Search results for "interleukin-1"

showing 10 items of 660 documents

Importance of the inducible costimulator molecule for the induction of allergic immune responses and its decreased expression on T helper cells after…

2004

The inducible costimulator (ICOS), a newly identified member of the CD28 receptor family that is induced after T-cell activation, and its ligand (ICOSL), being expressed on activated monocytes and dendritic cells play a key role in T-cell-mediated immune responses. As ICOS costimulation also seems to regulate T helper 2 effector cells, the aim of this study was to analyse the function of this molecule in allergic immune responses and their specific therapy, mainly venom immunotherapy (VIT). CD4+ T cells from grass pollen-, or bee or wasp venom-allergic donors were stimulated in the presence of autologous mature dendritic cells, which were pulsed with different allergen doses. In this system…

Antigens Differentiation T-Lymphocytemedicine.medical_treatmentImmunologyReceptors Antigen T-CellBiologyLymphocyte ActivationInducible T-Cell Co-Stimulator ProteinTh2 CellsImmune systemAntigenHypersensitivitymedicineHumansImmunology and AllergyInducible T-Cell Co-Stimulator ProteinReceptorArthropod VenomsHistocompatibility Antigens Class IICD28InterleukinOriginal ArticlesDendritic CellsImmunotherapyAllergensTh1 CellsCoculture TechniquesInterleukin-10CytokineDesensitization ImmunologicImmunologyImmunotherapyImmunology
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The interplay between the immune system and chemotherapy: emerging methods for optimizing therapy.

2013

Preclinical studies have revealed an unexpected ability of the immune system to contribute to the success of chemotherapy and radiotherapy. Anticancer therapies can trigger immune system activation by promoting the release of danger signals from dying tumor cells and/or the elimination of immunosuppressive cells. We have, however, recently discovered that some chemotherapies, such as 5-fluorouracil and gemcitabine, exert conflicting effects on anticancer immune responses. Although 5-fluorouracil and Gem selectively eliminated myeloid-derived suppressive cells in tumor-bearing rodents, these chemotherapies promoted the release of IL-1β and the development of pro-angiogenic IL-17-producing CD…

Antimetabolites Antineoplasticmedicine.medical_treatmentImmunologyInterleukin-1betaDeoxycytidineImmune systemImmunityNeoplasmsmedicineImmunology and AllergyAnimalsHumansImmunologic FactorsChemotherapyImmunity Cellularbusiness.industryImmunosuppressionInflammasomeChemoradiotherapyGemcitabineGemcitabineRadiation therapyImmunologyMyeloid-derived Suppressor CellCancer researchTh17 CellsFluorouracilbusinessmedicine.drugExpert review of clinical immunology
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Interleukin 27 induces differentiation of neural C6-precursor cells into astrocytes.

2007

Interleukin 6 (IL6)-type cytokines are major regulators of inflammation and thereby contribute to the neuropathology and pathophysiology associated with inflammation of the central nervous system (CNS). Furthermore, astrocyte development which is a key process in the development of the CNS is also controlled by cytokines of the IL6-family. Interleukin 27 (IL27) is a recently identified member of this family and has been implicated in the inhibition of TH17 T-cell-responses. Here we show that IL27 and the HHV8 encoded viral IL6 (vIL6) induce C6 glioma cells to differentiate into an astrocyte-like state. Cytokine stimulation led to STAT-factor phosphorylation and consequently to protein expre…

B-LymphocytesInterleukin-6Interleukin-17BiophysicsInterleukinCell DifferentiationCell BiologyBiologyBiochemistryCell biologyCell LineInterleukin 33Astrocyte differentiationInterleukin 32MiceInterleukin 20AstrocytesImmunologyAnimalsInterleukin 27Molecular BiologyInterleukin 4Interleukin 3Biochemical and biophysical research communications
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STAT6: its role in interleukin 4-mediated biological functions.

1997

Interleukin (IL) 4 is known to be a cytokine which plays a central role in the regulation of immune response. Studies on cytokine signal transduction have clarified the mechanism by which IL4 exerts its functions. Two cytoplasmic proteins, signal transducer and activator of transcription (STAT) 6 and IL4-induced phosphotyrosine substrate/insulin receptor substrate 2 (4PS/IRS2), are activated in IL4 signal transduction. Recent studies from STAT6-deficient mice have revealed the essential role of STAT6 in IL4-mediated biological actions. In addition, STAT6 has also been demonstrated to be important for the functions mediated by IL13, which is related to IL4. IL4 and IL13 have been shown to in…

