Search results for "degenerative diseases"

showing 10 items of 158 documents

Fractal analyses reveal independent complexity and predictability of gait

2017

Locomotion is a natural task that has been assessed since decades and used as a proxy to highlight impairments of various origins. Most studies adopted classical linear analyses of spatio-temporal gait parameters. Here, we use more advanced, yet not less practical, non-linear techniques to analyse gait time series of healthy subjects. We aimed at finding more sensitive indexes related to spatio-temporal gait parameters than those previously used, with the hope to better identify abnormal locomotion. We analysed large-scale stride interval time series and mean step width in 34 participants while altering walking direction (forward vs. backward walking) and with or without galvanic vestibular…

MalePhysiologyEffect of gait parameters on energetic costlcsh:MedicineWalkingMotor Neuron Diseases0302 clinical medicineElderlyMedicine and Health SciencesMastoid Processlcsh:ScienceMusculoskeletal SystemGaitMathematicsMultidisciplinary05 social sciencesNeurodegenerative DiseasesFractalsNeurologyPhysical SciencesFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]AnatomyGait AnalysisResearch ArticleAdultSTRIDEGeometryFOS: Physical sciencesSurgical and Invasive Medical ProceduresFractal dimension050105 experimental psychology03 medical and health sciencesYoung AdultFractalHumans0501 psychology and cognitive sciencesPredictabilityGalvanic vestibular stimulationSkeletonHurst exponentFunctional Electrical Stimulationbusiness.industryBiological LocomotionAmyotrophic Lateral SclerosisSkulllcsh:RBiology and Life SciencesPattern recognitionPhysics - Medical PhysicsAge GroupsGait analysis[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]People and PlacesPopulation Groupingslcsh:QArtificial intelligenceMedical Physics (physics.med-ph)business030217 neurology & neurosurgeryMathematics
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The hematopoietic factor G-CSF is a neuronal ligand that counteracts programmed cell death and drives neurogenesis.

2005

G-CSF is a potent hematopoietic factor that enhances survival and drives differentiation of myeloid lineage cells, resulting in the generation of neutrophilic granulocytes. Here, we show that G-CSF passes the intact blood-brain barrier and reduces infarct volume in 2 different rat models of acute stroke. G-CSF displays strong anti-apoptotic activity in mature neurons and activates multiple cell survival pathways. Both G-CSF and its receptor are widely expressed by neurons in the CNS, and their expression is induced by ischemia, which suggests an autocrine protective signaling mechanism. Surprisingly, the G-CSF receptor was also expressed by adult neural stem cells, and G-CSF induced neurona…

Malemedicine.medical_specialtyProgrammed cell deathNeutrophilsCellular differentiationApoptosisBiologyLigandsBrain IschemiaBrain ischemiaInternal medicineGranulocyte Colony-Stimulating FactormedicineAnimalsHumansGranulocyte Precursor CellsNerve TissueRats WistarReceptorAutocrine signallingStem CellsNeurogenesisCell DifferentiationNeurodegenerative DiseasesGeneral Medicinemedicine.diseaseNeural stem cellCell biologyRatsStrokeDisease Models AnimalEndocrinologyBlood-Brain BarrierReceptors Granulocyte Colony-Stimulating FactorStem cellResearch ArticleThe Journal of clinical investigation
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High-yield Production of Amyloid-β Peptide Enabled by a Customized Spider Silk Domain

2020

AbstractDuring storage in the silk gland, the N-terminal domain (NT) of spider silk proteins (spidroins) keeps the aggregation-prone repetitive region in solution at extreme concentrations. We observe that NTs from different spidroins have co-evolved with their respective repeat region, and now use an NT that is distantly related to previously used NTs, for efficient recombinant production of the amyloid-β peptide (Aβ) implicated in Alzheimer’s disease. A designed variant of NT from Nephila clavipes flagelliform spidroin, which in nature allows production and storage of β-hairpin repeat segments, gives exceptionally high yields of different human Aβ variants as a solubility tag. This tool e…

Models Molecular0301 basic medicineProtein domainBiophysicslcsh:MedicinePeptideBiosynthesis010402 general chemistryBiochemistry01 natural sciencesArticlelaw.invention03 medical and health sciencesProtein DomainslawAnimalsSpider silkAmino Acid SequenceNeurodegenerationlcsh:SciencePeptide sequencechemistry.chemical_classificationAmyloid beta-PeptidesMultidisciplinarybiologySpidroinlcsh:RNeurodegenerative diseasesNephila clavipesProteinsbiology.organism_classification0104 chemical sciences030104 developmental biologyBiochemistrychemistryYield (chemistry)Recombinant DNAlcsh:QPeptidesFibroinsScientific Reports
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Molecular topology as novel strategy for discovery of drugs with aβ lowering and anti-aggregation dual activities for Alzheimer's disease.

