Search results for "hepatocyte"

showing 10 items of 369 documents

Effects of a high-fat diet on energy metabolism and ROS production in rat liver.

2011

International audience; BACKGROUND & AIMS: A high-fat diet affects liver metabolism, leading to steatosis, a complex disorder related to insulin resistance and mitochondrial alterations. Steatosis is still poorly understood since diverse effects have been reported, depending on the different experimental models used. METHODS: We hereby report the effects of an 8 week high-fat diet on liver energy metabolism in a rat model, investigated in both isolated mitochondria and hepatocytes. RESULTS: Liver mass was unchanged but lipid content and composition were markedly affected. State-3 mitochondrial oxidative phosphorylation was inhibited, contrasting with unaffected cytochrome content. Oxidative…

Mitochondrial ROSMaleTranscription GeneticMESH : Reactive Oxygen SpeciesMitochondria LiverMESH : HepatocytesMitochondrionOxidative PhosphorylationMESH: Hepatocytes0302 clinical medicineMESH: Membrane Potential MitochondrialCitrate synthaseMESH: AnimalsBeta oxidationMESH : Electron Transport2. Zero hungerMembrane Potential Mitochondrial0303 health sciencesMESH : RatsAdenine nucleotide translocatorMESH: Energy MetabolismMESH: Reactive Oxygen SpeciesLipidsBiochemistryLiverMESH: Dietary FatsMitochondrial matrix030220 oncology & carcinogenesisBody CompositionMESH : Oxidative PhosphorylationATP–ADP translocaseMESH: Mitochondria LiverMESH: RatsMESH : Body CompositionMESH : MaleOxidative phosphorylationBiologyMESH : Rats WistarElectron Transport03 medical and health sciencesMESH: Oxidative Phosphorylation[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyRats WistarMESH: Electron Transport[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular Biology030304 developmental biologyHepatologyMESH: Transcription GeneticMESH : Transcription GeneticMESH : LiverMESH : LipidsMESH: Body CompositionMESH: Rats WistarMESH: LipidsDietary FatsMESH: MaleRatsMESH : Energy MetabolismMESH : Membrane Potential MitochondrialMESH : Mitochondria Liverbiology.proteinHepatocytesMESH : AnimalsEnergy MetabolismReactive Oxygen SpeciesMESH : Dietary FatsMESH: Liver
researchProduct

Intracellular glutathione in human hepatocytes incubated with S-adenosyl-L-methionine and GSH-depleting drugs

1991

Abstract The present study was undertaken to investigate (a) whether S- adenosyl- L -methionine (SAMe) added to culture medium can increase intracellular glutathione (GSH) levels in human hepatocytes and (b) whether SAMe can prevent the GSH depletion found in human hepatocytes incubated with GSH-depleting drugs (paracetamol, opiates, ethanol). Incubation of hepatocytes with increasing concentrations of SAMe resulted in a dose-dependent elevation of intracellular GSH content, which reached its maximum (35% increase) at 30 μM after 20 h. SAMe, as the only sulfur source in the medium, was efficient in repleting GSH-depleted hepatocytes following treatment with diethyl maleate. Incubation of hu…

NarcoticsS-Adenosylmethioninemedicine.medical_treatmentPharmacologyToxicologychemistry.chemical_compoundmedicineHumansAntidoteIncubationCells CulturedAcetaminophenEthanolMethionineDose-Response Relationship DrugEthanolGlutathioneGlutathioneHeroinmedicine.anatomical_structureLiverchemistryBiochemistryHepatocyteToxicityMethadoneIntracellularToxicology
researchProduct

Emerging Therapeutic Strategies for Traumatic Spinal Cord Injury

2020

Spinal cord injury (SCI) is a debilitating neurologic condition with tremendous socioeconomic impact on affected individuals and the health care system. The treatment of SCI principally includes surgical treatment and marginal pharmacologic and rehabilitation therapies targeting secondary events with minor clinical improvements. This unsuccessful result mainly reflects the complexity of SCI pathophysiology and the diverse biochemical and physiologic changes that occur in the injured spinal cord. Once the nervous system is injured, cascades of cellular and molecular events are triggered at varying times. Although the cascade of tissue reactions and cell injury develops over a period of days …

