Search results for "lcsh:R5-920"

showing 10 items of 408 documents

The fear-defense system, emotions, and oxidative stress

2020

Psychosocial stress has a profound impact on well-being and health. The response to stress is associated mainly with the amygdala, a crucial structure of the fear-defense system, essential for social cognition and emotion regulation. Recent neuroimaging-studies demonstrated how an increased metabolic activity of the amygdala enhances inflammation, and leads to cardiometabolic disease. The development of therapeutic strategies depends on our understanding of both which factors activate the fear-defense system and the subsequent molecular mechanisms that translate emotional stress into cell damage. Fear of emotions as an aftermath of attachment trauma is the most important trigger of the mala…

0301 basic medicineFear-defense systemEmotionsClinical BiochemistryInflammationAnxietymedicine.disease_causeBiochemistryAmygdalaProinflammatory cytokine03 medical and health sciences0302 clinical medicineSocial cognitionmedicineHumanslcsh:QH301-705.5Cell damageInflammationlcsh:R5-920business.industryOrganic ChemistryFearAmygdalamedicine.diseaseGraphical ReviewOxidative Stress030104 developmental biologymedicine.anatomical_structurelcsh:Biology (General)Anxietymedicine.symptomlcsh:Medicine (General)businessNeuroscience030217 neurology & neurosurgeryGlucocorticoidOxidative stressmedicine.drugRedox Biology
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Prooxidative chain transfer activity by thiol groups in biological systems

2020

Cysteine is arguably the best-studied biological amino acid, whose thiol group frequently participates in catalysis or ligand binding by proteins. Still, cysteine's unusual biological distribution has remained mysterious, being strikingly underrepresented in transmembrane domains and on accessible protein surfaces, particularly in aerobic life forms (“cysteine anomaly”). Noting that lipophilic thiols have been used for decades as radical chain transfer agents in polymer chemistry, we speculated that the rapid formation of thiyl radicals in hydrophobic phases might provide a rationale for the cysteine anomaly. Hence, we have investigated the effects of dodecylthiol and related compounds in i…

0301 basic medicineFree RadicalsDNA damageLipid peroxidationClinical BiochemistryProtein oxidationBiochemistryLipid peroxidation03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCysteine oxidationAnimalsHumansCysteineSulfhydryl CompoundsCaenorhabditis eleganslcsh:QH301-705.5chemistry.chemical_classificationlcsh:R5-920Organic ChemistryAmino acidTransmembrane domain030104 developmental biologylcsh:Biology (General)Structural biologychemistryBiochemistryThiyl radicalsThiolRadical propagationlcsh:Medicine (General)Protein oxidation030217 neurology & neurosurgeryResearch PaperCysteineRedox Biology
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ESC-Derived BDNF-Overexpressing Neural Progenitors Differentially Promote Recovery in Huntington's Disease Models by Enhanced Striatal Differentiation

2016

Summary Huntington's disease (HD) is characterized by fatal motoric failures induced by loss of striatal medium spiny neurons. Neuronal cell death has been linked to impaired expression and axonal transport of the neurotrophin BDNF (brain-derived neurotrophic factor). By transplanting embryonic stem cell-derived neural progenitors overexpressing BDNF, we combined cell replacement and BDNF supply as a potential HD therapy approach. Transplantation of purified neural progenitors was analyzed in a quinolinic acid (QA) chemical and two genetic HD mouse models (R6/2 and N171-82Q) on the basis of distinct behavioral parameters, including CatWalk gait analysis. Explicit rescue of motor function by…

0301 basic medicineGene ExpressionBiochemistrychemistry.chemical_compoundMice0302 clinical medicineNeural Stem CellsNeurotrophic factorsGenes Reporterlcsh:QH301-705.5Neuronslcsh:R5-920NeurogenesisCell DifferentiationAnatomyembryonic stem cellsHuntington Diseaselcsh:Medicine (General)NeurogliaLocomotionNeurotrophinHuntington’s diseaseCell SurvivalBiologyMedium spiny neuronArticle03 medical and health sciencesHuntington's diseaseGeneticsmedicinestriatal differentiationAnimalsBrain-derived neurotrophic factorBrain-Derived Neurotrophic FactorCell Biologymedicine.diseaseCorpus StriatumTransplantationDisease Models Animal030104 developmental biologylcsh:Biology (General)chemistrynervous systembiology.proteinNeuroscience030217 neurology & neurosurgeryBiomarkersDevelopmental BiologyQuinolinic acidStem Cell TransplantationStem Cell Reports
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First Report on the Latvian SARS-CoV-2 Isolate Genetic Diversity

2021

Remaining a major healthcare concern with nearly 29 million confirmed cases worldwide at the time of writing, novel severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) has caused more than 920 thousand deaths since its outbreak in China, December 2019. First case of a person testing positive for SARS-CoV-2 infection within the territory of the Republic of Latvia was registered on 2nd of March 2020, 9 days prior to the pandemic declaration by WHO. Since then, more than 277,000 tests were carried out confirming a total of 1,464 cases of coronavirus disease 2019 (COVID-19) in the country as of 12th of September 2020. Rapidly reacting to the spread of the infection, an ongoing sequenci…