BiologyMediatorimmune system diseasesAntigens CDparasitic diseasesDrug DiscoveryAnimalsHumansskin and connective tissue diseasesGenetics (clinical)Interleukin 4STAT6Interleukin-13Interleukinhemic and immune systemsReceptors InterleukinIRS2Cell biologyReceptors Interleukin-4Interleukin 13ImmunologySTAT proteinTrans-ActivatorsMolecular MedicineInterleukin-4Signal transductionSTAT6 Transcription FactorSignal TransductionJournal of molecular medicine (Berlin, Germany)
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Are IL-10+ regulatory Th17 cells implicated in the sustained response to glucocorticoid treatment in patients with giant cell arteritis? Comment on t…

2013

We have read with interest the recently published paper of Espigol-Frigole et al 1 in which the authors confirmed that interleukin (IL)-17 is highly expressed in giant cell arteritis (GCA) lesions.1–3 They also demonstrated for the first time that IL-17 expression in temporal artery biopsies (TABs) was correlated with a better outcome. Among other interesting results, the identification of Foxp3+IL-17+ T cells by confocal microscopy in TAB made the authors to hypothesize that these cells could be induced regulatory T cells (Treg) that may facilitate the remission of the disease under steroid therapy. …

BiopsyGiant Cell ArteritisImmunologyGeneral Biochemistry Genetics and Molecular Biologylaw.inventionRheumatologyRecurrenceConfocal microscopylawBiopsymedicineHumansImmunology and AllergyGlucocorticoidsmedicine.diagnostic_testbusiness.industryInterleukin-17InterleukinFOXP3Forkhead Transcription Factorsmedicine.diseaseInterleukin-10Temporal ArteriesGiant cell arteritisInterleukin 10ImmunologyTh17 CellsInterleukin 17businessGlucocorticoidmedicine.drugAnnals of the Rheumatic Diseases
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Invariant NKT cells are expanded in peripheral blood but are undetectable in salivary glands of patients with primary Sjögren's syndrome

2016

OBJECTIVES: Invariant NKT (iNKT) cells play a role in regulating the function of autoreactive B cells before their entry into germinal centres. Absence and/or reduction of iNKT cells have been demonstrated in patients with systemic lupus erythematosus (SLE) together with an increase of autoreactive B cell activity. Primary Sjögren's syndrome (pSS) is a systemic autoimmune disease in which lymphocyte infiltration and organisation in lymphoid structures of inflamed salivary glands occurs. The aim of the study was to investigate the percentage and function of iNKT in the salivary glands and peripheral blood of patients with pSS. METHODS: Minor salivary gland biopsies were obtained from patient…

BiopsyReceptors Antigen T-CellFluorescent Antibody TechniqueEnzyme-Linked Immunosorbent AssayCell CommunicationLymphocyte ActivationReal-Time Polymerase Chain ReactionSalivary GlandsInterferon-gammastomatognathic systemHumansLymphocyte CountCells CulturedCell ProliferationB-LymphocytesInterleukin-17Flow CytometryCoculture TechniquesSettore MED/16 - Reumatologiastomatognathic diseasesIL-17Sjogren's SyndromeAntibodies AntinuclearCase-Control StudiesAntibody FormationNatural Killer T-CellsSjögren's syndromeNKT cellNKT cells Sjögren's syndrome IL-17
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Eicosapentaenoic acid modulates fatty acid metabolism and inflammation in Psammomys obesus.

2015

Abstract The desert gerbil, Psammomys obesus, is a unique polygenic animal model of metabolic syndrome (insulin resistance, obesity and type 2 diabetes), and these pathological conditions resemble to those in human beings. In this study, the animals were fed ad libitum either a natural diet (ND) which contained desertic halophile plants or a standard laboratory diet (STD) or a diet which contained eicosapentaenoic acid (EPA), hence, termed as EPA diet (EPAD). In EPAD, 50% of total lipid content was replaced by EPA oil. By employing real-time PCR, we assessed liver expression of key genes involved in fatty acid metabolism such as PPAR-α, SREBP-1c, LXR-α and CHREBP. We also studied the expres…

Blood GlucoseMalemedicine.medical_specialtymedicine.medical_treatmentInterleukin-1betaAdipose tissueGene ExpressionBiologyBiochemistrychemistry.chemical_compoundInsulin resistanceInternal medicinemedicineAnimalsInsulinPPAR alphaRNA MessengerTriglyceridesLiver X Receptorschemistry.chemical_classificationFatty acid metabolismReverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaInsulinBody WeightFatty AcidsGeneral Medicinebiology.organism_classificationmedicine.diseaseLipid MetabolismOrphan Nuclear ReceptorsEicosapentaenoic acidEndocrinologychemistryAdipose TissueEicosapentaenoic AcidLiverlipids (amino acids peptides and proteins)PsammomysMetabolic syndromeInflammation MediatorsGerbillinaeSterol Regulatory Element Binding Protein 1Polyunsaturated fatty acidBiochimie
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Risk profiles in type 2 diabetes (metabolic syndrome): integration of IL-10 polymorphisms and laboratory parameters to identify vascular damages rela…

