Search results for "inflammation"

showing 10 items of 2662 documents

Intestinal CD36 : A long chain fatty acid receptor which controls post prandial hypertriglyceridemia, endotoxemia and intestinal epithelium integrity

2014

Post prandial hypertriglyceridemia represents a risk factor for cardio-vascular diseases and it is associated with metabolic syndrom, obesity, and insulino-resistance. The intestine influences lipid bioavailibility and post prandial hypertriglyceridemia. It controls the quantity and the quality of secreted chylomicrons by adapting its metabolism according to the lipid content of the diet. Nevertheless, the mechanism of dietary lipid detection by the enterocyte is not understood. Our work demonstrates that the transmembrane glycoprotein CD36 is a Long Chain Fatty Acid (LCFA) receptor which triggers ERK1/2 activation. This activation is responsible for the induction of mRNA rate of 3 key prot…

InflammationDietary lipidsIntestin grêle[SDV.AEN] Life Sciences [q-bio]/Food and NutritionERK1/2ChylomicronsEndotoxémieLipides alimentairesCD36EndotoxemiaIntestine
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Exploring a regulatory role for mast cells: 'MCregs'?

2010

Regulatory cells can mould the fate of the immune response by direct suppression of specific subsets of effector cells, or by redirecting effectors against invading pathogens and infected or neoplastic cells. These functions have been classically, although not exclusively, ascribed to different subsets of T cells. Recently, mast cells have been shown to regulate physiological and pathological immune responses, and thus to act at the interface between innate and adaptive immunity assuming different functions and behaviors at discrete stages of the immune response. Here, we focus on these poorly defined, and sometimes apparently conflicting, functions of mast cells.

InflammationEffectorMast cell; Regulatory cells; cell-cell crosstalkImmunologyRegulatory cellModels ImmunologicalAutoimmunityAdaptive ImmunityBiologybiochemical phenomena metabolism and nutritionAcquired immune systemT-Lymphocytes RegulatoryImmunity InnateClassical complement pathwaycell-cell crosstalkImmune systemRegulatory cellsNeoplasmsImmunologyImmune ToleranceMAST CELLAnimalsHumansImmunology and AllergyMast Cells
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Post-COVID syndrome, inflammation, and diabetes

2022

The raging COVID-19 pandemic is in its third year of global impact. The SARS CoV 2 virus has a high rate of spread, protean manifestations, and a high morbidity and mortality in individuals with predisposing risk factors. The pathophysiologic mechanisms involve a heightened systemic inflammatory state, cardiometabolic derangements, and varying degrees of glucose intolerance. The latter can be evident as significant hyperglycemia leading to new -onset diabetes or worsening of preexisting disease. Unfortunately, the clinical course beyond the acute phase of the illness may persist in the form of a variety of symptoms that together form the so-called "Long COVID" or "Post-COVID Syndrome". It i…

InflammationEndocrinologyCOVID-19 Long COVID New-onset diabetes Newly diagnosed diabetes Post-COVID syndrome SARS CoV-2 Syndemia Type 2 diabetesSARS-CoV-2Endocrinology Diabetes and MetabolismHyperglycemiaInternal MedicineDiabetes MellitusHumansCOVID-19RNA ViralPandemics
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Hemocyte migration during inflammatory-like reaction of Ciona intestinalis (Tunicata, ascidiacea).

2000

Following an injuring factor such as the injection of erythrocytes into the tunic of Ciona intestinalis, an inflammatory-like response occurs and blood cells are massively involved in these mechanisms. Electron microscope observations illustrate the infiltration and migration of blood cells throughout the mantle epithelium into the tunic and show several prominent morphological changes.

InflammationErythrocytesHemocytesSheepInflammationAnatomyBiologybiology.organism_classificationmedicine.diseaseEpitheliumCell biologyCiona intestinalisInjectionsHemocyte migrationmedicine.anatomical_structureCell MovementmedicineAnimalsCiona intestinalisTunicamedicine.symptomMantle (mollusc)Infiltration (medical)Ecology Evolution Behavior and SystematicsAscidiaceaJournal of invertebrate pathology
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CD248 enhances tissue factor procoagulant function, promoting arterial and venous thrombosis in mouse models

2021

BACKGROUND: CD248 is a pro-inflammatory, transmembrane glycoprotein expressed by vascular smooth muscle cells (VSMC), monocytes/macrophages, and other cells of mesenchymal origin. Its distribution and properties are reminiscent of those of the initiator of coagulation, tissue factor (TF). OBJECTIVE: We examined whether CD248 also participates in thrombosis. METHODS: We evaluated the role of CD248 in coagulation using mouse models of vascular injury, and by assessing its functional interaction with the TF-factor VIIa (FVIIa)-factor X (FX) complex. RESULTS: The time to ferric chloride-induced occlusion of the carotid artery in CD248 knockout (KO) mice was significantly longer than in wild-typ…

InflammationFactor VIIa030204 cardiovascular system & hematologyInferior vena cavaArticleThromboplastin03 medical and health sciencesTissue factorchemistry.chemical_compoundMice0302 clinical medicineThrombinTissue factor pathway inhibitorAntigens CDAntigens NeoplasmmedicineAnimalsHumansMice KnockoutVenous Thrombosismedicine.diagnostic_testFactor XHematologyCoagulationchemistrymedicine.veinCancer researchProthrombin Timemedicine.symptommedicine.drugPartial thromboplastin time
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The multiple roles of dopamine receptor activation in the modulation of gastrointestinal motility and mucosal function

