Search results for "granulocytes"

showing 10 items of 63 documents

Effects of Short- and Long-term Ultraviolet B Irradiation on the Immune System of the Common Carp (Cyprinus carpio)¶

2005

Carp (Cyprinus carpio) were repeatedly exposed to 0, 60, 120 and 240 mJ/cm2 ultraviolet B (UVB) radiation three times in 1 week (short-term exposure) or 12 times in 4 weeks (long-term exposure). The effect of UVB on the functioning of the carp immune system was studied on day 2 after the final irradiation. After short-term UVB exposure, the whole-blood respiratory burst and cytotoxic activity were markedly enhanced, with parallel responses in both the number of circulating granulocytes and in the plasma cortisol concentration of the fish. These changes were not detectable after long-term exposure. The respiratory burst by head kidney granulocytes was suppressed dose dependently after both e…

Cytotoxicity Immunologicmedicine.medical_specialtyCarpsHydrocortisoneUltraviolet RaysLymphocyteKidneyBiochemistryCyprinusCommon carpImmune systemInternal medicinemedicineAnimalsCytotoxic T cellLymphocytesPhytohemagglutininsPhysical and Theoretical ChemistryCarpHead Kidneyintegumentary systembiologyChemistryGeneral Medicinebiology.organism_classificationRespiratory burstmedicine.anatomical_structureEndocrinologyImmunoglobulin MImmune SystemImmunologyGranulocytesPhotochemistry and Photobiology
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Transglutaminase and polyamine dependence of effector functions of human immunocompetent cells

1982

AbstractThe effects of the transglutaminase inhibitor dansyl cadaverine (DC) and the polyamine antagonist methyl glyoxal-bis-(guanylhydrazone) (MeGbG) on the response of lymphocytes towards allogeneic and lectin stimulation and on the zymosan-induced chemiluminescence of neutrophilic granulocytes was studied. Application of DC resulted in dose-dependent suppression of chemiluminiscence and lymphocyte proliferation; no difference of inhibitory potential occurred with variation of incubation time in the latter system. MeGbG was inactive in granulocytes, but inhibited lymphocyte proliferation; its effect increased with time. The experiments provide further evidence for the importance of transg…

Dansyl cadaverineDNA ReplicationPolyamineLuminescenceMitoguazoneTissue transglutaminaseLymphocyteBiophysicsStimulationLymphocyte proliferationDiaminesGranulocyteBiologyLymphocyte ActivationGuanidinesBiochemistrylaw.inventionMeGbGchemistry.chemical_compoundStructural BiologylawCadaverinePolyaminesGeneticsmedicineHumansLymphocytesMolecular BiologyChemiluminescenceCadaverineTransglutaminasesGranulocyteCell BiologyTransglutaminaseMolecular biologyKineticsmedicine.anatomical_structurechemistryBiochemistrybiology.proteinLymphocytePolyamineAcyltransferasesGranulocytesFEBS Letters
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IL-17A and IL-17F do not contribute vitally to autoimmune neuro-inflammation in mice

2009

The clear association of Th17 cells with autoimmune pathogenicity implicates Th17 cytokines as critical mediators of chronic autoimmune diseases such as EAE. To study the impact of IL-17A on CNS inflammation, we generated transgenic mice in which high levels of expression of IL-17A could be initiated after Cre-mediated recombination. Although ubiquitous overexpression of IL-17A led to skin inflammation and granulocytosis, T cell–specific IL-17A overexpression did not have a perceptible impact on the development and health of the mice. In the context of EAE, neither the T cell–driven overexpression of IL-17A nor its complete loss had a major impact on the development of clinical disease. Sin…

Encephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentT cellEncephalomyelitisPopulation610 Medicine & healthMice TransgenicInflammation2700 General Medicine10263 Institute of Experimental ImmunologyMyelin oligodendrocyte glycoproteinMicemedicineAnimalseducationCells CulturedGlycoproteinseducation.field_of_studybiologybusiness.industryInterleukin-17General MedicineTh1 Cellsmedicine.diseasePeptide FragmentsMice Inbred C57BLCytokinemedicine.anatomical_structureImmunologybiology.protein570 Life sciences; biologyExperimental pathologyFemaleMyelin-Oligodendrocyte GlycoproteinInterleukin 17medicine.symptombusinessGranulocytesResearch Article
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Characterization of a novel population of low-density granulocytes associated with disease severity in HIV-1 infection

2012

The mechanisms resulting in progressive immune dysfunction during the chronic phase of HIV infection are not fully understood. We have previously shown that arginase, an enzyme with potent immunosuppressive properties, is increased in HIV seropositive (HIV+) patients with low CD4(+) T cell counts. Here we show that the cells expressing arginase in peripheral blood mononuclear cells of HIV+ patients are low-density granulocytes (LDGs) and that whereas these cells have a similar morphology to normal-density granulocyte, they are phenotypically different. Importantly, our results reveal that increased frequencies of LDGs correlate with disease severity in HIV+ patients.