0301 basic medicineHCoV-19Coronavirus disease 2019 (COVID-19)Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2)medicine.disease_cause03 medical and health sciences0302 clinical medicinePandemicmedicineChinaOriginal ResearchCoronavirusGenetic diversitylcsh:R5-920SARS-CoV-2LatvianOutbreakCOVID-19General Medicinegenetic diversityVirologyLatvialanguage.human_language030104 developmental biologyGeography2019-nCoVlanguageMedicinenext-generation sequencingSevere acute respiratory syndrome coronaviruslcsh:Medicine (General)030217 neurology & neurosurgeryFrontiers in Medicine
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Modulating endothelial adhesion and migration impacts stem cell therapies efficacy

2020

Abstract Background Limited knowledge of stem cell therapies` mechanisms of action hampers their sustainable implementation into the clinic. Specifically, the interactions of transplanted stem cells with the host vasculature and its implications for their therapeutic efficacy are not elucidated. We tested whether adhesion receptors and chemokine receptors on stem cells can be functionally modulated, and consequently if such modulation may substantially affect therapeutically relevant stem cell interactions with the host endothelium. Methods We investigated the effects of cationic molecule polyethylenimine (PEI) treatment with or without nanoparticles on the functions of adhesion receptors a…

0301 basic medicineHomingCell- and Tissue-Based TherapyMedizinCCR4lcsh:MedicineStem cellsMiceChemokine receptor0302 clinical medicineCell MovementCells CulturedMigrationlcsh:R5-920Cell DifferentiationAdhesion; Glioma; Homing; Migration; Stem cells; StrokeGeneral MedicineGlioma3. Good healthCell biologyStrokemedicine.anatomical_structureCellular Microenvironment030220 oncology & carcinogenesisAdhesionStem celllcsh:Medicine (General)Research PaperEndotheliumBiologyMesenchymal Stem Cell TransplantationGeneral Biochemistry Genetics and Molecular BiologyCell LineImmunophenotyping03 medical and health sciencesIn vivoCell AdhesionmedicineAnimalsHumansEndotheliumMesenchymal stem celllcsh:RMesenchymal Stem CellsXenograft Model Antitumor AssaysIn vitroRatsDisease Models Animal030104 developmental biologyBiomarkersStem Cell TransplantationHoming (hematopoietic)EBioMedicine
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The role of oxidative stress in cardiovascular disease caused by social isolation and loneliness

2020

Loneliness and social isolation are common sources of chronic stress in modern society. Epidemiological studies have demonstrated that loneliness and social isolation increase mortality risk as much as smoking or alcohol consumption and more than physical inactivity or obesity. Loneliness in human is associated with higher blood pressure whereas enhanced atherosclerosis is observed in animal models of social isolation. Loneliness and social isolation lead to activation of the hypothalamic-pituitary-adrenocortical (HPA) axis, enhanced sympathetic nerve activity, impaired parasympathetic function and a proinflammatory immune response. These mechanisms have been implicated in the development o…

0301 basic medicineHypothalamo-Hypophyseal Systemmedicine.medical_specialtyClinical BiochemistryPituitary-Adrenal SystemDiseasemedicine.disease_causeBiochemistryProinflammatory cytokine03 medical and health sciences0302 clinical medicineInternal medicinemedicineAnimalsHumansChronic stressSocial isolationlcsh:QH301-705.5Social stressSocial isolationlcsh:R5-920business.industryLonelinessOrganic ChemistryLonelinessRostral ventrolateral medullaCardiovascular diseaseGraphical ReviewRatsDisease Models Animal030104 developmental biologyEndocrinologylcsh:Biology (General)Cardiovascular DiseasesOxidative stressmedicine.symptombusinesslcsh:Medicine (General)030217 neurology & neurosurgeryOxidative stressRedox Biology
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Role of subclinical gut inflammation in the pathogenesis of spondyloarthritis

2018

Subclinical gut inflammation occurring in patients affected by spondyloarthritis (SpA) is correlated with the severity of spine inflammation. Several evidences indicate that dysbiosis occurs in SpA, and that may modulate intestinal permeability and intestinal immune responses. The presence of intestinal dysbiosis is accompanied in SpA patients with the presence of zonulin-dependent alterations of gut-epithelial and gut-vascular barriers. The leakage of epithelial and endothelial surface layers is followed by the translocation of bacterial products, such as lipopolysaccharide and intestinal fatty acid binding protein, in the systemic circulation. These bacterial products may downregulate the…

0301 basic medicineInnate immune responseLipopolysaccharideenthesitis-related arthritisCD14Mini ReviewInflammationInflammation mediator03 medical and health scienceschemistry.chemical_compound0302 clinical medicineImmune systemmedicine030203 arthritis & rheumatologyEnthesitis-related arthritilcsh:R5-920Gut microbiomeInnate immune systemIntestinal permeabilitybusiness.industryInnate lymphoid cellGeneral Medicinemedicine.diseaseinflammation mediatorsSettore MED/16 - Reumatologia030104 developmental biologychemistryImmunologyMedicinemedicine.symptombusinesslcsh:Medicine (General)DysbiosisSpondylitis
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A selective inhibitor of the Polo-box domain of Polo-like kinase 1 identified by virtual screening

