Search results for "inbred c57bl"

showing 10 items of 1287 documents

A New Method for Extracting Skin Microbes Allows Metagenomic Analysis of Whole-Deep Skin

2013

In the last decade, an extensive effort has been made to characterize the human microbiota, due to its clinical and economic interests. However, a metagenomic approach to the skin microbiota is hampered by the high proportion of host DNA that is recovered. In contrast with the burgeoning field of gut metagenomics, skin metagenomics has been hindered by the absence of an efficient method to avoid sequencing the host DNA. We present here a method for recovering microbial DNA from skin samples, based on a combination of molecular techniques. We have applied this method to mouse skin, and have validated it by standard, quantitative PCR and amplicon sequencing of 16S rRNA. The taxonomic diversit…

Microbial DNAScienceComputational biologyBiologyPolymerase Chain ReactionGTP Phosphohydrolaseslaw.inventionMicelawRNA Ribosomal 16SPell -- MicrobiologiaAnimalsHumansMicrobiomePhylogenyPolymerase chain reactionSkinGeneticsGenètica bacterianaMultidisciplinaryBacteriaintegumentary systemMicrobiotaQRBacterial taxonomyHuman microbiomeDNADNA extractionMice Inbred C57BLMetagenomicsEarth Microbiome ProjectMedicineMetagenomicsResearch ArticleGensPLoS ONE
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Influence of aging on murine neutrophil and macrophage function against Candida albicans.

2008

Previous work by our group showed that aged C57BL/6 mice develop an altered innate and adaptive immune response to Candida albicans and are more susceptible to systemic primary candidiasis. In this work, we used young (2-3 months old) and aged (18-20 months old) C57BL/6 mice to study in vitro the influence of aging on (1) the fungicidal activity of neutrophils and macrophages, (2) the production of cytokines by resident peritoneal macrophages in response to C. albicans, and (3) cell surface Toll-like receptor (TLR) 2 expression on resident peritoneal macrophages. Our results indicate that murine phagocytes have a fungicidal activity well preserved with aging. In vitro production of proinfla…

Microbiology (medical)ChemokineAgingNeutrophilsmedicine.medical_treatmentImmunologyBiologyMicrobiologyProinflammatory cytokineMicrobiologyMicePhagocytosisCandida albicansmedicineImmunology and AllergyMacrophageAnimalsCandida albicansCells CulturedInnate immune systemMicrobial ViabilityToll-Like ReceptorsGeneral Medicinebiology.organism_classificationCorpus albicansMice Inbred C57BLInfectious DiseasesCytokineImmunologybiology.proteinMacrophages PeritonealCytokinesTumor necrosis factor alphaFemaleFEMS immunology and medical microbiology
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ALDH-2 deficiency increases cardiovascular oxidative stress--evidence for indirect antioxidative properties.

2007

Abstract Mitochondrial aldehyde dehydrogenase (ALDH-2) reduces reactive oxygen species (ROS) formation related to toxic aldehydes; additionally, it provides a bioactivating pathway for nitroglycerin. Since acetaldehyde, nitroglycerin, and doxorubicin treatment provoke mitochondrial oxidative stress, we used ALDH-2−/− mice and purified recombinant human ALDH-2 to test the hypothesis that ALDH-2 has an indirect antioxidant function in mitochondria. Antioxidant capacity of purified ALDH-2 was comparable to equimolar doses of glutathione, cysteine, and dithiothreitol; mitochondrial oxidative stress was comparable in C57Bl6 and ALDH-2−/− mice after acute challenges with nitroglycerin or doxorubi…

Mitochondrial ROSAntioxidantmedicine.medical_treatmentBiophysicsAldehyde dehydrogenaseAcetaldehydeMitochondrionPharmacologymedicine.disease_causeBiochemistryCardiovascular SystemModels BiologicalAntioxidantschemistry.chemical_compoundMiceNitroglycerinmedicineAnimalsHumansCysteineMolecular Biologychemistry.chemical_classificationReactive oxygen speciesbiologyDose-Response Relationship DrugAldehyde Dehydrogenase MitochondrialAcetaldehydeCell BiologyGlutathioneAldehyde DehydrogenaseGlutathioneMitochondriaMice Inbred C57BLDithiothreitolOxidative StresschemistryBiochemistryDoxorubicincardiovascular systembiology.proteinReactive Oxygen SpeciesOxidative stressBiochemical and biophysical research communications
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Heterozygous deficiency of manganese superoxide dismutase in mice (Mn-SOD+/-): a novel approach to assess the role of oxidative stress for the develo…

