Search results for "eps"

showing 10 items of 1777 documents

Análisis de procesos inmunorreguladores en helmintiasis intestinales mediante el modelo Echinostoma caproni-ratón

2021

En la presente Tesis Doctoral se analizan diferentes parámetros que intervienen en las interacciones parásito-hospedador en helmintiasis intestinales mediante el empleo del modelo experimental Echinostoma caproni-ratón ICR. Para ello, se realizan diferentes estudios en los que se analiza y compara la respuesta inmunitaria en infecciones primarias, asociadas a susceptibilidad, y secundarias, asociadas a resistencia. Los resultados obtenidos demuestran que la susceptibilidad del ratón a la infección primaria con E. caproni se debe a su incapacidad de responder con producción de IL-25 lo cual determina que se genere una respuesta Th1 asociada a susceptibilidad. Asimismo, estos resultados ponen…

proteómicacaptura de anticuerposcatepsinamicrobiota intestinalechinostomatidae:CIENCIAS DE LA VIDA [UNESCO]Th1Th2susceptibilidadcronicidadtrematodaechinostoma caproniUNESCO::CIENCIAS DE LA VIDAintestinomacrófagosdisbiosisinterleuquina-4activación alternativaresistenciainterleuquina-25interleuquina-13
researchProduct

Aplidin® induces JNK-dependent apoptosis in human breast cancer cells via alteration of glutathione homeostasis, Rac1 GTPase activation, and MKP-1 ph…

2006

Aplidin® is an antitumor agent in phase II clinical trials that induces apoptosis through the sustained activation of Jun N-terminal kinase (JNK). We report that Aplidin® alters glutathione homeostasis increasing the ratio of oxidized to reduced forms (GSSG/GSH). Aplidin® generates reactive oxygen species and disrupts the mitochondrial membrane potential. Exogenous GSH inhibits these effects and also JNK activation and cell death. We found two mechanisms by which Aplidin® activates JNK: rapid activation of Rac1 small GTPase and downregulation of MKP-1 phosphatase. Rac1 activation was diminished by GSH and enhanced by L-buthionine (SR)-sulfoximine, which inhibits GSH synthesis. Downregulatio…

rac1 GTP-Binding ProteinProgrammed cell deathSmall interfering RNAGlutathione reductaseDown-RegulationAntineoplastic AgentsApoptosisBreast NeoplasmsCell Cycle ProteinsBiologyPeptides CyclicImmediate-Early ProteinsMembrane Potentialschemistry.chemical_compoundMiceDownregulation and upregulationDepsipeptidesProtein Phosphatase 1Phosphoprotein PhosphatasesAnimalsHomeostasisHumansMolecular Biologychemistry.chemical_classificationReactive oxygen speciesGlutathione PeroxidaseGlutathione DisulfideJNK Mitogen-Activated Protein KinasesProtein phosphatase 1Dual Specificity Phosphatase 1Cell BiologyGlutathioneCell biologyEnzyme ActivationOxidative StressGlutathione ReductasechemistryMitochondrial MembranesGlutathione disulfideCalciumProtein Tyrosine PhosphatasesReactive Oxygen SpeciesCopperHeLa CellsCell Death and Differentiation
researchProduct

Muscle‐tendon morphology and function following long‐term exposure to repeated and strenuous mechanical loading

2020

We mapped structural and functional characteristics of muscle‐tendon units in a population exposed to very long‐term routine overloading. Twenty‐eight military academy cadets (age: 21.00 ± 1.1 yrs; height: 176.1 ± 4.8 cm; mass: 73.8 ± 7.0 kg) exposed for over 24 months to repetitive overloading were profiled via ultrasonography with a senior subgroup of them (n = 11; age = 21.4 ± 1.0 yrs; height = 176.5 ± 4.8 cm; mass = 71.4 ± 6.6 kg) also tested while walking and marching on a treadmill. A group of eleven ethnicity‐ and aged‐matched civilians (age = 21.6 ± 0.7 yrs; height = 176.8 ± 4.3 cm; mass = 74.6 ± 5.6 kg) was also profiled and tested. Cadets and civilians exhibited similar morphology…

rasitusultrasoundlihaksetylikuormitusfascicle mechanicsoverloadingjänteetmarchinghermo-lihastoimintamedial gastrocnemiustriceps suraefaskiatbiomekaniikkahuman activitiespatellar tendon
researchProduct

Synthesis and biological evaluation of novel peptidomimetics as rhodesain inhibitors

