Search results for "electroretinography"

showing 10 items of 58 documents

ApoB100,LDLR-/- mice exhibit reduced electroretinographic response and cholesteryl esters deposits in the retina

2008

International audience; PURPOSE. To evaluate the retinal phenotype of 7- and 14-month-old apoB100,LDLR–/– mice, a relevant animal model of lipid metabolism dysfunction. METHODS. Single-flash electroretinograms were obtained from 7- and 14-month-old apoB100,LDLR–/– and control mice fed a standard diet under both scotopic and photopic conditions. Visual cycle retinoids were analyzed in eyes from dark-adapted mice. Retinal and choroidal vascularization was evaluated with scanning laser ophthalmoscopy. Fatty acids were analyzed in the retina. Esterified and free cholesterol was detected in eye cryosections. RESULTS. Scotopic and photopic b-wave amplitudes were significantly reduced in apoB100,L…

MaleHUMAN BRUCHS MEMBRANEgenetic structuresHIGH-FAT DIETLipid Metabolism DisordersBasement MembraneAGE-RELATED MACULOPATHYchemistry.chemical_compoundMice0302 clinical medicine[SDV.IDA]Life Sciences [q-bio]/Food engineeringFluorescein AngiographyPigment Epithelium of EyeTRANSGENIC MICE0303 health sciencesmedicine.diagnostic_testROD OUTER SEGMENTSmedicine.anatomical_structureBiochemistryHUMAN APOLIPOPROTEIN-BApolipoprotein B-100Femalelipids (amino acids peptides and proteins)Cholesterol EstersPhotopic visionVisual phototransductionmedicine.medical_specialtyDark AdaptationMice TransgenicBiologyRetinaRECEPTOR-NEGATIVE MICE03 medical and health sciencesRetinoidsRetinal DiseasesBASAL DEPOSITSInternal medicinemedicineElectroretinographyAnimals[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringFilipinHUMAN ATHEROSCLEROTIC LESIONS030304 developmental biologyRetinaRetinal pigment epitheliumRetinalMacular degenerationmedicine.diseaseMACULAR DEGENERATIONeye diseasesMice Inbred C57BLOphthalmoscopyEndocrinologychemistryReceptors LDLLDL receptor030221 ophthalmology & optometrysense organsPhotic StimulationElectroretinography
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Metabolic syndrome triggered by high-fructose diet favors choroidal neovascularization and impairs retinal light sensitivity in the rat

2014

Diabetic retinopathy and age-related macular degeneration are the leading causes of blindness in Western populations. Although it is a matter of controversy, large-scale population-based studies have reported increased prevalence of age-related macular degeneration in patients with diabetes or diabetic retinopathy. We hypothesized that metabolic syndrome, one of the major risk factors for type 2 diabetes, would represent a favorable environment for the development of choroidal neovascularization, the main complication of age-related macular degeneration. The fructose-fed rat was used as a model for metabolic syndrome in which choroidal neovascularization was induced by laser photocoagulatio…

MaleOrganes des sensmedicine.medical_treatment[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionVisual Acuitylcsh:MedicineGene ExpressionType 2 diabetesinduced insulin-resistanceanimal-modelscholesterol homeostasis0302 clinical medicineRetinal Rod Photoreceptor CellsRats Inbred BNHyperinsulinemiaMedicine and Health Sciencesanimal modèleratlcsh:Science2. Zero hungerMetabolic Syndrome0303 health scienceseducation.field_of_studyMultidisciplinaryLaser Coagulationsyndrome métaboliqueReverse Transcriptase Polymerase Chain Reactionhepatic steatosisFatty AcidsAngiographyDiabetic retinopathyChoroidal neovascularizationAdipose Tissue[ SDV.MHEP.OS ] Life Sciences [q-bio]/Human health and pathology/Sensory OrgansAlimentation et NutritionRetinal Disordersmedicine.symptomLaser coagulationResearch Articlediabètemedicine.medical_specialtymacular degenerationPopulationSensory Organselectroretinographic oscillatory potentials;induced insulin-resistance;fatty-acid profile;macular degeneration;diabetic-retinopathy;animal-models;cholesterol homeostasis;hepatic steatosis;mouse;associationAntigens Differentiation MyelomonocyticMédecine humaine et pathologieFructoseBiologyRetina03 medical and health sciencesAntigens CDDiabetes mellitusInternal medicine[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologymedicineElectroretinographyelectroretinographic oscillatory potentialsAnimalsHumansFood and Nutrition[SDV.MHEP.OS]Life Sciences [q-bio]/Human health and pathology/Sensory OrganseducationRetinopathymouse030304 developmental biologyNutritiondiabetic-retinopathylcsh:RassociationBiology and Life Sciencesdégénérescence maculaireMacular degenerationmedicine.diseaseChoroidal Neovascularizationeye diseasesDietFatty LiverOphthalmologyEndocrinologyMetabolic Disordersfatty-acid profile030221 ophthalmology & optometrylcsh:QInsulinomaHuman health and pathologysense organs[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Further delineation of eye manifestations in homozygous 15q13.3 microdeletions including TRPM1: a differential diagnosis of ceroid lipofuscinosis.