2005

Nitroglycerin (GTN)-induced tolerance was reported to be associated with increased levels of reactive oxygen species (ROS) in mitochondria. In the present study, we further investigated the role of ROS for the development of nitrate tolerance by using heterozygous manganese superoxide dismutase knock-out mice (Mn-SOD+/-). Mn-SOD is acknowledged as a major sink for mitochondrial superoxide. Vasodilator potency of mouse aorta in response to acetylcholine and GTN was assessed by isometric tension studies. Mitochondrial ROS formation was detected by 8-amino-5-chloro-7-phenylpyrido[3,4-d]pyridazine-1,4-(2H,3H)dione sodium salt (L-012)-enhanced chemiluminescence and mitochondrial aldehyde dehydro…

Mitochondrial ROSHeterozygoteAldehyde dehydrogenaseMitochondrionPharmacologymedicine.disease_causeMitochondria HeartSuperoxide dismutaseMiceNitroglycerinDrug tolerancemedicineAnimalsEndothelial dysfunctionAortaPharmacologychemistry.chemical_classificationReactive oxygen speciesbiologySuperoxide DismutaseDrug ToleranceAldehyde Dehydrogenasemedicine.diseaseEnzyme ActivationMice Inbred C57BLOxidative StresschemistryBiochemistrycardiovascular systembiology.proteinMolecular MedicineOxidative stresscirculatory and respiratory physiologyMolecular pharmacology
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Manganese superoxide dismutase and aldehyde dehydrogenase deficiency increase mitochondrial oxidative stress and aggravate age-dependent vascular dys…

2008

AimsImbalance between pro- and antioxidant species (e.g. during aging) plays a crucial role for vascular function and is associated with oxidative gene regulation and modification. Vascular aging is associated with progressive deterioration of vascular homeostasis leading to reduced relaxation, hypertrophy, and a higher risk of thrombotic events. These effects can be explained by a reduction in free bioavailable nitric oxide that is inactivated by an age-dependent increase in superoxide formation. In the present study, mitochondria as a source of reactive oxygen species (ROS) and the contribution of manganese superoxide dismutase (MnSOD, SOD-2) and aldehyde dehydrogenase (ALDH-2) were inves…

Mitochondrial ROSMaleAgingPhysiologyVasodilator AgentsMitochondrionVascular dysfunctionmedicine.disease_causeMitochondria HeartMuscle Smooth Vascularchemistry.chemical_compoundMiceEndothelial dysfunctionAortachemistry.chemical_classificationMice KnockoutbiologySuperoxideAldehyde Dehydrogenase MitochondrialAge FactorsVasodilationBiochemistryCardiology and Cardiovascular MedicineMitochondrial aldehyde dehydrogenasemedicine.medical_specialty8-oxodGOxidative phosphorylationDNA MitochondrialSuperoxide dismutaseManganese superoxide dismutaseddc:570Physiology (medical)Internal medicinemedicineAnimalsReactive oxygen speciesDose-Response Relationship DrugSuperoxide DismutaseMitochondrial oxidative stressOriginal ArticlesAldehyde Dehydrogenasemedicine.diseaseMice Inbred C57BLOxidative StressEndocrinologychemistrybiology.proteinEndothelium VascularReactive Oxygen SpeciesOxidative stressDNA DamageCardiovascular research
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Lack of GDAP1 induces neuronal calcium and mitochondrial defects in a knockout mouse model of Charcot-Marie-tooth neuropathy

2015

27 páginas, 9 figuras.

Mitochondrial proteinCancer Researchlcsh:QH426-470Nerve Tissue ProteinsBiologyMitochondrionCharcot-Marie-Tooth diseaseGDAP1 geneMiceGeneticsAutophagyAnimalsCalcium SignalingMolecular BiologyGenetics (clinical)Ecology Evolution Behavior and SystematicsCytoskeletonCalcium signalingGeneticsVoltage-dependent calcium channelEndoplasmic reticulumAutophagyBiología y Biomedicina / BiologíaAxonsCell biologyMitochondriaMitochondrialMice Inbred C57BLAlpha tubulinlcsh:Geneticsmitochondrial fusionKnockout mouseMitochondrial fissionCalcium ChannelsAnimal cellGene DeletionResearch Article
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Computational analysis of lung deformation after murine pneumonectomy. [corrected].

