Search results for "differentiation"

showing 10 items of 1605 documents

Management of acute promyelocytic leukemia: updated recommendations from an expert panel of the European LeukemiaNet

2019

Abstract Since the comprehensive recommendations for the management of acute promyelocytic leukemia (APL) reported in 2009, several studies have provided important insights, particularly regarding the role of arsenic trioxide (ATO) in frontline therapy. Ten years later, a European LeukemiaNet expert panel has reviewed the recent advances in the management of APL in both frontline and relapse settings in order to develop updated evidence- and expert opinion–based recommendations on the management of this disease. Together with providing current indications on genetic diagnosis, modern risk-adapted frontline therapy, and salvage treatment, the review contains specific recommendations for the …

Acute promyelocytic leukemiamedicine.medical_specialtymedicine.medical_treatmentImmunologyMEDLINETretinoinDiseaseHemorrhagic DisordersBiochemistryHemorrhagic disorderEuropean LeukemiaNetArsenic TrioxideLeukemia Promyelocytic AcutePregnancyRecurrenceHumansMedicineDisease management (health)Intensive care medicineSpecial ReportNeoadjuvant therapyAPL Differentiation SyndromeAgedbusiness.industryDisease ManagementCell BiologyHematologymedicine.diseasePractice Guidelines as TopicFemalebusinessBlood
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Immunolocalization of integrins in the normal and neoplastic colonic epithelium.

1993

Cryosections of normal colon (NC), tubular and villous adenomas (TA, VA), and variably differentiated colon adenocarcinomas (CA) were immunostained with monoclonal antibodies to alpha 1-6 and alpha v, and beta 1-4 integrin subunits; select samples were stained for cytokeratin (Ck) 20 and villin. In NC, alpha 2 staining was strongest in crypt cells; alpha 1,3 and alpha v, and beta 1,3 and beta 4, and Ck 20 and villin predominated in superficial enterocytes. In TA and VA, monolayered glands showed integrin, Ck 20 and villin patterns that differed slightly from both crypt and superficial enterocytes. Complex glands in VA showed decreased integrin staining and basal polarization; Ck 20 and vill…

AdenomaPathologymedicine.medical_specialtyIntegrinsColonCryptIntegrinFluorescent Antibody TechniqueAdenocarcinomadigestive systemEpitheliumExtracellular matrixImmunoenzyme Techniques03 medical and health sciencesCytokeratin0302 clinical medicineKeratinmedicineHumansTissue Distribution030304 developmental biologychemistry.chemical_classification0303 health sciencesbiologyCarcinomaMicrofilament ProteinsAntibodies MonoclonalCell DifferentiationGeneral MedicineEpitheliummedicine.anatomical_structurechemistry030220 oncology & carcinogenesisColonic Neoplasmsbiology.proteinImmunohistochemistryKeratinsVillinCarrier ProteinsVirchows Archiv. B, Cell pathology including molecular pathology
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Epithelial markers and differentiation in adnexal neoplasms of the skin: an immunohistochemical study including individual cytokeratins

1995

Applying immunohistochemical procedures for the detection of eight different cytokeratin (CK) polypeptides and other differentiation markers, we compared the staining patterns of normal cutaneous structures with those of benign adnexal tumors (n = 65). Syringomas exhibited a marker pattern highly reminiscent of that seen in normal dermal eccrine ducts (EMA in peripheral cells, CK 10 in intermediate cells, and CK 6, CK 19, and CEA in luminal cells). Nodular hidradenomas exhibited complex patterns suggesting relationship between tumor cells, including clear cells, and normal secretory coil cells (CK 7, CK 8, CK 19, and EMA); however, dermal-duct and epidermoid differentiation were also detect…

AdenomaPathologymedicine.medical_specialtySkin NeoplasmsHistologyHidradenomaCellular differentiationDermatologyBiologyBinding CompetitiveInner root sheathPathology and Forensic MedicineCytokeratinReference ValuesBiomarkers TumormedicineCarcinomaHumansNeoplasms Basal CellAdenoma Sweat GlandSyringomaMyoepithelial cellAntibodies MonoclonalCell Differentiationmedicine.diseaseCarcinoma Adenoid CysticImmunohistochemistrySweat GlandsStainingEpidermal CellsKeratinsImmunohistochemistryEpidermisJournal of Cutaneous Pathology
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Grades of atypia in tubular and villous adenomas of the human colon. An electron microscopic study.

