Search results for "marker"

showing 10 items of 3799 documents

Serology in adults with celiac disease: limited accuracy in patients with mild histological lesions.

2010

Celiac disease (CD) is a gluten-triggered enteropathy, presenting with insidious clinical patterns. It can occasionally be diagnosed in asymptomatic subjects. Our aim was to define the relationship among symptoms at diagnosis, serological markers [tissue transglutaminase antibodies (tTGA), anti-endomysium antibodies (EMA) anti-actin antibodies (AAA)] and degree of mucosal damage. A total of 68 consecutive adult patients with CD were enrolled. Intestinal biopsies were scored according to the Marsh classification modified by Oberhuber: I–II minimal lesions or absent villous atrophy; IIIA partial villous atrophy; IIIB–C total villous atrophy (TVA). HLA-typing was done for all patients. No asso…

AdultGenetic MarkersMalePathologymedicine.medical_specialtyAdolescentTissue transglutaminaseEnzyme-Linked Immunosorbent AssayDiseaseSeverity of Illness IndexAsymptomaticStatistics NonparametricSerologyYoung AdultInternal MedicinemedicineHumansEnteropathyIn patientProspective StudiesIntestinal MucosaVillous atrophyAgedAged 80 and overbiologybusiness.industryMiddle Agedmedicine.diseaseImmunoglobulin AItalyEmergency Medicinebiology.proteinFemaleAntibodymedicine.symptombusinessceliac disease
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Intra- and Interhemispheric Electroencephalogram Coherence in Siblings Discordant for Schizophrenia and Healthy Volunteers

1997

Former studies had pointed to an increased electroencephalogram (EEG) coherence in schizophrenics, but it remained unsolved whether this deviation represents the premorbid state or is only a consequence of the current or previous schizophrenic episodes. To clarify this question, we tested the hypothesis that subjects at elevated risk also reveal higher coherences compared to healthy controls. For that, intra- and interhemispheric EEG coherences were investigated in untreated schizophrenics, their healthy siblings, and healthy controls. Differences were only found regarding the intrahemispheric coherences. Both in schizophrenics and, even though to a lesser degree, in their siblings signific…

AdultGenetic MarkersMalePsychosismedicine.medical_specialtyAudiologyElectroencephalographyReference ValuesRisk FactorsMaldevelopmentHealthy volunteersmedicineHumansAttentionSiblingDominance CerebralPsychiatryBiological PsychiatryCerebral CortexFourier Analysismedicine.diagnostic_testElectroencephalographySignal Processing Computer-AssistedCoherence (statistics)medicine.diseasePsychotic DisordersSchizophreniaCerebral hemisphereSchizophreniaFemaleArousalPsychologyBiological Psychiatry
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Wisconsin Card Sorting Test: an indicator of vulnerability to schizophrenia?

1992

The Wisconsin Card Sorting Test (WCST) is a neuropsychological test, hypothesized to be an indicator of dorsolateral prefrontal cortex (DLPFC) functioning. The performance of schizophrenic patients in our sample (off medication) was worse than the performance of healthy controls in all variables of the WCST, including perseverative responses (PR) as well as non-perseverative responses (NPR). The rate of perseverative and non-perseverative responses was neither a function of the severity of the illness (measured by SANS/SAPS scales) nor the duration of the disease. Healthy siblings of schizophrenic probands revealed more perseverative responses than healthy controls, but did not show any dif…

AdultGenetic MarkersMalePsychosismedicine.medical_specialtyPsychometricsNeurocognitive DisordersNeuropsychological TestsAudiologySocial Environmentbehavioral disciplines and activitiesDevelopmental psychologyWisconsin Card Sorting TestRisk FactorsSchizophrenic PsychologymedicineHumansNeuropsychological assessmentPrefrontal cortexBiological Psychiatrymedicine.diagnostic_testGenetic Carrier ScreeningNeuropsychological testmedicine.diseaseDorsolateral prefrontal cortexPsychiatry and Mental healthmedicine.anatomical_structureSchizophreniaSchizophreniaFemaleSchizophrenic PsychologyPsychologySchizophrenia Research
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Lung microenvironments and disease progression in fibrotic hypersensitivity pneumonitis

2022

Rationale: Fibrotic hypersensitivity pneumonitis (fHP) is an interstitial lung disease caused by sensitization to an inhaled allergen. Objectives: To identify the molecular determinants associated with progression of fibrosis. Methods: Nine fHP explant lungs and six unused donor lungs (as controls) were systematically sampled (4 samples/lung). According to microcomputed tomography measures, fHP cores were clustered into mild, moderate, and severe fibrosis groups. Gene expression profiles were assessed using weighted gene co-expression network analysis, xCell, gene ontology, and structure enrichment analysis. Gene expression of the prevailing molecular traits was also compared with idiopathi…