2014

Background and Purpose: In this study, we demonstrate the use of Molecular topology (MT) in an Alzheimer's disease (AD) drug discovery program. MT uses and expands upon the principles governing the molecular connectivity theory of numerically characterizing molecular structures, in the present case, active anti-AD drugs/agents, using topological descriptors to build models. Topological characterization has been shown to embody sufficient molecular information to provide strong correlation to therapeutic efficacy. Experimental Approach: We used MT to include multiple bioactive properties that allows for the identification of multifunctional single agent compounds, in this case, the dual func…

Models MolecularDrug Evaluation Preclinicallcsh:MedicineDiseaseProtein aggregationBioinformaticsBiochemistryMechanical Treatment of SpecimensAnimal CellsMolecular Cell BiologyDrug DiscoveryMedicine and Health Scienceslcsh:ScienceTopology (chemistry)NeuronsMultidisciplinaryDrug discoveryMedicine (all)Anti aggregationNeurodegenerative DiseasesAnimal ModelsElectroporationTreatment OutcomeNeurologySpecimen DisruptionDatabases as TopicFemaleMolecular topologyAlzheimer's diseaseCellular TypesResearch ArticleDrug Research and DevelopmentMouse ModelsMice TransgenicComputational biologyBiologyResearch and Analysis MethodsProtein AggregatesModel OrganismsAlzheimer DiseaseMental Health and PsychiatrymedicineAnimalsHumansPharmacologyAmyloid beta-PeptidesBiochemistry Genetics and Molecular Biology (all)lcsh:RBiology and Life SciencesProteinsComputational BiologyCell BiologyDUAL (cognitive architecture)medicine.diseaseDisease Models AnimalAgricultural and Biological Sciences (all)Specimen Preparation and TreatmentFeasibility StudiesDementialcsh:QClinical MedicineProtein MultimerizationPLoS ONE
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Neurodegeneration in multiple sclerosis: novel treatment strategies.

2012

In recent years it has become clear that the neuronal compartment already plays an important role early in the pathology of multiple sclerosis (MS). Neuronal injury in the course of chronic neuroinflammation is a key factor in determining long-term disability in patients. Viewing MS as both inflammatory and neurodegenerative has major implications for therapy, with CNS protection and repair needed in addition to controlling inflammation. Here, the authors' review recently elucidated molecular insights into inflammatory neuronal/axonal pathology in MS and discuss the resulting options regarding neuroprotective and regenerative treatment strategies.

Multiple SclerosisInflammationNeuroprotectionmedicineAnimalsHumansPharmacology (medical)In patientMolecular Targeted TherapyNeuroinflammationNeuronsEvidence-Based Medicinebusiness.industryGeneral NeuroscienceMultiple sclerosisNeurodegenerationAnti-Inflammatory Agents Non-SteroidalNeurodegenerative Diseasesmedicine.diseasePathology of multiple sclerosisNeuroprotective AgentsTreatment strategyEducation Medical ContinuingNeurology (clinical)medicine.symptombusinessNeuroscienceExpert review of neurotherapeutics
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Beta-amyloid monomers are neuroprotective

2009

The 42-aa-long β-amyloid protein—Aβ1-42—is thought to play a central role in the pathogenesis of Alzheimer's disease (AD) (Walsh and Selkoe, 2007). Data from AD brain (Shankar et al., 2008), transgenic APP (amyloid precursor protein)-overexpressing mice (Lesné et al., 2006), and neuronal cultures treated with synthetic Aβ peptides (Lambert et al., 1998) indicate that self-association of Aβ1-42monomers into soluble oligomers is required for neurotoxicity. The function of monomeric Aβ1-42is unknown. The evidence that Aβ1-42is present in the brain and CSF of normal individuals suggests that the peptide is physiologically active (Shoji, 2002). Here we show that synthetic Aβ1-42monomers support …

N-MethylaspartateStimulationPeptideNeuroprotectionNeuro-degenerative diseasePathogenesismental disordersNitrilesmedicineAmyloid precursor proteinButadienesExcitatory Amino Acid AgonistsAnimalsEnzyme InhibitorsReceptorCells CulturedPodophyllotoxinchemistry.chemical_classificationCerebral CortexNeuronsAnalysis of VarianceAmyloid beta-PeptidesbiologyCell DeathDose-Response Relationship DrugGeneral NeuroscienceNeurodegenerationβ-Amyloid proteinNeurotoxicityself-assemblyTyrphostinsmedicine.diseaseEmbryo MammalianPeptide FragmentsCell biologyRatsNeuroprotective Agentschemistrybiology.proteinBrief CommunicationsNeuroscienceβ-Amyloid protein; Neuro-degenerative diseases; self-assembly
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Neurons as targets for T cells in the nervous system

2013

International audience; Accumulating evidence shows that T cells penetrate the central nervous system (CNS) parenchyma in several autoimmune, infectious, and degenerative neurological diseases. The structural and functional consequences for CNS neurons of their encounter with activated T cells have been investigated in several experimental systems, including ex vivo co-cultures, electrophysiology, and in vivo imaging. Here, we review the modalities of neuron/T cell interactions. We substantiate the contention that T cells are directly responsible for neuronal damage in a large number of neurological diseases and discuss mechanisms of neuronal damage mediated by distinct T cell subsets, the …

