Search results for "ilium"

showing 10 items of 131 documents

Emerging roles of extracellular vesicles in the nervous system.

2014

Information exchange executed by extracellular vesicles, including exosomes, is a newly described form of intercellular communication important in the development and physiology of neural systems. These vesicles can be released from cells, are packed with information including signaling proteins and both coding and regulatory RNAs, and can be taken up by target cells, thereby facilitating the transfer of multilevel information. Recent studies demonstrate their critical role in physiological processes, including nerve regeneration, synaptic function, and behavior. These vesicles also have a sinister role in the propagation of toxic amyloid proteins in neurodegenerative conditions, including …

Nervous systemSymposiumAmyloidGeneral NeuroscienceVesicleCiliumRegeneration (biology)2800 General NeuroscienceBrain610 Medicine & healthNeurodegenerative Diseases11359 Institute for Regenerative Medicine (IREM)BiologyExosomesMicrovesiclesNerve Regenerationmedicine.anatomical_structureTumor progressionmedicineAnimalsHumansNeoplasm InvasivenessCiliaNeuroscienceNeuroinflammationThe Journal of neuroscience : the official journal of the Society for Neuroscience
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Loss of Dishevelleds disrupts planar polarity in ependymal motile cilia and results in hydrocephalus.

2014

Defects in ependymal (E) cells, which line the ventricle and generate cerebrospinal fluid flow through ciliary beating, can cause hydrocephalus. Dishevelled genes (Dvls) are essential for Wnt signaling, and Dvl2 has been shown to localize to the rootlet of motile cilia. Using the hGFAP-Cre;Dvl1(-/-);2(flox/flox);3(+/-) mouse, we show that compound genetic ablation of Dvls causes hydrocephalus. In hGFAP-Cre;Dvl1(-/-);2(flox/flox);3(+/-) mutants, E cells differentiated normally, but the intracellular and intercellular rotational alignments of ependymal motile cilia were disrupted. As a consequence, the fluid flow generated by the hGFAP-Cre;Dvl1(-/-);2(flox/flox);3(+/-) E cells was significant…

Neuroscience(all)Dishevelled ProteinsMice TransgenicBiologyTransgenicArticleMiceEpendymaCell polarityFLOXGeneticsmedicinePsychologyAnimalsCiliaAdaptor Proteins Signal Transducingchemistry.chemical_classificationNeurology & NeurosurgeryGeneral NeuroscienceCiliumSignal TransducingNeurosciencesWnt signaling pathwayAdaptor ProteinsCell PolarityPhosphoproteinsDishevelledCell biologymedicine.anatomical_structurechemistryMotile ciliumCognitive SciencesEpendymaIntracellularHydrocephalusSignal TransductionNeuron
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Coupling of Azomethine Ylides with Nitrilium Derivatives ofcloso-Decaborate Clusters: A Synthetic and Theoretical Study

2012

The azomethine ylides p-R3C5H4N+CH−COC6H4R2-p (3 a: R3=H, R2=H, X=Br; 3 b: R3=H, R2=Me, X=I; 3 c: R3=H, R2=OMe, X=I; 3 d: R3=H, R2=F, X=I; 3 e: R3=Me, R2=Me, X=Br) react with the nitrile functionality of the closo-decaborate clusters [Bun4N][B10H9(NCR1)] (1 a: R1=Me; 1 b: R1=Et; 1 c: R1=Ph) in a CH3NO2 solution under mild conditions (20–25 °C, 2 min) to afford selectively products of the nucleophilic addition (ca. quantitative yields based on NMR analysis in [D6]DMSO, 71–87 % yield of isolated products). These products are the borylated enamino ketones as the salts bearing exclusively a tetrabutylammonium cation [Bun4N][B10H9{NCR1=C(N+C5H4R3-p)COC6H4R2-p}] (4 a–h,k–n) or the mixed salts [Bu…

Nucleophilic additionNitrileAzomethine ylideBoranesGeneral ChemistryPhotochemistryMedicinal chemistryCycloadditionchemistry.chemical_compoundchemistryNucleophilePyridiniumNitriliumta116ChemPlusChem
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Intraflagellar transport proteins in ciliogenesis of photoreceptor cells

2011

Background information. The assembly and maintenance of cilia depend on IFT (intraflagellar transport) mediated by molecular motors and their interplay with IFT proteins. Here, we have analysed the involvement of IFT proteins in the ciliogenesis of mammalian photoreceptor cilia. Results. Electron microscopy revealed that ciliogenesis in mouse photoreceptor cells follows an intracellular ciliogenesis pathway, divided into six distinct stages. The first stages are characterized by electron-dense centriolar satellites and a ciliary vesicle, whereas the formations of the ciliary shaft and the light-sensitive outer segment discs are features of the later stages. IFT proteins were associated with…