AdultGenetic MarkersMalePulmonary and Respiratory MedicinePathologymedicine.medical_specialtyExtrinsic Allergic Alveolitisextrinsic allergic alveolitisCritical Care and Intensive Care MedicineSeverity of Illness IndexTranscriptome03 medical and health sciences0302 clinical medicineFibrosisPulmonary fibrosisMedicineHumansLungAged030304 developmental biology0303 health sciencesLungmedicine.diagnostic_testpulmonary fibrosisbusiness.industryGene Expression ProfilingInterstitial lung diseaseReproducibility of ResultsOriginal ArticlesMiddle Agedrespiratory systemmedicine.diseaseFibrosisIdiopathic Pulmonary Fibrosis3. Good healthrespiratory tract diseasesmedicine.anatomical_structureBronchoalveolar lavage030228 respiratory systemCase-Control StudiesDisease ProgressionLinear ModelsFemaleHuman medicineTranscriptomebusinesstranscriptomeHypersensitivity pneumonitisAlveolitis Extrinsic AllergicAmerican journal of respiratory and critical care medicine
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A novel Angiogenin gene mutation in a sporadic patient with amyotrophic lateral sclerosis from southern Italy

2007

Mutations in the Angiogenin gene (ANG) linked to 14q11.2 have been recently discovered to be associated with Amyotrophic Lateral Sclerosis (ALS) in Irish and Scottish populations. In our study we investigated the role of ANG gene in ALS patients from southern Italy. We found a novel mutation in the signal peptide of the ANG gene in a sporadic patient with ALS (SALS). The molecular analysis of the ANG gene also demonstrated an allelic association with the rs11701 single nucleotide polymorphism (SNP) in familial ALS (FALS) but not in SALS patients. Our finding supports the evidence that the ANG gene is involved in ALS.

AdultGenetic MarkersMaleSignal peptideAngiogenin geneAngiogeninGenetic LinkageDNA Mutational AnalysisSingle-nucleotide polymorphismGene mutationBiologyPolymorphism Single NucleotidemedicineHumansSNPGenetic Predisposition to DiseaseGenetic TestingAlleleAmyotrophic lateral sclerosisGeneGenetics (clinical)AgedChromosomes Human Pair 14Motor NeuronsGeneticsAmyotrophic Lateral SclerosisChromosome MappingRibonuclease PancreaticMiddle Agedmedicine.diseaseAssociation studyAmino Acid SubstitutionItalyNeurologyCytoprotectionMutationNerve DegenerationPediatrics Perinatology and Child Healthcardiovascular systemCancer researchFemaleNeurology (clinical)ALShormones hormone substitutes and hormone antagonistsNeuromuscular Disorders
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Forty-two supernumerary marker chromosomes (SMCs) in 43,273 prenatal samples: chromosomal distribution, clinical findings, and UPD studies.

2005

Fluorescence in situ hybridization (FISH) analyses were performed on supernumerary marker chromosomes (SMCs) detected in 43 273 prenatal diagnoses over a period of 11 years, 1993–2003. A total of 42 pregnancies with SMC were identified, indicating a prevalence of one in 1032. A total of 15 SMCs were endowed with detectable euchromatin (prevalence, 1/2884), including six SMCs containing the cat eye critical region (CECR) on chromosome 22q11.21 (1/7212). De novo SMCs were found in 29 pregnancies (1/1492), including 14 euchromatic SMCs (48.2%). Follow-up studies were available for 24 cases. Nine pregnancies (37.5%) were terminated; two children (8.3%) were born with Pallister–Killian syndrome …

AdultGenetic MarkersMalemedicine.medical_specialtyAdolescentAneuploidyPrenatal diagnosisBiologyFetusPregnancyPrenatal DiagnosisGeneticsmedicineHumansSupernumeraryAbnormalities MultipleGenetic TestingChildGenetics (clinical)In Situ Hybridization FluorescenceGynecologyGeneticsChromosome AberrationsPregnancymedicine.diagnostic_testInfantUniparental Disomymedicine.diseaseAneuploidyUniparental disomyCat eye syndromeChorionic Villi SamplingChild PreschoolKaryotypingPopulation SurveillanceCytogenetic Analysiscardiovascular systemAmniocentesisFemaleChromosome 22Fluorescence in situ hybridizationEuropean journal of human genetics : EJHG
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Association between C1019T polymorphism of connexin37 and acute myocardial infarction: a study in patients from Sicily

2003

Abstract During atherogenesis, a critical role is played by intercellular communication via gap junctions, cell membrane channels linking the cytoplasmic compartments of adjacent cells. The component protein subunits of these channels, called connexin (Cx), belong to a multigene family. Cx37 is involved in growth, regeneration after injury and ageing of the endothelial cells, suggesting its role in atherosclerosis. The C1019 single nucleotide polymorphism (SNP) of Cx37 gene was associated with thickening of the carotid intima in Swedish men and was also associated with coronary artery disease in a Taiwanese population. On the other hand, in two more recent studies performed in male Japanese…

