Search results for "fibrin"

showing 10 items of 508 documents

Prediction of cardio- and cerebro-vascular events in patients with subclinical carotid atherosclerosis and low HDL-cholesterol.

2007

Low HDL-cholesterol concentrations are associated with increased cardiovascular risk and recent evidences suggest that HDL may aggravate the atherosclerotic process promoting inflammation: HDL are anti-inflammatory in the absence of inflammation but can become proinflammatory in the presence of atherosclerosis. Yet, no data is available on the cardiovascular outcome in subjects with low HDL-cholesterol and early stages of atherosclerosis. Therefore, we included in a prospective 5-year follow-up study 150 subjects with low HDL-cholesterol concentrations and subclinical carotid atherosclerosis, as assessed by carotid colour doppler, evaluating at baseline all the established traditional cardi…

Carotid Artery DiseasesMalemedicine.medical_specialtyPathologyBlood PressureFibrinogenAsymptomaticBody Mass IndexCoronary artery diseaseRisk FactorsInternal medicineDiabetes mellitusMedicineHumansProspective StudiesFamily historySubclinical infectionAgedInflammationbusiness.industryVascular diseasecarotid atherosclerosislow HDL-cholesterolCholesterol HDLMiddle Agedmedicine.diseaseAtherosclerosisSettore MED/11 - Malattie Dell'Apparato Cardiovascolaremedicine.anatomical_structureCerebrovascular CirculationCardiologyFemalemedicine.symptomCardiology and Cardiovascular Medicinebusinessmedicine.drugArteryAterosclerosiFollow-Up StudiesAtherosclerosis
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An assessment of the hemorheological profile in patients with subclinical carotid atherosclerosis divided in relation to the number of cardiovascular…

2021

We present a cohort of 100 subjects [43 men and 57 women; median age 66.00(25)] who were tested using carotid ultrasound to identify subclinical carotid atherosclerosis (SCA). We have evaluated the behaviour of whole blood viscosity (WBV) at high (450 s–1) and low (0.51 s–1) shear rates, plasma viscosity (450–1), hematocrit and mean erythrocyte aggregation. When compared to normal control subjects, using the Mann-Whitney test, we observed in SCA patients a significant increase in WBV only. The results were substantial after having divided the SCA subjects according to the cardiovascular risk factors (CRFs) and the degree of insulin resistance; the research was performed using two surrogate …

Carotid Artery DiseasesMalemedicine.medical_specialtyPhysiologyCardiovascular risk factors030204 cardiovascular system & hematologyHematocritFibrinogenErythrocyte aggregation030218 nuclear medicine & medical imaging03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInsulin resistanceuric acidRisk FactorsPhysiology (medical)Internal medicinemedicineHumanstraditional cardiovascular risk factorsSubclinical infectionAgedSubclinical carotid atherosclerosismedicine.diagnostic_testbusiness.industryhemorheological determinantsHematologymedicine.diseaseBlood ViscositychemistryCardiovascular DiseasesHeart Disease Risk FactorsCohortCardiologyUric acidFemaleInsulin Resistanceinsuline-resistenceCardiology and Cardiovascular Medicinebusinessmedicine.drugClinical hemorheology and microcirculation
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Association of elevated fibrinogen and C-reactive protein levels with carotid lesions in patients with newly diagnosed hypertension or type II diabet…

2006

BACKGROUND: Inflammation may be important for accelerated progression of atherosclerosis in patients with hypertension or diabetes, but few studies included subjects with early stages of atherosclerosis such as those with asymptomatic carotid lesions. METHODS: We studied 100 patients with newly diagnosed hypertension and another 100 patients with newly diagnosed type II diabetes to evaluate in such groups the association of two markers of inflammation, fibrinogen and C-reactive protein (CRP), with carotid atherosclerosis, beyond traditional cardiovascular risk factors (e.g., older age, male gender, obesity, smoking, family history of CAD, dyslipidemia). RESULTS: We found positive correlatio…