2016

Novel rhodesain inhibitors were developed by combining an enantiomerically pure 3-bromoisoxazoline warhead with a 1,4-benzodiazepine scaffold as specific recognition moiety. All compounds were proven to inhibit rhodesain with Ki values in the low-micromolar range. Their activity towards rhodesain was found to be coupled to an in vitro antitrypanosomal activity, with IC50 values ranging from the mid-micromolar to a low-micromolar value for the most active rhodesain inhibitor (R,S,S)-3. All compounds showed a good selectivity against the target enzyme since all of them were proven to be poor inhibitors of human cathepsin L. Novel rhodesain inhibitors were developed by combining an enantiomeri…

rhodesainPharmacologychemistry.chemical_classificationCathepsinPeptidomimetic010405 organic chemistryChemistryPeptidomimeticProton Magnetic Resonance SpectroscopyenPeptidomimetics; rhodesain; trypanosomaGeneral Medicine01 natural sciencesCombinatorial chemistryIn vitro0104 chemical sciencesCysteine Endopeptidases010404 medicinal & biomolecular chemistryEnzymeDrug DiscoveryIc50 valuesMoietyPeptidomimeticsCarbon-13 Magnetic Resonance SpectroscopytrypanosomaBiological evaluationJournal of Enzyme Inhibition and Medicinal Chemistry
researchProduct

Epilepsy in neuropathologically verified Alzheimer's disease

2018

Purpose Subjects with Alzheimer’s disease (AD) have been shown to be at a higher risk for epilepsy. The vast majority of the previous studies have not included a full neuropathological examination. Methods The objective of this study was to assess the prevalence of epilepsy and clinicopathological characteristics in a well-defined study group of 64 subjects with AD. We evaluated the clinicopathological findings in 64 subjects (mean age at death 85 ± 8.6 years) from a longitudi-nal study cohort of patients with dementia. Results Eleven out of the 64 subjects (17%) had a history of epilepsy, which is comparable to previous studies. The subjects with AD and epilepsy were significantly younger …

ruumiinavausalzheimerneurodegenerationAlzheimerin tautiepilepsiadementianeurodegeneratiiviset sairaudet
researchProduct

Tapausselostus CP-vammaisen lapsen musiikkiterapiakuntoutuksesta

2000

sensomotoriset aistimuksetCP-vammaisetsensomotorinen kehitysmusiikkiterapiamoniaistillinen kuntoutusepilepsia
researchProduct

Laboratoriskie rādītāji agrīna mirstības riska novērtēšanā multidisciplināra stacionāra intensīvās terapijas nodaļā pacientiem ar sadzīvē sākušos sep…

2022

Ievads. Sepse ir dzīvību apdraudoša akūta saslimšana, kas raksturojas ar pastiprinātu un disfunkcionālu imūnsistēmas atbildi uz infekciju. Tā izpaužas ar orgānu bojājumu, kam nepieciešama ātra terapijas uzsākšana. Ir būtiski identificēt sepses pacientus ar strauju slimības progresiju, kuriem ir agrīns mirstības risks. Ir pieejami dažādi ātri veicami, lēti laboratoriskie izmeklējumi, ko varētu izmantot agrīnas septiņu dienu mirstības riska novērtēšanā. Mērķis. Noskaidrot dažādu laboratorisku rādītāju saistību septiņu dienu mirstībai un novērtēt, kā tos varētu izmantot sepses mirstības rādītāju paredzēšanā intensīvās terapijas nodaļā (ITN). Metodes. Retrospektīvā kohorta pētījumā no 2020. gad…

sepsesadzīvē iegūtslaktātssadzīvē sāciesintensīvā terapijaMedicīna
researchProduct

Hyperoxia in critically ill patients with sepsis and septic shock: a systematic review

2023

Abstract Background In septic patients, hyperoxia may help with its bactericidal effects, but it may cause systemic impairments. The role of hyperoxia and the appropriate oxygen target in these patients is unknown. The aim of this systematic review was to summarize the available literature. Methods We conducted a systematic search screening PubMed and Cochrane Library. Studies on adult patients with sepsis or septic shock and admitted to ICU addressing the topic of hyperoxia were included and described. Results We included 12 studies, for a total of 15.782 included patients. Five studies were randomized controlled trials (RCTs) or analyses from RCTs, three were prospective observational stu…

sepsis
researchProduct

Antithrombin III and Activated Protein C for the treatment of severe sepsis with coagulopathy

2006

sepsisAT IIISettore MED/41 - AnestesiologiaaPC
researchProduct

Clostridium difficile Induced Inflammasome Activation and Coagulation Derangements

2022

C. difficile enterocolitis (CDAC) is the most common hospital infection, burdened by an increased incidence of coagulation-related complications such as deep vein thrombosis (DVT) and disseminated intravascular coagulation (DIC) as well as a significant sepsis-related mortality. In this review, we analyzed the available data concerning the correlation between coagulation complications related to C. difficile infection (CDI) and inflammasome activation, in particular the pyrin-dependent one. The little but solid available preclinical and clinical evidence shows that inflammasome activation increases the risk of venous thromboembolism (VTE). As proof of this, it has been observed that in vitr…

sepsisMicrobiology (medical)enterocolitiinduced inflammasome activationcoagulation derangementVirologyClostridium difficileMicrobiologyMicroorganisms
researchProduct