OrganogenesisFluorescent Antibody TechniqueBiologyFlagellumCiliary shaftPhotoreceptor cellRetina03 medical and health sciencesMice0302 clinical medicineMicroscopy Electron TransmissionIntraflagellar transportCiliogenesisMolecular motormedicineAnimalsHumansPhotoreceptor CellsCilia030304 developmental biology0303 health sciencesCiliumCell BiologyGeneral MedicineCell biologymedicine.anatomical_structureCytoplasmsense organsCarrier Proteins030217 neurology & neurosurgeryBiology of the Cell
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Human brain organoids assemble functionally integrated bilateral optic vesicles

2021

During embryogenesis, optic vesicles develop from the diencephalon via a multistep process of organogenesis. Using induced pluripotent stem cell (iPSC)-derived human brain organoids, we attempted to simplify the complexities and demonstrate formation of forebrain-associated bilateral optic vesicles, cellular diversity, and functionality. Around day 30, brain organoids attempt to assemble optic vesicles, which develop progressively as visible structures within 60 days. These optic vesicle-containing brain organoids (OVB-organoids) constitute a developing optic vesicle's cellular components, including primitive corneal epithelial and lens-like cells, retinal pigment epithelia, retinal progeni…

OrganogenesisInduced Pluripotent Stem Cellsretinal pigment epitheliumiPSCsEmbryonic DevelopmentBiology03 medical and health sciencesDiencephalonchemistry.chemical_compoundProsencephalon0302 clinical medicineGeneticsOrganoidmedicineHumansInduced pluripotent stem cell030304 developmental biology0303 health sciencesforebrain organoidsRetinal pigment epitheliumbrain organoidsVesicleprimordial eye fieldsOVB-organoidsCell DifferentiationRetinalCell BiologyOptic vesicleHuman brainCell biologyOrganoidsmedicine.anatomical_structurenervous systemchemistryMolecular MedicineFOXG1; OVB-organoids; brain organoids; forebrain organoids; iPSCs; optic vesicles; primary cilium; primordial eye fields; retinal pigment epitheliumoptic vesiclesFOXG1030217 neurology & neurosurgeryprimary ciliumCell Stem Cell
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Immunofluorescence Analysis as a Diagnostic Tool in a Spanish Cohort of Patients with Suspected Primary Ciliary Dyskinesia

2020

Primary ciliary dyskinesia (PCD) is an autosomal recessive rare disease caused by an alteration of ciliary structure. Immunofluorescence, consisting in the detection of the presence and distribution of cilia proteins in human respiratory cells by fluorescence, has been recently proposed as a technique to improve understanding of disease-causing genes and diagnosis rate in PCD. The objective of this study is to determine the accuracy of a panel of four fluorescently labeled antibodies (DNAH5, DNALI1, GAS8 and RSPH4A or RSPH9) as a PCD diagnostic tool in the absence of transmission electron microscopy analysis. The panel was tested in nasal brushing samples of 74 patients with clinical suspic…

PCD antibody cilia immunofluorescence primary ciliary dyskinesiaPathologymedicine.medical_specialtyPrimary Ciliary DyskinesiaImmunofluorescencelcsh:MedicineImmunoglobulinsImmunofluorescenceArticleImmunofluorescència03 medical and health sciences0302 clinical medicinePrimary ciliary dyskinesiaCiliary axonemeantibodymedicineotorhinolaryngologic diseasesCiliaRespiratory systemAntibody030304 developmental biologyPrimary ciliary dyskinesia0303 health sciencesmedicine.diagnostic_testbiologybusiness.industryCiliumlcsh:RciliaGeneral Medicinemedicine.diseasePCD030228 respiratory systemDiscinesia ciliar primàriaCohortbiology.proteinAntibodybusinessImmunoglobulinesRare disease
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An siRNA-based functional genomics screen for the identification of regulators of ciliogenesis and ciliopathy genes

2015

Item does not contain fulltext Defects in primary cilium biogenesis underlie the ciliopathies, a growing group of genetic disorders. We describe a whole-genome siRNA-based reverse genetics screen for defects in biogenesis and/or maintenance of the primary cilium, obtaining a global resource. We identify 112 candidate ciliogenesis and ciliopathy genes, including 44 components of the ubiquitin-proteasome system, 12 G-protein-coupled receptors, and 3 pre-mRNA processing factors (PRPF6, PRPF8 and PRPF31) mutated in autosomal dominant retinitis pigmentosa. The PRPFs localize to the connecting cilium, and PRPF8- and PRPF31-mutated cells have ciliary defects. Combining the screen with exome sequen…

