Search results for "hormones"

showing 10 items of 1169 documents

Chronological Age Interacts with the Circadian Melatonin Receptor 1B Gene Variation, Determining Fasting Glucose Concentrations in Mediterranean Popu…

2020

Gene-age interactions have not been systematically investigated on metabolic phenotypes and this modulation will be key for a better understanding of the temporal regulation in nutrigenomics. Taking into account that aging is typically associated with both impairment of the circadian system and a decrease in melatonin secretion, we focused on the melatonin receptor 1B (MTNR1B)-rs10830963 C&gt

Blood GlucoseMale0301 basic medicinePhysiologyType 2 diabetestype-2 diabetesDiabetis no-insulinodependentMediterranean population0302 clinical medicineRisk FactorsPolymorphism (computer science)MedicineNon-insulin-dependent diabetesmelatonin receptorAged 80 and overeducation.field_of_studyNutrition and DieteticsMediterranean RegionAge FactorsDiabetis en l'embaràsFastingMiddle AgedGestational diabetesMTNR1B polymorphismCohortFemalepregnancywomengestational diabeteslcsh:Nutrition. Foods and food supplyage-interactionhormones hormone substitutes and hormone antagonistsAdultDiabetes riskAdolescentPopulationlcsh:TX341-641030209 endocrinology & metabolismPolymorphism Single NucleotideRisk AssessmentArticleYoung Adult03 medical and health sciencesDiabetes mellitusHumanseducationAgedRetrospective StudiesReceptor Melatonin MT2business.industrymedicine.diseaseCross-Sectional Studies030104 developmental biologyDiabetes Mellitus Type 2Melatonin receptor 1BSpainheterogeneitybusinessDiabetes in pregnancyfasting glucoseFood ScienceNutrients
researchProduct

Serum Dehydroepiandrosterone Sulfate and Risk for Type 2 Diabetes in Older Men and Women: The Pro.V.A Study

2016

Objective: A large body of clinical data suggests the importance of endogenous sex hormones in the pathogenesis of diabetes, but very little is known about the possible relationship between dehydroepiandrosterone sulfate (DHEAS) and diabetes, particularly in the elderly. We aimed, therefore, to examine whether high serum levels of DHEAS have any protective effects on the incidence of type 2 diabetes and to elucidate the possible role of gender in a cohort of older subjects. Methods: We followed 1258 community-dwelling subjects aged ≥65 years without type 2 diabetes who belonged to the Progetto Veneto Anziani (Pro.V.A.) for 4.4±1.2 years. DHEAS were measured at baseline and categorized into …

Blood GlucoseMale0301 basic medicinemedicine.medical_specialtyDHEASEndocrinology Diabetes and MetabolismBlood sugarPhysiology030209 endocrinology & metabolismType 2 diabetesNOsex hormones in the pathogenesis of diabeteCohort Studies03 medical and health scienceschemistry.chemical_compoundElderly0302 clinical medicineEndocrinologyDehydroepiandrosterone sulfateRisk FactorsInternal medicineDiabetes mellitusInternal MedicineHumansMedicineAgedGlycated HemoglobinDehydroepiandrosterone Sulfatebusiness.industryIncidence (epidemiology)Type 2 diabetesGeneral MedicinePrognosismedicine.diseaseCommunity-dwelling adultsDiabetes and Metabolism030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2chemistrydehydroepiandrosterone sulfate (DHEAS)CohortCommunity-dwelling adults; DHEAS; Elderly; Type 2 diabetes; Internal Medicine; Endocrinology; Endocrinology Diabetes and MetabolismFemaleGlycated hemoglobinbusinessBiomarkersFollow-Up StudiesCohort studyCanadian Journal of Diabetes
researchProduct

Transgenerational effects on intestinal inflammation status in mice perinatally exposed to bisphenol S

2020

International audience; Increasing evidence has highlighted the critical role of early life environment in shaping the future health outcomes of individuals in subsequent generations. Bisphenol S (BPS) has been widely used as a substitute for various plastic materials due to the limited application of Bisphenol A (BPA) which is an endocrine disruptor. However, the lack of efficient evaluation of BPS leaves doubts about the relevant substitute of BPA. Few studies of transgenerational inheritance have examined the effects of environmental exposures to endocrine disruptors on the immune system. In this study, we analyzed the transgenerational effects of BPS on intestinal inflammation and its c…