Enzyme Metabolismlcsh:MedicineHIV InfectionsBiochemistryACTIVATION0302 clinical medicineImmunophenotypingImmunodeficiency VirusesRENAL-CELL CARCINOMAHIV SeropositivityMedicineSUPPRESSOR-CELLSlcsh:ScienceImmune ResponseCD180303 health scienceseducation.field_of_studyMultidisciplinarymedicine.diagnostic_testT Cellsvirus diseasesMiddle Aged3. Good healthEnzymesSEROPOSITIVE PATIENTSArginasemedicine.anatomical_structurePhenotypeHIV epidemiologyDisease ProgressionMedicineInfectious diseasesScience & Technology - Other TopicsNEUTROPHILResearch ArticleAdultGeneral Science & TechnologyT cellImmune CellsPopulationImmunologyCD18Viral diseasesGranulocytePeripheral blood mononuclear cellMicrobiologyFlow cytometryImmunophenotyping03 medical and health sciencesADHERENCEVirologyMD MultidisciplinaryHumanseducationBiology030304 developmental biologyScience & TechnologyArginasebusiness.industryTetraspanin 30MULTIDISCIPLINARY SCIENCESlcsh:RARGINASE-IHIVVirologyENDOTHELIAL-CELLSAntigens CD63ImmunologyLeukocytes Mononuclearlcsh:Qbusiness030215 immunologyGranulocytes
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Binding of Escherichia coli hemolysin and activation of the target cells is not receptor-dependent.

2005

Abstract Production of a single cysteine substitution mutant, S177C, allowed Escherichia coli hemolysin (HlyA) to be radioactively labeled with tritiated N-ethylmaleimide without affecting biological activity. It thus became possible to study the binding characteristics of HlyA as well as of toxin mutants in which one or both acylation sites were deleted. All toxins bound to erythrocytes and granulocytes in a nonsaturable manner. Only wild-type toxin and the lytic monoacylated mutant stimulated production of superoxide anions in granulocytes. An oxidative burst coincided with elevation of intracellular Ca2+, which was likely because of passive influx of Ca2+ through the toxin pores. Competi…

ErythrocytesAcylationMutantBacterial ToxinsBiologymedicine.disease_causeBiochemistryHemolysin ProteinsSuperoxidesmedicineEscherichia coliHumansReceptorMolecular BiologyEscherichia coliRespiratory BurstSequence DeletionBinding SitesToxinHemolysinBiological activityCell BiologyMolecular biologyLymphocyte Function-Associated Antigen-1Respiratory burstBiochemistryAmino Acid SubstitutionMutationMutagenesis Site-DirectedbacteriaCalciumK562 CellsIntracellularGranulocytesThe Journal of biological chemistry
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Immunohistological differential diagnosis of inflammatory colonic diseases.

1984

Immunohistological investigations were carried out on human colonic tissue from, I healthy mucosa, 2 slightly inflamed mucosa, 3 mucosa with ulcerative colitis, 4 mucosa with Crohn's colitis, using antibodies against immunoglobulins and complement components. All our antibodies, including F(ab')2 fragments, demonstrated a progressive increase of labelled cells from healthy mucosa through slightly inflamed mucosa to mucosa with ulcerative colitis, in contrast to a complete absence of labelled cells in cases of Crohn's disease. The results are discussed with regard to their pathogenesis and their clinical significance for the differentiation of ulcerative colitis and Crohn's colitis.

Immunoglobulin Amedicine.medical_specialtyPathologyHistologyComplement Activating EnzymesGastroenterologyPathology and Forensic MedicinePathogenesisDiagnosis DifferentialCrohn DiseaseInternal medicinemedicineHumansColitisCrohn's diseasebiologybusiness.industryHistocytochemistryComplement C1qImmunochemistryComplement C4General MedicineComplement C3medicine.diseaseColitisUlcerative colitisdigestive system diseasesImmunoglobulin AImmunoglobulin MImmunoglobulin Mbiology.proteinImmunohistochemistryColitis UlcerativeAntibodybusinessGranulocytesHistopathology
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Induction of interferon regulatory factors, 2′‐5′ oligoadenylate synthetase, P68 kinase and RNase L in chronic myelogenous leukaemia cells and its re…

