozygous mice with a null mutation in the perlecan gene exhibit normal formation of BMs. However, BMs deteriorate in regions with increased mechanical stress such as the contracting myocardium and the expanding brain vesicles showing that perlecan is crucial for maintaining BM integrity. As a consequence, small clefts are formed in the cardiac muscle leading to blood leakage into the pericardial cavity and an ar…

Heart Defects Congenitalcardiac muscleMesenchymeSchwartz–Jampel syndromeRestriction MappingPerlecanBasement MembraneExtracellular matrixMiceMice CongenicchondrodysplasiaCalcification PhysiologicexencephalyLamininmedicineAnimalsNeural Tube DefectsCells CulturedBasement membranebiologyCartilageOssification HeterotopicHomozygoteCell Biologymedicine.diseaseMice Mutant StrainsBasement membrane assemblyCell biologyperlecanMutagenesis Insertionalmedicine.anatomical_structureCartilageBiochemistryGene Targetingbiology.proteinOriginal ArticleGenes LethalProteoglycansCollagenHeparitin SulfateExostoses Multiple HereditaryHeparan Sulfate ProteoglycansThe Journal of Cell Biology
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Transcatheter radiofrequency perforation and stent implantation for palliation of pulmonary atresia in a 3060-g infant

1995

In a 3060-g infant with fibromuscular pulmonary atresia an open right ventricular outflow tract was created by means of interventional cardiological methods. Following two inadvertent perforations without sequelae or clinical symptoms, radiofrequency perforation and subsequent balloon dilatation were successfully performed. The implantation of a Palmaz iliac stent led to a predictable communication between the right ventricle and pulmonary artery.

Heart Septal Defects VentricularCardiac Catheterizationmedicine.medical_specialtymedicine.medical_treatmentPerforation (oil well)Coronary Angiographymedicine.arterymedicineHumansVentricular outflow tractmedicine.diagnostic_testbusiness.industryInfant NewbornStentmedicine.diseaseSurgerymedicine.anatomical_structureEchocardiographyPulmonary AtresiaVentricleAtresiaAngiographyPulmonary arteryCatheter AblationFemaleStentsRadiologyCardiology and Cardiovascular MedicinePulmonary atresiabusinessCatheterization and Cardiovascular Diagnosis
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Congenital anomalies among live births in a high environmental risk area—A case-control study in Brindisi (southern Italy)

2013

Maternal exposure to ambient pollution has been increasingly linked to the risk of congenital anomalies (CAs) in the fetus and newborns. Recently, a descriptive study in the high environmental risk city of Brindisi (Italy) revealed an increased prevalence of total CAs, especially congenital heart disease (CHD) and ventricular septal defects (VSDs), both at the local level and in comparison with the pool of EUROCAT registries. This paper concerns a population-based case control study to investigate the association between maternal exposure to air pollutants - sulfur dioxide (SO2) and total suspended particulate (TSP) matter - and the risk of CA. Cases were newborns up to 28 days of age, born…

Heart Septal Defects VentricularCongenital anomaliesPediatricsmedicine.medical_specialtyPercentilePopulationAir pollutionAir pollutionmedicine.disease_causeBiochemistryIndustrial exposureEnvironmental riskPregnancyEnvironmental healthBrindisimedicineHumansCitieseducationGeneral Environmental Scienceeducation.field_of_studyPregnancybusiness.industryInfant NewbornCase-control studyOdds ratiomedicine.diseaseIncreased riskItalySulfur dioxideMaternal ExposureCase-Control StudiesFemaleParticulate MatterbusinessEnvironmental Research
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VATER/VACTERL association: clinical variability and expanding phenotype including laryngeal stenosis.

1992

Vertebral abnormalities and anorectal and tracheoesophageal defects are the main manifestations in the VATER/VACTERL association. Radial defects vary from radial aplasia to thumb duplication. Heart and renal defects are reported with lower frequency. Additional malformations, such as the laryngeal stenosis described in the present patient, may expand the phenotype of the association. The wide spectrum of congenital abnormalities confirms the high clinical variability of VATER/VACTERL association which seems to be due to a disruption of blastogenesis. © 1992 Wiley-Liss, Inc.

Heart Septal Defects VentricularMalemedicine.medical_specialtyRadial aplasiaTerminology as TopicmedicineHumansEsophageal AtresiaLungGenetics (clinical)Polydactylybusiness.industryVATER/VACTERL ASSOCIATIONInfant NewbornLaryngostenosisAnatomySyndromemedicine.diseaseAnusPhenotypeVACTERL associationStenosismedicine.anatomical_structurePhenotypeRadiologyLaryngeal StenosisbusinessTracheoesophageal FistulaAmerican journal of medical genetics
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