Blood GlucoseMaleBisphenol SHealth Toxicology and Mutagenesis[SDV]Life Sciences [q-bio]0208 environmental biotechnologyPhysiology02 engineering and technologyEndocrine Disruptors010501 environmental sciences01 natural sciencesFecesMicePregnancySulfonesPerinatal ExposureGeneral MedicinePollutionIntestine[SDV] Life Sciences [q-bio][SDV.TOX] Life Sciences [q-bio]/ToxicologyIntestinesEndocrine disruptorPrenatal Exposure Delayed Effects[SDV.TOX]Life Sciences [q-bio]/ToxicologyCytokinesGestationFemalemedicine.symptomhormones hormone substitutes and hormone antagonistsEnvironmental EngineeringOffspringInflammationImmune systemPhenolsmedicineAnimalsEnvironmental ChemistryWeaningEndocrine system0105 earth and related environmental sciencesInflammationbusiness.industryBody WeightPublic Health Environmental and Occupational HealthGeneral Chemistry020801 environmental engineeringPerinatal exposureMice Inbred C57BLMetabolismbusiness
researchProduct

Sequential Intensification of Metformin Treatment in Type 2 Diabetes With Liraglutide Followed by Randomized Addition of Basal Insulin Prompted by A1…

2012

OBJECTIVE We evaluated the addition of liraglutide to metformin in type 2 diabetes followed by intensification with basal insulin (detemir) if glycated hemoglobin (A1C) ≥7%. RESEARCH DESIGN AND METHODS In 988 participants from North America and Europe uncontrolled on metformin ± sulfonylurea, sulfonylurea was discontinued and liraglutide 1.8 mg/day added for 12 weeks (run-in). Subsequently, those with A1C ≥7% were randomized 1:1 to 26 weeks’ open-label addition of insulin detemir to metformin + liraglutide (n = 162) or continuation without insulin detemir (n = 161). Patients achieving A1C <7% continued unchanged treatment (observational arm). The primary end point was A1C change bet…

Blood GlucoseMaleEXENATIDEendocrine system diseasesdiabetes liraglutide metfortmin hypoglycemiaEndocrinology Diabetes and Metabolismmedicine.medical_treatmentType 2 diabetesTHERAPYGastroenterologyMELLITUSInsulin DetemirGlucagon-Like Peptide 1GLYCEMIC CONTROLOriginal ResearchInsulin detemirAged 80 and overClinical Care/Education/Nutrition/Psychosocial ResearchTREATED PATIENTSMiddle AgedMetforminMetforminNPH INSULINInsulin Long-ActingFemaleLife Sciences & Biomedicinehormones hormone substitutes and hormone antagonistsmedicine.drugAdultmedicine.medical_specialtyPARALLEL-GROUPAdolescentmedicine.drug_classHypoglycemiaEndocrinology & MetabolismDiabetes mellitusInternal medicineInternal MedicinemedicineHumansHypoglycemic AgentsCOMBINATIONAgedGlycated HemoglobinAdvanced and Specialized NursingScience & TechnologyLiraglutidebusiness.industryInsulin26-WEEKnutritional and metabolic diseasesLiraglutideEFFICACYmedicine.diseaseSulfonylureaEndocrinologyDiabetes Mellitus Type 2business
researchProduct

Adiponectin, resistin and leptin in paediatric chronic renal failure: Correlation with auxological and endocrine profiles

2013

Introduction: Chronic renal failure (CRF) compromises nutrition, growth, puberty, glycometabolic homeostasis, and adipokine secretion (i.e. adiponectin, resistin, and leptin). Adipokines play a role in the clinical outcome, but data in paediatric patients is scant. Aim: To evaluate the link between kidney function, adiponectin, resistin, leptin, hormonal status, nutritional state and late outcome of CRF children. Materials and methods: We studied leptin, adiponectin and resistin levels in 31 CRF patients (19 males, 12 females, aged 12.1 ± 4.47 years) managed conservatively, and 30 healthy age- and gender-matched controls. Clinical, auxological, biochemical, hormonal data, glucose and insuli…

Blood GlucoseMaleLeptinmedicine.medical_specialtyTime FactorsAdolescentAdolescent Nutritional Physiological Phenomenamedicine.medical_treatmentNutritional StatusRenal functionAdipokineHOMA-IRchemistry.chemical_compoundSettore MED/38 - Pediatria Generale E SpecialisticaRisk FactorsInternal medicineAdipokinemedicineChronic renal failureHumansInsulinResistinObesityChildGlycated HemoglobinCreatinineAdiponectinbusiness.industryInsulinLeptinAge Factorsnutritional and metabolic diseasesPrognosismedicine.diseaseObesityEndocrinologychemistryCardiovascular DiseasesNephrologyCase-Control StudiesDisease ProgressionKidney Failure ChronicFemaleResistinAdiponectinInsulin ResistancebusinessBiomarkershormones hormone substitutes and hormone antagonists
researchProduct