1996

The genes crucially determining the therapeutic response of chronic myelogenous leukaemia (CML) to interferon-alpha (IFN-alpha) are unknown. Recently, two independent IFN-alpha signalling pathways were identified: the classic pathway mediates induction of 2'-5' oligoadenylate synthetase (2-5 OAS), p68 kinase and IFN regulatory factor-2 (IRF-2), whereas the alternate pathway leads to activation of IFN regulatory factor-1 (IRF-1). We investigated whether deficient or imbalanced expression of components of these two pathways is associated with resistance of CML cells to antiproliferative action of IFN alpha/beta. Constitutive and IFN-induced transcript levels of IFN-dependent genes in mononucl…

Interferon Regulatory Factor 2T-LymphocytesCellular differentiationmedicine.medical_treatmentProtein Serine-Threonine KinaseseIF-2 KinaseLeukemia Myelogenous Chronic BCR-ABL PositiveEndoribonucleases2'5'-Oligoadenylate SynthetasemedicineHumansRNA MessengerTreatment FailureInterferon alfaEIF-2 kinasebiology2'-5'-OligoadenylateInterferon-alphaHematologyBlotting NorthernHematopoietic Stem CellsPhosphoproteinsDNA-Binding ProteinsGene Expression Regulation NeoplasticRepressor ProteinsCytokineIRF1Cancer researchbiology.proteinInterferon Regulatory Factor-2GranulocytesInterferon Regulatory Factor-1Transcription Factorsmedicine.drugInterferon regulatory factorsBritish Journal of Haematology
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Unsaturated Fatty Acids Drive Disintegrin and Metalloproteinase (ADAM)-dependent Cell Adhesion, Proliferation, and Migration by Modulating Membrane F…

2011

The disintegrin-metalloproteinases ADAM10 and ADAM17 mediate the release of several cell signaling molecules and cell adhesion molecules such as vascular endothelial cadherin or L-selectin affecting endothelial permeability and leukocyte transmigration. Dysregulation of ADAM activity may contribute to the pathogenesis of vascular diseases, but the mechanisms underlying the control of ADAM functions are still incompletely understood. Atherosclerosis is characterized by lipid plaque formation and local accumulation of unsaturated free fatty acids (FFA). Here, we show that unsaturated FFA increase ADAM-mediated substrate cleavage. We demonstrate that these alterations are not due to genuine ch…

KeratinocytesMembrane FluidityADAM10Lipid BilayersVascular permeabilityBiologyADAM17 ProteinBiochemistryCapillary PermeabilityADAM10 ProteinCell MovementMembrane fluidityCell AdhesionAnimalsHumansCell adhesionMolecular BiologyCell ProliferationCell adhesion moleculeCell growthFluorescence recovery after photobleachingEndothelial CellsMembrane ProteinsCell BiologyAtherosclerosisADAM ProteinsCell biologyLipoproteins LDLADAM ProteinsHEK293 CellsFatty Acids UnsaturatedCholesterol EstersRabbitsAmyloid Precursor Protein SecretasesGranulocytes
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Effects of ultraviolet light on immune parameters of the roach

2000

Ultraviolet B radiation penetrates into water and can affect fish health and the immune system, as is the case with mammals. Teleost fish, the roach, were exposed to UVB irradiation in aquariums and a panel of immune parameters was determined. In addition to altered blood picture and respiratory burst by blood leukocytes, changes were noted also in major lymphatic organs. Respiratory burst and natural cytotoxicity activity of head kidney granulocytes and mitogen-activated proliferation of splenic lymphocytes were suppressed. Although mostly transitory, some parameters remained suppressed for the following 2 weeks. Ultraviolet A radiation had only minor effects. The stress induced by UVB may…

KidneyUltraviolet RaysFishesGeneral MedicineBiologyKidneyToxicologyUltraviolet A RadiationRespiratory burstLymphatic systemmedicine.anatomical_structureImmune systemImmune SystemImmunopathologyImmunologymedicineUltraviolet lightAnimalsHumansLymphocytesCytotoxicityHeadGranulocytesRespiratory BurstToxicology Letters
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Essential role of surface-bound chemoattractant in leukocyte migration

1977

MANY chemotactic factors, usually proteins or peptides, have been isolated and studied, but little is known about the basic mechanism of leukocyte migration. This movement is termed chemotaxis if its direction is determined by substances in the cells' environment1. The chemotactic agent is assumed to convey information to the leukocytes by interaction with receptors. The subsequent sequence of events thus triggered in the cells is unknown but metabolic changes such as activation of an esterase have been reported as occurring as the cells move forward (for review see ref. 2). A role for surface-bound chemoattractant in cell locomotion was suggested by the observation that mouse fibroblasts m…

Leukocyte migrationBinding SitesMultidisciplinaryChemistryPhagocytosisGuinea PigsCellCaseinsChemotaxisIn Vitro TechniquesCell biologyChemotaxis Leukocytemedicine.anatomical_structureMembraneSolubilityCaseinLeukocytesmedicineAnimalsReceptorOpsoninGranulocytesNature
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