1980

Of a total of 544 tubular, villous and tubulo-villous adenomas of the human colon which were investigated by light microscopy, six tubular and six villous adenomas were examined under the electron microscope. It was shown that the two types of adenoma differ in their tissue architecture, but not in their cytological appearance. Different grades of epithelial atypia occur in both types of adenoma. These are designated as grades I to III, correspond to mild, moderate and severe atypia respectively. Whereas adenoma cells with atypia grade I clearly show a cytological relationship with crypt epithelia of the normal colonic mucosa under the electron microscope, adenoma cells with atypia grade II…

Adenomamedicine.medical_specialtyPathologyendocrine system diseasesAdenomaCellular differentiationCryptBiologyGastroenterologyMalignant transformationlaw.inventionlawInternal medicinemedicineAtypiaHumansElectron microscopicCell Differentiationmedicine.diseasedigestive system diseasesstomatognathic diseasesMicroscopy ElectronColonic NeoplasmsElectron microscopeHuman colonGlycogenVirchows Archiv. B, Cell pathology including molecular pathology
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STEM CELLS AND COLON CANCER

2012

The current concept of tumorigenesis suggests that cancers arise and are “driven” by cells with stem cell-like properties, known as cancer stem cells (CSCs), which share many functional and molecular features with normal stem cells. Self-renewal key pathways (e.g., Wnt, Notch, and Hedgehog) are tightly regulated in normal stem cells, but are impaired in CSCs. For instance, active Wnt pathway plays a crucial role in colon cancer pathophysiology, where deregulation of the adenomatous polyposis coli (APC) gene, a negative regulator of Wnt signaling, represents one of the earliest alterations in the multistep process of colon carcinogenesis, causing early adenoma formation. Normal colon stem ce…

Adenomatous polyposis coliCellular differentiationWnt signaling pathwayStem cell factorBiologymedicine.disease_causeEndothelial stem cellCancer stem cellImmunologyCancer researchmedicinebiology.proteinStem cellCarcinogenesis
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CD73-generated extracellular adenosine in chronic lymphocytic leukemia creates local conditions counteracting drug-induced cell death

2011

Abstract Extracellular adenosine (ADO), generated from ATP or ADP through the concerted action of the ectoenzymes CD39 and CD73, elicits autocrine and paracrine effects mediated by type 1 purinergic receptors. We have tested whether the expression of CD39 and CD73 by chronic lymphocytic leukemia (CLL) cells activates an adenosinergic axis affecting growth and survival. By immunohistochemistry, CD39 is widely expressed in CLL lymph nodes, whereas CD73 is restricted to proliferation centers. CD73 expression is highest on Ki-67+ CLL cells, adjacent to T lymphocytes, and is further localized to perivascular areas. CD39+/CD73+ CLL cells generate ADO from ADP in a time- and concentration-dependen…

AdenosineCellular differentiationChronic lymphocytic leukemia5'-Nucleotidase; Adenosine; Adenosine Diphosphate; Adenosine Triphosphate; Antigens CD; Antineoplastic Agents Phytogenic; Apyrase; Autocrine Communication; Cell Death; Cell Differentiation; Cell Movement; Cell Survival; Etoposide; Extracellular Space; GPI-Linked Proteins; Humans; Leukemia Lymphocytic Chronic B-Cell; Paracrine Communication; Receptor Adenosine A2A; Tumor Cells Cultured; Biochemistry; Immunology; Hematology; Cell BiologyMICROENVIRONMENTCD38BiochemistryACTIVATIONAdenosine TriphosphateCell MovementPhytogenichemic and lymphatic diseasesTumor Cells CulturedChronic5'-NucleotidaseEtoposideLeukemiaCulturedCell DeathTUMOR-GROWTHApyrasePurinergic receptorCell DifferentiationHematologyLymphocyticCDTumor CellsCell biologyAdenosine DiphosphateAutocrine CommunicationLeukemiaReceptorIMMUNE SUPPRESSIONReceptor Adenosine A2ACell SurvivalImmunologyAntineoplastic AgentsAdenosinergicBiologyGPI-Linked ProteinsDAMAGE-INDUCED APOPTOSISAdenosine A2AParacrine signallingAntigens CDParacrine CommunicationmedicineHumansAntigensAutocrine signallingImmunobiologyB-CellCell BiologyDAMAGE-INDUCED APOPTOSIS; T-CELLS; IMMUNE SUPPRESSION; ZAP-70 EXPRESSION; TUMOR-GROWTH; RECEPTOR; CD73; ACTIVATION; CD38; MICROENVIRONMENTmedicine.diseaseAntineoplastic Agents PhytogenicLeukemia Lymphocytic Chronic B-CellSettore MED/15 - MALATTIE DEL SANGUET-CELLSCD73Extracellular SpaceZAP-70 EXPRESSIONCD38Blood
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A Good Breath of Oxygen for Beta-Like Cells Obtained From Porcine Exocrine Pancreatic Tissue

2011

Ischemia is the most important factor that affects organ survival during harvesting. The two-layer method (TLM) is one of several cold storage solutions that seeks to preserve organs and cells avoiding in vivo and in vitro ischemia. We compared the retrieval of beta-like elements from exocrine pancreatic cells using TLM versus University of Wisconsin (UW) solutions. For this purpose pancreata laparoscopically harvested from 20 female pigs were preserved in UW solution or TLM before digestion. The resulting exocrine cells were divided into 2 groups: the first was cultured in a designed medium to allow differentiation into beta-like cells and the second was cryopreserved before the differenti…