Vinblastine-induced autophagocytosis: effects on liver glycogen

1983

The possible similarities of the mechanism by which vinblastine induces autophagocytosis in liver were compared with the known effects of glucagon in glucagon-induced autophagocytosis. A single intraperitoneal injection of vinblastine produced a wave of autophagocytosis in less than 0.5 h in mouse hepatocytes. Liver glycogen content decreases simultaneously and blood glucose first increased and then decreased below control values. Both liver cAMP concentration and the activity of glycogen phosphorylase remained unchanged. These findings provide evidence that the induction of autophagocytosis after vinblastine injection is not mediated by cAMP. The increased degradation of glycogen may occur…

Blood GlucoseMaleendocrine systemmedicine.medical_specialtyPhosphorylasesAutophagocytosismedicine.medical_treatmentIntraperitoneal injectionBiophysicsBiologyVinblastineBiochemistryGlucagonMicechemistry.chemical_compoundPhagocytosisStructural BiologycAMPInternal medicineAutophagyCyclic AMPGeneticsmedicineAnimalsMolecular BiologyGlycogendigestive oral and skin physiologyVinoblastineCell BiologyVinblastineMicroscopy ElectronEndocrinologyLiverchemistryGlycogenhormones hormone substitutes and hormone antagonistsmedicine.drugFEBS Letters
researchProduct

Involvement of prostacyclin and potassium channels in the diabetes-induced hyporeactivity of the rabbit carotid artery to B-type natriuretic peptide

2012

The relation between diabetes and stroke is bidirectional: diabetes is an important risk factor for ischemic stroke, and acute stroke frequently induces hyperglycemia. On the other hand, plasma B-type natriuretic peptide (BNP) levels are raised in diabetes and stroke. The purpose was to study how alloxan-induced diabetes might modify the effects of BNP in rabbit carotid arteries and the mechanisms involved in such actions. To do this, isometric tension in isolated rabbit carotid artery was recorded and prostanoids release and plasma NT-proBNP were measured by enzyme immunoassay. BNP induced a relaxation of phenylephrine-precontracted carotid arteries, and this relaxation was lower in diabet…

Blood GlucoseMalemedicine.medical_specialtyPotassium ChannelsEndotheliummedicine.drug_classProstacyclinNitric OxideGlibenclamideThromboxane A2chemistry.chemical_compoundDiabetes mellitusInternal medicineNatriuretic Peptide BrainDiabetes MellitusmedicineNatriuretic peptideAnimalscardiovascular diseasesEndothelial dysfunctionStrokePharmacologyDose-Response Relationship Drugbusiness.industryBody Weightmedicine.diseaseEpoprostenolPeptide FragmentsCarotid Arteriesmedicine.anatomical_structureEndocrinologychemistryPotassiumcardiovascular systemRabbitsbusinessReceptors Atrial Natriuretic Factorhormones hormone substitutes and hormone antagonistsmedicine.drugEuropean Journal of Pharmacology
researchProduct

Impact of Fasting Glycemia on Short-Term Prognosis after Acute Myocardial Infarction

2007

The prognosis of patients with acute myocardial infarction (MI), according to the new criteria for impaired fasting glucose (IFG) (FG 100-126 mg/dl), has not been evaluated.A total of 2353 patients with acute MI and surviving at d 5 after admission were analyzed for short-term morbidity and mortality. FG was obtained at d 4 and 5. Patients were classified as diabetes mellitus (known diabetes or FGor = 126 mg/dl), high IFG (110or = FG126 mg/dl), low IFG (100or = FG110 mg/dl), and normal fasting glucose (NFG) (FG100 mg/dl).Among the 2353 patients, 968 (41%) had diabetes mellitus, 262 (11%) had high IFG, 332 (14%) had low IFG, and 791 (34%) had NFG. Compared with NFG patients, 30-d cardiovascu…

Blood GlucoseMalemedicine.medical_specialtyendocrine system diseasesHeart diseaseEndocrinology Diabetes and MetabolismClinical BiochemistryMyocardial InfarctionSensitivity and Specificitybehavioral disciplines and activitiesBiochemistryCohort StudiesFasting glucoseEndocrinologyRisk FactorsInternal medicineDiabetes mellitusDiabetes MellitusPrevalencemedicineHumansMyocardial infarctionAcute miAgedCardiovascular mortalitybusiness.industryBiochemistry (medical)nutritional and metabolic diseasesFastingMiddle AgedPrognosismedicine.diseaseImpaired fasting glucoseEndocrinologyROC CurveHyperglycemiaHeart failureFemaleMorbiditybusinesshormones hormone substitutes and hormone antagonistspsychological phenomena and processesThe Journal of Clinical Endocrinology & Metabolism
researchProduct

Phenotyping of type 2 diabetes mellitus at onset on the basis of fasting incretin tone: Results of a two-step cluster analysis.