2010

Recently it has been reported that low serum IL-10 levels are associated with an increased susceptibility for metabolic syndrome and type 2 diabetes mellitus (T2DM). We investigated whether the -1087G/A (rs1800896), -824C/T (rs1800871), -597C/A (rs1800872) IL-10 polymorphisms were associated with type 2 diabetes in a study on a cohort of Italian Caucasians comprising 490 type 2 diabetic and 349 control subjects. Stratifying the data according to IL-10 genotypes, trends for the progressive increase of glucose and neutrophil levels were observed in -1087GG vs. -1087GA vs. -1087AA positive diabetic patients (-1087GG < -1087GA < -1087AA). In addition, evaluating the laboratory parameters accord…

Blood GlucoseMalemedicine.medical_specialtytype 2 diabetes mellituNeutrophilsPopulationMyocardial InfarctionType 2 diabetesGastroenterologyPolymorphism Single NucleotideCohort StudiesDiabetes ComplicationsLaboratory profile IL-10 levelRisk FactorsInternal medicineDrug DiscoverymedicineSettore MED/05 - Patologia ClinicaHumansIL-10 genotypeMyocardial infarctioneducationgrade of membershipBlood urea nitrogenPharmacologyMetabolic Syndromeeducation.field_of_studybusiness.industryVascular damage pronenessrisk profileType 2 Diabetes MellitusMiddle Agedmedicine.diseaseInterleukin-10EndocrinologyDiabetes Mellitus Type 2HaplotypesCohortKidney Failure ChronicIL-10 genotypesFemalegrade-of-membership analysitype 2 diabetesMetabolic syndromebusinessKidney disease
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Peroxisome Proliferator-Activated Receptor Deficiency Increases the Risk of Maternal Abortion and Neonatal Mortality in Murine Pregnancy with or with…

2006

We assessed the implication of peroxisome proliferator-activated receptor (PPAR) alpha deficiency in pregnancy outcome and neonatal survival and in the modulation of T cell differentiation in murine diabetic pregnancy and their offspring. Pregnant wild-type (WT) and PPAR alpha-null mice of C57BL/6J genetic background were rendered diabetic by five low doses of streptozotocin. We observed that, in the absence of diabetes, PPAR alpha deficiency resulted in an increase in abortion rate, i.e. 0% in WT mice vs. 20% in PPAR alpha-null mice [odds ratio (OR) = 14.33; P = 0.013]. Under diabetic conditions, the abortion rate was enhanced, i.e. 8.3% in WT mice vs. 50% in PPAR alpha-null mice (OR = 4.2…

Blood Glucosemedicine.medical_specialtyOffspringRatónT-LymphocytesPeroxisome proliferator-activated receptorBiologyPeroxisomeDiabetes Mellitus ExperimentalInterferon-gammaMiceEndocrinologyTh2 CellsDownregulation and upregulationPregnancyDiabetes mellitusInternal medicinemedicineAnimalsInsulinPPAR alphaLymphocyte CountRNA MessengerReceptorFetal Deathchemistry.chemical_classificationMice KnockoutPregnancy[SCCO.NEUR]Cognitive science/NeuroscienceCell DifferentiationTh1 CellsStreptozotocinmedicine.diseaseLipidsInterleukin-10Abortion SpontaneousMice Inbred C57BLPregnancy ComplicationsEndocrinologychemistry[ SCCO.NEUR ] Cognitive science/NeuroscienceCytokinesInterleukin-2FemaleInterleukin-4Spleenmedicine.drug
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Blood flow patterns spatially associated with platelet aggregates in murine colitis.

2009

In the normal murine mucosal plexus, blood flow is generally smooth and continuous. In inflammatory conditions, such as chemically-induced murine colitis, the mucosal plexus demonstrates markedly abnormal flow patterns. The inflamed mucosal plexus is associated with widely variable blood flow velocity as well as discontinuous and even bidirectional flow. To investigate the mechanisms responsible for these blood flow patterns, we used intravital microscopic examination of blood flow within the murine mucosal plexus during dextran sodium sulphate-and trinitrobenzenesulfonic acid-induced colitis. The blood flow patterns within the mucosal plexus demonstrated flow exclusion in 18% of the vessel…

Blood PlateletsPathologymedicine.medical_specialtyHistologyPlatelet AggregationInterleukin-1betaBiologyArticleMicrocirculationchemistry.chemical_compoundMiceIntestinal mucosamedicineAnimalsPlateletColitisIntestinal MucosaEcology Evolution Behavior and SystematicsPlexusMice Inbred BALB CGene Expression ProfilingMicrocirculationDextran SulfateBlood flowmedicine.diseaseColitisMice Inbred C57BLDextranchemistryTrinitrobenzenesulfonic AcidRegional Blood FlowAcute DiseaseEndothelium VascularAnatomyChemokinesIntravital microscopyBiotechnologyAnatomical record (Hoboken, N.J. : 2007)
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