AdenosineTime FactorsCell SurvivalSwineAllopurinolCellular differentiationOrgan Preservation Solutionsbeta-like-cells porcine esocrine pancreatic tisuueCold storageCell SeparationCryopreservationAndrologyPancreatectomyRaffinoseIn vivoSettore BIO/13 - Biologia ApplicataInsulin-Secreting CellsmedicineAnimalsInsulinViaspanCells CulturedCryopreservationFluorocarbonsTransplantationbusiness.industryCell DifferentiationAnatomyGlutathionePancreas ExocrineIn vitroCulture MediaOxygenTransplantationSettore MED/18 - Chirurgia GeneraleGlucosemedicine.anatomical_structureTissue and Organ HarvestingFemaleLaparoscopySurgeryPancreasbusiness
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Induction of B-cell development in adult mice reveals the ability of bone marrow to produce B-1a cells

2009

AbstractTo study B-cell development from bone marrow (BM), we generated recombination-activating gene 1 (Rag1)–targeted mice lacking mature lymphocytes. B-cell development can be induced in such mice by B cell–specific restoration of a functional Rag1 transcription unit. Follicular and marginal zone B cells populated the spleen when Rag1 expression was permitted. Notably, the peritoneal cavity was dominated by bona fide B-1a cells, as judged by surface markers and functional properties. These BM-derived B-1a cells exhibited a polyclonal VDJ repertoire with substantial N nucleotide insertions. Nevertheless, physiologic frequencies of phosphatidylcholine-specific B cells were detected. Import…

Adoptive cell transfer1303 BiochemistryGenes RAG-1Immunology2720 HematologyB-Lymphocyte SubsetsSpleenBone Marrow CellsEnzyme-Linked Immunosorbent AssayMice Transgenic610 Medicine & healthBiology10263 Institute of Experimental ImmunologyBiochemistryPolymerase Chain ReactionRecombination-activating gene1307 Cell BiologyPeritoneal cavityMicemedicineAnimalsB cellB-Lymphocytes2403 ImmunologyStem CellsCell DifferentiationCell BiologyHematologyMarginal zoneFlow CytometryMolecular biologyAdoptive Transfermedicine.anatomical_structureImmunoglobulin MImmunologybiology.protein570 Life sciences; biologyBone marrow
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Controversies on the role of Th17 in cancer: a TGF-β-dependent immunosuppressive activity?

2012

The immune system has important roles in limiting the spread of cancer and shaping the tumor microenvironment. Although the contributions of T helper 17 (Th17) cells (a subtype of CD4(+) T lymphocytes) to autoimmunity and allergy response are well known, their roles in cancer remain ambiguous. Despite adoptive transfer studies indicating that mouse Th17 cells support anticancer immunity, the Th17 cells that naturally infiltrate experimental tumors appear to have a tumor-promoting effect. These contradictory properties can be related to the high degree of plasticity inherent in Th17 cells and their capacity to differentiate into tumoricidal Th1-like cells. Mouse Th17 cells induced by transfo…

Adoptive cell transferAngiogenesisAntigen-Presenting Cellschemical and pharmacologic phenomenaBiologymedicine.disease_causeAutoimmunityMice03 medical and health sciences0302 clinical medicineImmune systemAntigenAntigens CDTransforming Growth Factor betaImmunityNeoplasmsImmune TolerancemedicineAnimals5'-NucleotidaseMolecular Biology030304 developmental biologyImmunity Cellular0303 health sciencesTumor microenvironmentNeovascularization PathologicApyraseModels ImmunologicalCell DifferentiationTh1 Cells3. Good health030220 oncology & carcinogenesisImmunologyCancer researchTh17 CellsMolecular MedicineTransforming growth factorTrends in Molecular Medicine
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Genetic proof for the transient nature of the Th17 phenotype

2010

IL-17-producing CD4(+) T cells (Th17) have been classified as a new T helper cell subset. Using an IL-17 fate mapping mouse strain, which genetically fixes the memory of IL-17 expression, we demonstrate that IL-17A/F-expressing T helper cells generated either in vitro or in vivo are not a stable T-cell subset. Upon adoptive transfer of IL-17F-reporter-positive Th17 cells to RAG-deficient or WT animals, encephalitogenic Th17 cells partially lose IL-17 expression and upregulate IFN-γ. Additionally, we show that Th1 cells can convert in vivo to IL-17A/IFN-γ-coexpressing cells in the mesenteric lymph nodes (mLN). Our data classify IL-17A and IL-17F as cytokines produced transiently in response …

Adoptive cell transferEncephalomyelitis Autoimmune ExperimentalGenes RAG-1TransgeneImmunologyReceptors Antigen T-CellMice TransgenicBiologyLymphocyte ActivationInterferon-gammaMiceInterleukin 21AntigenGenes ReporterT-Lymphocyte SubsetsIn vivomedicineAnimalsImmunology and AllergyCytotoxic T cellMesenteric lymph nodesMice KnockoutIntegrasesCell DifferentiationT helper cellTh1 CellsAdoptive TransferCell biologyMice Inbred C57BLmedicine.anatomical_structureGene Expression RegulationImmunologyTh17 CellsEuropean Journal of Immunology
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