2015

Aims/Introduction According to some authors, in type 2 diabetes there is a reduced postprandial action of glucagon-like peptide-1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP). However, little is known about the role of fasting incretins in glucose homeostasis. Our aim was to evaluate, through a two-step cluster analysis, the possibility of phenotyping patients with type 2 diabetes at onset on the basis of fasting GLP-1, GIP and ghrelin. Materials and Methods A total of 96 patients with type 2 diabetes within 6 months of onset (mean age 62.40 ± 6.36 years) were cross-sectionally studied. Clinical, anthropometric and metabolic parameters were evaluated. At fasting the follow…

Blood GlucoseMalemedicine.medical_treatmentEndocrinology Diabetes and MetabolismSettore MED/13 - Endocrinologiachemistry.chemical_compound0302 clinical medicineGlucagon-Like Peptide 1Glucose homeostasisCluster AnalysisHomeostasis030212 general & internal medicineGeneral MedicineArticlesMiddle AgedGlucagon-like peptide-1GhrelinGlucagon‐like peptide‐1PhenotypeClinical Science and CareFemaleOriginal Articlehormones hormone substitutes and hormone antagonistsGlucagon-like peptide-1medicine.medical_specialtyGlucose-dependent insulinotropic polypeptideIncretin030209 endocrinology & metabolismGastric Inhibitory PolypeptideGhrelin; Glucagon-like peptide-1; Glucose-dependent insulinotropic polypeptide; Endocrinology Diabetes and Metabolism; Internal MedicineIncretins03 medical and health sciencesInsulin resistanceGlucose‐dependent insulinotropic polypeptideInternal medicineDiabetes mellitusmedicineInternal MedicineHumansAgedAdiponectinbusiness.industryInsulinmedicine.diseaseEndocrinologyGlucosechemistryDiabetes Mellitus Type 2Glycated hemoglobinInsulin ResistancebusinessJournal of diabetes investigation
researchProduct

Prevalence of type 2 diabetes and impaired fasting glucose in patients affected by rheumatoid arthritis: Results from a cross-sectional study.

2017

Abstract Although the better management of rheumatoid arthritis (RA) has significantly improved the long-term outcome of affected patients, a significant proportion of these may develop associated comorbidities including cardiometabolic complications. However, it must be pointed out that a comprehensive cardiometabolic evaluation is still poorly integrated into the management of RA patients, due to a limited awareness of the problem, a lack of appropriate clinical studies, and optimal strategies for cardiovascular (CV) risk reduction in RA. In addition, although several studies investigated the possible association between traditional CV risk factors and RA, conflicting results are still av…

Blood GlucoseMalerheumatoid arthritisTime FactorsCross-sectional studyType 2 diabetesAdrenal Cortex HormoneBody Mass IndexArthritis Rheumatoid0302 clinical medicineimpaired fasting glucoseAdrenal Cortex HormonesRisk FactorsRheumatoidCardiovascular DiseasePrevalence030212 general & internal medicineMedicine (all)Diabetes MellituGeneral MedicineMiddle AgedC-Reactive ProteinCholesterolcardiovascular risk; impaired fasting glucose; inflammation; rheumatoid arthritis; type 2 diabetes;Cardiovascular DiseasesCohortHypertensionFemaletype 2 diabetesCase-Control Studiecardiovascular risk; impaired fasting glucose; inflammation; rheumatoid arthritis; type 2 diabetes; Adrenal Cortex Hormones; Adult; Aged; Arthritis Rheumatoid; Blood Glucose; Body Mass Index; C-Reactive Protein; Cardiovascular Diseases; Case-Control Studies; Cholesterol; Cross-Sectional Studies; Diabetes Mellitus Type 2; Female; Glucose Intolerance; Humans; Hypertension; Male; Middle Aged; Prevalence; Risk Factors; Time Factors; Medicine (all)Type 2Research ArticleArthritiHumanAdultcardiovascular riskmedicine.medical_specialtyTime FactorObservational StudyNO03 medical and health sciencesInternal medicineDiabetes mellitusGlucose Intolerancemedicinecardiovascular risk impaired fasting glucose inflammation rheumatoid arthritis type 2 diabetesHumansAged030203 arthritis & rheumatologyCross-Sectional Studietype 2 diabetebusiness.industryRisk Factor6900Case-control studyrheumatoid arthritimedicine.diseaseImpaired fasting glucoseSettore MED/16 - ReumatologiaCross-Sectional StudiesDiabetes Mellitus Type 2inflammationCase-Control StudiesMetabolic syndromebusinessBody mass index
researchProduct