2014

The 15q13.3 heterozygous microdeletion is a fairly common microdeletion syndrome with marked clinical variability and incomplete penetrance. The average size of the deletion, which comprises six genes including CHRNA7, is 1.5 Mb. CHRNA7 has been identified as the gene responsible for the neurological phenotype in this microdeletion syndrome. Only seven patients with a homozygous microdeletion that includes at least CHRNA7, and is inherited from both parents have been described in the literature. The aim of this study was to further describe the distinctive eye manifestations from the analysis in the three French patients diagnosed with the classical 1.5 Mb homozygous microdeletion. Patients…

MalePathologymedicine.medical_specialtygenetic structuresalpha7 Nicotinic Acetylcholine ReceptorEncephalopathyTRPM Cation ChannelsChromosome DisordersBiologyBlindnessEyePupilNeuronal Ceroid-LipofuscinosesNight BlindnessSeizuresIntellectual DisabilityRetinal DystrophiesGeneticsmedicineElectroretinographyMyopiaHumansEye AbnormalitiesChildGenetics (clinical)TRPM1Genetic Association StudiesCongenital stationary night blindnessGeneticsChromosomes Human Pair 15DystrophyEye Diseases HereditaryGenetic Diseases X-LinkedOptic NerveMicrodeletion syndromemedicine.diseasePenetranceChild PreschoolFemalesense organsDifferential diagnosisChromosome DeletionAmerican journal of medical genetics. Part A
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Metabolomics of the aqueous humor in the rat glaucoma model induced by a series of intracamerular sodium hyaluronate injection

2015

Glaucoma models are helpful to study disease characteristics and to design new therapeutic options. Metabolomic profiling approach have been used to elucidating the molecular characteristics of the aqueous humor. Juvenile male Wistar rats experimental (n = 15) and controls (n = 6) were used for these studies. Experimental rats received weekly intracamerular injection of 25 µl of sodium hyaluronate in the left eye and sterile saline solution in the right eye, consecutively for ten weeks. Rats were subjected to anterior/posterior eye segment examinations, intraocular pressure (IOP), and flash electroretinograms (ERG). The aqueous humor was collected at endpoints and analyzed by Nuclear Magnet…

MalePosterior Eye Segmentmedicine.medical_specialtyIntraocular pressureMagnetic Resonance Spectroscopygenetic structuresSodium hyaluronateCarbohydratesGlaucomaAqueous humorAqueous humor Glaucoma model Metabolomics Nuclear magnetic resonance Rats Sodium hyaluronateInjectionsAqueous HumorPathogenesisCellular and Molecular Neurosciencechemistry.chemical_compoundMetabolomicsAnterior Eye SegmentOphthalmologyElectroretinographymedicineAnimalsMetabolomicsAmino AcidsHyaluronic AcidRats WistarIntraocular PressureGlaucomamedicine.diseaseLipidseye diseasesSensory SystemsRatsSurgeryDisease Models AnimalOphthalmologychemistrysense organsErgFollow-Up StudiesExperimental Eye Research
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Synaptic vesicle alterations in rod photoreceptors of synaptophysin-deficient mice.

2001

Abstract The abundance of the integral membrane protein synaptophysin in synaptic vesicles and its multiple possible functional contributions to transmitter exocytosis and synaptic vesicle formation stand in sharp contrast to the observed lack of defects in synaptophysin knockout mice. Assuming that deficiencies are compensated by the often coexpressed synaptophysin isoform synaptoporin, we now show that retinal rod photoreceptors, which do not synthesize synaptoporin either in wild-type or in knockout mice, are affected by the loss of synaptophysin. Multiple pale-appearing photoreceptors, as seen by electron microscopy, possess reduced cytoplasmic electron density, swollen mitochondria, an…