Carotid Artery Diseasesmedicine.medical_specialtyInflammationFibrinogenAsymptomaticGastroenterologyInternal medicineDiabetes mellitusmedicineHumansC-reactive protein Hypertension Inflammation Events AtherosclerosisFamily historybiologybusiness.industryC-reactive proteinFibrinogenGeneral Medicinemedicine.diseaseObesityEndocrinologyC-Reactive ProteinCarotid ArteriesDiabetes Mellitus Type 2Hypertensionbiology.proteinmedicine.symptombusinessTunica MediaDyslipidemiamedicine.drugArchives of medical research
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Markers of inflammation are strong predictors of subclinical and clinical atherosclerosis in women with hypertension

2008

Cardiovascular diseases in women still rises and remains their leading cause of death in most developed countries; yet we have less sex-specific data in women than in men as a result of lower enrollment in clinical trials and low rates of sex-specific reporting. The aim of our study was to evaluate in hypertensive postmenopausal women the potential predictive role of markers of inflammation, for example, fibrinogen and C-reactive protein (CRP), on subclinical and clinical atherosclerosis, beyond that of the other established cardiovascular risk factors. We studied 127 asymptomatic hypertensive postmenopausal women with different degrees of carotid intima–media thickness, as examined by the …

Carotid Artery Diseasesmedicine.medical_specialtySettore MED/09 - Medicina InternaTime FactorsMEDLINERisk AssessmentSex FactorsPredictive Value of TestsRisk FactorsInternal medicineOdds RatiomedicineHumansUltrasonography Doppler ColoratherosclerosisMarkers of inflammationAtherosclerosis C-reactive protein events hypertension inflammationAgedSubclinical infectionCause of deathbusiness.industryFibrinogenGeneral MedicineOdds ratioMiddle AgedPostmenopauseClinical trialC-Reactive ProteinCardiovascular DiseasesPredictive value of testsHypertensionWomen's HealthFemaleInflammation MediatorsCardiology and Cardiovascular MedicineRisk assessmentbusinessDeveloped countryBiomarkersCoronary Artery Disease
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Candida albicans fibrinogen binding mannoprotein: expression in clinical strains and immunogenicity in patients with candidiasis

1998

A 58 kDa cell wall-associated fibrinogen binding mannoprotein (mp58), previously characterized by our group in a Candida albicans laboratory strain (ATCC 26555), was found to be also present in the cell wall of clinical isolates of this fungus. Most strains examined appear to have functional mp58 species, as detected by their ability to bind fibrinogen. Western immunoblot analysis, with a monovalent polyclonal antibody generated against the mp58 species from strain ATCC 26555, revealed differences in recognition patterns depending on the strain tested and the culture conditions used. Serum samples from normal and Candida infected individuals were examined for the presence of antibodies agai…

Cell wall:CIENCIAS DE LA VIDA::Microbiología [UNESCO]Candida albicansFibrinogen-binding mannoproteinClinical strainsSerologic responseUNESCO::CIENCIAS DE LA VIDA::MicrobiologíaCandida albicans; Clinical strains; Cell wall; Fibrinogen-binding mannoprotein ; Serologic response
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Interaction of inflammation, thrombosis, aspirin and enoxaparin in CNS experimental antiphospholipid syndrome

2008

Experimental antiphospholipid syndrome (eAPS) induced by immunization with beta(2)-glycoprotein I (beta(2)-GPI) causes behavioral hyperactivity. We assessed the role of thrombotic and inflammatory perivascular factors and standard APS therapies for CNS manifestations. Groups of mice (n=10 per group) were immunized once with beta(2)-GPI (eAPS) or adjuvant (controls) and treated daily from 1 month after immunization with either sham injections, aspirin (1.2 mg/kg) or enoxaparin (1 mg/kg) for 3 months. Serum antiphospholipid antibodies (aPL) and brain levels of tissue necrosis factor-alpha (TNF-alpha) and prostaglandin E (PGE) were then measured by ELISA and thrombin inhibitors by immunoblot. …