Nervous systemmedicine.medical_specialtyCordmedicine.medical_treatmentSpinal cord injuryRegenerative MedicineMesenchymal Stem Cell TransplantationNeuroprotection03 medical and health sciences0302 clinical medicineNeural Stem CellsGlyburideGranulocyte Colony-Stimulating FactormedicineHumansHypoglycemic AgentsIntensive care medicineErythropoietinSpinal cord injurySpinal Cord InjuriesNeuronal PlasticityRehabilitationCombination treatmentsHepatocyte Growth Factorbusiness.industryNeurological RehabilitationDecompression SurgicalSpinal cordmedicine.diseaseNeuroregenerationNeuroprotectionClinical trialFibroblast Growth FactorsClinical trialmedicine.anatomical_structure030220 oncology & carcinogenesisIntercellular Signaling Peptides and ProteinsSurgeryNeuroregenerationSchwann CellsNeurology (clinical)business030217 neurology & neurosurgeryStem Cell TransplantationWorld Neurosurgery
researchProduct

Uridine uptake inhibition as a cytotoxicity test for a human hepatoma cell line (HepG2 cells): comparison with the neutral red assay

2001

International audience; This study describes a sensitive microassay for measuring cytotoxicity based on the degree of inhibition of RNA synthesis in HepG2 cells. RNA synthesis is measured by the kinetic uptake of radiolabeled uridine. A large number of compounds were tested in a wide range of concentrations. The concentration required to induce 50% inhibition of HepG2 uridine uptake rates (IC50) was determined for each compound and used to rank its potency. These IC50s were compared with IC50s measured with the neutral red assay. 2-acetylaminofluorene, benzo[a]pyrene and methylnitrosourea were not cytotoxic in the neutral red assay. Uridine uptake was always inhibited at lower concentrations…

Neutral redCarcinoma Hepatocellular[SDV.TOX.TCA]Life Sciences [q-bio]/Toxicology/Toxicology and food chainToxicologyXenobiotics03 medical and health scienceschemistry.chemical_compoundInhibitory Concentration 500302 clinical medicineNeutral redToxicity TestsTumor Cells CulturedPotencyCytotoxic T cellHumansBenzopyrenesCytotoxicityColoring AgentsUridine030304 developmental biology0303 health sciencesReproducibility of ResultsMethylnitrosourea2-AcetylaminofluoreneUridine uptakeIn vitroUridineKineticschemistryBiochemistryCytotoxicity-helpG2 cell line[SDV.TOX.TCA] Life Sciences [q-bio]/Toxicology/Toxicology and food chain030220 oncology & carcinogenesisToxicityCarcinogensHepatocytesPyreneRNARegression AnalysisWater Pollutants Chemical
researchProduct

Comparative analysis of eight cytotoxicity assays evaluated within the ACuteTox Project.

2013

A comparative analysis of eight cytotoxicity assays [the 3T3 and normal human keratinocytes Neutral Red Uptake (NRU) assay, the primary rat hepatocytes, human HepG2 and 3T3 MU assay, and the human A.704, SH-SY5Y and HepG2 cells propidium iodide (PI) assay] included in several work packages of the EU Integrated Project ACuteTox, has been carried out. The aim was to evaluate whether cells originating from liver, kidney and brain provided different in vitro acute toxicity results, and the influence of primary liver cells versus cell lines originated from the same tissue. Spearman rank correlation analysis and Hierarchical Cluster Analysis were performed based on the IC50 (50% inhibitory concen…

Neutral redCell SurvivalCytotoxicityBiologyToxicologyConcentration-response analysischemistry.chemical_compoundMiceCell Line TumorToxicity Tests AcuteAnimalsHumansMTT assayPropidium iodideCytotoxicityCells CulturedAcute toxicityCytotoxinsIn vitro toxicologyCorrelation analysisGeneral Medicine3T3 CellsMolecular biologyAcute toxicityRatsHierarchical Cluster AnalysischemistryCell cultureToxicityImmunologyIn vitro assaysHepatocytesToxicology in vitro : an international journal published in association with BIBRA
researchProduct