Nervous systemMultiple SclerosisT cell[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyAntigen presentationCentral nervous systemInflammationAdaptive ImmunityBiology[SDV.BC.IC] Life Sciences [q-bio]/Cellular Biology/Cell Behavior [q-bio.CB]Nervous System03 medical and health sciences0302 clinical medicineT-Lymphocyte Subsets[SDV.BC.IC]Life Sciences [q-bio]/Cellular Biology/Cell Behavior [q-bio.CB]medicineAnimalsHumansEncephalitis Viral030304 developmental biologyNeuronsAntigen PresentationImmunity Cellular0303 health sciencesGeneral NeuroscienceHistocompatibility Antigens Class Iapoptosis[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyT cellNeurodegenerative DiseasesAcquired immune systemcentral nervous systemneuron3. Good healthmedicine.anatomical_structurenervous system[SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunologyinflammation[SDV.IMM.IA] Life Sciences [q-bio]/Immunology/Adaptive immunologyencephalomyelitisNeuronNervous System Diseasesmedicine.symptomNeuroscience030217 neurology & neurosurgeryEx vivo
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Extracellular Vesicle-Mediated Cell–Cell Communication in the Nervous System: Focus on Neurological Diseases

2019

Extracellular vesicles (EVs), including exosomes, are membranous particles released by cells into the extracellular space. They are involved in cell differentiation, tissue homeostasis, and organ remodelling in virtually all tissues, including the central nervous system (CNS). They are secreted by a range of cell types and via blood reaching other cells whose functioning they can modify because they transport and deliver active molecules, such as proteins of various types and functions, lipids, DNA, and miRNAs. Since they are relatively easy to isolate, exosomes can be characterized, and their composition elucidated and manipulated by bioengineering techniques. Consequently, exosomes appear…

Nervous systemReviewCell CommunicationTheranostic NanomedicineCatalysilcsh:Chemistry0302 clinical medicineCell–cell interactionlcsh:QH301-705.5Tissue homeostasisSpectroscopyDrug Carriers0303 health sciencesnervous systemCell DifferentiationNeurodegenerative DiseasesComputer Science Applications1707 Computer Vision and Pattern RecognitionGeneral MedicineExtracellular vesicleComputer Science ApplicationsCell biologymedicine.anatomical_structureTheranostics toolExtracellular vesicleextracellular vesiclesneurological diseasesCell signalingCell typecell–cell interactionexosomesBiologyCatalysisInorganic Chemistry03 medical and health sciencesExtracellularmedicineCell-cell interactionHumansPhysical and Theoretical ChemistryMolecular Biology030304 developmental biologytheranostics toolsOrganic ChemistrybiomarkersBiomarkercentral nervous systemMicrovesiclesExosomelcsh:Biology (General)lcsh:QD1-999030217 neurology & neurosurgeryNeurological diseaseInternational Journal of Molecular Sciences
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Emerging roles of extracellular vesicles in the nervous system.

2014

Information exchange executed by extracellular vesicles, including exosomes, is a newly described form of intercellular communication important in the development and physiology of neural systems. These vesicles can be released from cells, are packed with information including signaling proteins and both coding and regulatory RNAs, and can be taken up by target cells, thereby facilitating the transfer of multilevel information. Recent studies demonstrate their critical role in physiological processes, including nerve regeneration, synaptic function, and behavior. These vesicles also have a sinister role in the propagation of toxic amyloid proteins in neurodegenerative conditions, including …

Nervous systemSymposiumAmyloidGeneral NeuroscienceVesicleCiliumRegeneration (biology)2800 General NeuroscienceBrain610 Medicine & healthNeurodegenerative Diseases11359 Institute for Regenerative Medicine (IREM)BiologyExosomesMicrovesiclesNerve Regenerationmedicine.anatomical_structureTumor progressionmedicineAnimalsHumansNeoplasm InvasivenessCiliaNeuroscienceNeuroinflammationThe Journal of neuroscience : the official journal of the Society for Neuroscience
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Knowledge and Attitudes towards Palliative Care : Validation of the Spanish Version of Questionnaire on Palliative Care for Advanced Dementia

2022

Background: Palliative care is essential in the care of people with advanced dementia, due to the increasing number of patients requiring care in the final stages of life. Nurses need to acquire specific knowledge and skills to provide quality palliative care. The Questionnaire on Palliative Care for Advanced Dementia (qPAD) is useful for assessing knowledge and attitudes toward palliative care, but its adaptation to the Spanish language and analysis of its effectiveness and usefulness for the Spanish culture is lacking. Objective: To report on the Spanish language adaptation and psychometric analysis of the qPAD. Methods: The Questionnaire on Palliative Care for Advanced Dementia Spanish v…

NeurologiaHealth Information ManagementLeadership and ManagementHealth PolicyTractament pal·liatiuHealth InformaticsAlzheimer’s disease; neurodegenerative diseases; pain; end-of-life care; nursing
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