Central Nervous Systemmedicine.medical_treatmentEnzyme-Linked Immunosorbent AssayInflammationPharmacologylcsh:RC321-571AnticoagulationMiceFibrinolytic AgentsAntiphospholipid syndromeAnimalsMedicineBeta 2-Glycoprotein IAlprostadilEnoxaparinlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryPhospholipidsInflammationBehaviorAnalysis of VarianceMice Inbred BALB CAspirinAspirinBehavior AnimalTumor Necrosis Factor-alphabusiness.industryThrombosisAntiphospholipid Syndromemedicine.diseaseThrombosisAnimal modelsDisease Models AnimalNeurologybeta 2-Glycoprotein IImmunologyExploratory BehaviorFemaleTumor necrosis factor alphamedicine.symptombusinessDiscovery and development of direct thrombin inhibitorsProstaglandin Emedicine.drugNeurobiology of Disease
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Associated arterial and venous cerebral manifestations in Behçet's disease

2018

Behcet's disease is a rare multisystemic vasculitis with an etiology that is still unknown. Neurological manifestations may be seen in approximately 5-15% of patients, and both parenchymal and extraparenchymal neurological involvement has been described. When cerebral venous thrombosis (CVT) is the main extraparenchymal manifestation of Behcet's disease, the condition is then dubbed "angio-Behcet's syndrome". However, arterial involvement is extremely rare, with only one reported case of vasculo-neuro-Behcet's disease, characterized by both venous and intracranial arterial involvement - until now. This report is of two patients diagnosed with Behcet's disease characterized by the concomitan…

Cerebral veinsmedicine.medical_specialtymedicine.diagnostic_testbusiness.industryBehcet's disease030204 cardiovascular system & hematologymedicine.diseaseeye diseasesstomatognathic diseases03 medical and health sciencesVenous thrombosis0302 clinical medicineIntracranial ThrombosisNeurologyInternal medicineEtiologymedicineCardiologyNeurology (clinical)Vasculitisbusiness030217 neurology & neurosurgeryFibrinolytic agentCerebral angiographyRevue Neurologique
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Oral Glycoprotein IIb/IIa Antagonists for Unstable Angina — Is There Still a Chance for the Oral Substances?

2001

The intravenous glycoprotein IIb/IIIa antagonists abciximab, tirofiban and eptifibatide are well accepted for the therapy of patients with unstable angina and/or as concomitant medication during coronary interventions. Despite the fact that these drugs are not used in all patients presenting with unstable angina during coronary interventions, the scientific evidence is overwhelming including the substantial reduction in mortality 3 years after utilisation of abciximab for coronary interventions in patients with unstable angina. In addition to these two indications, intravenous glycoprotein IIb/IIIa antagonists are currently being investigated for use in patients undergoing carotid artery in…

Clinical Trials as Topicmedicine.medical_specialtyUnstable anginaFibrinogen receptorbusiness.industryAdministration OralPlatelet Glycoprotein GPIIb-IIIa ComplexHematologyTirofibanmedicine.diseaseAnginaInternal medicinemedicineAbciximabCardiologyEptifibatideHumansAngina UnstableMyocardial infarctionbusinessStrokePlatelet Aggregation Inhibitorsmedicine.drugThrombosis Research
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Intramuscular tranexamic acid

2021

For many anaesthetists around the world, the mere mention of the word ‘pharmacokinetics’ is sufficient to make their eyes glaze over and their attention wander. Pharmacokinetics is seen as an art that is as obscure and esoteric as the art of divination (prediction) practiced by the likes of Professor Sybille Trelawny 1 but that has varying and mostly limited relevance to clinical practice. Although this hyperbole may have elements of truth, it is a fact that pharmacokinetic data are the essential foundation upon which rational drug dosing guidelines are developed for all drugs.

Clinical pharmacologybusiness.industryMORTALITYPOSTPARTUM HEMORRHAGEPostpartum haemorrhageAntifibrinolytic Agentstranexamic acidlaw.inventionAnesthesiology and Pain MedicinetraumaPharmacokineticsPregnancylawpostpartum haemorrhageAnesthesiaManagement of Technology and InnovationmedicineHumansFemaleclinical pharmacologybusinesspharmacokineticsTranexamic acidmedicine.drugBritish Journal of Anaesthesia
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Coagulation and fibrinolysis

2016

Coagulation fibrinolysis laboratory diagnosis
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