Sequential Hepatogenic Transdifferentiation of Adipose Tissue-Derived Stem Cells: Relevance of Different Extracellular Signaling Molecules, Transcrip…

2009

Adipose tissue contains a mesenchymal stein cell (MSC) population Known as adipose-derived stein cells (ASCs) capable of differentiating into different cell types. Our aim was to induce hepatic transdifferentiation of ASCs by sequential exposure to several combinations of cytokines, growth factors, and hormones. The most efficient hepatogenic protocol includes fibroblastic growth factors (FGF) 2 and 4 and epidermal growth factor (EGF) (step 1), hepatocyte growth factor (HGF), FGF2, FGF4, and nicotinamide (Nic) (step 2), and oncostatin M (OSM), dexamethasone (Dex), and insulin-tranferrin-selenium (step 3). This protocol activated transcription factors [GATA6, Hex, CCAAT/enhancer binding prot…

NiacinamideCellular differentiationBiomedical Engineeringlcsh:MedicineOncostatin MBiologyDexamethasoneSeleniumEpidermal growth factorEnhancer bindingHumansInsulinCells CulturedHepatocyte differentiationTransplantationHepatocyte Growth FactorGene Expression Profilinglcsh:RTransdifferentiationTransferrinMesenchymal Stem CellsHep G2 CellsCell BiologyFlow CytometryMolecular biologyCell biologyFibroblast Growth FactorsAdipose TissueHepatocyte Nuclear Factor 4Hepatocyte nuclear factor 4 alphaCell TransdifferentiationHepatocytesStem cellSignal TransductionTranscription FactorsAdult stem cellCell Transplantation
researchProduct

Nanoassemblies Based on Supramolecular Complexes of Nonionic Amphiphilic Cyclodextrin and Sorafenib as Effective Weapons to Kill Human HCC Cells

2015

Sorafenib (Sor), an effective chemiotherapeutic drug utilized against hepatocellular carcinoma (HOC), robustly interacts with nonionic amphiphilic cyclodextrin (aCD, SC6OH), forming, in aqueous solution, supramolecular complexes that behave as building blocks of highly water-dispersible colloidal nanoassemblies. SC6OH/Sor complex has been characterized by complementary spectroscopic techniques, such as UV-vis, steady-state fluorescence and anisotropy, resonance light scattering and H-1 NMR. The spectroscopic evidences and experiments carried out in the presence of an adamantane derivative, which competes with drug for CD cavity, agree with the entrapment of Sor in aCD, pointing out the role…

NiacinamideErythrocytesPolymers and PlasticsCell SurvivalAdamantaneDrug CompoundingSupramolecular chemistryBioengineeringNanotechnologyAdamantaneAntineoplastic AgentsBinding CompetitiveHemolysisAmphiphilic Cyclodextrins; Nanoparticles; Sorafenib; HCC cellsHCC cellsBiomaterialschemistry.chemical_compoundSurface-Active AgentsIn vivoCell Line TumorAmphiphileMaterials ChemistryHumanschemistry.chemical_classificationCyclodextrinsAqueous solutionCyclodextrinPhenylurea CompoundsSorafenibFluorescenceCombinatorial chemistrydigestive system diseasesNanostructuresBINDING INTERACTION THERAPY PHARMACOKINETICS BIOAVAILABILITY NANOPARTICLESDrug LiberationKineticsnanoassembliecyclodextrinchemistryDelayed-Action PreparationsProton NMRHepatocytes
researchProduct