AdultGenetic MarkersMalemedicine.medical_specialtyPathologyGenotypeHeart diseasePopulationMyocardial InfarctionSingle-nucleotide polymorphismPolymerase Chain ReactionPolymorphism Single NucleotideGastroenterologyConnexinsCoronary artery diseaseGene FrequencyRisk FactorsInternal medicineOdds RatioHumansMedicineSNPMyocardial infarctioneducationSicilyRetrospective Studieseducation.field_of_studybusiness.industryIncidenceCase-control studyDNAOdds ratioMiddle Agedmedicine.diseasePhenotypeCardiology and Cardiovascular MedicinebusinessInternational Journal of Cardiology
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Restless legs syndrome: Epidemiological and clinicogenetic study in a South Tyrolean population isolate.

2006

Genetic contributions to restless legs syndrome (RLS) have been consistently recognized from population and family studies. To determine the clinical and genetic features of RLS in a population isolate and explore linkage to three previously described susceptibility loci on chromosomes 12q, 14q, and 9p, respectively, an isolated population in the South Tyrolean Alps was identified and 530 adults participated in the study. Using a two-step strategy, 47 patients with idiopathic RLS were ascertained. The prevalence in the population was 8.9%. Twenty-eight patients (59.6%) had at least one affected first-degree relative and were classified as hereditary cases. In a single extended pedigree, lin…

AdultGenetic MarkersMalemedicine.medical_specialtyPopulationFamily studiesLocus heterogeneityGermanyRestless Legs SyndromeSurveys and Questionnairesmental disordersEpidemiologymedicineHumansRestless legs syndromeeducationLinkage (software)Geneticseducation.field_of_studyPolymorphism Geneticbusiness.industryChromosome MappingReproducibility of Resultsmedicine.diseaseSurgeryPedigreeNeurologyItalyHomogeneousSusceptibility locusFemaleNeurology (clinical)business
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Molecular characterization of 39 de novo sSMC: contribution to prognosis and genetic counselling, a prospective study.

2012

Small supernumerary marker chromosomes (sSMCs) are structurally abnormal chromosomes that cannot be characterized by karyotype. In many prenatal cases of de novo sSMC, the outcome of pregnancy is difficult to predict because the euchromatin content is unclear. This study aimed to determine the presence or absence of euchromatin material of 39 de novo prenatally ascertained sSMC by array-comparative genomic hybridization (array-CGH) or single nucleotide polymorphism (SNP) array. Cases were prospectively ascertained from the study of 65,000 prenatal samples [0.060%; 95% confidence interval (CI), 0.042-0.082]. Array-CGH showed that 22 markers were derived from non-acrocentric markers (56.4%) a…

AdultGenetic MarkersRiskEuchromatinKaryotypeContext (language use)Prenatal diagnosisSingle-nucleotide polymorphismGenetic CounselingBiologyPolymorphism Single NucleotideYoung AdultPregnancyPrenatal DiagnosisGeneticsmedicineSNPHumansGenetic Predisposition to DiseaseProspective StudiesGenetics (clinical)Genetic Association StudiesIn Situ Hybridization FluorescenceGeneticsChromosome AberrationsComparative Genomic Hybridizationmedicine.diagnostic_testKaryotypeMiddle AgedPrognosisMolecular biologyFemaleFranceSwitzerlandSNP arrayFluorescence in situ hybridizationGenome-Wide Association StudyClinical genetics
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Genome-wide analysis reveals DNA methylation markers that vary with both age and obesity

2014

AbstractThe combination of the obesity epidemic and an aging population presents growing challenges for the healthcare system. Obesity and aging are major risk factors for a diverse number of diseases and it is of importance to understand their interaction and the underlying molecular mechanisms. Herein the authors examined the methylation levels of 27578 CpG sites in 46 samples from adult peripheral blood. The effect of obesity and aging was ascertained with general linear models. More than one hundred probes were correlated to aging, nine of which belonged to the KEGG group map04080. Additionally, 10 CpG sites had diverse methylation profiles in obese and lean individuals, one of which wa…

AdultGenetic Markersmedicine.medical_specialtyAgingAlpha-Ketoglutarate-Dependent Dioxygenase FTOGenome-wide association studyBiologyMicroarrayBioinformaticsEpigenesis GeneticGeneticsmedicineHumansEpigeneticsObesityKEGGTelomeraseAgedMedicinsk genetikGeneticsProteinsGeneral MedicineMethylationDNA MethylationMiddle Agedmedicine.diseaseObesityCpG siteDNA methylationMedical geneticsCpG IslandsFemaleEpigeneticsMedical GeneticsGenome-Wide Association Study
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