2018

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0301 basic medicineLK Polo-like kinasePolo-like kinaseCell cycleIC50 50% inhibition concentrationVirtual drug screeningPLK103 medical and health sciences0302 clinical medicineNeoplasmsTargeted chemotherapylcsh:Science (General)MitosisComputingMethodologies_COMPUTERGRAPHICSCDK cyclin-dependent kinasePBD Polo-box domainPyRxNatural productslcsh:R5-920MultidisciplinaryMicroscale thermophoresisKinaseChemistryCell cycleCell biology030104 developmental biology030220 oncology & carcinogenesisCancer cellOriginal ArticleCAMKK2 calcium/calmodulin-dependent protein kinase kinase 2PC Polo-box caplcsh:Medicine (General)Multipolar spindleslcsh:Q1-390Journal of Advanced Research
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Early endocrine disruptors exposure acts on 3T3-L1 differentiation and endocrine activity

2017

International audience; Introduction: Data from last years suggested that early exposure to endocrine disruptors (EDs) can predispose newborns to endocrine dysfunction of adipocytes, obesity, and associated disorders. The implication of EDs at low doses on adipocyte development has been poorly investigated. For instance, vinclozolin (V) is a dicarboximide fungicide widely used in agriculture since the 90's, alone or in mixture with genistein (G), an isoflavonoid from Leguminosae. This study aims to identify the effect of vinclozolin alone or with genistein, on adipose tissue properties using cell culture.Methods: In steroid-free conditions, 3T3-L1 pre-adipocytes were induced to differentiat…

0301 basic medicineLeptin[ SDV.TOX.ECO ] Life Sciences [q-bio]/Toxicology/Ecotoxicologytissu adipeuxPharmaceutical ScienceGenisteinAdipose tissueoestradiolsourisTriglyceridechemistry.chemical_compound0302 clinical medicineAdipocyteratVinclozolinlcsh:QH301-705.5Original Researchperturbateur endocrinienlcsh:R5-920LeptinGeneral MedicineGenistein[ SDV.TOX.TCA ] Life Sciences [q-bio]/Toxicology/Toxicology and food chainobésité030220 oncology & carcinogenesisAlimentation et NutritionEndocrinologie et métabolismeleptine[SDV.TOX.ECO]Life Sciences [q-bio]/Toxicology/Ecotoxicologylcsh:Medicine (General)medicine.medical_specialtyAdipose tissue[SDV.TOX.TCA]Life Sciences [q-bio]/Toxicology/Toxicology and food chainBiologyadipocyteGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesInternal medicinemedicineFood and NutritionEndocrine systemEndocrine disruptorsEndocrinology and metabolism3T3-L1adipose tissue;endocrine disruptors;genistein;triglyceride;leptin;vinclozolin;promotes adipocyte differentiation;adipose-tissue development;rat;adipocytes;mice;obesity;expression;estradiol030104 developmental biologyEndocrinologylcsh:Biology (General)chemistryVinclozolinCell culture
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Protein tyrosine phosphatase 1b deficiency protects against hepatic fibrosis by modulating nadph oxidases

2019

Inflammation is typically associated with the development of fibrosis, cirrhosis and hepatocellular carcinoma. The key role of protein tyrosine phosphatase 1B (PTP1B) in inflammatory responses has focused this study in understanding its implication in liver fibrosis. Here we show that hepatic PTP1B mRNA expression increased after bile duct ligation (BDL), while BDL-induced liver fibrosis was markedly reduced in mice lacking Ptpn1 (PTP1B−/−) as assessed by decreased collagen deposition and α-smooth muscle actin (α-SMA) expression. PTP1B−/− mice also showed a significant increase in mRNA levels of key markers of monocytes recruitment (Cd68, Adgre1 and Ccl2) compared to their wild-type (PTP1B+…

0301 basic medicineLiver CirrhosisMaleClinical BiochemistryGene ExpressionApoptosisBiochemistryMice0302 clinical medicineFibrosisTransforming Growth Factor betaRNA Small Interferinglcsh:QH301-705.5Liver injuryProtein Tyrosine Phosphatase Non-Receptor Type 1lcsh:R5-920NADPH oxidaseProtein tyrosine phosphatase 1BbiologyChemistryNOX4Bile duct ligationImmunohistochemistry3. Good healthNOX1Femalelcsh:Medicine (General)hormones hormone substitutes and hormone antagonistsResearch PaperBone marrow transplantationKupffer CellsLiver fibrosisdigestive systemCell LineBile Acids and Salts03 medical and health sciencesmedicineHepatic Stellate CellsAnimalsInflammationOrganic Chemistrymedicine.diseaseMolecular biologyTransplantationDisease Models Animal030104 developmental biologylcsh:Biology (General)Culture Media ConditionedNADPH oxidasesHepatic stellate cellbiology.proteinHepatocytesHepatic fibrosisReactive Oxygen Species030217 neurology & neurosurgeryBiomarkersRedox Biology
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