PRPF31Pregnancy ProteinsInbred C57BLCiliopathiesMiceImmunologicCerebellumDatabases GeneticEye AbnormalitiesNon-U.S. Gov'tZebrafishExome sequencingMice KnockoutGeneticsResearch Support Non-U.S. Gov'tCiliumHigh-Throughput Nucleotide SequencingMetabolic Disorders Radboud Institute for Molecular Life Sciences [Radboudumc 6]GenomicsKidney Diseases CysticPhenotypeKidney DiseasesRNA InterferenceAbnormalitiesMultipleFunctional genomicsCiliary Motility DisordersGenetic MarkersEllis-Van Creveld SyndromeKnockoutJeune syndromeOther Research Radboud Institute for Molecular Life Sciences [Radboudumc 0]BiologyResearch SupportTransfectionRetinaArticlewhole-genome siRNA screenJoubert syndromeN.I.H.DatabasesCysticreverse geneticsResearch Support N.I.H. ExtramuralGeneticCerebellar DiseasesJoubert syndromeCiliogenesisSuppressor FactorsJournal ArticleSuppressor Factors ImmunologicmedicineAnimalsHumansAbnormalities MultipleGenetic Predisposition to DiseasePhotoreceptor CellsCiliaGenetic TestingCaenorhabditis elegansExtramuralMembrane ProteinsProteinsReproducibility of ResultsCell Biologymedicine.diseaseMice Inbred C57BLCytoskeletal ProteinsCiliopathyRenal disorders Radboud Institute for Molecular Life Sciences [Radboudumc 11]HEK293 CellsMutationciliopathiesGenome-Wide Association StudyNature Cell Biology
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C21orf2 is mutated in recessive early-onset retinal dystrophy with macular staphyloma and encodes a protein that localises to the photoreceptor prima…

2015

Background/aim We have noted a phenotype of early-onset retinal dystrophy with macular staphyloma but without high myopia. The aim of this study is to report the underlying genetic mutations and the subcellular localisation of the gene product in the retina. Methods Retrospective case series (2012–2015); immunohistochemical analyses of mammalian retina for in situ protein localisation. Results All three probands were first noted to have decreased vision at 3–6 years old which worsened over time. At ages 39, 37 and 12 years old, all had similar retinal findings: dystrophic changes (retinal pigment epithelium mottling, vessel narrowing), macular staphyloma (despite only mild myopia or high hy…

Pathologygenetic structuresSus scrofaPolymerase Chain ReactionPhotoreceptor cellchemistry.chemical_compoundConsanguinityMiceChildFrameshift MutationGeneticsmedicine.diagnostic_testMagnetic Resonance ImagingSensory SystemsTissue DonorsPedigreemedicine.anatomical_structureFemaleRetinal DystrophiesTomography Optical CoherenceDilatation PathologicAdultmedicine.medical_specialtyBlotting WesternMolecular Sequence DataMutation MissenseGenes RecessiveBiologyRetinaCellular and Molecular NeuroscienceRetinal DystrophiesmedicineElectroretinographyAnimalsHumansAmino Acid SequencePhotoreceptor Connecting CiliumRetrospective StudiesRetinaRetinal pigment epitheliumDystrophyProteinsRetinalmedicine.diseaseeye diseasesOphthalmologyCiliopathyCytoskeletal Proteinschemistrysense organsElectroretinographyThe British journal of ophthalmology
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Primary ciliary dyskinesia. Ciliopathies

2008

Abstract Primary ciliary dyskinesia is a genetically inherited syndrome characterised by ciliary immotility or dysmotility. Deficiency in mucociliary clearance produces chronic respiratory infections from birth, male sterility by spermatozoid immotility and situs inversus in 40%–50% of patients (Kartagener's syndrome). Diagnosis is made by analysing ciliary motility with high-speed digital video and ciliary ultrastructure. The wide distribution and functions of the cilia in the body mean that this dysfunction can generate other ciliopathies apart from primary ciliary dyskinesia.

Pathologymedicine.medical_specialtyBronchiectasisSterilityMucociliary clearancebusiness.industryCiliumGeneral MedicineAnatomymedicine.diseaseCiliopathiesSitus inversusRetinitis pigmentosaotorhinolaryngologic diseasesmedicineHumansbusinessCiliary Motility DisordersPrimary ciliary dyskinesiaActa Otorrinolaringologica (English Edition)
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Identification of Novel Molecular Components of the Photoreceptor Connecting Cilium by Immunoscreens

2002

Abstract The connecting cilium of photoreceptor cells is the only intracellular link between the morphologically, functionally and biochemically different compartments of the inner and outer segments. The non-motile modified cilium plays an important role in the organization and the function of photoreceptor cells, namely in delivery and turnover of enzymes and substrates of the visual transduction cascade, and the photosensitive membranes of the outer segment. The protein components of the cilium participate in the intracellular transport through the cilium, in the outer segment disk morphogenesis and in the maintenance of discrete membrane domains. In order to identify yet unknown cytoske…

Photoreceptor Connecting CiliumAdenomatous Polyposis Coli ProteinXenopus ProteinsBiologyPhotoreceptor cellRats Sprague-DawleyMiceCellular and Molecular NeurosciencemedicineAnimalsDrosophila ProteinsCiliaCloning MolecularCytoskeletonMicrotubule-Associated Protein 4CytoskeletonGene LibraryRetinaCiliumCalcium-Binding ProteinsDynactin ComplexSensory SystemsRatsCell biologyMice Inbred C57BLOphthalmologymedicine.anatomical_structureCentrinsense organsMicrotubule-Associated ProteinsPhotoreceptor Cells VertebrateVisual phototransductionExperimental Eye Research
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