2012

In many mammalian species, the removal of one lung (pneumonectomy) is associated with the compensatory growth of the remaining lung. To investigate the hypothesis that parenchymal deformation may trigger lung regeneration, we used microCT scanning to create 3-dimensional finite element geometric models of the murine lung pre- and post-pneumonectomy (24 hours). The structural correspondence between models was established using anatomic landmarks and an iterative computational algorithm. When compared with the pre-pneumonectomy lung, the post-pneumonectomy models demonstrated significant translation and rotation of the cardiac lobe into the post-pneumonectomy pleural space. 2-dimensional maps…

Models AnatomicPathologymedicine.medical_specialtyX-ray microtomographymedicine.medical_treatmentFinite Element AnalysisBiomedical EngineeringCompensatory growth (organ)BioengineeringBiologyDeformation (meteorology)ArticlePneumonectomyMiceParenchymamedicineAnimalsRegenerationComputational analysisPneumonectomyLungLungGeneral MedicineAnatomyX-Ray Microtomographyrespiratory systemLoberespiratory tract diseasesComputer Science ApplicationsHuman-Computer InteractionMice Inbred C57BLmedicine.anatomical_structureComputer methods in biomechanics and biomedical engineering
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Indicaxanthin, a multi-target natural compound from Opuntia ficus-indica fruit: From its poly-pharmacological effects to biochemical mechanisms and m…

2019

Abstract Over the latest years phytochemical consumption has been associated to a decreased risk of both the onset and the development of a number of pathological conditions. In this context indicaxanthin, a betalain pigment from Opuntia ficus-indica fruit, has been the object of sound research. Explored, at first, for its mere antioxidant potential, Indicaxanthin is now regarded as a redox-active compound able to exert significant poly-pharmacological effects against several targets in a number of experimental conditions both in vivo and in vitro. This paper aims to provide an overview on the therapeutical effects of indicaxanthin, ranging from the anti-inflammatory to the neuro-modulatory…

Models MolecularPyridinesOpuntia ficusPhytochemicalsContext (language use)Antioxidant potential01 natural sciencesMiceStructure-Activity Relationship03 medical and health scienceschemistry.chemical_compoundMulti targetCell Line TumorNeoplasmsSettore BIO/10 - BiochimicaBetalainDrug DiscoveryAnimalsHumansCell Proliferation030304 developmental biologyInflammationIndicaxanthin Multi-target compound Poly-pharmacology Antioxidant Antiinflammatory Antitumoral Antiproliferative Neuromodulator Molecular modellingPharmacologyBiological Products0303 health sciencesDose-Response Relationship DrugMolecular StructureTraditional medicine010405 organic chemistryNatural compoundOrganic ChemistryOpuntiaGeneral MedicineAntineoplastic Agents PhytogenicSettore CHIM/08 - Chimica FarmaceuticaBetaxanthins0104 chemical sciencesMice Inbred C57BLNeuroprotective AgentsPhytochemicalchemistryBlood-Brain BarrierFruitDrug Screening Assays AntitumorIndicaxanthinEuropean Journal of Medicinal Chemistry
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Strain-specific development of the mossy fiber system in organotypic cultures of the mouse hippocampus

1988

Abstract The postnatal development of the hippocampus of the inbred mice strains BALB/c, C57BL/6, and DBA/2 was studied in organotypic explant cultures using the roller-tube technique. In vivo, mice exhibit strain-specific mossy fiber distribution patterns. As a main result we found, that after cultivation of 3–4 weeks, similar strain-specific patterns became apparent in vitro, as visualized by a modified Timm staining. From this experiment we can conclude that a postnatal extrinsic influence cannot be the cause of the strain-specific hippocampal features.

Mossy fiber (hippocampus)Mice Inbred BALB CNeuronal PlasticityStrain (chemistry)General NeuroscienceHippocampusMice Inbred StrainsHippocampal formationBiologyHippocampusIn vitroCell biologyMice Inbred C57BLMiceSpecies SpecificityInbred strainMice Inbred DBAIn vivoAnimalsNeuroscienceCells CulturedExplant cultureNeuroscience Letters
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Immunohistochemical analysis of KCNQ2 potassium channels in adult and developing mouse brain

2005

The syndrome of benign familial neonatal convulsions (BFNC) is characterized by seizures starting within the first days of life and disappearing within weeks to months. BFNC is caused by loss-of-function mutations in the potassium channels KCNQ2 and KCNQ3 which can well explain the resulting neuronal hyperexcitability. However, it is not understood why seizures predominantly occur in the neonatal period. A potential explanation might be a change in the expression pattern of these channels during development. We therefore performed an immunohistochemical analysis of mouse brain slices at different stages of postnatal development using an antibody recognizing the C-terminus of the KCNQ2 chann…

Mossy fiber (hippocampus)medicine.medical_specialtyThalamusCentral nervous systemHippocampusBiologyHippocampal formationHippocampusMidbrainMiceEpilepsyInternal medicineNeural PathwaysmedicineAnimalsKCNQ2 Potassium ChannelTissue DistributionMolecular BiologyGeneral NeuroscienceBrainGene Expression Regulation Developmentalmedicine.diseaseImmunohistochemistryAxonsMice Inbred C57BLGlobus pallidusEndocrinologymedicine.anatomical_structurenervous systemMossy Fibers HippocampalNeurology (clinical)Developmental BiologyBrain Research
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