MalePresynaptic TerminalsSynaptophysinAction PotentialsFluorescent Antibody TechniqueDark AdaptationBiologyRibbon synapseSynaptic vesicleSynaptic TransmissionExocytosisExocytosisMiceRetinal Rod Photoreceptor CellsElectroretinographySynaptic vesicle recyclingAnimalsMice KnockoutSex CharacteristicsGeneral NeuroscienceVesicleMembrane ProteinsClathrin-Coated VesiclesSynaptoporinCell biologyMice Inbred C57BLMicroscopy ElectronProtein TransportKnockout mouseSynaptophysinbiology.proteinFemaleSynaptic VesiclesNeurosciencePhotic StimulationNeuroscience
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Retinas of the Diurnal RodentArvicanthis ansorgeiAre Highly Resistant to Experimentally Induced Stress and Degeneration

2011

International audience; PURPOSE. Environmentally induced stress plays a significant role in retinal degeneration and blindness both in animals and in humans. Among such sources of stress, phototoxicity is well studied and has been shown to lead to photoreceptor-specific loss in a number of species. However, the vast majority of studies have been conducted in nocturnal, albino rod-dominant rat and mouse strains, and the pertinence of such findings to human pathology and cone loss is debatable. The authors examined retinal vulnerability to damage in the diurnal murid rodent Arvicanthis ansorgei, a pigmented species with a large number of cones. METHODS. The authors used established protocols …

MaleRetinal degenerationLightRodentsprague dawlayFatty Acids Nonesterifiedbright cyclic lightMicechemistry.chemical_compound0302 clinical medicine[SDV.IDA]Life Sciences [q-bio]/Food engineeringoxidative stressmethyl-N-nitrosoufrea0303 health sciencesbiologymedicine.diagnostic_testmouse retinaRetinal DegenerationMethylnitrosoureaAnatomydocosahexaenoic acidCircadian Rhythmmedicine.anatomical_structureDocosahexaenoic acidRetinal Cone Photoreceptor CellsN-3 fatty acidsPhototoxicityAlkylating Agentsmedicine.medical_specialtylight-induced degeneration03 medical and health sciencesSpecies SpecificityStress Physiologicalbiology.animalInternal medicineElectroretinographymedicineAnimals[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process Engineering030304 developmental biologyRetinaRetinalmedicine.diseaseMice Inbred C57BLMuridaeratsTissue DegenerationDisease Models AnimalEndocrinologyrhodopsinchemistryregenerationinduced photoreceptor apoptosis030221 ophthalmology & optometrysense organsElectroretinographyInvestigative Opthalmology & Visual Science
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Intravitreal Docosahexaenoic Acid in a Rabbit Model: Preclinical Safety Assessment

2014

PurposeThe purpose of the present study was to evaluate the retinal toxicity of a single dose of intravitreal docosahexaenoic acid (DHA) in rabbit eyes over a short-term period.MethodsSixteen New Zealand albino rabbits were selected for this pre-clinical study. Six concentrations of DHA (Brudy Laboratories, Barcelona, Spain) were prepared: 10 mg/50 µl, 5 mg/50 µl, 2'5 mg/50 µl, 50 µg/50 µl, 25 µg/50 µl, and 5 µg/50 µl. Each concentration was injected intravitreally in the right eye of two rabbits. As a control, the vehicle solution was injected in one eye of four animals. Retinal safety was studied by slit-lamp examination, and electroretinography. All the rabbits were euthanized one week a…

MaleRetinal degenerationgenetic structuresÀcids grassosPharmacologyBiochemistryMicechemistry.chemical_compoundCorneaMedicine and Health SciencesRatolinsExperimentació animalDegeneració (Patologia)Multidisciplinarymedicine.diagnostic_testQFatty AcidsChromatographic TechniquesRDegeneration (Pathology)Animal ModelsLipidsChemistrymedicine.anatomical_structureNeurologyDocosahexaenoic acidPhysical SciencesMedicineRetinal DisordersRabbitsSafetyAnatomyResearch Articlemedicine.medical_specialtyHistologyDrug Research and DevelopmentDocosahexaenoic AcidsNew Zealand AlbinoScienceOcular AnatomyResearch and Analysis MethodsRetinaInjectionsAqueous HumorModel OrganismsOcular SystemElectroretinographyToxicity Tests AcutemedicineAnimalsFatty acidsGas ChromatographyPharmacologyDose-Response Relationship Drugbusiness.industrySignificant differenceBiology and Life SciencesRetinalmedicine.diseaseSurgeryVitreous BodyOphthalmologychemistryNeuro-OphthalmologyAnimal experimentationRabbit modelClinical MedicinebusinessElectroretinographyPLoS ONE
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In vivo consequences of cholesterol-24S-hydroxylase (CYP46A1) inhibition by voriconazole on cholesterol homeostasis and function in the rat retina