2022

Dopamine (DA) is a catecholamine regulatory molecule with potential role in physiology and physiopathology of the intestinal tract. Various cellular sources of DA have been indicated as enteric neurons, immune cells, intestinal flora and gastrointestinal epithelium. Moreover, DA is produced by nutritional tyrosine. All the five DA receptors, actually described, are present throughout the gut. Current knowledge of DA in this area is reviewed, focusing on gastrointestinal function in health and during inflammation. Research on animal models and humans are reported. A major obstacle to understanding the physiologic and/or pharmacological roles of enteric DA is represented by the multiplicity o…

InflammationGastrointestinal inflammationCellular and Molecular NeuroscienceCatecholaminesGastrointestinal motilityEndocrine and Autonomic SystemsDopamineHumansAnimalsGastrointestinal transportNeurology (clinical)Dopamine receptorsReceptors DopamineAutonomic Neuroscience
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Correction to: Depletion of regulatory T cells increases T cell brain infiltration, reactive astrogliosis, and interferon-γ gene expression in acute …

2019

Traumatic brain injury (TBI) is a major cause of death and disability. T cells were shown to infiltrate the brain during the first days after injury and to exacerbate tissue damage. The objective of this study was to investigate the hitherto unresolved role of immunosuppressive, regulatory T cells (Tregs) in experimental TBI."Depletion of regulatory T cell" (DEREG) and wild type (WT) C57Bl/6 mice, treated with diphtheria toxin (DTx) to deplete Tregs or to serve as control, were subjected to the controlled cortical impact (CCI) model of TBI. Neurological and motor deficits were examined until 5 days post-injury (dpi). At the 5 dpi endpoint, (immuno-) histological, protein, and gene expressio…

InflammationGeneral NeuroscienceImmunologyCorrectionBrainT-Lymphocytes Regulatorylcsh:RC346-429Lymphocyte DepletionCellular and Molecular NeuroscienceDisease Models AnimalInterferon-gammaMiceNeurologyAstrocytesBrain Injuries TraumaticAnimalsGliosislcsh:Neurology. Diseases of the nervous systemJournal of Neuroinflammation
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Inflammation and Life-Span

2005

In their Review “Inflammatory exposure and historical changes in human life-spans” (17 Sept., p. [1736][1]), C. E. Finch and E. M. Crimmins reinforce earlier suggestions that many diseases and disabilities of older age have their roots in previous exposures to infectious agents and other sources

InflammationGerontologyPolymorphism GeneticMultidisciplinarybiologyLife spanmedia_common.quotation_subjectLongevityLongevityInflammationBiological evolutionInfectionsBiological EvolutionLife ExpectancyCardiovascular Diseasesbiology.animalmedicineLife expectancyCytokinesHumansmedicine.symptomAllelesFinchmedia_commonScience
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Association of redox and inflammation-related biomarkers with prognosis in IgA nephropathy: A prospective observational study.

2022

IgA nephropathy (IGAN) has a variable prognosis. Risk stratification tools are usually based on clinical parameters combined with histologic Oxford-MEST-C score. Circulating redox- and inflammation-related biomarkers may be related to histological changes in IGAN. Therefore, we studied the performance of these biomarkers in predicting the rate of GFR-loss in IGAN.This was an observational prospective study. Fifty-seven stable patients with IGAN were examined at baseline and after a mean observational time of 5.9 ± 1.1 years. The main outcome measure was eGFR-loss per year with predefined groups, stable (1.5 ml/min/1,73 mFifteen patients were in the progressive, 11 in the intermediate, and 3…

InflammationGlomerulonephritis IGABiochemistryAdvanced Oxidation Protein ProductsReceptors Tumor Necrosis Factor Type IPhysiology (medical)Disease ProgressionHumansVDP::Medisinske Fag: 700OsteopontinCysteineProspective StudiesOxidation-ReductionBiomarkersGlomerular Filtration RateFree radical biologymedicine
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Intraosseous foreign-body granuloma in the mandible subsequent to a 20-year-old work-related accident

2008

Made available in DSpace on 2013-08-12T18:41:16Z (GMT). No. of bitstreams: 0 Previous issue date: 2008-10-01 Made available in DSpace on 2013-08-12T18:41:16Z (GMT). No. of bitstreams: 0 Previous issue date: 2008-10-01 Made available in DSpace on 2013-09-30T18:34:07Z (GMT). No. of bitstreams: 0 Previous issue date: 2008-10-01 Made available in DSpace on 2013-09-30T18:34:07Z (GMT). No. of bitstreams: 0 Previous issue date: 2008-10-01 Submitted by Vitor Silverio Rodrigues (vitorsrodrigues@reitoria.unesp.br) on 2014-05-20T14:04:30Z No. of bitstreams: 0 Submitted by Vitor Silverio Rodrigues (vitorsrodrigues@reitoria.unesp.br) on 2014-05-20T14:04:30Z No. of bitstreams: 0 Made available in DSpace …

InflammationGranulomaUNESCO::CIENCIAS MÉDICASForeign-bodyMandible:CIENCIAS MÉDICAS [UNESCO]Giant cells
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