Biological effects of high molecular weight lignin derivatives

2010

Abstract A number of high molecular weight (HMW) lignin derivatives possessing varied chemical properties were screened for their biological effects in order to obtain more information on the possible structural features of HMW lignin-related effects. The studied compounds were both commercial and in-house extracted lignin derivatives. Bioassays used include reverse electron transport (RET), Vibrio fischeri, Daphnia magna , and juvenile rainbow trout ( Oncorhynchus mykiss) hepatocytes. The studied lignin derivatives inhibited the in vitro systems and luminescence of V. fischeri bacteria to some extent–daphnids were not affected. It seems that, at least in the RET assay, certain pH-dependent…

Paperendocrine systemHealth Toxicology and MutagenesisDaphnia magnaLigninWaste Disposal Fluidcomplex mixturesMicrobiologyElectron Transportchemistry.chemical_compoundToxicity TestsCytochrome P-450 CYP1A1AnimalsLigninBioassayFinlandVibriobiologyfungitechnology industry and agriculturePublic Health Environmental and Occupational Healthfood and beveragesBiological activityGeneral Medicinebiology.organism_classificationPollutionIn vitroVibrioReverse electron flowDaphniachemistryBiochemistryOncorhynchus mykissHepatocytesBiological AssayWater Pollutants ChemicalBacteriaEcotoxicology and Environmental Safety
researchProduct

Expression of T-cadherin in tumor cells influences invasive potential of human hepatocellular carcinoma

2006

Overexpression of T-cadherin (T-cad) transcripts occurs in approximately 50% of human hepatocellular carcinomas (HCCs). To elucidate T-cad functions in HCC, we examined T-cad protein expression in normal and tumoral human livers and hepatoma cell lines and investigated its influence on invasive potential of HCC using RNA interference silencing of T-cad expression in Mahlavu cells. Whereas T-cad expression was restricted to endothelial cells (EC) from large blood vessels in normal livers, it was up-regulated in sinusoidal EC from 8/15 invasive HCCs. Importantly, in three of them (38%) T-cad was detected in tumor cells within regions in which E-cadherin expression was absent. Among six hepato…

Pathologymedicine.medical_specialtyCarcinoma HepatocellularTranscription GeneticLiver cytologyCell Culture TechniquesMotilityBiologyTransfectionBiochemistryRNA interferenceCell MovementCell Line TumorGeneticsmedicineGene silencingAnimalsHumansNeoplasm Invasivenesscardiovascular diseasesRNA Small InterferingMolecular BiologyDNA PrimersWound Healingprimary tumors cadherin switch cell invasion hepatoma cell lines RNA interferenceLiver NeoplasmsEndothelial CellsTransfectionHCCSFibroblastsCadherinsdigestive system diseasesT-cadherinLiverCell cultureCancer researchHepatocytesRabbitsCell DivisionBiotechnology
researchProduct

Hepatic progenitors for liver disease: current position

2010

Alice Conigliaro1, David A Brenner2, Tatiana Kisseleva21University “La Sapienza”, Dipartimento di Biotecnologie Cellulari ed Ematologia Policlinico Umberto I, V Clinica Medica, Rome, Italy; 2Department of Medicine, University of California, San Diego, La Jolla, CA, USAAbstract: Liver regeneration restores the original functionality of hepatocytes and cholangiocytes in response to injury. It is regulated on several levels, with different cellular populations contributing to this process, eg, hepatocytes, liver precursor cells, intrahepatic stem cells. In response to injury, mature hepatocytes have the capability to proliferate and give rise to new hepatocytes and cholangi…

Pathologymedicine.medical_specialtyoval cellsLiver cytologyMedicine (miscellaneous)cholangiocytes; hepatic progenitor; hepatocytes; intrahepatic stem cells; liver disease; liver precursor cells; oval cellsReviewBiologyhepatocytemedicinecholangiocytesProgenitor cellliver precursor cellQH573-671Mesenchymal stem cellintrahepatic stem cellCell Biologyhepatic progenitorliver precursor cellsintrahepatic stem cellsLiver regenerationCell biologyHaematopoiesisAmniotic epithelial cellsHepatic stellate cellhepatocytesStem cellCytologyliver diseasecholangiocyteStem Cells and Cloning: Advances and Applications
researchProduct