2014

International audience; Cholesterol 24S-hydroxylase (CYP46A1) converts cholesterol into 24S-hydroxycholesterol in neurons and participates in cholesterol homeostasis in the central nervous system, including the retina. We aimed to evaluate the consequences of CYP46A1 inhibition by voriconazole on cholesterol homeostasis and function in the retina. Rats received daily intraperitoneal injections of voriconazole (60 mg/kg), minocycline (22 mg/kg), voriconazole plus minocycline, or vehicle during five consecutive days. The rats were submitted to electroretinography to monitor retinal functionality. Cholesterol and 24S-hydroxycholesterol were measured in plasma, brain and retina by gas chromatog…

Malegenetic structuresgliaBiochemistrycholesterol homeostasischemistry.chemical_compoundHomeostasisEnzyme Inhibitorsretinal ganglion cellmedicine.diagnostic_testAnatomyUp-RegulationCYP46A1medicine.anatomical_structureCholesterolRetinal ganglion cellCytokineslipids (amino acids peptides and proteins)MicrogliaNeurogliamedicine.medical_specialtyCentral nervous systemBiophysicsNerve Tissue ProteinsBiologyRetinal ganglionRetinaIn vivoInternal medicinemedicineCholesterol 24-HydroxylaseElectroretinographyvoriconazoleAnimalsRats Wistar[SDV.MHEP.OS]Life Sciences [q-bio]/Human health and pathology/Sensory OrgansMolecular BiologyRetinaCholesterolRetinalCell BiologyTriazolesHydroxycholesterolseye diseasesRatsEndocrinologyPyrimidineschemistrySteroid Hydroxylasessense organs[SDV.AEN]Life Sciences [q-bio]/Food and NutritionElectroretinography
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Electroretinographic response in WAG/Rij rats after low-intensity cyclic light exposure.

1993

In order to investigate the combined influence of age and light, the b-wave and oscillatory potentials (OPs) of the electroretinogram (ERG) were recorded in 1.5-, 7- and 12-month-old WAG/Rij rats, reared under homogenous low-intensity cyclic light exposure. Wistar albino rats of the same ages, reared under the same conditions, served as controls. The b-wave amplitude decreased, and its implicit time increased in the older age groups significantly more in WAG/Rij than in Wistar rats. Statistical analysis indicated that the b-wave amplitude is a more suitable parameter than implicit time in differentiating the ERG variations of one rat strain from the other. The added amplitude of the OPs als…

Malemedicine.medical_specialtyAgingPeriodicitygenetic structuresOscillatory potentialsLightDark AdaptationRetinaCellular and Molecular NeuroscienceOpticsAge groupsInternal medicinemedicineElectroretinographyAnimalsStatistical analysisRats WistarLight exposuremedicine.diagnostic_testChemistrybusiness.industryRetinal DegenerationRat strainRats Inbred StrainsGeneral MedicineSensory SystemsIntensity (physics)RatsOphthalmologyEndocrinologyFemalebusinessErgElectroretinographyOphthalmic research
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Early lipoic acid intake protects retina of diabetic mice

2008

The aim of this study was to test the effect of lipoic acid treatment on the retina after a short diabetic insult. Diabetes was induced by alloxan and mice were divided into sub-groups; control, diabetic, diabetic+insulin and all groups received+/-lipoic acid (100 mg/kg body weight) for 3 weeks. GSH content, MDA concentration, GPx activity were measured and electroretinograms (ERG) were recorded. Early administration of lipoic acid to diabetic mice prevented the statistically significant decreases of GSH content and GPx activity and normalized MDA concentration. Moreover, lipoic acid restored electroretinogram b-wave amplitude of diabetic animals to control values. Lipoic acid has a protect…

Malemedicine.medical_specialtyAntioxidantgenetic structuresmedicine.medical_treatmentmedicine.disease_causeBiochemistryAntioxidantsDrug Administration ScheduleRetinaDiabetes Mellitus ExperimentalMicechemistry.chemical_compoundMalondialdehydeInternal medicineDiabetes mellitusAlloxanElectroretinographymedicineAnimalsHypoglycemic AgentsInsulinGlutathione PeroxidaseDiabetic RetinopathyThioctic Acidbusiness.industryInsulinGeneral MedicineGlutathionemedicine.diseaseGlutathioneeye diseasesOxidative StressLipoic acidEndocrinologychemistrylipids (amino acids peptides and proteins)sense organsbusinessErgOxidative stressFree Radical Research
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